- 更多网络例句与蛋白样变性相关的网络例句 [注：此内容来源于网络，仅供参考]
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Abstract］ objective to study the pathological features and histopathological type and differential diagnosis of hepatic focal nodular hyperplasia.methods the clinicopathological characteristics of 40 cases of fnh were studied.all were evaluted by use of paraffin embedded sections and he staining before light microcope observation.results there were 28 females and 12 males fnh patients whose age were from 16 to 62 years（median 41.3）,all alpha-fetoprotein was negative and had no hepatitis history.25 cases were classic type showed characteristic central stellate fibrotic scar,composed of fibrous connestive tissue and tortuous blood vessels.8 cases were telangiectic type,the left were mixed type and adenomatoid type.conclusion fnh is a reactive proliferation of hepatic cells to local blood vessel anomalies,it is not realy a tumor.its differential diagnosis includes hepatic adenomatous hyperplasia nodule,hepatic anaplasia nodular hyperplasia,fibrolamellar hepatocellular carcinoma and hepatocellular adenoma.
目的 探讨肝局灶性结节性增生的病理形态特点、组织分型及鉴别诊断。方法分析40例肝局灶性结节性增生的临床资料，并采用石蜡包埋he染色光镜下观察其组织学特点。结果 40例肝局灶性结节性增生患者中，女28例，男12例，年龄18~62岁，平均年龄41.3岁，所有病例术前均无肝炎病史，甲胎蛋白阴性，组织学上25例为经典型，有特征性的中央纤维瘢痕，由纤维结缔组织及扭曲血管组成。8例为毛细血管扩张型，其余为混合型及腺瘤样增生型。结论肝结节性增生是一种肝细胞对局部血管的异常反应性增生，并非真性肿瘤，主要与肝腺瘤样增生性结节、肝间变性结节状增生、肝纤维板层癌及肝细胞腺瘤鉴别。
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Objective To observe the effect of Shenghui Keli on AD encephalon neural degeneration induced by A β(β-Amyloid protein) from neural function, biochemical and gene expression. Research the degeneration related to apoptosis of cell and gene expression correlate to apoptosis. Then investigate the mechanisms of Shenghui keli treating AD .
目的 从神经功能形态、生化与基因表达等方面，观察生慧颗粒对β-淀粉样蛋白(β-Amyloid protein，Aβ)诱导脑神经细胞变性的干预作用，以及与细胞凋亡、凋亡相关基因表达的关系，探讨生慧颗粒治疗Alzheimer病的作用机制。
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PartⅡ:Effect of bicyclol on hepatocyte apoptosis in alcohol-intoxicated mice and related mechanismsThree consecutive intragastric administration of alcohol(6g/kg)in 12h interval resulted in an obvious liver injury and inflammation in mice,as indicated by the elevation of serum alanine transaminase(2.2 fold of control),increased hepatic COX-2 protein expression(2.2 fold of control),as well as liver pathological changes characterized by myelonic degeneration,vacuolar degeneration and inflammatory infiltration.
双环醇对酒精中毒小鼠肝脏细胞凋亡的保护作用及机制研究小鼠连续三次酒精(6g/kg)灌胃后可导致明显肝损伤，变现为血清ALT水平升高(为对照组的2.2倍)，肝脏炎症相关蛋白环氧合酶-2(cyclooxygenase-2，COX-2)表达增加(为对照组的2.2倍)，肝组织出现髓样变性、小空泡变性和炎性细胞浸润。
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Changes in Red Cell Enzyme Activity in Relation to Red Cell Survival in Infancy Antibody Deficiency Syndrome with Increased Gamma1 M(B2M) Globulins and Absent Gamma1 A(B2A) Globulins and Gamma2 Globulins. Needle Biopsy of the Liver in Various Forms of Childhood Tuberculosis Jaundice Associated with Severe Bacterial Infection in Young Infants Effectt of Hypoxia on Newborn Animals Renal Amyloidosis Resulting From a Chronically Infected Burn. Skin Testing of Allergic and Normal Children with Purified Milk Proteins. Pericarditis in Juvenile Rheumatoid Arthritis Cardiac Complication of Diphtheria Adenovirus Complement-fixing Titers from Birth through First Year Congenital Coronary A-V Fistula Narcotic and Methamphetamine Use During Pregnancy Purulent Pericarditis Transfer of Bilirubin-C^4 Across Monkey Placenta Plasma Antidiuretic Hormone in Nephrogenic Diabetes Insipidus Studies of Anabolic Steroids:Ⅰ.
红血球酵素活性度之变化与婴儿红血球生存期之关系 Gamma1 M(B2M)球蛋白增加而无Gamma1 A(B2A)球蛋白与Gamma2球蛋白之抗体缺乏症候羣儿童期各型结核病之肝藏针管活体切片检查幼儿细菌严重感染并发之黄疸缺氧对於新生动物之影响及其与肺玻璃膜病之关系烧伤处慢性感染引起之肾性淀粉样变性以精制奶粉蛋白对过敏症与正常小儿作皮肤试验年幼性类风湿性关节为合并的心包炎白喉之心脏合并症自出生至一岁之腺病毒补体结合滴定度先天性冠状动静脉瘻管怀孕时使用麻醉剂与Methamphetamine之结果化脓性心囊炎胆红素C^14标猴子胎盘之转送肾因性尿崩症患儿之血清抗利尿荷尔蒙之证明同化类固醇之研究Ⅰ。
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Amyloidosis is characterized by slow deposition over years of increasing amounts of an amorphous proteinaceous material in one or more tissues.
图示淀粉样变性，特征是不定形的蛋白性质物质多年慢慢沉积在一个或更多的组织中，见在暗红心肌纤维间有浅粉红色淀粉样沉积物。
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The expressions of VEGF mRNA in renal cortex in B and group C increased greatly compared with A group at 8th week(P.01), And the expression was decreased more in C group than that in B group at the 8th week;④The light microscopes results showed that no pathological changes in group A; pathological changes were much obvious in group B:glomerular capillary lumen tumbling,lumens blocked,mesangial region widened,basal lamina thicking,mesenterium base inceased,the volume of glomerulus become large,cell population increased,renal tubule vacuolization, renal interstitium was infiltrated by lots of lymphocyte and mononuclear macrophage; pathological changes in group C was light,only see glomerular capillary lumen lightly stegnosis,few lymphocyte infiltrating.
免疫组化结果显示第8周B组大鼠肾皮质VEGF蛋白含量较A组显著增加(P.01)，C组VEGF含量较B组有明显减少(P.01)，C组较A组表达量仍然增加(P.01)；③第8周B组肾皮质VEGF mRNA表达较A组有明显上调(P.01)，与B组相比，辛伐他汀可以明显减少C组肾皮质VEGF mRNA表达；④光学显微镜下A组肾小球毛细血管腔均匀一致，无狭窄，肾小管-间质无炎症细胞浸润。B组则病变较明显：大鼠肾小球毛细血管袢塌陷，管腔闭塞，系膜区增宽，基膜增厚和系膜基质增多，肾小球体积增大，出现玻璃样变；肾小管尤其是近区小管肿胀、变性、空泡形成，肾间质可见大量淋巴细胞和单核巨噬细胞浸润。C组病变较轻，可见肾小球毛细血管管腔轻度狭窄，肾小管-间质见少量淋巴细胞浸润。
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Results: CPCC treatment was found to effectively attenuate the degenerative changes observed in alloxan-induced animals, such as hypertrophy of liver, adhesion phenomenon between adjacent hepatic lobules, crimpling and partial necrosis of hepatocytes, inflammatory cell infiltration, vacuolation, endothelial cell injury and the nuclear damage etc.
结果：CPCC处理使四氧嘧啶处理后小鼠肝脏肿大、肝小叶粘连现象明显改善；肝指数降低；减轻肝细胞变形、肝窦淤血、炎症细胞浸润及肝细胞空泡样变、血管内皮细胞损伤，核变性程度同时CPCC能有效防止四氧嘧啶所致小鼠肝脏可溶性蛋白的变化。
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Pathologic examination included: tubulointerstitial lesion which was the main pathologic presentation, tubular epithelial cell becoming flat, TEC necrosis and wither, departure between the cytoplasm and basis membrane, drops of protein in cytoplasm, broken basic membrane of TEC, inflammatory cells infiltrating in tubulointerstitium, tubulointerstitium fibrosis, plasm cell infiltration.
MM肾损害主要以小管-间质病变为主，病理表现为肾小管上皮细胞扁平，存在不同程度变性、坏死或萎缩，胞浆与基膜分离，胞浆内可见蛋白滴，甚至小管基膜断裂，间质炎性细胞浸润、纤维化，部分有淀粉样物质沉积，较少见浆细胞浸润。
- 更多网络解释与蛋白样变性相关的网络解释 [注：此内容来源于网络，仅供参考]
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amyloid：淀粉样物质
Virchow称其为淀粉样物质(amyloid),并将沉积该物质的组织,称为淀粉样变性. 现已证明,所谓淀粉样物质,实为由不同成分组成的蛋白质. 故称其为淀粉样蛋白更为合适. 目前普遍认为淀粉样变病(amyloid disease)属于免疫性疾病.
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amyloid degeneration：淀粉样变性
24.淀粉样变性(Amyloid degeneration) 在组织内有淀粉样蛋白的沉积. 后者是一种醣蛋白,对结晶紫呈异染性(Metachromasia)即染成与结晶紫颜色不同的紫红色. 见于皮肤淀粉样变性等. 25.结缔组织纤维蛋白样变性(Fibrinoid degeneration of connective tissue)病变处因纤维蛋白渗透到胶原内,
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hyaline degeneration：透明变性
⑷玻璃样变或透明变性:玻璃样变或透明变性(hyaline degeneration)是指在组织内或细胞内出现玻璃样半透明的均性物质,即所谓透明蛋白. 苏木精-伊红染色呈均-淡红色,具折光性,见于瘢痕疙瘩等.
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hyaline degeneration：玻璃样变性
hemosiderin 含铁血黄素,血铁黄蛋白 | hyaline degeneration 玻璃样变性 | hyaline membrane 透明膜
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lipoid proteinosis：类脂质蛋白沉积症
类脂质蛋白沉积症(lipoid proteinosis)又称Urbach-Wiethe病、皮肤黏膜透明蛋白变性(hyalinosis cutis et mucosae),是一种罕见的遗传性疾病. 常发生于婴儿,主要在皮肤、黏膜或内脏有无定形嗜伊红透明物质沉积,临床以眼睑增厚、肘膝黄色瘤样斑块伴声音嘶哑为特征.
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slow virus：慢病毒
有人怀疑AD与慢病毒(Slow Virus)或朊蛋白(Prion Protein)有关. 这是由于AD与皮层-纹体-脊髓变性(CJD)的某些病理特点相似,例如CJD患者大脑中也有淀粉样蛋白沉积(与AD不是同一种),AD患者大脑中某些变化与CJD病变相似. 但是,
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mucinoid：粘蛋白样的 类粘蛋白
mucinogengranule 粘蛋白原粒 粘蛋白原粒 | mucinoid 粘蛋白样的 类粘蛋白 | mucinoiddegeneration 类粘蛋白变性