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We conclude that postbiopsy anuria associated with a subcapsular hematoma and acute absence of diastolic flow on doppler ultrasound should be considered pathognomonic of APK.
我们总结认为,活检后无尿合并包膜下血肿与多普勒超声检查提示急性移植肾舒张期无血流时应该考虑APK的特异病症。
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Methods:After 1-week washout and 2-week placebo run-in period,patients with seated diastolic blood pressure 95-115 mmHg (n=76) were administrated with losartan 50 mg once-daily or losartan 50 mg/hydrochlorothiazide 12.5 mg once daily for 4 weeks,followed by double doses once daily for another 4 weeks in those patients with SeDBP≥90 mmHg.
坐位舒张压95~115 mmHg(1 mmHg=0.133 kPa)的76例原发性高血压患者,经1周药物洗脱期,2周安慰剂期后,随机服用氯沙坦50 mg(氯沙坦组,n=37),每日1次或氯沙坦50 mg与双氢克尿噻12.5 mg合剂(氯沙坦+双氢克尿噻合剂组,n=39),每日1次。4周末坐位舒张压≥90 mmHg者,剂量分别加倍,继续服用4周。
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Primary atrial tumor has obvious characteristic on RT-3DE: All the atrial tumor display the same size of stereoimages as the tumor themselves. There is a clear boundary between the stereoimage of the myxoma and the atrial wall and there exists a short pedicle connecting the atrial myxoma to the atrial wall or the interatrial septum. The body of the atrial myxoma moves to and fro through the atrium and the atrioventricular valve with the contraction and relaxation of the heart with a fixative pedicle. Atrial myxoma leads to relative atrioventricular valve incompetence during systolic phase and relative atrioventricular valve stenosis during diastolic phase, but doesn't result in pathological changes of atrioventricular valve which restore to normal after exsection of the atrial myxoma. Malignant atrial tumor closely and extensively contact with atrial wall without obvious boundary, pedicle and movement with the contraction and relaxation of the heart.
所有心房肿瘤RT-3DE均实时显示与肿瘤大小相同的立体形态图像;心房黏液瘤立体形态图像与心房壁界限明显,都通过一短蒂与房间隔或心房壁相连,瘤体以蒂为固定点随心脏收缩、舒张在心房与房室瓣口之间往返运动;心房黏液瘤收缩期瘤体导致房室瓣相对关闭不全,舒张期瘤体导致房室瓣相对狭窄,但瘤体不引起房室瓣器质性病变,瘤体摘除后房室瓣功能即恢复正常;恶性心房肺瘤立体形态图像与心房壁关系密切,界限不明显,接触范围广泛,无蒂,瘤体不随心脏收缩、舒张运动。
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It was suggested that aging did not only influence diastolic filling period, but also influence STI. 3. With increasing age, whether at rest or during exercice, fractional shortening of minor semiaxis, mean velocity of circumferential fiber shortening, cardiac index and ejection fraction did not change, but immediately post pacing, PEP prolonged and PEP/ LVET increased in elderly group and did not change in young group. It was suggested that STI was sensitivity in detecting systolic function of LV. 4. There was a good correlation between aging and decreased compliance and diastolic function of left ventricle which was manifested by a reduction of early diastolic peak folw velocity, time-velocity integral of early diastole, 1/3 filling fraction (1/3FF)(r=-0.958,-0.875 and -0.937, P<0.01~0.001) and a significant compansatory increase in peak flow velocity of atrial contraction, time-velocity integral of atrial contraction, the ratio between APFV and EPFV and the ratio of ATVI and Total TVI (r=0.958, 0.956, 0.966 and 0.986, P<0.001) and the prolongation of Dec T and RFP with increasing age. It was particularly true for the subgroups above the age of 50 years.
静息状态下收缩时间间期与年龄无相关性,负荷后即刻,老年组射血前期延长(P<0.05),射血前期与射血期比值加大(P<0.05),青年组无变化,提示年龄因素亦影响STI,但由于静息状态下得到代偿,STI变化往往表现不明显。3、静息及负荷状态左室短轴缩短率、平均周径缩短率、心指数及射血分数均不随年龄增长而变化,而负荷后即刻老年组PEF延长,PEP/LVET加大,提示STI是检测左室收缩功能的敏感指标。4、随年龄增长,心房收缩期充盈峰值流速、流速积分、充盈分数及心房收缩期与舒张早期充盈峰值流速之比值显著增高(r=0.958、0.956、0.986及0.966,P均<0.001),舒张早期充盈峰值流速及流速积分、1/3充盈分数(1/3FF)下降(r=-0.958、-0.875及-0.937,P<0.01~0.001)。
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There was no statistically significant difference in parameters of IVS motion between obstructive and nonobstructive HCM.The data of hypertrophic LVPW motion were lower than those of normal subjects,as well as those of nonhypertrophic LVPW in HCM group.
左室后壁肥厚者收缩期及舒张早期的DTI测值多低于正常,而左室后壁不肥厚者收缩期指标无明显变化,舒张早期指标低于正常;两者相比,前者收缩期及舒张早期各参数多低于后者。
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We study longitudinal motion characteristics during isovolumic contraction , ejection , isovolumic relaxation , early relaxation , atrial systole phases in left ventricular total and regional myocardium of myocardial infarction patients with three vessels lesions by Quantitative Tissue Velocity Imaging , assess systolic and diastolic function in left ventricular regional myocardium, discuss postsystolic shortening in isovolumic relaxation phase and its clinical meanings.
目的 利用超声评价三支病变的心肌梗死患者左室整体和局部心肌在心动周期各时相的纵向运动特点,评价左室局部心肌收缩和舒张功能,探讨其在等容舒张期出现的收缩后收缩现象及临床意义。
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Lactated Ringer's or 7.5% NaCl+6% dextran 70 was given for resuscitation 6h postburn.The volumes and rates of fluid infusion were controlled basically on the urinary output of 1.0ml*kg-1*h-1 and cardiac output of 70%~80% of preburn values.The volume load,+dp/dtmax,-dp/dtmax,CI,DO2 and VO2 were obtained to evaluate the effect of HSD resuscitation. Results The resuscitated volume of HSD was 30.56% less during first 24h postburn and 59.50% less at 4h after resuscitation than LR's.
采用犬35%TBSA Ⅲ度烧伤模型,伤后6 h分别用乳酸林格液及HSD进行复苏,并以每h尿量为1.0ml/kg及心输出量为伤前值的70%~80%来调整输液速度及输液量,观察HSD在复苏中容量负荷、左心室等容收缩期最大压力变化速率及左心室舒张期压力下降最大变化速率、心脏指数、氧供给(DO2)及氧消耗(VO2)等的变化。
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The volumes and rates of fluid infusion were controlled basically on the urinary output of 1.0ml*kg-1*h-1 and cardiac output of 70%~80% of preburn values.The volume load,+dp/dtmax,-dp/dtmax,CI,DO2 and VO2 were obtained to evaluate the effect of HSD resuscitation. Results The resuscitated volume of HSD was 30.56% less during first 24h postburn and 59.50% less at 4h after resuscitation than LR's.
采用犬35%TBSA Ⅲ度烧伤模型,伤后6 h分别用乳酸林格液及HSD进行复苏,并以每h尿量为1.0ml/kg及心输出量为伤前值的70%~80%来调整输液速度及输液量,观察HSD在复苏中容量负荷、左心室等容收缩期最大压力变化速率及左心室舒张期压力下降最大变化速率、心脏指数、氧供给(DO2)及氧消耗(VO2)等的变化。
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L-arginine, an NO precursor, reduced the contractile amplitude and end-diastolic cell length in the cardiomyocyte; but reversibly increased LVDP,±dp/dtmax, and coronary flow in isolated perfused rat heart.
2NO的前体L-精氨酸(L-arginine, L-Arg)引起心肌细胞舒张期细胞长度缩短、收缩幅度降低。离体灌流心脏LVDP、冠脉流量、和±dp/dtmax增高,用K-H液复灌后可恢复正常。
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There was some difference with Diastolic filling time of mitral valve under load after hypoxic training in both groups.
在运动负荷状态下,HiHiLo组与LoHi组二尖瓣舒张期充盈时间低氧训练后的变化具有显著性差异(p.05)。
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ejaculation:(射精)
4.射精(ejaculation)由性反应周期的持续期转入性高潮 期后,交感神经紧张性进一步增高,引起外尿道括约肌舒张,而尿道内括约肌仍保持紧张性收缩状态,以防止精液逆流入膀胱.