血缺氧的

基本解释 (translations)

anoxemic

与血缺氧的相关的网络例句 [注：此内容来源于网络，仅供参考]

In the survival group, patients' blood dynamics were stable gradually, DO2 and VO2 increased obviously, the blood lactic acid level was low, after complex therapy the patients were survival, however, in the death group the data demonstrated that patients' blood dynamics were unstable, the DO2 and VO2 was difficult to increase, and the blood lactic acid kept at high level, it showed that the tissues were seriously inthe condition of ischemia and anoxemia, the cell function failure was irreversible, although many kinds of complex therapy were carried on, the patient still died finally.

生存组中，患者血液动力学逐渐平稳，DO2和VO2均明显升高，血乳酸水平较低，经综合治疗后患者得于存活，而死亡组数据显示，患者血液动力学不稳定，DO2和VO2的提高非常困难，且血乳酸水平持续较高，提示组织缺血缺氧严重，细胞功能衰竭不可逆，虽采取多种措施进行综合治疗，但患者最终仍然死亡。

Sanchez ET ,Botella LM ,Velasco B ,et al. Synergistic cooperation between hypoxia and transforming growth factor2beta pathways on humanvascular endothelial growth factor gene expression. J Biol Chem ,2001,19,276:38527238535.[3] Blancher C,Moore JW,Talks kl,et al.Relationship of hyposia-inducible factor HIF-1alpha expression to vascular endothelial growth factor I nduction and hypoxia survival in human breast cancer cell lines.

肝细胞癌中缺氧诱导因子-1α、热休克蛋白70的表达】；综上所述，肝细胞癌发生后，由于血供不足诱导HIF-1α过度表达，应激状态下产生的HSP70通过减轻缺血性损伤，增加毛细血管的生成及减轻细胞凋亡等途径使肿瘤细胞的增殖、移行加快，促进恶性肿瘤的生长、浸润和转移，使肿瘤细胞在适应缺氧、能量代谢、细胞凋亡及转移中起重要作用。

The blood pressure of.1 of pathology physiology mechanism that SAS causes CVD breathes time-out to be able to bring about airframe in elevatory Morpheus anoxic, sympathetic is excessive and excited, element of serous catechu phenolic amine, kidney and hemal endodermis element are elevatory, bring about hemal easy of hemal convulsion; to shrink the function is disorder, can make blood-vessel flowing flesh happening reframes and fleshy, systemic blood-vessel obstruction adds element of the kidney when; is chronic and anoxic, system of hemal and nervous element is activationed, bring about blood pressure to lift. Obstruction of way of energy of life of patient of OSAS of disease of blood of 1.2 low oxygen increases, cause air current to interrupt, breath pauses, at the same time airframe gets used to low oxygen environment gradually, breathing centre drops to low oxygen and sensitivity of disease of blood of tall carbonic acid, breath suspends a frequency increasing, farther aggravating airframe is anoxic. 1.3 heads are self-adjusting the function drops normal person changes quickly in systematic circulation blood pressure when, the head can be passed adjust independently functional generation protects effect, make change of cerebral blood flow not big.

SAS诱发CVD的病理生理机制。1血压升高睡眠中呼吸暂停可导致机体缺氧，交感神经过度兴奋，血浆儿茶酚胺、肾素以及血管内皮素升高，导致血管痉挛；血管舒缩功能紊乱，可使血管平滑肌发生重构和肥厚，全身血管阻力增加；慢性缺氧时肾素—血管紧张素系统被激活，导致血压升高。1.2低氧血症OSAS患者气道阻力增加，造成气流中断、呼吸暂停，同时机体逐渐适应低氧环境，呼吸中枢对低氧和高碳酸血症敏感性下降，呼吸暂停次数增加，进一步加重机体缺氧。1.3脑自动调节功能下降正常人在体循环血压快速变化时，脑可通过自主调节功能产生保护效应，使脑血流量变化不大。

The clinical manifestations except HIE's features are combined irregular, indefinite krauomania on the face and limbs; eyeball gaze, fremitus, respiratory temporary arrest,and blood calcium descending.

现将36例HIE其中合并抵钙血症23例分析如下。临床资料一、诊断标准缺氧缺血性脑病的诊断及分度参考韩玉昆等的新生儿缺氧缺血性脑病临床诊断依据和分度方法［'］。

Prior administration of "Styrax Pill for Coronary Disease" proper (6 components viz., styrax liquidus, Borneolum, Lignum santali, Aristolochia, Olibanum and Cinnabaris) or the mixture of Styrax liquidus, Borneolum, Lignum Santali and Olibanum to mice prolonged the survival time of the animals subjected to low pressure hypoxia. Styrax liquidus and Borneolum administered separately showed the same effect, whereas none of the other four components showed significant protection.

用小鼠耐缺氧试验和麻醉狗冠状窦血流量及心脏动-静脉血氧差(MA-VO_2)的实验研究冠心苏合丸及其成分的作用，发现：(1)冠心苏合丸能延长小鼠耐缺氧的时间；(2)冠心苏合丸能使心肌梗塞狗的CSF回升，减少心率和MA-VO_2，对非心肌梗塞狗不能提高CSF但亦能减少心率和MA-VO_2；(3)在本实验条件下，苏合香脂和冰片似为冠心苏合丸中起作用的两个成分，其余成分如檀香、青木香、乳香或其油均未能证实其效果。

The left carotid artery was expose and placed over an ultrasonic flow probe.The brain flow volume was recorded with an ultra-sonic volume meter.The pursiness time of mice was observed to evaluate the effect of tianma xingnao capsule on cere-bral ischemia and anoxia.Methods The permeability of normal blood-cerebral barrier was investigated in mice.

目的 应用二道血流速度流量仪超声探头测定大鼠颈总动脉，以反映云南省昭通制药有限公司提供的天麻醒脑胶囊对脑血流量的影响；方法记录小鼠断头后张口喘气时间，观察天麻醒脑胶囊的抗脑缺血缺氧作用；小鼠尾静脉注射Evans溶液，测定脑组织Evans的吸光度以反映药物的血脑通透性。

One, the end that domestic oxygen cure treats to COPD patient and meaning are anoxic can cause cellular loss quickly, correct anoxic main to having the COPD patient that has disease of blood of sexual low oxygen effect, research shows LTOT ameliorable COPD accompanies the survival rate that breathes exhaustion patient chronically.

一、家庭氧疗对COPD患者治疗的目的及意义缺氧可迅速引起细胞损伤，纠正缺氧对具有进行性低氧血症的COPD患者具有重要功能，探究表明LTOT可改善COPD伴慢性呼吸衰竭患者的生存率。

Part 1: Permeability of Exogenetic BDNF through placental barrier of pregnant rats and blood-brain barrier of fetus with transient uteroplacental ischemiaObjective To investigate the potential of BDNF to penetrate the placental barrier and get into the fetus and pass through the blood-brain barrier of fetus to play neuroprotective function in the condition of transient ischemia.

第一部分：外源性脑源性神经营养因子在宫内缺氧环境中对胎盘及胚鼠血脑屏障通透性的实验研究目的：探讨外源性脑源性神经营养因子在宫内缺氧环境下能否通过胎盘屏障进入胎鼠体内，再通过胎鼠血脑屏障进入胎鼠脑组织而发挥其生物功能。

The transition from the systemic inflammatory response syndrome to seere sepsis and septic shock inoles a myriad of pathogenic changes, including circulatory abnormalities that result in global tissue hypoxia.1,2 These pathogenic changes hae been the therapeutic target of preious outcome studies.12 Although this transition occurs oer time, both out of the hospital and in the hospital, in outcome studies interentions hae usually been initiated after admission to the intensie care unit.12 In studies of goal-directed hemodynamic optimization, in particular, there was no benefit in terms of outcome with respect to normal and supranormal hemodynamic end points, as well as those guided by mixed enous oxygen saturation.9,13 In contrast, een though we enrolled patients with lower central enous oxygen saturation and lower central enous pressure than those studied by Gattinoni et al.9 and with a higher lactate concentration than those studied by Hayes et al.,13 we found significant benefits with respect to outcome when goal-directed therapy was applied at an earlier stage of disease.

从全身炎症反应综合征转为严重脓毒症和脓毒性休克牵涉大量发病的变化，包括导致全身性缺氧的循环紊乱。这些发病的变化成为先前结果研究的治疗学目标。尽管这些转变随着时间的流失而逐渐出现，既可以在院外，也可在院内，在结果研究中，干预措施往往在进入ICU之后才启动。特别的，在那些目标导向血流动力学最优化研究中，依照结果，那些以正常或超正常血流动力学为终点的措施未能显示出益处，以混合静脉血氧饱和度为导向的研究同样如此。

This is because:① hypertension caused by macrovascular disease, microvascular disease, the body part can hypoxia, blood lactate levels, leading to uric acid by inhibiting renal tubular secretion, coupled with increased synthesis of uric acid in patients, renal clearance of uric acid will reduce the functionality;② some long-term use in patients with hypertension Thiazide diuretics, the result of hypovolemia, which was repeated absorption of uric acid, thus leading to hyperuricemia.

这是因为：①高血压引起的大血管病变、微血管病变，可使机体部分组织缺氧、血乳酸水平升高，导致肾小管分泌尿酸受到抑制，加之病人体内尿酸合成增加，肾脏清除尿酸的功能就会降低；②部分高血压患者在长期使用噻嗪类利尿剂后，造成血容量减少，致使尿酸被重复吸收，从而引起高尿酸血症。

与血缺氧的相关的网络解释 [注：此内容来源于网络，仅供参考]

anemic hypoxia：贫血性缺氧

(2)贫血性缺氧(anemic hypoxia),在贫血或一氧化碳中毒时,虽然动脉血的Po2正常,但可资运用的血红素减少. (3)滞留或局部缺血性缺氧(stagnant or ischemic hypoxia),某一部份组织之血流过慢,虽然血中Po2及血红素的含量均正常,

anoxemia：缺氧血症

是由于空气的氧分压降低(hypoxia)而造成的吸气的氧不足所引起的缺氧血症(anoxemia),或者由于血行障碍以及血中的血红蛋白发生变性等,进而造成的体内缺氧.

ischemic anoxia：缺血性缺氧

由于动脉狭窄或阻塞,致动脉血灌流不足而引起的缺氧,又称缺血性缺氧(ischemic anoxia);由于静脉血回流受阻,血流缓慢,微循环淤血,导致动脉血灌流减少而引起的缺氧,称淤血性缺氧(stagnant anoxia).

isotonic hypoxemia：等张性低氧血症

由于以物理状态溶解在血液内的氧不受血红蛋白的影响,这型缺氧的pao2正常,属于等张性低氧血症(isotonic hypoxemia). (一)原因 1、贫血(anemia)各种原因引起的贫血,单位容积血液内红细胞数和血红蛋白量减少,虽然pao2和氧饱和度正常,但氧容量降低,

anoxemic：缺氧血症的

anoxemia 缺氧血症 | anoxemic 缺氧血症的 | anoxia 缺氧症

anoxemic：血缺氧的

anoxemic 缺氧血症的 | anoxemic 血缺氧的 | anoxia 缺氧症

anoxemic：缺氧血症的, 血缺氧的

anoxemia /血缺氧/缺氧血症/ | anoxemic /缺氧血症的/血缺氧的/ | anoxia /缺氧症/

congestive hypoxia：淤血性缺氧

循环性缺氧还可以分为缺血性缺氧( ischemic hypoxia )和淤血性缺氧( congestive hypoxia ). 缺血性缺氧是由于动脉供血不足所致;淤血性缺氧是由于静脉回流受阻所致. 组织性缺氧( histogenous hypoxia )是指由于组织、细胞利用氧障碍所引起的缺氧.

hypokinetic hypoxia：低动力性缺氧

74.循环性缺氧(circulatory hypoxia)指组织血流量减少使组织氧供应减少所引起的缺氧,又称低动力性缺氧(hypokinetic hypoxia). 75.缺血性缺氧是指 ...74.循环性缺氧(circulatory hypoxia)指组织血流量减少使组织氧供应减少所引起的缺氧,

hypotonic hypoxemia：低张性低氧血症

当PaO2低于8kPa(60mmHg)时,可直接导致CaO2和SaO2明显降低,因此低张性缺氧也可以称为低张性低氧血症(hypotonic hypoxemia). 血液性缺氧(hemic hypoxia)指Hb量或质的改变,使CaO2减少或同时伴有氧合Hb结合的氧不易释出所引起的组织缺氧.