查询词典 alveolitis

Infrequent cases (about 1%) of interstitial lung disease (ILD-described as interstitial pneumonia, pneumonitis, and alveolitis) have been observed in patients receiving IRESSA.

罕见情况下（约1％），间质性肺疾病（电信，描述为间质性肺炎，肺炎，以及肺泡炎）中已观察病人接受易瑞沙。

Results (1) The degrees of alveolitis and pulmonary fibrosis of the model group was increased remarkably on day 7,14,28 compared with the control group (P.05), The expression of TNF-α, TGF-β1, FN protein of model group on day 7, 14, 28 was remarkably increased compared with the control group (P.05);(2)The degrees of fibrosis and the expression of TNF-α, TGF-β1 , FN protein are decreased significantly in Group B compared with Group A( P.05) but not significantly in Group C.

结果：(1)模型组肺泡炎、肺纤维化程度都明显重于对照组，肺泡炎、肺纤维化第7、14、28d差异显著(P＜0.05)；TNF-α、TGF-β_1、FN蛋白表达明显上调(P＜0.01)；(2)治疗组B组第7、14、28d肺泡炎、肺纤维化程度较模型组明显减轻，TNF-α、TGF-β_1、FN蛋白表达明显下调(P＜0.05)：(3)治疗组C组第28d肺泡炎、肺纤维化程度与模型组相似，TNF-α、TGF-β_1、FN蛋白表达与A组比较无统计学意义(P＞0.05)。

AIM: To study the inhibitory effect of IGF binding protein-4 (IGFBP-4), especially in corporation with heparin on the pathopoiesis of insulin-like growth factor-1 (IGF-1) in alveolitis and fibrosis.

目的：探讨类胰岛素生长因子(IGF-1)及其结合蛋白－4(IGFBP-4)、肝素在肺纤维化形成过程中的相互关系。

Group T has the same tendency as group B; but the degree of alveolitis and pulmonary fibrosis decreased. Compared to group B, the expression of TGF-β1, Smad3 was decreased significantly in group T, so did the index of lung cells apoptosis.

干预组各时间点TGF-β1和Smad3表达明显低于模型组(P.05)，28天时大鼠肺组织中细胞凋亡指数干预组明显低于模型组。

Idiopathic pulmonary fibrosis, caused by unidentified reasons, is a kind of disease featured in diffuse alveolitis and the disordered structure of alveolus that finally lead to the fibrosis of pulmonary interstitial substance.

特发性肺纤维化(Idiopathic pulmonary fibrosis ，IPF)是一种原因不明、以弥漫性肺泡炎和肺泡结构紊乱最终导致肺间质纤维化为特征的疾病。

Methods: 60 SD rats were divided into 4 groups randomly, the nomal group, the model group, QZHJ group, and the hydrocortisone group. Rats were sacrificed in bdtch on the 3rd, 7th and 21st day after treatment, then the concentration of interleukin-8(IL-8) in bronchoalveolar lavage fluid and the content of transforming growth factor-β1 (TGF-β1) of lung tissue were deected, while the severity of alveolitis and fibrosis were measured.

将60只健康SD大鼠随机分为正常组、模型组、芪术合剂组和强的松组，经气管滴入博莱霉素复制肺纤维化大鼠模型，并分组予以治疗，于治疗后第3、7、21日分批处死大鼠，每组每次6只；用免疫组化法观察肺组织转化生长因子-β1的表达，并测定支气管肺泡灌洗液中中性粒细胞和白介素－8的水平。

Moreover, our group could show increased oxidation of extracellular glutathione in vitro by cultivated alveolar macrophages from patients with fibrosing alveolitis, both spontaneoulyand after PMA stimulation.

而且，我们对从纤维性小肺泡炎患者中获得的肺泡巨噬细胞在体外进行培养，并应用自发和PMA进行刺激，研究结果显示：细胞外谷胱甘肽的氧化增加。

Results Beginning from alveolitis, PF in different diseases has similar pathological changes in a progressive and reparative process. About 7-8% of SARS convalescent patients showed apparent PF, which affected their life quality and increased medical treatment costs.

结果 不同病种的肺间质纤维化性改变都是从肺泡炎开始，在发展和修复过程中导致的肺纤维化倾向有共同之处。

The result showed that the experimental group can restrain thephosphorylation activity of P38MAPK and down-regulate the amount ofⅢtypecollagen hyaluronic acid and IL-6, improve the degree of alveolitis and pulmonaryfibrosis, decrease the expression of TGF-β1 in the lung tissue compared with themode group at all times.

研究结果显示：在各个时间点，肺痹汤各剂量组较模型组可以抑制P38MAPK磷酸化活性和下调Ⅲ型胶原透明质酸和IL-6细胞因子含量，改善肺泡炎及肺纤维化的程度，降低TGF-β_1在肺组织中的表达。

Amelioration the pulmonary alveolitis and fibrosis were shown after treatment by BYHUD.Conclusion: BYHUD has definite effect in treating PIF.

补阳还五汤能有效改善实验性肺纤维化大鼠的症状，并延缓其进程。

The labia have now been sutured together almost completely.The drains and the Foley catheter come out at the top.

此刻阴唇已经几乎完全的缝在一起了，排除多余淤血体液的管子和Foley导管从顶端冒出来。

To get the business done, I suggest we split the difference in price.

为了做成这笔生意，我建议我们在价格上大家各让一半。