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ARG相关的网络例句
与 ARG 相关的网络例句 [注:此内容来源于网络,仅供参考]

XEX ~ Arg min{FIy E ~2} where the upper level is about topological structure optimization(a~ is a topological structure varable), and the lower level is a constrained programming about continuous variable y. 2 Apply graph, the action of permutation group on a graph, equiva- lent class, orbit etc.

取得的主要成果有: 1 依点到集的映射、集值映射、二元映射和分离定理等理论首次建立了具有拓扑结构双层规划的数学模型 min F s.t.x∈X y∈Arg min{F|y∈Ω}其上层规划为关于离散变量x的拓扑结构优化,下层规划是关于连续变量的约束规划。

As a model peptide, Arg-Tyr-AcOH was also investigated in its interaction with deprotonated radical cations of purine.

作为肽的模型,对Arg-Tyr-AcOH与脱质子的嘌呤自由基之间的相互作用也进行了研究。

L-Arg, L-NNA and aminoguanidine had no effect on the iNOS mRNA expression.

而L-Arg、NOS抑制剂L-NNA及氨基胍均不影响胰腺组织iNOS基因水平的表达。

L-Arg(1 mmol/L)has a positive inotropic and chronotropic effect on isolated rat cardiac function.

L-Arg 1 mmol/L对大鼠离体心脏功能有正性变时,变力作用。

The third arg is the maximum limit to the growable size.

第三arg是最高限额可增长的大小。

In the L-Arg group, the amount of NO and SOD increased significantly (P.01, P.01), whereas that of MDA decreased (P.05), and the damage in the nephric tissue and renal function was alleviated,as compared with the control group.On the contrary, the amount of NO in the L-NAME group decreased significantly (P.05) compared with the control group.

给予L-Arg组大鼠肾组织中NO含量较对照组增加显著(P.01),SOD活性增高(P.01),MDA水平降低(P.05),肾功能改善,肾脏组织结构损伤减轻。

Results: Small round or linear haemorrhagic erosions were induced in the oxyntic glandular area after rats exposed to WRS. Meanwhile, GMBF decreased and NO contents in plasma and gastric mucosa increased significangly compared with those without WRS. L-NAME not only further decreased GMBF and inhibited the increases of NO contents but also aggravated the extent of gastric mucosal lesions. In contrast, L-Arg increased both GMBF and NO contents resulting in the lessening of mucosal injuries. All these changes were significant compared with control group.

结果:浸水束缚应激4 h可致严重的胃粘膜损伤,同时粘膜血流量明显下降,血浆及粘膜NO含量明显升高,与正常对照组相比差异非常显著(P<0.01);L-NAME明显加重粘膜损伤,并阻抑了应激所致的血浆及粘膜内NO含量的升高,使粘膜血流量更趋下降,与实验对照组相比差异显著(P<0.05);L-Arg可明显升高粘膜血流量及血浆、粘膜内NO含量,明显减轻粘膜损伤程度,与实验对照组相比差异非常显著(P<0.01)。

The effect was attenuated by preadministration of L-arg.

提示L-arg:NO通路参与睡-醒周期节律的调节。

L-Arg therapy in rever sing acute hypoxia is better than Dex therapy.

L-Arg较地塞米松能改善急性缺氧的病理生理反应。

L-Arg-NO pathway may tonically inhibit blood pressure,which may be mediated through r-aminobutyric acidreceptor.

结论PAG内L-Arg-NO通路对动物血压有紧张性抑制作用,可能是通过GABA受体介导。

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