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The expressions of ICAM-1 on bronchial and alveolar epithelium, arteriolar and venular endothelium of lung were examined by immunohistochemical method.

近年来国外有数篇哮喘患者及动物模型气管上皮细胞及粘膜下层气管内皮细胞粘附分子ICAM-1增加的报道。

Only weak expression of ICAM-1 was found on bronchial and alveolar epithelium, pulmonary arteriolar and venular endothelium in normal and sensitized rat lung. The expression of ICAM-1 in all tissues mentioned above were increased in asthmatic rats.

免疫组化结果显示，哮喘大鼠支气管上皮细胞、肺动静脉内皮细胞及肺泡上皮细胞ICAM-1表达均明显高于对照组。

The significantly increase of ICAM-1 mRNA expression on alveolar epithelium and pulmonary venular endotheium was also found in asthmatic rats compared with those of normal and sensitized control, but the expression of ICAM-1 mRNA on pulmonary arteriolar endothelium and bronchial epithelium showed a inceasing tendency.

原位杂交结果表明，哮喘组大鼠肺静脉内皮细胞、肺泡上皮细胞ICAM-1 mRNA表达量明显多于对照组；支气管上皮细胞、肺动脉内皮细胞粘附分子表达也有增加的趋势。

Cytokines found to have up-regulated in cells were measured in serum and lung tissue of rats exposed to large tidal volume ventilation. For investigating the intracellular pathway of cytokine up-regulation in VILI, exogenous TNF-α or H2O2 was added to culture media of alveolar epithelial cells. Cytokines were then measured. To explore the role of oxidant stress in VILI, N-acetylcysteine, as an anti-oxidant, was used in vitro and in vivo.

我们在体外培养的肺上皮细胞中加入外源性肿瘤坏死因子、过氧化氢或N-乙酰半胱氨酸，观察细胞因子的变化。N-乙酰半胱氨酸亦被投入大鼠呼吸机诱导肺损伤模型，观察用药后细胞因子的变化。

METHODS: Decalcified material including the alveolar bone was sectioned and stained by means of immunohistochemistry for detection of the neurofilament protein.

大鼠磨牙及其牙槽骨脱钙后切片并行神经丝蛋白免疫组织化学染色。

The results showed that:(1) In COPD group the alveolar walls were broken. The average area of alveolars was 2.46 times as big as the control group, and the average diameter was 1.42 times as compared with the control group. As the diameter of bronchioles was less than 100μm and 100～200μm respectively, the average area of smooth muscle in bronchioles increased to 3.06-fold and 2.13-fold respectively. By electron microscopic, the proportion of Clara cells of respiratory bronchioles in COPD group decreased, the Clara cells were denaturalized, the proportion of Clara cells containing low density secretory granules increased. The airway resistance was 1.84 times while the dynamic compliance was only 70.45% comparing with the control group.

结果表明：(1)光镜下，COPD组大鼠与正常对照组比较，肺泡壁破坏、肺泡腔扩大，平均肺泡面积和平均肺泡直径分别为正常对照组的2.46倍和1.42倍，在直径＜100μm及直径100～200μm的小支气管，COPD组大鼠支气管平滑肌面积分别为正常对照组的3.06倍和2.13倍；电镜下可见，在呼吸性细支气管，Clara细胞占上皮细胞的比例下降，细胞变性，含低电子密度颗粒的Clara细胞比例增加；呼吸功能测定可见COPD组大鼠呼气相气道阻力是正常对照组的1.84倍、动态呼吸系统总顺应性为正常对照组的70.45％。

DISCUSSION:Extensive interstitial and alveolar fibrosis results in the characteristic HRCT finding of honeycombing or honeycomb lung.

讨论：广泛的肺间质及肺泡纤维化在HRCT上的特征性表现为蜂窝样改变，或称为蜂窝肺。

PaO2/FiO2 of both patients was less than 225 mm Hg.In both patients, high-resolution computed tomography scans at the exacerbation showed typical signs of IPF including peripheral predominant, basal predominant reticular abnormality, with honeycombing and traction bronchiectasis and bronchiolectasis,and newly developing alveolar opacity.

呼吸困难分别在1周和半月内加重；2例患者氧合指数均小于225 mm Hg；急性加重时2例患者HRCT表现为两下肺分布的网状影、蜂窝影、牵拉性细支气管扩张和支气管扩张等典型的IPF表现，并出现新的磨玻璃影。

In conscious yogic breathing a much larger quantity of air reaches the lungs and inflates more alveolar tissue.

在瑜伽的呼吸中，有更多的空气进入肺部和肺的微小的气泡中。

The study aims to synthesize a new denture base material to complemete the electret of teeth-extracted alveolar bone and stimulate bone growth towards the denture base when masticating and inhibit or rimit the RRR.

本课题研制新型改性义齿基托，在修复牙列缺损或缺失时通过驻极体效应和压电效应，补充患者失牙区局部驻极态的丧失，利用咀嚼运动产生的微电流刺激骨向基托方向定向生长，抵御或缓解牙槽骨的吸收。

I didn't watch TV last night, because it .

昨晚我没有看电视，因为电视机坏了。

Since this year, in a lot of villages of Beijing, TV of elevator liquid crystal was removed.

今年以来，在北京的很多小区里，电梯液晶电视被撤了下来。