髓周膜

基本解释 (translations)

perimyelis

与髓周膜相关的网络例句 [注：此内容来源于网络，仅供参考]

The notable proliferation was not observed by eyes in the local of injection. The infiltration of inflammation cells and mild proliferation of fibrocyte around dura mater was observed by HE stained in 4 and 8 weeks after injection. Infiltration and exudation of inflammation cells was observed by HE stained in epidural nerve root. Compared with group A, no changes of group B, C and D were observed under specific stained. Proliferation of type Ⅱ collagen fibers around dura mater was seen under immunohistochemical stained in 4 and 8 weeks after injection. There is no significant demyelination changes under LFB stained. The thickness and shape of the myelin sheath in epidural nerve root was not regular under transmission electronic microscopy in 4 and 8 weeks after injection. Fibroblast was also seen there. In nerve endometrium, macrophage could be seen under TEM, myelinated nerve fiber changed significantly, but nonmyelinated nerve fiber changed mildly. When 8 weeks, the changes of group D is smaller than the group B and C.

给药局部肉眼观察未见明显的纤维组织增生；HE染色可见B、C、D三组给药后四周及八周时硬膜内外均有炎细胞浸润，纤维细胞轻度增生，硬膜外神经根内有炎细胞浸润及炎性渗出；特殊染色B、C、D三组同A组相比未见有脊髓及神经根的改变；免疫组化染色，给药后四周及八周时，硬膜内外均有Ⅱ型胶原纤维增生；固兰染色B、C、D三组未见有明显脱髓鞘改变，与A组相比无明显异常改变；电镜观察B、C、D三组在给药后的四周及八周时，表现为硬膜外神经根内髓鞘厚薄不一，形状不规则，可见成纤维细胞，神经内膜中可见有巨噬细胞；粗大的有髓神经纤维变化明显，无髓神经纤维受累较轻；八周时电镜下D组改变较B、C两组为轻。

Pathological examination showed the obvious edema in myelin sheath,axons and perineurium in 2 w group,and disintegrated myelin sheath,degenerated axons and hyperplastic fibrous connective tissues in the other model groups.

病理学观察：卡压2周组神经束内髓鞘、轴索和神经束膜纤维出现明显水肿，余卡压组可见髓鞘崩解、轴索变性，纤维结缔组织增生逐渐明显。

The intact secretory granules (100 -300 nm) coated with membrane existed not only in the endings of the unmyelinated nerve fibers but also occasionally in perivascular space.

完整的大型膜包分泌颗粒(100～300 nm)不仅大量存在于无髓神经纤维内，而且也见于血管周隙内。

Methods: 50 normal Wistar male rats were randomly divided into five geoups: the sham operated group, injury model group, shenjindan capsule treat group, high dose and low dose group (duzhongyaotong pill treat, D and E)(n=10each). Autogenous nucleus pulposus was removed from the coccygeal vertebral and placed into the back of left L5~6 nerve root, The modle of the non-compressive nucleus pulposus protrusion was created by transplanting autogenous nucleus pulposus in the back of left L5~6 nerve roots. At tow weeks, the SP in dorsal root ganglion was determined by immunohistochemistry.

取健康Wistar大鼠50只，随机分为假手术组、模型组、对照组、高剂量组（D组、腰腿理痛散高剂量组）、低剂量组（E组、腰腿理痛散低剂量组），每组10只，将大鼠自身的尾椎髓核取出移植于左侧硬膜外腔L5、L6神经根背侧，造成非压迫性大鼠自体髓核移植硬膜外模型，通过免疫组化的方法测定大鼠2周时DRG中CGRP的变化。

We compare it with the Votalin treating that have been proved effective on clinic during the experiment, and detect intraosseous pressure in the upper part of tibia and the content of nitrogen monoxidum in articular cartilages、 synovial membranes、 synovias of knee joint, we can initially identify the mechanism of action that this theropy treat on gonarthritis, supply the theoretical and experimental evidence for making general use of it on clinic.

以致骨内高压的方式造模，于兔腹股沟中点以下10mm处结扎并切断右侧股静脉建立兔膝骨关节炎模型，造模10周后各组采用相应的方法进行治疗，4周后将骨髓活检穿刺针穿刺达胫骨上端髓腔内，以三通管与传感器相连检测骨内压，后将动物处死，取膝关节软骨、滑膜、关节液，检测一氧化氮含量。

Results:According to their anatomic location and vascular morphology,there were SDAVF in 3 cases,giant perimedullary AVF in one and intramedullary AVM in two.

结果：根据病变位置和血管形态、血流动力学改变，本组有3例符合硬膜动静脉瘘，1例为髓周的巨大AVF，2例为髓内型。

Results: According to their anaomic location and vascular morphology, there were SDAVF in 3 cases, giant Perimedullary AVF in one and intramdullare AVM in tWo.

结果：根据病变位置和血管形态、血流动力学改变，本组有3例符合硬膜动静脉瘘，l例为髓周的巨大AVF，2例为髓内型。

Materials and Methods: Of 62 patients with spinal AVMs which confirmed by MRI, DSA and/ or pathology, 30cases had intramedullary AVMs(13 occult AVMs, 5 glopular AVMs, and 12 juvenile AVMs), 24 cases had perimedullary AVF (5 type I , 12 type Ⅱ, and 7 type Ⅲ), and 8 cases had dural AVF.

材料和方法：收集了62例经MRI、DSA和手术病理证实的髓血管畸形患者，其中髓内隐匿型动静脉畸形13例、球形AVMs 5例、幼稚型AVMs 12例：髓周膜内动静脉瘘I型5例、Ⅱ型12例、Ⅲ型7例；硬膜型AVF8例。

Results: The male patients appeared more than females in spinal cord AVMs, but the occult intramedullary AVMs had the same morbidity The patients with intramedullary AVMs and perimdeullary AVF were younger, in contrast, patients with dural AVF were usually older than 40 years of age. The intra - medullary AVMs were most common located in the cervical and thracic - lumbar spinal cord, the perimedullary AVF and dural AVF were most seen in the low thracic of lumbar region. The most common initial symptom associated with intramedullary lesions was accute onset where as the progressive aggravation was the most common presenting symptom in cases of dural AVF. The prognosis of the patients with intramedullary lesions had hemorrhage was worse than who without hemorrhage.

结果：脊髓AVM s中，隐匿型AVMs无明显的性别差异，余各类型男性均多于女性；髓内AVMs、髓周AVF以青少年多见，硬膜型AVF发生于中老年；髓内AVMs多见于颈髓及胸腰段脊髓，髓周AVF及硬膜型AVF多见于胸腰段；髓内AVMs以急性起病多见，髓周AVF表现为进行性加重，少数可急性发作，硬膜型AVF以慢性起病多见；髓内出血者预后较无出血者差。

While the tissue spaces surrounding a few blood vessels wasAl and Fg positive,no Al or Fg positive cells were observed.In antemortem injurygroup,diffuse subarachnoid hemorrhage,cerebral edema,swelling or pyknotic neu-rons could be observed.The axons showed irregular swelling and disconnection at1～3h,marked swelling and disconnection at 6h,and retraction ball at 15h whichwas more remarkable at 24h after injury.The space between myelin sheaths andaxons was increased at 3～6h after injury.Tortuous and wavelike myelin sheathswhich adhered on axons incompletely,or even peeled off could be found from 15hto 24h after injury.Perinuclear lysis of Nissl bodies began at 24h after injury.Thenumber of GFAP positive cells in cerebrum and brain-stem increased significantlyfollowed by decrease,and then increased again,but the time courses of the changesin different areas of brain were not same.Al and Fg positive neural cells,mainlysurrounded blood vessels,with diffuse or peripherally distributed positive matter incytoplasm could be observed at 0.5h after injury.The number of Al or Fg positivecells and the intensity of immunoreaction increased with the time of injury.The areaof SYN positivity in medulla oblongata and pons decreased notably 3～6h afterinjury,then return to normal levels and continued to 24h after injury.

生前损伤组，可见广泛蛛网膜下腔出血，脑组织水肿，神经细胞肿胀，晚期神经元固缩；伤后1～3h见部分神经轴突不规则增粗、断裂，伤后6h断端膨大，伤后15h可见收缩球，至伤后24h更为明显；伤后3～6h可见部分神经髓鞘与轴突之间的间隙增宽，伤后15h髓鞘明显曲折，不完全附着在轴突两侧，甚至剥脱，持续到伤后24h；核周尼氏体减少在伤后24h才开始出现；同一部位的GFAP阳性细胞数目随损伤时间发生改变，先增多（最早在伤后0.5h），达到高峰后减少，其后又有增多趋势，但不同部位的GFAP阳性细胞数目增减的时间过程不尽相同，同时，大脑中的GFAP阳性细胞数目也有改变；伤后0.5h，可在脑干组织中见到Al和Fg阳性神经细胞，主要位于血管周围，阳性物在胞浆中呈弥散性分布，但部分细胞的阳性物仅分布于靠近胞膜的胞浆中而呈环状，随损伤时间延长，阳性细胞数目增多，反应强度增加；伤后3～6h，延髓及桥脑中的SYN阳性物面积减少，其后恢复到正常水平，并持续到伤后24h。