查询词典 AHR

Their data demonstrated that IL-9's effect on AHR depended on the proliferation and maturation of mast cells.

他们的数据证实：IL9对AHR的作用，依赖于肥大细胞的增殖和成熟。

We have investigated whether AhR involved in the effects of BP on hypoxia-induced angiogenesis.

因此，本篇的研究目的欲探讨在苯芘前处理的人类脐静脉内皮细胞是否透过AhR去影响缺氧状态下的血管新生作用。

Overall, expression of the AhR protein apparently is involved in adipocyt differentiation.

综合上述观察，说明了 AhR 基因参与调控脂肪细胞分化的可能性。

These results suggest that AHR effects are dependent on Kv channel slow inactivation.

这样的结果说明了AHR阻断Kv1.2电流的效果需依赖C-型钝化的加速。

Besides, we also used AhR shRNA to observe the effects of BP on the hypoxia-induced tube formation.

此外，也利用AhR shRNA将人类脐静脉内皮细胞进行基因静默的方式来进行实验，观察AhR静默后对於苯芘所减少缺氧状态下的管状构造形成是否有影响。

In addition,% block of Kv currents by AHR was independent of the intracellular K+ concentration.

实验结果显示，在细胞外侧加入AHR可加速Kv电流C-型钝化的现象，并且其作用必须是在通道开启的阶段，而自细胞内加入AHR并不能加速电流的C-型钝化。

Besides, AhR shRNA also can reverse the inhibition of BP on the hypoxia-induced tube formation.

此外，利用AhR的拮抗剂alpha-NF及AhR shRNA可以回复苯芘所减少缺氧状态下所诱导的管状构造形成。

The toxicity of dioxins is dependent on their affinity to AhR. And if this affinity data would be gotten, we could know their harm degree and have ability to analyze and control these toxins quickly and effectively.

Dioxins与受体AhR相互作用的强弱决定着化合物毒性的大小，如能获得化合物对AhR的亲和性，就可以了解其危害程度，从而为快速有效对毒物进行分析及控制提供条件。

As well, neither AhR nor CYP1B1 overexpression was associated with cancer survival of lung adenocarcinomas.

从肺腺癌存活与AhR或CYP1B1过度表现分析，AhR或CYP1B1蛋白质表现与肺腺癌预后无关。

In response to TCDD exposure, AhR nuclear translocator forms a heterodimer with the aryl hydrocarbon receptor after the ligand-dependent translocation of AhR to the nucleus, and eventually the complex binds to the xenobiotic response elements, which promote transcription of several specific genes, such as CYP1A1.However, the diverse effects of TCDD could not be simply explained by the AhR pathway.

对於戴奥辛的毒性机制，以往的研究认为与芳香基碳氢化合物受体（arylhydrocarbon receptor－AhR）的活化有关；TCDD 活化了AhR，被活化的AhR 从细胞质转移到细胞核内，在细胞核与另一个蛋白质AhR nuclear translocator形成二聚体，并与DNA 上的xenobiotic regulatory element结合，诱发某些基因（如：CYP1A1）的转译与表现；不过，单从AhR的代谢途径并不能解释所有生物上的毒性现象。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。