查询词典 aldehyde group

ARL-1 gene can reduce the cytotoxicity of anti-cancer drug consisting of active aldehyde group and suppress apoptosis induced by anti-cancer drug consisting of active aldehyde group.

ARL-1基因能降低含有活性醛基的抗肿瘤药物的细胞毒性，和抑制其所诱导的细胞凋亡。

Result: The nm-HAP-sol present the dispersed and uniformity needle. A431cells inhibition ratio was maximizing at the 5 day in the 400mg/L nm-HAP-sol group, the 200mg/L nm-HAP-sol group and the 100mg/L nm-HAP-sol group, inhibition ratio of the 400mg/L nm-HAP-sol group, the positive control group and the 50mg/L nm-HAP-sol group was respectively (77.89±6.29)%,(77.46±8.26)% and (1.23±0.15)%. It was detected that the number of A431 cells were persistently increasing in the 50mg/L nm-HAP-sol groups while decreasing in the 400mg/L nm-HAP-sol group, the positive control group and abidingly increasing for 4 and 3 days then decreasing in the 100mg/L and 200mg/L nm-HAP-sol group by inverted microscope.

结果：超声乳化法配制的HAP纳米溶胶呈均匀分散的针状；400mg/L、200mg/L和100mg/L溶胶组对A431细胞抑制率在第5天达到各组最大值，此时400mg/L溶胶组与阳性对照组的抑制率分别为（77.89±6.29）%和（77.46±8.26）%， 50mg/L溶胶组抑制率为（1.23±0.15）%；倒置显微镜下见50mg/L溶胶组细胞数目持续增加，聚集成簇，细胞呈现多角形或梭形，胞浆透亮，而100mg/L 和200mg/L溶胶组的细胞数目分别增加至第4天和第3天开始下降，400mg/L溶胶组和阳性对照组细胞数目持续减少，细胞体积缩小，漂浮细胞增多。

There are the normal group,the AS group,the pure swimming group,the swimming plus AS group,the simvastatin group(simvastatin plus AS of overstrain),the large dose Tongxinluo group,the small dose Tongxinluo group,the bulbus allii macrostemonis four drugs group and the ginseng group.

目的对络气虚滞型动脉粥样硬化模型中9个编号组即正常组、动脉粥样硬化组、单纯游泳组、游泳加动脉粥样硬化组、辛伐他汀组(即辛伐他汀+过劳加动脉粥样硬化，下同)、通心络大剂量组、通心络小剂量组、薤白四味组、人参组进行综合评价。

Result: The ROM of group A had no significant difference with that of the control group. The ROM of group B had no significant difference with that of the control group except rotation, each ROM of group C and group D was significant difference with the control group, and the ROM of group D was the biggest. The ROM of group E was the smallest and had statistically significant difference in each motion compared with the control.

结果：A组ROM值与对照组无显著性差异；B组除旋转外与对照组无显著性差异，旋转稳定性也达到了对照组的61.86%；C组与对照组均有显著性差异；D组ROM值最大，与对照组有显著性差异；E组除后伸外ROM值均明显小于对照组，差异有显著性(P.05)。

12H treatment group compared with 12h model group: the level of MCP-1 of the Gardenoside group decreased and MIP-1 is no different with the model group; the level of the MCP-1, MIP-1 of the Baicalin group are no different with the model group; the level of MCP-1, MIP-1 of the Concha Margatitifera Usta decreased than the model; the level of MCP-1 of the Cholic Acid group is no different and the level of MIP-1 decreased than the model; the level of MCP-1 of the Hefang Group decreased and the level of MIP-1 of it decreased than the model group.

合方治疗脑缺血24小时组缺血脑组织nNos光密度值较缺血24小时模型组明显降低（p＜0.01）。细胞的死亡分为坏死和凋亡两种方式，凋亡是细胞为避免对机体造成更大损伤而采取的一种程序性的死亡方式。珍珠母、胆酸、栀子苷、黄芩苷、合方治疗组较脑缺血24小时模型组可明显降低P53阳性表达，考虑药物抑制细胞凋亡可以起到对缺血脑组织的保护作用。

24H treatment group compared with 24h model group: the level of MCP-1 of the Gardenoside group decreased, but the level of MIP-1 of it is no different with the model group; the level of the MCP-1 of the Baicalin group increase and the level of MIP-1 of it all decreased; the level of MCP-1 and MIP-1 of the Concha Margatitifera Usta all decreased; the level of MCP-1 of the Cholic Acid group decreased and the level of MIP-1 is no different with the model group; the level of MCP-1 and MIP-lof the Hefang Group decreased than the model group.

在本实验中，脑缺血能够诱导P53蛋白表达，P53蛋白可能参与调控缺血后神经细胞延迟性死亡的病理生理过程，药物通过抑制细胞凋亡，从而发挥治疗作用。五、大鼠局灶性脑缺血12小时模型组缺血脑组织HSP70光密度值含量较正常组明显增高（p＜0.05）。珍珠母治疗脑缺血12小时组缺血脑组织HSP70光密度值较缺血12小时模型组无显著性差异（p＞0.05）。胆酸治疗脑缺血12小时组缺血脑组织HSP70光密度值较缺血12小时模型组无显著性差异（p＞0.05）。

12H treatment group compared with 12h model group: the density of light of nNos of the Gardenoside group decreased and P53 decreased with the model group; the density of light of the nNos of the Baicalin group decreased and which of the P53 is no different with the model group, the density of nNos of the Concha Margatitifera Usta decreased and which of P53 decreased than the model; the density of of nNos of the Cholic Acid group is no different and the density of P53 decreased than the model; the density of the light of nNos of the Hefang Group is no different and which of P53 decreased than the model group.

珍珠母治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组无显著性差异（p＞0.05）。胆酸治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组无显著性差异（p＞0.05）。栀子苷治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组明显增高（p＜0.01）。黄芩苷治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组明显增高（p＜0.01）。合方治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组无显著性差异（p＞0.05）。

Methods: Diabetic nephropathy rats induced by intraperitoneal injection of STZ were randomized into PN model group, n=10, Vitamin B6 group (group B, n=10) and aminoguanidine group (group A, n=10), contrasted to anormal control group (group C, n=10). Aminoguqnidine and VitB6 with 1 g dissolved in 1 L distilled water were given to group A and B, while group C and D only had 1 L disstided water. All groups drunk water freely after 24 hours.

利用链脲菌素腹腔注射法诱导建立DN大鼠模型，将其随机分为模型组、维生素B6治疗组、氨基胍治疗组，并与正常对照组比较，每组10只大鼠；A、B组每天分别用氨基胍、维生素B61g溶于1L蒸馏水中，C、D组仅给予1L蒸馏水，24h后自由饮用。

After antituberculotic treatment for 6 months.The rates of sputum coversion of group 1,group 2 and group 3 were 76.92%,94.79% and 100% respectively.The mean sputum coversion time of group 1,group 2 and group 3 were 12.46 weeks 5.0 weeks and 3.0 weeks respectively and the rates of focus absorption of group 1 ,group 2 and group 3 were 61.54%. 86.46% and 100% respectively.

经过6个月抗痨治疗后，Ⅰ组痰菌转阴率为76.92%，平均转阴时间为12.46周，病人疗程结束时，肺内病灶X线吸收占61.54%；Ⅱ组痰菌转阴率为94.79%，平均转阴时间为5.0周，病人疗程结束时，肺内病灶X线吸收占86.46%；Ⅲ组痰菌转阴率为100%，平均转阴时间为3.0周，病人疗程结束时，肺内病灶X线吸收占100%。

Methods: 1. Rats in the study were divided into six groups: normal control group, hyperlipidemia group (fed with high-cholesterol diet), atherosclerosis group (vitamin D〓 intramuscular injection, endothelium denudate and fed with high-cholesterol diet with vitamin D〓), L-arginine group (besides treatment as atherosclerosis group, 2.25％ L-arginine was added in the drinking water), chitosan group (besides treatment as atherosclerosis group, 5％ chitosan was added in the diet), simvastatin group (besides treatment as atherosclerosis group, 0.03％ simvastatin was added in the diet). The study lasted 90 days.

1。实验大鼠分为6组：正常对照组、高脂血症组、动脉粥样硬化组(维生素D〓肌肉注射加动脉球囊损伤手术加含维生素D〓的高脂饲料饲养)、L-精氨酸组(动脉粥样硬化组基础上饮水中加入2.25％L-精氨酸)、几丁糖组(动脉粥样硬化组基础上饲料中加入5％几丁糖)、辛伐他汀组(动脉粥样硬化组基础上饲料中加入0.03％辛伐他汀)，90天后取标本检测。2。

This was not understood at the time, and therefore went unaddressed, until ascension became so distorted that we chose to stop ascending and wait for a time when all records would be revealed.

这在当时没有被了解，因此进行着无改编，直到提升变得如此扭曲，我们选择去停止提升并等待所有记录能被揭示的那一天。

The attack＇s nuisance value is increased because mobile phone networks also send notifications of new voicemail by sms,so an attacked phone may stop advising of new voice messages too,it warned.

它警告：这种攻击的危害增长是因为移动电话网络也会通过短信发送新语音邮件的通知。所以受到攻击的手机可也以停止新的语音信息的提示。