血管扩张神经

基本解释 (translations)

vasodilator

与血管扩张神经相关的网络例句 [注：此内容来源于网络，仅供参考]

Objective To investigate the clinical features, causes of blindness and diagnosis of Vogt Koyanagi Harada syndrome Methods The data of 157 patients with VKH syndrome were reviewed and analyzed Patients were carefully examined with slit lamp, ophthalmoscope, three mirror lens, fundus fluorescein angiography, indocyanine green angiography and HLA typing Results Headache was noted in 73 5% of these patients Simultaneous involvement of both eyes occurred in 80 8% of these patients Chroiditis,papilledema and edema of the retina adjacent to the optic nerve were noted in 100% of these patients in the posterior uveitis stage, whereas recurrent granulomatous anterior uveitis (98 4%),"sunset glow" fundus (95 8%) and Dalen Fuchs nodules (71 2%) were the common ocular findings in the recurrent anterior uveitis stage The common causes of blindness were papillitis, exudative retinal detachment and complicated cataract in the posterior uveitis stage, anterior uveal involvement stage and its recurrent stage Poliosis (36 3%) and alopecia (35 0%) were the most common extraocular findings Early irregular patches of fluorescence, followed by localized hyperfluorescent spots were the typical findings of FFA Dilation of choroidal vessels and leakage of ICG from the choroidal vessels were the common ICGA findings The prevalence of HLA DR4 and HLA DRw53 in patients (54 9% and 71 8% respectively) was significantly higher than that in controls (14 7% and 38 2% respectively) Conclusions VKH syndrome is characterized by chroiditis, papillitis or neuroretinitis in the posterior uveitis stage, followed by a generalized uveitis with a typical recurrent granulomatous anterior uveitis Extraocular findings and relevant examinations including FFA, ICGA and HLA typing are helpful to the diagnosis of VKH syndrome

目的探讨Vogt-Koyanagi-Harada综合征患者的临床特征、盲目原因及诊断等有关问题。方法对在1996年1月至2000年12月间就诊资料完整的157例VKH综合征患者进行回顾性分析，并对裂隙灯、眼底镜、三面镜、荧光素眼底血管造影(fundus fluorescein angiography，FFA)、吲哚青绿血管造影(indocyanine green angiography，ICGA)及人类白细胞抗原分型等检查结果进行分析。结果 VKH综合征最常见的前驱症状为头痛(102例，73.5%)，双眼同时患病118例(80.8%)；后葡萄膜炎期眼部主要表现为脉络膜炎、视乳头及附近视网膜水肿(100.0%)；前葡萄膜炎反复发作期眼部表现为复发性肉芽肿性前葡萄膜炎(128例，98.4%)、晚霞状眼底改变(95.8%)及Dalen-Fuchs结节(71.2%)；后葡萄膜炎期、前葡萄膜受累期及前葡萄膜炎反复发作期导致盲目的主要原因分别为视乳头炎、视网膜脱离及并发性白内障；毛发变白(36.3%)及脱发(35.0%)是最常见的眼外表现；炎症活动期FFA典型表现为斑驳状高荧光，ICGA发现脉络膜血管扩张、通透性增高等改变；VKH综合征患者HLA-DR4及HLA-DRw53的阳性率(54.9%及71.8%)显著高于正常对照组(14.7%及38.2%)。结论 VKH综合征患者在后葡萄膜炎期眼部典型表现为双侧脉络膜炎、视乳头炎或神经视网膜炎，随后出现以反复发作的肉芽肿性前葡萄膜炎为特征的全葡萄膜炎。眼外症状及相关的辅助检查包括FFA、ICGA 及HLA分型等有助于VKH综合征的诊断。

Parotid region skin shows vasodilatation, flush and diaphoresis in meal. The mechanism is aberration-regenerate theory. The treatments include drug treatment, local treatment and operation treatment. The obviate is to put interval stuff in operation, skin flap can prevent nerves wrong to unite on parotid raw surface. It can barricade insulate the wrong unite of parasympathetic nerve and reign skin interneural if insert apo-cell subcuticula ground substance in operation.

腮腺区皮肤在进餐时出现血管扩张、潮红、出汗现象，其机制为迷走再生学说，其治疗有药物治疗、局部治疗、手术治疗，预防主要是术中放置间隔材料，皮瓣在腮腺创面上防止神经错联发生；如术中植入脱细胞真皮基质，屏障隔离副交感神经与支配皮肤神经间的错联。

Dilation of a blood vessel, as by the action of a nerve or drug.

血管扩张血管的扩张，如由于神经或药品

A vasodilator used to treat severe hypertension; one side effect is hirsutism so it is also sold as a treatment for male-patterned baldness.

用来治疗严重高血压的血管扩张神经药物；此药物的副作用是多毛症，因此也被作为治疗秃头。

This drug is a vasodilator that increases blood supply and accelerates healing and hair regrowth in most patients.

这一种药物是增加血补给，而且加速的血管扩张神经痊愈和在大部分病人中的头发再生长。

A vasodilator that is sometimes used to treat angina pectoris.

用来治疗心绞痛的血管扩张神经剂。

A vasodilator that is released by cells in the human immune system as part of an allergic reaction; also affects the amount of stomach acid released.

被人类免疫系统的细胞释放的血管扩张神经药物；也能影响胃酸释放的含量。

Results Denatured necrosis occurred on the surface of hypothalamic periosteum with no cells and there was a large amount of starchy bodies and hydrops in foci with angiectasis of choroid plexus and necrosis and calcification in tunicaintima of vesicles,and mitochondria in the nerve synapse disappeared and part of anteroposterior membranes ...

结果 慢性SZ患者下丘脑部室管膜表面变性坏死、无细胞，病灶中有大量淀粉样小体和水肿，脉络丛血管扩张，管壁内膜有坏死和钙化；神经突触内大部分突触小泡和线粒体消失，部分前后膜模糊不清。

The pressor effect of adenosine was due to carotid body cheoreceptor activation. However, a slight increase of ABP induced by intracarotid bolus injection of adenosine following the section of carotid sinus nerves was observed in a few rats, which implied that the central effect might be involved in the pressor effect of adenosine. The delayed effect of adenosine may be attributed to the direct actions (including the negative chronotropic action, thenegative dromotropic action, the negative inotropic action and the vasodilator effect) of adenosine on the heart and blood vessels.

前一效应主要与颈动脉体化学感受器的激活有关，但在少数动物，颈动脉内注射腺苷的升压效应在切除窦神经后并未完全消除，表明腺苷有可能进入神经中枢而发挥作用；后一效应可能由腺苷直接作用于心脏和血管，通过其负性变力、变时、变传导和血管扩张等作用，导致心脏自律性降低，输出量减少和外周阻力下降。

Vasodilators may step up the heart rate and cause headaches.

血管扩张神经会加速心跳并引起头痛。

与血管扩张神经相关的网络解释 [注：此内容来源于网络，仅供参考]

active hyperemia：主动性充血

动脉性充血(arterial hyperemia)又称主动性充血(active hyperemia),简称充血. 一般指器官或局部组织细动脉血输入量增多. 病因血管舒张神经兴奋性增高或血管收缩神经兴奋性降低、舒血管活性物质释放增加等,引起细动脉扩张、血流加快,

vasovagal syncope：血管迷走性晕厥

一、血管迷走性晕厥 血管迷走性晕厥(Vasovagal syncope)是一种神经反射性晕厥,系由多种因素触发不同类型的神经反射,引起周围血管扩张、低血压与心动过缓所致的自限性晕厥发作.

telangiectasis：毛细血管扩张

毛细血管扩张(Telangiectasis)是皮肤和粘膜上的表浅小血管因种种原因呈持续性扩张、从毛细血管扩张症图片看出,它的形成色红或紫红色斑状、点状、线状或星芒状损害,其分布可为局限性或广泛性,或与血管神经相一致,或呈一侧性,多无自觉症状,

Vasodilation：血管扩张,血管舒张

血管紧缩,血管缩小 vasoconstriction | 血管扩张,血管舒张 vasodilation | 血管扩张的,血管扩张药,血管扩张神经 vasodilator

sympathetic vasodilation：交感神经性血管扩张

sympathetic vasoconstrictor nerve 交感缩血管神经 | sympathetic vasodilation 交感神经性血管扩张 | sympathetic vasodilator nerve 交感舒张神经

vasodilator nerve：血管扩张神经,血管舒缩神经,血管舒张神经

vasoconstrictor nerve 血管收缩神经 | vasodilator nerve 血管扩张神经,血管舒缩神经,血管舒张神经 | vasodilator nerve fibers 血管舒张神经纤维

neurogenic shock：神经原性休克

(五)神经原性休克(neurogenic shock) 神经原性休克是动脉阻力调节功能严重障碍,血管张力丧失,引起血管扩张,导致外周血管阻力降低,有效循环血容量减少所致的休克,单纯由于神经因素引起的休克少见,可见于外伤,剧痛,脑脊髓损伤,药物麻醉,

shunting：分流

因动静脉分流(shunting)使周围脑组织缺血,导致癫痫发作或局部神经学障碍,或因出血引发临床症状. 海绵状血管瘤由扩张的血管窦与结缔组织构成,血管壁无平滑肌及弹性组织,且病变内不含脑实质组织为其特点. 易伴随小出血,

vasculolymphatic：血管淋巴管的

血管扩张神经vasodilator | 血管淋巴管的vasculolymphatic | 血管瘤angiomahaemangiomahemangioma