门静脉

基本解释 (translations)

janitrix

词组短语

portal vein · vena portae

与门静脉相关的网络例句 [注：此内容来源于网络，仅供参考]

Oeeki Y reported a method of partial arterialization of portal vein with ileo-colon arteriovenous anastomosis in 1997, he thought this can prevent theliver failure caused by embolism of hepatic artery after liver operation.

但过量动脉血(门静脉内动脉血灌流量达到门静脉正常血流量的一倍以上时)灌流肝内门静脉则对肝脏有害，如导致肝内门静脉分支增宽，内膜纤维变性，以及肝细胞微粒体酶活力的改变。

Methods Eight-four rats were randomly divided into 7 groups. PDR2/TK or buffer TE was injected through the artery/portal vein of the left live lobe. Ganciclovir or normal saline was injected intraperitoneally.

84只大鼠随机分为动脉TK、动脉TK+更昔洛韦、动脉对照、门静脉TK、门静脉TK+GCV、门静脉对照及空白对照等7组。

Methods The portal venous flow and hepatic arterial flow were measured with Doppler ultrasonic instrument in 25 patients with portal hypertension.The free portal venous pressure was measured by manometric method.The hepatic function was measured by indocynine green retention rate at 15 minutes during perioperative period.

应用超声多普勒分别检测25例门静脉高压症患者手术前后门静脉血流量、肝动脉血流量，采用水柱法测量自由门静脉压力的变化，应用吲哚氰绿15分钟潴留率(R15ICG)分别估测患者手术前后的肝功能。

Results Spiral CT features of cavernous transformation of portal vein were mainly showed: main portal vein or branch expanding,circuiting,mussy;one to pass abnor dabble in liver parenchyma at arterial phase; primary lesion.

结果 门静脉海绵样变性的螺旋CT主要表现为：门静脉主干或其分支扩张增粗，门静脉走行区可见迂曲扩张、紊乱的静脉血管影；肝实质动脉期一过性异常灌注；显示原发病灶。

Results Along with the formation of liver cirrhosis and PHG, the gastric mucosa became thicker and more congestive. The portal pressure elevated gradually, followed by the decrease of endothelin-1 levels in both the peripheral and the portal blood. ETA mRNA expression was significantly elevated in the postcapillary venule and the precapillary arteriole, and predominantly in the latter.

结果：Wistar大鼠在肝硬化门静脉高压形成过程中，其胃黏膜厚度增加，黏膜淤血；门静脉压力呈梯度性升高；外周及门静脉血浆ET水平随门静脉压力升高而降低；胃黏膜ETA mRNA主要在黏膜基底部的微循环前微动脉和后微静脉血管壁表达，其表达水平随ET浓度的降低和门静脉压力的升高而增加，且其在小动脉表达明显强于小静脉。

Radiography of veins or a vein after injection of a radiopaque substance.

门静脉门静脉的，门静脉系统的，与门静脉系统有关的

And There was no serious complication;②In the course ofinterventional therapy, Direct portal vein angiography demonstrated vena coronaria ventriculi(100%)andgastricveins(65.26%)andvenagastricaposterior38.43%, Angiography demonstrated venacoronariaventriculi communicate esophagus varicose veins, gastric veins and vena gastrica posterior communicategastric varicose veins. vena coronaria ventriculi had only a small percentage of double vein, about30.57%. The sites of vena coronaria ventriculi arising from the portal vein, splenic vein, portosplenic junction, were found in 52.06%、27.39%、20.55% respectively.③12 extrahepaticprotosystemic shunts were found in these patients. Include gastro-nephrosshuntof 3 cases, 7 caseswere splenetic- nephros shunt and 2 cases shown recanalization of umbilical vein .④The averageportal pressure before and after the procedure were 3.87±1.82kPa and 3.64±1.14kPa in 73patients, but to the time of rebleeding, it was 3.96±0.23kPa in the 11 cases.⑤There werethree kinds of variceal outcome: disappearance (54,low degree (19).⑥Spearman logisticanalyse and ANOVAtest shown liver function class, variceal degree of the splenic necrosis area,the blood direction in portal vein before operation and remain small collateral routes were thesignificant factors concerning outcome of varices.⑦The bleeding volume and portalhypertensive gastropathy are main risk factors of rebleeding.⑧The course of livercirrhosis is the risk factor of survival and extrahepatic portosystemic shunt , fine varices are thebeneficial factors to survival.⑨During all cases'followed-up data, the 1, 2, 3, 4, 5 yearcumulative survival rates and rebleeding rates were 17.81%, 28.77%, 38.36%, 43.84%, 47.95%and93.15%,91.78%,86.30%,83.56%,80.82%respectively. Conclusion The interventional disconnection treatment for liver cirrhosis and portalhypertension was designed suitability. It rapidlycontrol bleeding,butpressure of portal vein was notobvious high, perfusion was not low .it was compared with surgery therapeutic that interventionaldisconnection treatment was safe and had a significant clinical effect to hemorrhage and preventfrom rebleeding.

结果：①术后一过性发热62例(84.9%)，腹痛腹胀48例(65.8%)是介入断流术常见的并发症，未发生严重并发症；②门静脉造影显示胃冠状静脉、胃短静脉和胃后静脉的曲张分流的出现率是100%、65.26%和38.34%；显示食管静脉曲张主要由胃冠状静脉供血，胃静脉曲张主要由胃短静脉和胃后静脉供血；胃冠状静脉大多数为单支，少数为双支，其双支的出现率分别为30.57%；胃冠状静脉开口于门静脉主干的为52.06%，开口于脾静脉主干的为27.39%和开口于门脾静脉交汇处的为20.55%；③发现胃肾分流3例，脾肾分流7例、腹膜后门腔静脉分流2例，以及CTA检查发现脐静脉开放者2例；④73例患者介入断流术前和术后平均自由门静脉压力分别为3.87±1.82kpa和3.64±1.14kpa，前后比较存在显著性差异；11例再次介入手术患者的术前、术后和复发后的自由门静脉压力分别为4.02±0.24kpa、3.82±0.25kpa和3.93±0.23kpa ，前后比较发现首次术前与术后存在显著性差异，首次术前和复发出血术前门静脉压力比较无显著性差异；⑤介入术后复查曲张静脉转归基本消失54例，轻度19例；⑥Spearman相关分析和Logistic多因素回归分析，肝功能分级、静脉曲张程度、门脉血流方向和残存小侧支四个因素对曲张静脉转归有影响；Spearman相关分析和Logistic多因素回归分析门脉高压性胃病和出血量等因素对复发出血时间有影响；⑦COX回归分析，门体分流和曲张静脉转归两个因素对术后生存有影响；⑧术后随访6-70月，1、2、3、4、5年的累计复发出血率和累计生存率分别为17.81%、28.77%、38.36%、43.84%、47.95%和93.15%、91.78%、86.30%、83.56%、80.82%；结论：介入断流术治疗门脉高压食管胃底静脉曲张有独特的优点，可以快速直接控制曲张静脉出血而门静脉压力无显著增高，保证了肝脏灌注；与外科分流术相比适应证广、损伤轻、术后恢复快，不易遗漏曲张静脉；肝功能分级、曲张静脉程度、门脉血流方向和残存侧支血管对食管胃曲张静脉转归有影响；门脉高压性胃病对复发出血时间有影响；门体分流和曲张静脉转归对生存时间有影响。

The first to tertiary ramification of portal vein and their significance. The first order branch of portal vein had four patterns: namely, normal branching pattern, 90 cases (90%); trifurcation, 5 cases (5%); independent branching of right posterior branch from main portal vein, 4 cases (4%); and complicated branching pattern, 1 case (1%).

门静脉第一到第三级分支的解剖和变异门静脉第一级分支有四种类型，即正常分支型，90例（90％）；右前支、右后支和左门静脉同时分支型，5例（5％）；右后支独立分支型，4例（4％）；及复杂分支型，1例（1％）。15例（15％）右门静脉不形成单独右后支，而是右后下支P6和右后上支P7分别从右门静脉分出，这种变异并非少见而文献中却很少描述。

Portal braches of 4-5 orders were found in all 19 cases of Child Grade I, with esophageal varices and paragastric varices and no fistula of hepatic artety-portal vein or portal vein embolus; Portal braches of 3-4 orders were displayed in all 16 cases of Child Grade II, besides esophageal varices and paragastric varices, cirsomphalos, retroperitoneal varices, paraesophageal varices, dilated azygos vein were also noted, some patients had fistula and portal vein embolus; in Child Grade III, portal branches of 3~4 orders were displayed with more collateral circulations, dilated left renal vein and paragallbladder varices were appeared, with fistula and portal vein embolus or not.

Child I级19例，肝内门静脉显影4~5级，侧枝循环以食管及胃周静脉曲张为主，没有肝动脉－门静脉瘘或门静脉血栓形成；Child Ⅱ级16例，肝内门静脉显影3~4级，侧枝循环除食管及胃周静脉外，脐周静脉、腹膜后静脉、食管周围静脉、奇静脉也有所开放，部分有肝动脉－门静脉瘘或门静脉血栓形成；Child Ⅲ级6例，肝内门静脉显影3~4级，侧枝循环开放较Ⅱ级增多，可出现左肾静脉扩张或胆囊周围静脉曲张，伴或不伴有肝动脉－门静脉瘘或门静脉血栓形成。16排螺旋CT门静脉造影对上述病例分级为一级17例，二级18例，三级6例，与Child分级结果高度相关。

This operation is suitable for the following portal hypertension: simple occlusion of hepatic vein (a pathological type of Budd-Chiari syndrome)thrombosis in portal vein or prehepatic portal hypertension due to cavernous transformation; intrahepatic portal hypertension with rebleeding after treatment of splenectomy or without operation, and the patients with liver function in Child A or B class.

本手术主要适应于以下原因引起的门静脉高压：①单纯化肝静脉闭塞(布-加综合征的一种病理类型)；②门静脉主干血栓形成成海绵样变性引起的肝前性门静脉高压；③肝内型门静脉高压行断流术后再发出血或未行手术，肝功能为Child A和B级者。

与门静脉相关的网络解释 [注：此内容来源于网络，仅供参考]

portal hypertension：门静脉高压

门静脉高压(portal hypertension)是一组由门静脉压力持久增高引起的症候群. 绝大多数病人由肝硬化引起,少数病人继发于门静脉主干或肝静脉梗阻以及一些原因不明的因素. 当门静脉血不能顺利通过肝脏回流入下腔静脉就会引起门静脉压力增高.

portal vein：肝门静脉

在正常人中,食道及胃的静脉血管是经肝门静脉(Portal Vein)跑到肝脏的. 当肝脏因肝炎或其它因素导致肝硬化时,肝门静脉血管内的血压便会上升. 正常肝门静脉血压是3mmHg,当它上升到12mmHg时,食道及胃的静脉便慢慢地曲张,最后会破裂而出血,

Portal vein embolization：门静脉栓塞

门静脉栓塞:Portal vein embolization | 门静脉癌栓:Portal vein tumor thrombi | 选择性门静脉栓塞:selective portal vein embolization

portal pyemia：门静脉脓血症

门静脉高压, portal hypertension? | 门静脉脓血症, portal pyemia? | 门静脉血栓形成, thrombosis of portal vein?

pylephlebitis：门静脉炎

pylephlebectasis 门静脉扩张 | pylephlebitis 门静脉炎 | pylethrombophlebitis 门静脉血栓静脉炎

pylethrombophlebitis：门静脉血栓静脉炎

pylephlebitis 门静脉炎 | pylethrombophlebitis 门静脉血栓静脉炎 | pylethrombosis 门静脉血栓形成

pylethrombophlebitis：门静脉血栓性静脉炎

pylephlebitis门静脉炎 | pylethrombophlebitis门静脉血栓性静脉炎 | pylethrombosis门静脉血栓形成

pylethrombophlebitis：门静脉血栓静脉炎无忧雅思网

pylephlebectasis 门静脉扩张 | pylethrombophlebitis 门静脉血栓静脉炎无忧雅思网,_|7O[@Z9J QW | pylethrombosis 门静脉血栓形成无忧雅思网R\\^(f&RZ;y;WU#p

pylethrombosis：门静脉血栓形成

pylethrombophlebitis 门静脉血栓静脉炎 | pylethrombosis 门静脉血栓形成 | pylic 门静脉的

pylethrombosis：门静脉血栓形成无忧雅思网

pylethrombophlebitis 门静脉血栓静脉炎无忧雅思网,_|7O[@Z9J QW | pylethrombosis 门静脉血栓形成无忧雅思网R\\^(f&RZ;y;WU#p | pylic 门静脉的无忧雅思网mH]MNT