酸缺乏

基本解释 (translations)

anacidity

与酸缺乏相关的网络例句 [注：此内容来源于网络，仅供参考]

The application form of acidifier has developed from single acid to compound acids. But reasonable compound acidifier is insufficiency in theoretics to sustain, this is the weakness aspect in acidifier research. The evolution about these aspects have been summarized in this article.

酸化剂应用形式也经历了由单一酸到复合酸的过程，但复合酸的合理配伍缺乏相应的理论基础，目前关于各种酸化剂的互作关系以及酸化剂和其他添加剂间的互作效应的研究正逐步展开，必将为酸化剂的进一步发展提供理论支持。

In most known examples, the affected proteins are enzymes, as in phenylketonuric oligophrenia which affects 1 in 10,000 of the population. It is due to deficiency in the liver cells of phenylalanine hydroxylase, which normally converts phenylalanine to tyrosine, and brain damage apparently results from raised blood and cerebrospinal fluid levels of phenylalanine and its metabolites.

在已知的大部分病例中受损的蛋白质是酶，如苯丙酮尿酸精神幼稚病，患者占人口的万分之一，是由于在正常情况下使苯基氨基酸转变为酪氨酸的肝细胞苯基丙氨酸羟化酶缺乏所致，因此血液和脑脊液的苯基丙氨酸和其代谢产物水平升高而引起脑损伤。

Generally, its difficult to determin VPA in serum with HPLC because VPA lacks nitrogen and a ring moiety and therefore has no chromophoric characteristics.

丙戊酸缺乏氮和共轭环，因此紫外吸收弱，一般HPLC法难以实现。

ResultsThe commonest symptoms and t heir frequency in sub-he althy people were fatigue 78.7%, poor quality of sleep 73.4%, amnesia 59.9 %, fatigue irrelievable after rest 59.1%, dry throat 59.0%, dizziness 58.6%, dr y and xerotic eyes 58.3%, ache with distension eyes 57.8%, ache 56.4%, ear ly awakening 52.7%, difficulty in falling asleep 52.5%...

结果亚健康人群中发生频数及均数较高的症状：疲劳78·7%，睡眠质量差73·4%，健忘59·9%，疲劳在休息后不能缓解59·1%，咽干59·0%，头昏58·6%，眼睛干涩58·3%，眼睛酸胀57·8%，身体疼痛56·4%，早醒52·7%，入睡困难52·5%，易怒51·8%等；常见危险因素：缺乏休闲活动60·4%，缺乏体育锻炼58·0%，经常加班56·7%，空气污染56·2%，噪音51·3%等；亚健康人群中常见中医证候：脾气虚证10·2%，肝郁脾虚证10·1%，心脾两虚证9·7%，脾虚湿困证7·4%，气虚证4·2%等。

In addition, it also leads to the increase of T〓 and FT〓 content, the decrease of T〓 and FT〓 content, the increase of TSH content in the plasma, which seriously influences the whole body's growth and metabolism of nutrimental materials and is one of the mechnism of nutritional anemia that is caused by lack of matter of making blood.

首次揭示雏鸡硒缺乏不但对甲状腺造成损伤，而且还特异性地降低肝脏、肾脏等组织中ID-1活性，使血浆3-5-3'-5'-四碘甲状腺原氨酸、游离T〓含量升高，3-5-3'-三碘甲状腺原氨酸、游离T〓含量下降，促甲状腺激素含量升高，严重影响整个机体的生长发育及营养物质代谢，并是导致造血物质缺乏所致的营养性贫血的机理之一。

In conclusion,lacking for choline and methionine led to decrease growth performance decrease and tibia with short disease; Basal diet supplemented with 750 ppm – 1250 ppm Cholin improved Growth performance ,and prevented the occurrence of perosis. Higher dietary choline can alleviate the adverse effects of methionine and choline deficiency.

总结而论，缺乏胆碱与甲硫胺酸会导致生长性能下降和胫骨粗短症；饲粮中添加750~1250 ppm之胆碱可以改善生长性能及防止胫骨粗短症的发生；饲粮添加高剂量的胆碱，可以缓和甲硫胺酸和胆碱缺乏之不良影响。

The protein interaction domains was delineated at the N-terminal 50-amino-acid fragment of HCV core protein and the C-terminus of p53. Confocal analysis also revealed that these two proteins colocalize in subnuclear granules and peri-nuclear region. Transfection experiments using a p53-responsive reporter plasmid in HuH-7, Hep3B, HepG2 and H1299 demostrated that full-length HCV core protein could elicit a positive or negative effect on the p53-mediated transcriptional activation depending on the concentration of the HCV core protein.

更且利用p53蛋白C端删除55-95个胺基酸之质体与含p53蛋白结合区之报导基因和表现全长HCV核心蛋白三者之质体於H1299细胞株进行共同转染时，与全长p53蛋白比较，发现HCV核心从增强全长p53蛋白之转活化能力转为抑制p53蛋白缺乏C端55-95个胺基酸之转活化能力，因实验室已有结果证实活体外及活体内缺乏C端55个及75个胺基酸之p53蛋白无法与HCV核心蛋白进行结合(Kao， unpublished data)。

Results （1） Clinical characteristics included abdominal pain,diarrhea,abdominal distension,nausea,vomiting,low heat and weight,etc.Clinical characteristic symptom relieved with rectification of ketosis and ideally controlling of blood sugar;（2）The counts of acidophil leukocyte in blood and marrow dropped with the relief of symptom ;（3） Endoscopic features concluded mucosal erosion and hydrops,involved the whole stomach,lack of specificity,gastric antrum and ileocecum were more invaded,lots of acidophil leukocytes were seen in biopsy;（4）Hormone,insulin were the first choice to treat eosinophilic gastroenteritis,which could relieve symptom rapidly and reduce acidophil leukocyte to normal;（5）If the diseases recur again and again,the patients should adopt more treatment time,use insulin long time or use immuno-suppressive agent.

结果 （1）患者的临床表现样，包括腹痛、腹泻、腹胀、恶心、呕吐、低热及体重下降等糖尿病酮症的表现；临床症状的缓解随酮症的纠正和血糖控制理想而缓解；（2）外周血和骨髓中嗜酸细胞计随着症状的缓解而下降；（3）内镜下表现多黏膜糜烂和水肿，累及全胃，缺乏特异性，以胃窦和回盲部最常受累，活检可见量嗜酸细胞浸润；（4）激素、胰岛素应用为治疗的线药物，可迅速缓解症状，并使嗜酸细胞恢复正常；（5）病情反复复发的患者可考虑延长激素的治疗时间、长期应用胰岛素或免疫抑制剂。

Results （1） Clinical characteristics included abdominal pain,diarrhea,abdominal distension,nausea,vomiting,low heat and weight,etc.Clinical characteristic symptom relieved with rectification of ketosis and ideally controlling of blood sugar;（2）The counts of acidophil leukocyte in blood and marrow dropped with the relief of symptom ;（3） Endoscopic features concluded mucosal erosion and hydrops,involved the whole stomach,lack of specificity,gastric antrum and ileocecum were more invaded,lots of acidophil leukocytes were seen in biopsy;（4）Hormone,insulin were the first choice to treat eosinophilic gastroenteritis,which could relieve symptom rapidly and reduce acidophil leukocyte to normal;（5）If the diseases recur again and again,the patients should adopt more treatment time,use insulin long time or use immuno-suppressive agent.

结果 （1）患者的临床表现多样，包括腹痛、腹泻、腹胀、恶心、呕吐、低热及体重下降等糖尿病酮症的表现；临床症状的缓解随酮症的纠正和血糖控制理想而缓解；（2）外周血和骨髓中嗜酸细胞计数随着症状的缓解而下降；（3）内镜下表现多为黏膜糜烂和水肿，累及全胃，缺乏特异性，以胃窦和回盲部最常受累，活检可见大量嗜酸细胞浸润；（4）激素、胰岛素应用为治疗的一线药物，可迅速缓解症状，并使嗜酸细胞恢复正常；（5）病情反复复发的患者可考虑延长激素的治疗时间、长期应用胰岛素或免疫抑制剂。

Clinical characteristic symptom relieved with rectification of ketosis and ideally controlling of blood sugar;（2）The counts of acidophil leukocyte in blood and marrow dropped with the relief of symptom ;（3） Endoscopic features concluded mucosal erosion and hydrops,involved the whole stomach,lack of specificity,gastric antrum and ileocecum were more invaded,lots of acidophil leukocytes were seen in biopsy;（4）Hormone,insulin were the first choice to treat eosinophilic gastroenteritis,which could relieve symptom rapidly and reduce acidophil leukocyte to normal;（5）If the diseases recur again and again,the patients should adopt more treatment time,use insulin long time or use immuno-suppressive agent.

结果 （1）患者的临床表现多样，包括腹痛、腹泻、腹胀、恶心、呕吐、低热及体重下降等糖尿病酮症的表现；临床症状的缓解随酮症的纠正和血糖控制理想而缓解；（2）外周血和骨髓中嗜酸细胞计数随着症状的缓解而下降；（3）内镜下表现多为黏膜糜烂和水肿，累及全胃，缺乏特异性，以胃窦和回盲部最常受累，活检可见大量嗜酸细胞浸润；（4）激素、胰岛素应用为治疗的一线药物，可迅速缓解症状，并使嗜酸细胞恢复正常；（5）病情反复复发的患者可考虑延长激素的治疗时间、长期应用胰岛素或免疫抑制剂。

与酸缺乏相关的网络解释 [注：此内容来源于网络，仅供参考]

alkaptonuria：尿黑酸尿症

另一代谢疾患为尿黑酸尿症(alkaptonuria). 酪氨酸在分解代谢中生成中间产物尿黑酸,如尿黑酸氧化酶缺乏,则尿黑酸裂环降解受阻,大量尿黑酸排入尿中,经空气氧化为相应的对醌,后者可聚合为黑的色素. 此种代谢性疾患一般无严重后果. 此外,

alkaptonuria：黑酸尿症

另一代谢疾患为尿黑酸尿症(alkaptonuria). 酪氨酸在分解代谢中生成中间产物尿黑酸,如尿黑酸氧化酶缺乏,则尿黑酸裂环降解受阻,大量尿黑酸排入尿中,经空气氧化为相应的对醌,后者可聚合为黑的色素. 此种代谢性疾患一般无严重后果. 此外,

Tyrosine：酪氨酸

种氨基酸与醣类长期受热的结果:(A)色氨酸 (B)离氨酸 (C)白氨酸(D)甲硫氨酸29、消瘦症(Marasmus)是饮食中缺乏:(A)脂肪和蛋白质 (B)能量 (C)蛋白质 (D)蛋白质及能量33、(A)苯丙氨酸(Phenylalanine) (B)酪氨酸(Tyrosine) (C)撷氨酸(V

achylia：胃液缺乏,消化液缺乏

achromycin 消色霉素,四环素 | achylia 胃液缺乏,消化液缺乏 | acid ? 酸;酸性的

anacid：酸缺乏的, 无酸的

anachronously | 不合时代地 | anacid | 酸缺乏的, 无酸的 | anacidity | 酸缺乏, 胃酸缺乏症

anacid：微酸的

anachronous /时代错误的/ | anacid /微酸的/ | anacidity /酸缺乏/

anacidity：酸缺乏

anachronously 不合时代地 | anacidity 酸缺乏 | anaclasis 长短音换位

anacidity：酸缺乏, 胃酸缺乏症

anacid | 酸缺乏的, 无酸的 | anacidity | 酸缺乏, 胃酸缺乏症 | anaclasimeter | 屈光检查计 眼折光计

homogentisic acid：尿黑酸

硌氨酸(tyrosin)主导荷尔蒙的新陈代谢,可与身体里的讯息传迅因子-多巴氨联络(Dopamin),...因病患体内缺乏尿黑酸氧化脢(homogentisic acid oxidase),此脢是参与酪胺酸(tyrosin)代谢途径中尿黑酸(homogentisic acid)的氧化,若缺乏会造成尿

methionic acid：亚甲基二磺酸,甲二磺酸

methiodal sodium 碘甲磺钠 | methionic acid 亚甲基二磺酸,甲二磺酸 | methionine deficiency 蛋氨酸缺乏