诱导抵抗

基本解释 (translations)

reactance

与诱导抵抗相关的网络例句 [注：此内容来源于网络，仅供参考]

Objective To observe the effects of berberine and taurine on PAI-1 mRNA expression in epiploon adipose tissue of high fat rats model of insulin resistance. and to study their effects on blood glucose, blood lipids, insulin levels, insulin sensitivity indexand body weight.

目的 观察黄连素、牛磺酸分别与联合应用对高脂饲料诱导的胰岛素抵抗大鼠脂肪组织 PAI-1 mRNA 表达的影响以及对大鼠空腹血糖、胰岛素、血脂、胰岛素敏感指数、大网膜脂肪细胞面积和体重的影响。

To determine whether the hepatic insulin resistance of obesity and type 2 diabetes is due to impaired insulin-induced suppression of glycogenolysis as well as gluconeogenesis, 10 lean nondiabetic, 10 obese nondiabetic, and 11 obese type 2 diabetic subjects were studied after an oernight fast and during a hyperinsulinemic-euglycemic clamp.

为了确定肥胖和2型糖尿病的肝胰岛素抵抗是否是由于胰岛素诱导的糖原分解作用的抑制和糖原合成作用的损伤引起，我们纳入了10位体瘦非糖尿病者、10位肥胖肥糖尿病者以及11肥胖2型糖尿病患者，对他们禁食一夜后以及高血糖-正常血糖钳夹中的状态进行研究。

We determine model rat FBG,fasting insulin,sugar tolerance,insulin tolerance,insulin sensitivity index,and insulin resistance index,lipid metabolism and free fatty acids content of serum and hepatic,HE staining of pancreas,to observe the effects of Huang Qi San on glycolipid metabolism and insulin resistance in T2DM-IR.

黄芪散干预实验性2型糖尿病胰岛素抵抗的作用及机制研究通过建立STZ所致DM小鼠模型、氢化可的松琥珀酸钠诱导的IR小鼠模型初步评价黄芪散对DM及IR的干预作用。

The result of this study will be a fundamental in treatment of HCC via the way inducing apoptosis.

研究结果为弄清肝癌抵抗机体的细胞凋亡中的作用机制奠定一定的实验和理论基础，有望为肝癌的诱导凋亡治疗开辟一个全新的研究领域。

However, the reports on target tissue of myoblast in insulin resistance skeletal muscle cell models are few.

背景：在胰岛素抵抗的细胞模型中，以游离脂肪酸诱导3T3-L1脂肪细胞和高胰岛素诱导的HepG2肝癌细胞最为常见，对分布最为广泛靶组织骨骼肌的胰岛素抵抗模型报道较少。

It means that cisplatin-induced RPTC apoptosis mainly is in mitochondrial signal pathway but not in endoplasmic reticulum.

说明RPTC抵抗顺铂诱导的细胞凋亡的主要作用是通过线粒体途径而不是通过内质网起作用的。

AIM: To investigate the effects of TNF-α: induced insulin resistance on INSIGI, INSIG2, SCAP and SREBP expressions in mice. METHODS: Male C57BL/6J mice were randomly divided into 4 groups.

目的：探讨肿瘤坏死因子α诱导的胰岛素抵抗对脂代谢相关基因INSIGI、INSIG2、SCAP和SREBP表达的影响。

Thus, we have demonstrated that in response to a HFD, brain GLP-1 signaling induces hyperinsulinemia and insulin resistance and decreases energy expenditure by reducing metabolic thermogenesis and ambulatory activity.

因此，我们认为脑中的GLP-1信号诱导的高胰岛素血症、胰岛素抵抗和能量消耗的降低（通过减少代谢产热和基础活动）是对高脂饮食的一种适应。

Although ultraviolet-absorbing compounds were induced by solar ultraviolet radiation, they could not fully counteract the damage to H. fusiformis induced by UVR.

此外，阳光紫外辐射也诱导产生了一定量的紫外吸收物质，但并不足以抵抗紫外辐射对藻体的伤害作用。

The results showed as:(1)Gegen decreases fasting insulin and insulin resistance index and increases insulin sensitive index of rats of insulin resistance induced by dexamethasone significantly;(2)Gegen can improve glucose tolerance of rats of insulin resistance but not to alloxan indued diabetes mice;(3)Gegen increases glucose intake of 3T3-L1 adipocytes and improves the insulin resistance of 3T3-L1 adipocytes induced by dexamethasone significantly;(4)Gegen can activate peroxisome proliferator- activated receptor gamma, make it bind to PPAR response elements;(5)Gegen can up-regulate mRNA expression of PPAR v ,aP2 and adiponectin.

结果表明：①葛根显著降低地塞米松诱导的胰岛素抵抗大鼠血清胰岛素水平和胰岛素抵抗指数，升高胰岛素敏感指数，改善大鼠的胰岛素抵抗；②葛根显著改善胰岛素抵抗大鼠的糖耐量，但对四氧嘧啶糖尿病小鼠的糖耐量没有明显的作用；③葛根显著增加胰岛素抵抗3T3-L1脂肪细胞对葡萄糖的摄取，改善地塞米松诱导的3T3-L1脂肪细胞的胰岛素抵抗；④葛根能促进HepG2肝癌细胞过氧化物酶体增殖物激活受体γ与靶基因的特异DNA序列结合，对PPARγ及其下游基因的转录具有激活作用；⑤葛根能够促进PPARγ及其下游基因脂肪酸结合蛋白(aP2)、脂联素mRNA的表达。