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The enhanced cell surface expression of CTLA-4 is likely responsible for the induction of T cell anergy.

结论CTLA-4表面表达水平的升高很可能与T细胞无能状态的诱导有关。

About the mechanism of HBV persistence, there are many hypothesizes such as anergy and exhaustion of T cells, decreased quality and function of dendrtic cells etc.

关于乙型肝炎病毒感染后的慢性化假说很多，既往的研究提示可能与T细胞的无能或耗竭，树突状细胞的数量和功能的下降及机体对病毒的免疫耐受有关。

The results from the present study may have important implications for the study of the mechanism underlying T cell anergy induction in vivo and for applications of immune tolerance based therapy.

这对体内T细胞免疫耐受产生的机制研究和临床应用都有重要意义。

The lack of antigen-presenting cell activity of CB MNCs in primary mixed lymphocyte culture did not induce allogeneic T cell anergy.

缺乏抗原提呈细胞活性的脐带血单个核细胞在初级混合淋巴细胞培养下不能诱导同种异体反应T细胞的无反应性。

In io, mTOR inhibition promotes T cell anergy under conditions that would normally induce priming.

体内研究中，在正常可激发免疫反应的条件下，抑制mTOR可促使T细胞无能。

In the absence of appropriate costimulation, partial activation of T cells can be lead to a state of long-lasting antigen-specific unresponsiveness or anergy.

在TCR识别MHC-抗原肽的同时，B7和CD28分子对的共刺激作用将使T细胞增殖并上调细胞因子表达，启动免疫应答反应。

Importantly, T cells engineered to express a rapamycin-resistant mTOR construct are resistant to anergy induction caused by rapamycin.

重要地是，T细胞表达人为构造的耐受雷帕霉素mTOR可对抗雷帕霉素诱导的无能。

While the TCR engagement with specific antigen, the administration of CTLA-4Ig can induce T cell anergy in vitro or tolerance in vivo.

在TCR识别外来抗原的同时给予可溶性CTLA-4，在体外可诱导T细胞无应答，在体内则可诱导特异性免疫耐受。

The results were analyzed with the statistical software package spss10.0.results ①between two groups,there was no significant difference about the affection of age,sex and the destribution and location of aneurism on prognosis outcome(p＜0.05).however,the grading of hunt hess and fisher in group a(gos＞3) showed significantly lower than in group b(gos≤3),（p＜0.001）;② there was no significant difference in the frequency of heart rate, st elevation, st depression,t wave inversion,u wave inversion and ventricular arrhythmia(p＞0.05).the frequency of abnormal q wave, supraventricular arrhythmia and the ecg scores in group a showed significantly lower than in group b （p＜0.05,p＜0.001）;③the ecg scores was the most powerful multivariate risk stratified(ecg ≥5 vs ecg＜5; p=0.002,or=0.010,95%ci 0.001~0.173).conclusion the ecg score, a new and simple method of semi quantification, was a powerful risk predictor for patients with sah.

应用spss10.0 统计包分析结果。结果①在年龄、性别、动脉瘤生长部位影响动脉瘤性sah预后方面，两组病例无明显差异（p＞0.05）。而按sah后hunt hess分级和fisher分级，gos＞3病例的分级要明显低于gos≤3的病例的分级（p＜0.01）；②两组在心率、st段抬高、st段压低、t波倒置、u波倒置、室性心律失常出现的频率无明显差异（p＞0.05）；在异常q波和室上性心律失常出现频率及心电图评分方面，gos＞3的组别明显低于对照组（p＜0.05，p＜0.001）；③心电图评分是sah患者预后最显著影响的变量（p=0.002，or=0.010，95%的可信区间0.001~0.173）。结论心电图评分作为一种新型、简便的半定量方法，能有效的预测蛛网膜下腔出血病人的预后。关键词：心电图；预后；评分；蛛网膜下腔出血

How was I supposed to know Carmine T. Angelo...

我怎么会应该知道卡麦T安杰罗。。。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。