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amygdaloid相关的网络例句

查询词典 amygdaloid

与 amygdaloid 相关的网络例句 [注:此内容来源于网络,仅供参考]

Effect of estrogen on dopamine release evoked by electric stimulation of central amygdaloid nucleus. Acta Physiol Sin, 2001, 53(3):170~174.

而且在线粒体呼吸链中,氧接受一个电子O·〖TX-*82, O·〖TX-*82在过氧化物歧化酶的作用下可生成H2O2,而O·〖TX-*82本身可促进Fe3+由其铁结合蛋白上的释放,从而加速了OH·的生成[13],机体氧化与抗氧化机制平衡的破坏同样加速了这一反应。

ABSTRACT Aim and Methods: Using fast cyclic voltammetry and carbon fibre mi croelectrode, we monitored dopamine release from caudate putamen ,nucleus accumbens and central amygdaloid nucleus of normal rats in v ivo, in order to find out the suitable parameters to evoke DA release of certa in area. DA release was evoked by electrical stimulation of medial forebrain bundl e or ventral tegmental area.

目的和方法:采用快速周期伏安法在体研究电刺激内侧前脑束或腹侧背盖区诱发的纹状体、伏核或中央杏仁核多巴胺释放的特点,探索电刺激诱发不同核团DA释放的适宜刺激参数。

Results:(1) The rats produced amphetamine CPP after treated with 2 mgkg^(-1) amphetamine for 4 days. Ketamine and low, middle, high dose rhynchophylline all could eliminate the CPP effect, and the effect of rhychophylline was increased in a dose-dependent manner. Rhychophylline itself could not induce CPP in normal rats.(2) When compared with normal rats, the NR2B expression in nucleus accubens and amygdaloid of the model rats was obviously increased. Ketamine and middle, high dose rhynchophylline could decrease NR2B expression induced by amphetamine, while low dose rhynchophylline did not affect NR2B expression. Rhynchophylline had no effect on RN2B expression in normal rats.

结果:(1)建立了苯丙胺(2mgkg^(-1),连续4d)诱导的位置偏爱模型,氯胺酮及钩藤碱低、中、高剂量均可消除苯丙胺诱导的位置偏爱效应,随钩藤碱剂量增加其效应加强,且本身无精神依赖性;(2)苯丙胺模型组大鼠伏核和杏仁核NR2B蛋白表达增加,氯胺酮及钩藤碱中、高剂量抑制NR2B表达,低剂量及本身对NR2B表达无影响。

Three hours following seizures there was an evident expression of c-fos proteins in the hippocampus (CA1 area), dentate gyrus, piriform cortex, dorsal part of entorhinal cortex, and amygdaloid nucleus, and there was a dramatic increase of c-fos proteins in CA3 area and the areas mentioned above except the CA1 area where c-fos proteins apparently decreased after electroacupuncture treatment.

在海马青霉素致痫模型上观察了致痫及致痫加电针后脑内c-fos蛋白的变化,结果表明致痫时海马〓区、齿状回、梨状皮层、背侧内嗅皮层、杏仁核见到较多的c-fos表达;电针后c-fos在〓区明显减少,在〓区及上述其余各区则显著增加。

Hypothalamus, paraventricular nucleus, supraoptic nucleus and amygdaloid nucleus. Conclusion: Above mentioned nuclei might involve in rat abdomino pain which is caused by peritoneal noxious stimulation, which offers groundwork of experiment for further studying killing pain with nervous biology.

T6~L3脊髓后角、延髓孤束核、臂旁核、蓝斑核、下丘脑、室旁核、视上核、杏仁核与大鼠腹膜伤害性刺激造成的腹痛有关,本研究为进一步进行神经生物学研究镇痛机制提供了实验依据。

Methods: After intranasal or intraperitoneal administration of r-HuEPO, the behavioral and electroencephalographic changes were observed in pentylenetetrazol and maximal electroshock induced seizure or electrical amygdaloid-kindled seizure of rats.

另外,大鼠经鼻给r-HuEPO后,观察对最大电休克诱导的惊厥发作级别,前肢或后肢强直性伸展时间的影响;对杏仁核电点燃后阈刺激诱导的行为等级,大发作持续时间及后放电的影响。

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