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The results Showed that twenty isolates have the following biological aspects:① Capability of forming capsule,② Growth at 37℃ within 3days,③ Forming brown colonies on Niger seed agar, CACA agar,④ Urea hydrolysis test positive within 96 hours.⑤ No blue colonies were found on CGB agar.⑥ The genus could be identified by ID32 identification kid, but the species could not.⑦ The sequence of the LSUrDNA D1/D2 region analyzed confirmed to be C. neoformans var neoformans.⑧ Serotype A and AD were found in these isolates, Serotype A was predominant(83.33%),⑨ Some biodiversities of isolates existed.

结果 20株菌皆具有以下特征：①形成英膜，②37℃3天内生长试验皆为阳性，③咖啡酸玉米琼脂和油菊籽培养基皆可以产生褐色菌落，④尿素酶试验96h皆呈阳性，⑤CGB培养基未见蓝色菌落生长，⑥ID32鉴定板可以鉴定到属，种的鉴定结果各不相同，⑦大亚基核糖体DNA的D1/D2区域序列分析证实皆为新生隐球菌的新生变种，⑧血清型仅见A和AD二型，以A型为多数(83.33%)，⑨20株临床分离株随机扩增DNA多态性分析有一定的生态多样性。

Then we examine whether NMDA receptor is involved in the protective effect of carnosine on Aβ42-induced neurotoxicity because of the important role of NMDA receptor in AD.

由于NMDA受体在AD病理过程中的重要地位，本实验进一步检测NMDA受体的表达变化。

Meanwhile, as a putative neurotransmitter in the brain, carnosine has a protective effect in many diseases such as AD, because of its abundant functions such as an anti-oxidant, in anti-glycation, anti-aging.

同时，有研究表明肌肽作为脑内一种重要的神经递质，对多种神经系统疾病如AD具有保护作用。

In this paper, in order to solve the load balance problem of Ad hoc network, works on different MAC protocol performance analyse, theoretical modeling, multi-path routing load balance protocol and cocurrent multipath transfer protocol are represented.

为了满足自组织网络对于负载均衡传输的需求，本文重点对解决Ad hoc网络负载均衡相关问题在MAC层协议性能分析，理论建模以及适用于负载均衡的新型路由协议和新型传输协议等几个方面展开了研究。

AD-AM is familial involve velar confluence, cell factor and grow the control with the desquamation of factor, migratory cell, and the decision that certain course is like muscle development, be fertilized and cellular ending.

AD-AM家族涉及到膜融合、细胞因子和生长因子的脱落、细胞迁移的控制，以及某些进程如肌肉发育、受精和细胞结局的决定。

A copyright was written before me before, collect fees the article that station and free station are faced with heaven and earth, although inserted the Ad that publicizes octave stationmaster net among them, but, because that is me,do station of 3 years of films to realize the thing that give, it is the experience that the summary in I and other center communication comes out, so, the center that it has implication with copyright to be being done people have very good directiveness, after that article is given out, the QQ that 20 no less than stationmaster add me directly seeks advice from relevant matters concerned.

我前面写了一篇版权面前，收费站和免费站面临天壤的文章，尽管其中插入了宣传八度站长网的ad，但是，由于那是我做三年电影站悟出的东西，是我和别的站长交流中总结出来的经验，所以，它对正在做与版权有瓜葛的站长们具有很好的指导性，那篇文章发出后，有不下20个站长直接加我的QQ咨询相关事宜。

Objective: To explore the inhibition effects of ectogenic wild-type p53 cDNA (Ad-wtp53) on colorectal carcinoma cell lines with different p53 gene status and search for the role of wild type p53 tumor suppressor gene in occurrenc and progress of malignant tumor.

目的：探索重组腺病毒介导的外源性野生型p53cDNA(Ad-wtp53)对内源性p53基因状态不同结直肠癌细胞的抑制作用是否存在差异，研究p53基因抑制肿瘤细胞增殖的机制。

The cells expressing nAchR clearly decrease in the areas of CA1 and cortex of rats when their fimbria/fornix is transected, but the cell number increases significanly if ectogenic estrogen is added. The change in nAchR is closely relative with AD whose pathological course can be influenced by the intervention of estrogen.

穹隆海马伞切断组大鼠脑内海马CA1区、皮层区nAchR的阳性细胞显著减少，补充外源性雌激素治疗后，上述各脑区内的nAchR表达显著增加。nAchR的变化与AD的病理变化过程密切相关，补充雌激素可以干预这种病理过程。

Objective To investigate the effect of Ad-METH1 on endotheliocyte secretion activity and approach the possible mechanism of preventing hypertrophic scar by angiogenesis inhibition.

目的：研究基因重组血管抑制剂Ad-METH1（metalloprotease and thrombospondin1）对培养的内皮细胞分泌活性的影响，探讨血管生成抑制防治增生性瘢痕的可能机制。

Inhibition of Ad-METH1 on secretion of ET-1 and bFGF from endotheliocyte might have act an important role in preventing the form of hypertrophic scar.

Ad-METH1对内皮细胞ET-1、bFGF分泌活性有影响，此是其早期参与抑制增生性瘢痕形成的可能机制之一。

But we don't care about Battlegrounds.

但我们并不在乎沙场中的显露。

Ah! don't mention it, the butcher's shop is a horror.

啊！不用提了。提到肉，真是糟透了。