查询词典 C-polymer

There are four kind of isoforms of endothelin in human and other mammals named ET-1, ET-2, ET-3 and ET-β, which are slightly different in construction and pharmacological effect. In human beings, ET-1 is the dominative subtypes. ET-1 remains in blood at a low level about 5ng per liter on physical state. It is synthesized, stored, released and metabolized locally. The half-life of ET-1 is about 1 hour. ET-1 is the most potent vasoconstrictive factor till now, and it is more functional in vein than in artery. In vascular bed, there are two kind of ET receptors. Type A mainly located in smooth muscle cells, whereas type B in endothelial cells. The latter can stimulate intimal hyperplasia via a parasecretion way and activate some oncogenes such as c-fos and c-myc and then enhance their expression. These alterations result in constriction of blood vessels, thus the SMC steps into proliferate state from silent state.

人及哺乳动物体内有四种结构及药理学性质略有差异的异物体，分别为ET-1、ET-2、ET-3、ET-β，而在人主要是ET-1，在生理条件下，ET-1在血浆中含量较低，约为5ng〓，故ET-1不是一个循环激素，而是局部合成释放，局部起作用的活性物质，半衰期约1小时，ET-1是目前已知的最强的血管收缩剂，对静脉的作用比动脉强，在血管床，ET受体有A、B两型，A型主要分布在平滑肌细胞，B型主要分布在内皮细胞，它可以通过旁分泌途径刺激内膜增生，具有有丝分裂原效应，可以激活某些癌基因如C-fos、C-myc使其表达增强引起血管收缩，使静止期SMC进入增殖期，还可以通过信号传导途径，与bFGF、GTF-β、PDGF等生长因子协同作用，起共有丝分裂原作用。

Methods The method of replenishing qi to reinforce the spleen and dissolving phlegm to remove turbidity was used,and eight periumbilical points (Tianshu,Huaroumen,Wailing,Yinjiao and Shuifen),points Guanyuan and Zusanli were selected in cooperation with auricular-plaster therapy.In the corresponding period,30 healthy persons were selected for comparison.Changes in FBG,INS,ISI,TC,TG,LDL-C and HDL-C were evaluated after acupuncture in simple obesity patients.Also were changes in waist circumference,hip circumference,waist-to-hip ratio,body weight and BMI,and differences in them between the curative effects on degree Ⅰ and degree Ⅱ obesity.

方法采用&益气健脾，化痰消浊&法，取脐周八穴(天枢、滑肉门、外陵、阴交、水分)、关元、足三里等穴，配合耳穴贴压，并与同期30例健康体检者比较，观察针刺前后单纯性肥胖症患者FBG、INS、ISI、TC、TG、LDL-C、HDL-C的变化；还观察针刺前后单纯性肥胖症患者腰围、臀围、腰臀比、体重、BMI的变化，及其Ⅰ度肥胖、Ⅱ度肥胖的疗效差异。

Methods Mammary adenocarcinoma EMT6 was cultured and propagated, then implanted on female BALB/c mice. Tea polyphenols was peroral administrated and local injected, C-Jun in tumor tissue was detected by immunohistochemical method.

应用小鼠可移植性乳腺癌EMT6细胞系，经培养传代后，以纯系BALB/c小鼠为荷瘤动物进行肿瘤移植，并采用茶多酚灌胃及局部注射两种干预措施，以免疫组化方法检测小鼠乳腺癌组织C-Jun表达。

After titanium was phosphated with H_3PO_4 and TEP, the elements of P, C, 0, Ti and the radicles of P-O-C,-PO_3,-PO_4 were detected by X-ray photoelectron spectroscopy and the Fourier transformed infrared spectroscopy. It presents that the phosphorus had been bonded onto the titanium surface with chemical combination.

纯钛表面经过磷化处理，通过X光电子能谱和红外光谱分析可知，表面分别检测到了P、O、C、Ti元素和P-O-C、-PO_3、-PO_4等原子团，说明磷化处理可以有效的使磷元素以化学键合的方式引入表面。

Moreoer, c-Fos and phospho–c-Jun protein expression was inhibited by PPARα agonists, indicating that PPARα ligands suppress OPN expression through negatie cross talk with AP-1–dependent transactiation of the OPN promoter.

另外，c-Fos及磷酸化c-Jun蛋白表达可被PPARα激动剂抑制，说明PPARα配体通过OPN启动子AP-1依赖型转录激活的负交互作用而抑制OPN表达。

Comparing the nucleotide sequences of 16S rRNA gene and ribosomal protein gene, we could find that these two strains have the highest homology of 99.4% and 99.8%, respectively. The nucleotide sequences of 16S rRNA gene for the CWB-YN strain shared higher homology (98.5%,98.9% and 98.0%) with phytoplasma strains in Tomato big bug 16SrI-A group, Western aster yellow (16SrI-B group and Clover phyllody (16SrI-C group). But it is obviously under 97.0% with other phytoplasma groups. The CWB-GZh strain shared higher homology (98.7%,99.1% and 98.3%) with phytoplasma strains in Tomato big bug(16SrI-A group ),Western aster yellow (16SrI-B group) and Clover phyllody (16SrI-C group).

苦楝丛枝病植原体云南株系与其它组16S rRNA基因序列的同源率进行比较，结果与16SrI-A组中的番茄巨芽病植原体(Tomato big bug，BB)、16SrI-B组中的西方翠菊黄化病植原体(Western aster yellow，SAY)和16SrI-C组中的三叶草变病植原体(Clover phyllody，CPh)同源率达最高，分别为98.5%、98.9%和98.0%，而与其它组的植原体16S rRNA基因序列的同源率均低于95%；苦楝丛枝病植原体贵州株系与16Sr I-A组的番茄巨芽病植原体(Tomato big bug，BB)、16Sr I-B组的西方翠菊黄化病植原体(Western aster yellow， SAY)和16SrI-C组中的三叶草变叶病植原体(Clover phyllody，CPh)的同源率达最高，分别为98.7%、99.1%和98.3%。

The vibrational spectra of two isotopic derivatives of γ-picolinic acid (-C^1^6O~2D,-C^1^8O~2H) have been predicted with the SQM force field resulting from the transferred scaling factors. The calculated isotopic shifts are in good agreement with experimental data.

用这组校正因子得到的力场预测了γ-吡啶甲酸的两个同位素衍生物(-C^1^8O~2H和-C^1^6O~2D)的振动光谱，所得同位素位移值与实验数据符合良好。

Methods The pathological features of EPS were observed in 17 cases, and the expressions of HCG, HPL, EMA, PRL, PLAP, Vim, actin, c-erbB-2 and c-myc were analyzed immunohistochemically.

对17例经病理检查诊断为EPS的病例进行病理学形态观察，采用免疫组化方法标记滋养细胞HCG、HPL、EMA、PRL、PLAP、Vim、actin、c-erbB-2和c-myc。

Results The neurological defects score was singificiant lower in D group (41.50±8.18) than that in group A(82.20±8.08), B(63.60±6.22) and C (61.00±6.58)(all P.01). Compared with groups A, C, group D showed that the cellular nucleus was shrinkaged slightly, the gap of cells was nomal roughly and the degree of rarefaction in neurofibra net was not serious under the light microscope. Under the electron microscope, the neuron was degenerated, the cellular membrane was integrated and the plastosome was swelled lightly in the group D. The physiological demage were slighter in D group.

结果 D组神经功能缺损评分(41.50±8.18)明显低于A组(82.20±8.08)、B组(63.60±6.22)和C组(61.00±6.58)（均P.01）；光镜下D组神经细胞核轻度皱缩，细胞周围间隙大致正常，神经纤维网疏程度不严重；电镜下D组神经元轻度变性，细胞核无皱缩，核膜完整，线粒体轻度肿胀；脑组织病理损害程度较B组和C组明显减轻。

Outcome:(1) Acute ischemic damage may induce the expression of c-fos protein in Cerebral Cortex quickly. Also, the expression increases by the ischemia time delay. It may hint that ischemia injury can rise the ability of nerve cells to tolerate ischemia and anoxia. And the expression level of c-fos can be up-regulated by putting blood of 12 hand Jing Points. So we think that treated by this therapy can rise the irritability of brain to resist the development of ischemia and enhance the ability to plerosis.(2) Acute ischemic damage may induce the expression of HspVO in Cerebral Cortex quickly. Also, the expression increases by the ischemia time delay. It may hint that ischemia injury can rise the ability of nerve cells to tolerate ischemia and anoxia.

三 结果(1)急性缺血性脑损伤可快速诱导c-fos蛋白在皮层缺血区脑组织的表达，其表达有随缺血时间延长而逐渐增多的趋势，提示脑的缺血性损伤可应激性地增强神经细胞对缺血、缺氧的耐受和适应能力，而应用井穴放血的干预治疗可明显上调缺血区皮层脑组织c-fos的表达水平，进一步增强脑组织对抗缺血性损伤的应激能力，抵抗缺血性脑损害的进一步发展。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。