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I. R.相关的网络例句

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与 I. R. 相关的网络例句 [注:此内容来源于网络,仅供参考]

Omputation of Car group PMN is remarkable when R2 and R6 under;PMN of I/R group bandicoot breath erupts the metabolic rule of intensity and PMN computation are consistent.

ar组PMN计数在R2和R6时显著低于I/R组大鼠;PMN呼吸爆发强度的变化规律和PMN计数一致。

CASI has protective effect on myocardial I/R injury in rats by reducing oxidative damage of free radicals on myocardium, enhancing activity of antioxidase and correcting disequilibrium of PGI2/TXA2 etc.

CASI对大鼠心肌I/R损伤具有明显保护作用,可能与其减少自由基对心肌的氧化损伤,增强抗氧化酶活性,纠正PGI2/TXA2失衡等机制有关。

METHODS: In vivo I/R injury of rabbit was induced by blocking the hilum of left lung.

建立兔在体I/R损伤模型,实验分为对照组,I/R组和IPC组。

I/R could induce the increased AIF positive cell expression and nerve cell apoptosis.

I/R可诱导AIF阳性细胞表达增加,并引起神经细胞凋亡。

The staining of F-actin in the ischemia reperfusion group was decreased significantly (P.05) compared with the normal control group. There was a marked loss of canalicular microvilli and distension of canalicular lumen in the ischemia reperfusion group. The staining of F-actin in the LA group was significantly increased than those in the I/R group (P.05). Conclusions: Ischemia reperfusion induces an disruption of bile canalicular F-actin microfilaments and a loss of microvilli. Alpha-lipoic acid can protect bile canalicular F-actin microfilaments against IRI.

肝脏I/R可造成胆小管F-actin微丝破坏、微绒毛丧失,导致胆小管收缩减弱,胆汁排泄功能受损,这可能是大鼠肝脏I/ R后缺血型胆道病变发生的主要机制;α-硫辛酸通过清除氧自由基,降低脂质过氧化反应,调节细胞内氧化剂-抗氧化剂的含量,保护胆小管F-actin微丝结构免遭缺血再灌注损伤破坏,进而预防和治疗缺血型胆道病变。

3NE、MTT、LDH、ET-1、ICAM-1 and Tie-2experiment show: in the process of oxygen glucose deprivation/reoxygenation ,BMEC have subjected great damagement, I/R groups have significant differences compared with NC groups (p.05);polymorphonuclear leukocyte can aggregrate this injury, I/R+PMN groups have significant differences compared with I/R groups (p.05); after Sivelestat Sodium treatment, the effect of I/R injury has been decreased, I/R+PMN+Siv groups have significant differences compared with I/R+PMN groups (p.05), and NE、MTT、LDH experiment show that the three dose groups there was a significant dose-effect relationship.

3测定NE、MTT、LDH、ET-1、ICAM-1、Tie-2 mRNA均显示:氧糖剥夺/复氧复糖过程能对BMEC造成损伤,I/R组与NC组比较有明显的统计学差异(p.05);中性粒细胞能加重对BMEC的损伤,I/R组与I/R+PMN组之间比较亦有统计学差异(p.05);西维来司钠处理后,血管内皮细胞的损伤明显减轻,I/R+PMN组与I/R+PMN+SIR组比较有统计学差异(p.05),并且NE测定以及MTT、LDH实验提示在西维来司钠三个剂量组之间存在明显的量-效关系,其对BMEC的保护作用有剂量依赖性。

RESULTS: As compared with group I-R, Fas/FasL mRNA slightly expressed in intima and extima of small pulmonary artery, alveoli, and bronchiole epithelia in group Pur. The values of AI, W/D and IQA showed significantly lower than that in group I-R at 60, 180, 300 minutes after reperfusion in lung tissue (P.01 and P.05). Meanwhile, abnormal changes of the lung tissue in morphologically were lessen markedly in group Pur.

结果:肺再灌注1h、3h、5h, Pur组Fas/FasL mRNA在肺小动脉内膜、肺小静脉内膜、肺泡上皮及肺支气管上皮呈弱阳性表达,明显低于同一时点I-R组(P.05);AI、W/D和IQA值显著低于I-R组(P.01和P.05);肺组织形态学异常改变不同程度减轻。

Results: Both Pur and Hup-A improved learning memory ability of I/R mice, the effect of Pur group was better and the effect of combination group was the most significant. Both Pur and Hup-A decreased neuron apoptosis and raised content of monoamine nervous transmitter of I/R mice. The cortex neuron apoptosis rate in combination group was lower than that in Hup-A group,the homovanillic acid level of cerebral cortex in combination group was higher than that of Pur group.

结果:Pur(120 mg/kg)或Hup-A(400 μg/kg)都可提高I/R小鼠的学习记忆成绩,Pur组的作用优于Hup-A组,联合用药组(Hup-A 200 μg/kg+Pur 60 mg/kg)作用最强;各用药组都可降低大脑皮层细胞凋亡率,升高大脑皮层某些单胺类递质水平,其中,联合用药组的大脑皮层细胞凋亡率低于Hup-A组,大脑皮层高香草酸水平高于Pur组。

TSRP could decrease brain hydrated amount and markedly lower permeability of blood-brain barrier subjected to 2 h MACO followed by 24 h reperfusion, and this may be a mechanism of TSRP alleviating brain edema during I/R.

茅莓总皂苷各剂量组显著降低缺血2h再灌注6,24,48,72 h脑水肿和其对血脑屏障通透性,这可能是其减轻I/R时脑水肿的机制之一。

The results showed: there is the apoptosis of the myocardial cells during the period of I/R. The further study showed that there is the express of Bcl-2, Bax and Fas in the model of the cultured myocardial cells which were treated with ischemia-reperfusion The express of Bax, Fas proteins increase obviously. The overexpress of Bcl-2 is weak positive which shows that there is certain relationship between the apoptosis of the myocardial cells and the higher express of Fas and the ratio descent of the Bcl-2/Bax.

结果表明:在I/R过程中确有心肌细胞凋亡现象,而且进一步研究发现在培养的心肌细胞缺血-再灌注的模型中均见Bcl-2、Bax、Fas的表达,Bax、Fas蛋白的过度表达明显增加,Bcl-2的过度表达仅呈弱阳性,表明心肌细胞的凋亡与Fas的高表达,Bcl-2/Bax的比例下调有一定的必然联系。

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