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FN相关的网络例句
与 FN 相关的网络例句 [注:此内容来源于网络,仅供参考]

We obtain new lower bounds for the sum of degrees of simple and distinctirreducible factors of the polynomial f1 +···+ fn, where fi(1≤i≤n) are pairwise relatively prime polynomials of several variables with coefficients in C.

七我们对形如f1 +···+ fn的多项式的单重不可约因子的次数和及不同不可约因子的次数和的下界分别作出新的估计,其中fi(1≤i≤n)为系数在C中的两两互素的不可约多项式。

Result: 286 restrain the secretion of TGF-β〓 and decrease the quantity ofα-SMA in liver cells, and the accumulation of FN, LN, type Ⅰand Ⅲ collagen in ECM. The degree of liver cell damage was alleviated and the adverse effect what the purtenance received was lightened. The store function of liver was enhanced.

结果:"268方"可抑制TGF-β〓的分泌,减少α-SMA在肝细胞间存在的量,减少ECM之FN、LN、Ⅰ、Ⅲ型胶原等的沉积,同时可减轻肝细胞受损程度、增强肝脏贮备功能、减轻内脏受到的不良影响,从而实现预防及治疗慢性肝炎的目标。

FN and LN (the non-collagenous index) out of ECM were measured by immuno-histochemistry mean, and the accumulation of ECM was measured by typeⅠand Ⅲ collagen ,α-SMA was chosen as a symbol of the activated reposit fat cell. TGF-β〓 was chosen as a symbol of the cellular factor made the reposit fat cell activated. We observed the mechanism of 286 through the variety of the upper indexes during the experiment.

用免疫组化方法测评肝细胞外基质中的非胶原指标FN、LN,胶原指标Ⅰ、Ⅲ型胶原等在实验过程中的变化作为判定ECM沉积增多的事实指标;用a-平滑肌肌动蛋白作为贮脂细胞被激活的标志物指标;用转化生长因子-β〓作为激活贮脂细胞的细胞因子指标;通过观测上述指标在实验中的变化,来探讨"268方"的作用机理。

FN and LN(the non-collagenous index) out of ECM were measured by immuno-histochemistry mean, and the accumulation of ECM was measured by type I amd III coHagen, a -SMA was chosen as a symbol ef .the activated reposit fat cell.TGF- P1 was chosen as a symbol of th ieccellular factor made the reposit fat cell activated.We observed the mi sclnanism of 286 through the variety of the upper indexes during the experiment.

用免疫组化方法测评肝细胞外基质中的非胶原指标FN、LN,胶原指标Ⅰ、Ⅲ型胶原等在实验过程中的变化作为判定ECM沉积增多的事实指标;用a-平滑肌肌动蛋白作为贮脂细胞被激活的标志物指标;用转化生长因子-β_1(TGF-β_1)作为激活贮脂细胞的细胞因子指标;通过观测上述指标在实验中的变化,来探讨"268方"的作用机理。

The expressions of FN mRNA and TGFβ1 mRNA were detected by semiquantitative RTPCR.

用半定量RTPCR法检测该细胞中FN mRNA和TGFβ1 mRNA 的表达。

As applications, we prove the following results: If / has pointwise pseudo-orbit tracing property, for any k ∈ Z+, and fk is chain transitive, then for any k ∈ Z+, fk has open set transitive ; If f has pointwise pseudo-orbit tracing property, and for any n ∈ Z+,fn is chain transitive, then f has sensitive dependence on initial conditions; If f is open set mixing and has pointwise pseudo-orbit tracing property, then f has property P; Let f :→ be a homeomophism, then f is pointwise pseudo-orbit tracing property if and only if the shift map σf is pointwise pseudo-orbit tracing property.

作为应用,证明如下结论:若f具有逐点伪轨跟踪性质,且对任意k∈Z+,fk为链转换的,那么对任意k∈Z+,fk为开集转换;若f具有逐点伪轨跟踪性质,且对任意n∈Z+,fn为链转换的,则f具有初始敏感依赖性质;若f为开集混合的,且具有逐点伪轨跟踪性质,那么f具有性质P;设f:→是同胚映射,那么f具有逐点伪轨跟踪性质当且仅当移位映射σf具有逐点伪轨跟踪性质。

Methods The serum Fn and HA levels in 50 cases of HCC 14 cases of benign hepatic disease and 100 healthy persons were determined by enzyme-labelled turbidimetry and radioimmuoassay.

方法应用酶标仪比浊法及放免技术对50例肝癌病人血清Fn和HA测定,并以肝良性病变和正常人作对照。

The study showed the recombinant wt/mCREG protein depressed the VSMC proliferation depending on dose and the optimal concentration was 400nM;2biologic function of CREG protein and the membrance receptor mechanism:①effect on VSMC migration: the wound healing experiment showed the OB2 cells migration was slower significantly after added wt/mCREG(400Nm) in supernatant. The HITASY cells migration were very slowly and no remarkable change. The gelatinase digestion and Western blot analysis showed the matrix metalloproteinase was decreased and TIMPs was increased;②effect on differentiation: after added wt/mCREG(400nM), the expression of myocardin, SMα-actin, MHC and caldesmin were increased and that of LM-1 and FN were decreased in OB2 cells. These effects were more significant when adding wtCREG.;③effect on VSMC proliferation: Cell cycle assay and BrDU stain showed: after added the wtCREG and mCREG protein, the ratio of cell in G0/G1 phase increased to 0.5773 and 0.5572 from 0.5308 respectively in OB2 group, which increased to 0.7369 and 0.7034 respectively from 0.6297 in HITASY group;3Role of M6P/IGF2R in CREG biologic function:①ELISA and co-immunoprecipitation showed the wt/mCREG binding to M6P/IGF2R directly.②antibody blocking test: when the anti-IGF2R was added to medium at the same time with wt/mCREG at different concentration(2μg/mL、4μg/mL、8μg/mL),the effects of CREG protein which depressing proliferation, migration, secretion and promoting differentiation were blocked, which had the positive correlation to the concentration of added anti body. The studies showed two combinant CREG promoted VSMC switch to differentiation phaenotype, at the same time, depress VSMC proliferation, migration and secreting extracellular matrix.

上述实验结果证实:两种重组CREG蛋白对VSMC增殖均有剂量依赖性的抑制作用,并且相同浓度的糖基化的CREG蛋白对细胞增殖的抑制效应更为显著,最佳效应浓度为400nM;2两种重组CREG蛋白添加后对HITASY和OB2细胞生物学行为的影响:①CREG蛋白对VSMC迁移的影响:刮伤实验发现,加入最佳效应浓度的wtCREG和mCREG蛋白24h后,OB2组迁移能力下降,HITASY组无明显变化;细胞外基质金属蛋白酶-2,9(Matrix metallo-proteinase 2,9,MMP2 ,9)明胶酶电泳检测和Western blot检测结果证实,两种CREG蛋白均可以使OB2细胞合成细胞外基质MMP2,9减少,而组织金属蛋白酶抑制物(Tissue Inhibitors of Metalloproteinases,TIMPs)增加;②CREG蛋白对VSMC分化的影响:加入400nM的wtCREG和mCREG蛋白12h后,OB2细胞myocardin、SMα-actin、MHC、caldesmin表达增加,LM-1、FN表达减少;③流式细胞仪分析细胞周期和BrDU染色分析证实,加入400nM的wtCREG和mCREG蛋白后,OB2组G0/G1期细胞由0.5308分别增加至0.5773和0.5572,HITASY组G0/G1期细胞由0.6297分别增加至0.7369和0.7034;3M6P/IGF2R在重组CREG蛋白的生物学功能中的调控作用:①免疫共沉淀和免疫荧光双染色分析结果显示,CREG蛋白与M6P/IGF2R存在直接结合;②应用抗体阻断实验:将不同浓度的anti- M6P/IGF2R(2、4、8μg /mL)与两种CREG蛋白同时加入培养液中,CREG蛋白抑制VSMC增值、迁移和合成细胞外基质、促进分化的效应减弱,而且与加入anti- M6P/IGF2R浓度正相关。

The thickness of alveolar wall, the areas of mesenchyma in lung tissue were measured by HE staining. Immunohistochemical evaluation was to detect the expression of TNF-α, TGF-β1, FN protein.

灌药后第7、14、28天分组处死动物,提取肺组织,HE染色行肺泡炎、肺纤维化程度分级及免疫组化测定肺组织TNF-α、TGF-β_1、FN蛋白表达。

FN-301s is F-Si multi-functioned penetrating sealer designed for the special needs such as oil resistant, excellent water repellency.

FN-301s 是为有防油,出色防水性要求而设计的氟硅多功能渗透性防水剂。

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