查询词典 acid group

And alkyl or alkylene,an amino acid eater having an extremely high reactivity,sulfonic acid group can be added into side chain of polyaspartic acid to form modified polyaspartic acid.

许多药物具有—NH2基团，能够与PASP的羧酸形成酰胺基团，被嵌入到PASP分子链上，改变PASP的性质。

In some embodiments where adhesion and alkali removability need to be independently controlled it is preferable that the carrier vehicle comprises two or more types of metal chelating groups wherein at least one of the metal chelating groups is an acid group and at least one of the metal chelating groups is not an acid group.

在有些体现在附着力和碱取出需要独立控制，这是可取的，承运人车辆由两个或两个以上的金属类型螯合组其中至少有金属螯合集团之一，是一种酸组和至少一个金属螯合组不是一个酸组。

12H treatment group compared with 12h model group: the density of light of HSP70 of the Gardenoside group is not different and which of it of TGFβ〓 increased with the model group; the density of light of the HSP70 of the Baicalin group in not different and which of the TGF-β〓 is no different with the model group, the density of HSP70 of the Concha Margatitifera Usta is not different and which of TGFβ〓 increased than the model group; the density of the HSP70 of the Cholic Acid group is no different and the density of TGF-β〓 increased than the model; the density of the light of HSP70 of the Hefang Group increased and which of TGF-β〓 increased than the model group.

合方治疗脑缺血12小时组缺血脑组织TGF-β1光密度值较缺血12小时模型组明显升高（p＜0.01）。大鼠局灶性脑缺血24小时模型组缺血脑组织TGF-β1光密度值含量较正常组明显增高（p＜0.01）。珍珠母治疗脑缺血24小时组缺血脑组织TGF-β1光密度值较缺血24小时模型组明显升高（p＜0.01）。胆酸治疗脑缺血24小时组缺血脑组织TGF-β1光密度值较缺血24小时模型组明显升高（p＜0.01）。栀子苷治疗脑缺血24小时组缺血脑组织TGF-β1光密度值较缺血24小时模型组无显著性差异（p＞0.05）。黄芩苷治疗脑缺血24小时组缺血脑组织TGF-β1光密度值较缺血24小时模型组明显升高（p＜0.01）。

12H treatment group compared with 12h model group: the level of MCP-1 of the Gardenoside group decreased and MIP-1 is no different with the model group; the level of the MCP-1, MIP-1 of the Baicalin group are no different with the model group; the level of MCP-1, MIP-1 of the Concha Margatitifera Usta decreased than the model; the level of MCP-1 of the Cholic Acid group is no different and the level of MIP-1 decreased than the model; the level of MCP-1 of the Hefang Group decreased and the level of MIP-1 of it decreased than the model group.

合方治疗脑缺血24小时组缺血脑组织nNos光密度值较缺血24小时模型组明显降低（p＜0.01）。细胞的死亡分为坏死和凋亡两种方式，凋亡是细胞为避免对机体造成更大损伤而采取的一种程序性的死亡方式。珍珠母、胆酸、栀子苷、黄芩苷、合方治疗组较脑缺血24小时模型组可明显降低P53阳性表达，考虑药物抑制细胞凋亡可以起到对缺血脑组织的保护作用。

12H treatment group compared with 12h model group: the level of SOD, MDA of the Gardenoside group is no different with the model group; the level of the SOD of the Baical in group increase and the level of MDA is no different; the level of SOD of the Concha Margatitifera Usta is no different and the MDA of it decreased than the model; the level of SOD of the Cholic Acid group increased and the level of MDA decreased; the level of SOD of the Hefang Group increased and the level of MDA of it decreased than the model group.

考虑nNos可能只是参与缺血早期的神经毒性作用。到缺血24小时这一时程已经减低或耗竭。大鼠局灶性脑缺血12小时模型组缺血脑组织P53光密度值含量较正常组明显增高（p＜0.01）。珍珠母治疗脑缺血12小时组缺血脑组织P53光密度值较缺血12小时模型组明显降低。胆酸治疗脑缺血12小时组缺血脑组织P53光密度值较缺血12小时模型组明显降低（p＜0.05）。栀子苷治疗脑缺血12小时组缺血脑组织P53光密度值较缺血12小时模型组明显降低（p＜0.05）。

24H treatment group compared with 24h model group: the level of MCP-1 of the Gardenoside group decreased, but the level of MIP-1 of it is no different with the model group; the level of the MCP-1 of the Baicalin group increase and the level of MIP-1 of it all decreased; the level of MCP-1 and MIP-1 of the Concha Margatitifera Usta all decreased; the level of MCP-1 of the Cholic Acid group decreased and the level of MIP-1 is no different with the model group; the level of MCP-1 and MIP-lof the Hefang Group decreased than the model group.

在本实验中，脑缺血能够诱导P53蛋白表达，P53蛋白可能参与调控缺血后神经细胞延迟性死亡的病理生理过程，药物通过抑制细胞凋亡，从而发挥治疗作用。五、大鼠局灶性脑缺血12小时模型组缺血脑组织HSP70光密度值含量较正常组明显增高（p＜0.05）。珍珠母治疗脑缺血12小时组缺血脑组织HSP70光密度值较缺血12小时模型组无显著性差异（p＞0.05）。胆酸治疗脑缺血12小时组缺血脑组织HSP70光密度值较缺血12小时模型组无显著性差异（p＞0.05）。

12H treatment group compared with 12h model group: the density of light of nNos of the Gardenoside group decreased and P53 decreased with the model group; the density of light of the nNos of the Baicalin group decreased and which of the P53 is no different with the model group, the density of nNos of the Concha Margatitifera Usta decreased and which of P53 decreased than the model; the density of of nNos of the Cholic Acid group is no different and the density of P53 decreased than the model; the density of the light of nNos of the Hefang Group is no different and which of P53 decreased than the model group.

珍珠母治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组无显著性差异（p＞0.05）。胆酸治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组无显著性差异（p＞0.05）。栀子苷治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组明显增高（p＜0.01）。黄芩苷治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组明显增高（p＜0.01）。合方治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组无显著性差异（p＞0.05）。

Started from D-tyrosine, we obtained the tetramic acid 96 using Jouin reaction. The assembly of the second fragment 109 was stared from 1,4-butandiol,using asymmetric alkylation reaction developed by Oppolzer as a key step. Coupling of the tetramic acid 96 with 109 provided the key intermediate 110 according to Yoshii\'s procedure. After remove protective group and Sharpless epoxidation, we obtained the key intermediate 113. Its macrocyclization to form the final product is in progress.

本论文的第二章主要对Macrocidin A开展了合成工作，我们从D构型的酪氨酸出发，经过官能团转换；利用Jouin反应生成Tetramic acid 96，另外一个片断从1，4-丁二醇开始经过多步反应生成碘化物104，利用Oppolzer发展的手性樟脑磺酰胺为辅基参与的不对称烷基化反应，引入手性甲基，生成关键中间体109，接着通过Yoshii发展的方法连接96和109生成110，经过Sharpless不对称环氧化反应建立环氧化合物，然后脱去保护基得到关环前体113，分子内的Mitsunobu反应进行关大环反应正在进行之中。

Models of rotator cuff injury were created at the synovium. 0.05 mL hyaluronic acid was injected into the subacromial bursa mucosa in the hyaluronic acid group, whereas 0.05 mL corticosteroids in the corticosteroids group.

透明质酸治疗组肩峰下滑囊注射0.05 mL透明质酸钠；皮质激素治疗组肩峰下滑囊注射倍他米松0.05 mL。

Thus we conclude that JA might be one of the potent factors, other than auxin, involving in the regulation of LR formation.3. Relationship between JA and SA on LR formation in rice seedlingMouse monoclonal antibodies against salicylic acid was produced and characterized for the first time. The McAb S2-8F, were derived from an immunogen in which 5-aminosalicylic acid (5-ASA) was conjugated to KLH (hemocyanin from keyhole limpets) through its-NH2 group.

实验结果还表明IBA对水稻幼苗侧根发生的促进作用可能并非是通过转化为吲哚乙酸(indole-3-acetic acid，IAA)而实现，因为外源IAA不能促进水稻幼苗侧根的发生，荣莉酸对水稻幼苗侧根发生的诱导效应及其与生长素和水杨酸的关系却和 IBA一样可以抑制种于根伸长及促进侧根伸长，只是IAA具有这种效应需要的浓度是IBA的20多倍。3。

For a big chunk of credit-card losses; the number of filings (and thus charge-off rates) would be rising again, whether

年美国个人破产法的一个改动使得破产登记急速下降，而后引起了信用卡大规模的亏损。

Eph. 4:23 And that you be renewed in the spirit of your mind

弗四23 而在你们心思的灵里得以更新