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NOS相关的网络例句
与 NOS 相关的网络例句 [注:此内容来源于网络,仅供参考]

CuteSEO information list the word that if the webpage is not,must use frame act against one's will so, so the way that settles search engine respect has two: The first, use Nos undertakes optimizing

CuteSEO资讯列表那么如果网页非不得已一定要用框架的话,那么解决搜索引擎方面的办法就有两个:第一、使用Nos进行优化

The NoS positive fibers and terminals were densely found in laminae Ⅰand Ⅱ and scattered in other lami-nae.

在此三部位分别有18.7%、11.6%和22.6%和NOS阳性神经元同时呈FOS阳性;有5.56%、2.67%和3%FOS阳性神经元同时是NOS阳性。

⑴There are NOS/ER positive neurons in the hypothalamic paraventricular nucleus, supraoptic nucleus, periventricular nucleus and lateral hypothalamic area.⑵The double-labeled neurons were mostly distributed in the medial dorsal and lateral dorsal of supraoptic nucleus and in the medial parvicellular, ventral zone. They were extensive slightly,but scattered in lateral hypothalamic area, sporadic in the periventricular nucleus.

1在大鼠下丘脑视上核、室旁核、下丘脑外侧区及室周核均有NOS与ER双染神经元分布。2 NOS与ER双染神经元主要集中分布在视上核的背内侧和背外侧部及室旁核小细胞部腹内侧区,在下丘脑外侧区分布较广但比较分散,室周核呈散在分布。

Results: The number of labelled NOS-positive neurons and NGF-positive neurons was respectively increased significantly (P.01) in pterygopalatine ganglion of the allergic rhinitis rats.

结果:实验组和正常组大鼠翼腭神经节内均有NOS和NGF表达,但实验组大鼠翼腭神经节内NOS和NGF表达均较正常组显著性增加(P.01)。

Methods The rat model of middle cerebral artery ischemia 2h/reperfusion 22h was established.The neurological scale, cerebral infracted volume, cerebral water content, activities of NOS and SOD were measured. ResultsThe average neurological scote,cerebral infracted volume,cerebral water content,activity of NOS,content of MDA in Naokangling Capsule group significantly decreased.

采用大鼠大脑中动脉缺血2 h/再灌注22 h模型,神经病学评分,脑梗塞范围及脑组织水含量变化,观察脑康灵胶囊抗脑缺血/再灌注损伤的效应;通过测定大鼠脑组织中一氧化氮合酶(nitric oxide synthase,NOS),超氧化物歧化酶(superoxide disumtase,SOD)活性及丙二醛(malondialdehyde,MDA)含量的变化以探讨药物作用的机制。

MethodsThe rat model of middle cerebral artery ischemia 2h/reperfusion 22h was established.The neurological scale, cerebral infracted volume, cerebral water content, activities of NOS and SOD were measured. ResultsThe average neurological scote,cerebral infracted volume,cerebral water content,activity of NOS,content of MDA in Naokangling Capsule group significantly decreased.

方法采用大鼠大脑中动脉缺血2 h/再灌注22 h模型,神经病学评分,脑梗塞范围及脑组织水含量变化,观察脑康灵胶囊抗脑缺血/再灌注损伤的效应;通过测定大鼠脑组织中一氧化氮合酶(nitric oxide synthase,NOS),超氧化物歧化酶(superoxide disumtase,SOD)活性及丙二醛(malondialdehyde,MDA)含量的变化以探讨药物作用的机制。

The inducible nitric oxide synthase is obviously expressed in speen of rats under the predator stress, suggesting important function for iNOS in speen physiology. However, the expression and subcellular localization for NOS in speen development and differentiation are largely unknown. In the present study, the changes in iNOS activity in spleen of rats under predator stress were studied to explore the role of iNOS in the decreased immunoactivity of the stressed rats. The changes in iNOS were observed with immunobistochemical method, and the contents of NOS and NO in spleen with biochemical method.

在捕食压力下,大鼠脾脏诱导型一氧化氮合酶明显表达,说明iNOS起到了重要的生理功能,然而,NOS的表达及在亚细胞水平的定位以及相应的差异变化等都很不明确,本试验采用捕食应激模型,利用免疫组织化学方法和生化测定,检测心理性应激对大鼠脾脏iNOS活性的影响及探讨这种影响与应激动物免疫力下降之间的关系。

After administration of L-N〓-nitro-Arginine, a specific antagonist of NOS, it was found that the basal and stimulated NO release of blood vessels (aorta, internal carotid, renal and mensenteric artery) was decreased further. The increase of coronary blood flow stimulated by Ach was also reduced further, while the platelet aggregation degree and erythrocyte aggregation index increased significantly. Pathological examination indicated that aortic endothelium was destroyed, the blood vessels in cerebral, cardiac and renal tissues were hypertrophied further and some were almost occluded. The above fin dings resulted in SHR feeded with L-NNA having high incidence of stroke rate and high degree of renal cirrhosis when compared to control SHR. The infarct and hemorrhagic focuci in cerebral tissues, infarct focuci in cardiac tissues, atrophy and hyalinedegeneration of renal glomeruli and degeneration even necrosis of renal tubuli in renal tissues were al so found. The increase ratio of brain and heart to body weight and decrease ratio of kidney to body wei ght further demonstrated that the cerebral, cardiac and renal injuries were aggravated.

在上述结果的基础上进一步给予SHR NOS特异性拮抗剂左旋亚硝基精氨酸后,血管(胸主、颈内、肾及肠系膜动脉)NO基础释放及激动剂刺激释放进一步减少,Ach刺激冠脉流量的增加和胸主动脉内膜NOS活性也进一步减少,血小板聚集程度及红细胞聚集指数显著上升,病理切片显示胸主动脉内皮细胞破坏明显,脑心肾组织内的血管进一步增厚甚至几乎阻塞,导致饲L-NNA的SHR缺血性及出血性脑卒中发生率显著高于对照SHR,脑体重比显著升高;心肌内出现明显的缺血梗塞疤痕灶,心体重比显著升高;肾小球明显萎缩、玻璃样变性,肾小管也变性甚至坏死,使肾硬化程度显著增加呈颗粒性固缩肾,肾体重比也显著减少。

The vasodilating mechanism of AE from CM ma he related to promoting the release of NO from endothelium of blood vessel and synthesis of NOS and NOS in aortic tissue.

桑白皮丙酮提取物的舒张血管作用机制可能与血管释放NO和促进cNOS、NOS合成有关。

Thereafter, both NO content and NOS activity of testis homogenates were increased.

结果 与对照组比较,糖尿病组血清NO含量升高,NOS活性先升后降;睾丸组织在诱模后NO含量下降,而NOS活性变化不明显,随后两者均呈升高趋势。

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相关中文对照歌词
Se Nos Muere El Amor
No Nos Moveran
Acá Entre Nos
Nos Estorbó La Ropa
Deus Nos Surpreende
Cuando Nos Volvamos A Encontrar
Si Nos Quedara Poco Tiempo
No Nos Alcanzó La Noche
Nos Da Cariad
Aca Entre Nos
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