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Results Multisystem were damaged,such as peripheral, hepatomegaly, splenomegaly,lymphadenectasis, endocrinopathy, skin changes, edema and plastcytosis in bone marrow are all more common damages, all of them peripheral neuropathy is the most characteristic manifestation.
结果本病是累及多系统的疾病，以周围神经病最具有特征性，肝、脾和淋巴结肿大，内分泌的病变，皮肤改变，水肿及骨髓浆细胞增多比较常见。
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Results In the medium and high dose F0 groups, it was observed that the atrophy and incrassation of seminiferous tubule, decrease of spermatogenesis, hyperplasia of interstitial tissue, especially in high dose groups spermatozoon abnormality and nucleolus concentration in the rats testis after DU ingestion for 14 months. The changes became more severe with the prolongation of DU ingestion. Such changes occurred in filial rats (F1) after DC ingestion for 5 months. In the medium and high dose F0 groups, it was observed that a little atrophy of kidney glomerulus, hyperplasia of interstitial tissue after DC ingestion for 14 months, and kidney glomerulus fibrosis happened after DC ingestion for 20 months, such changes occurred in filial rats (F1) after DC ingestion for 5 months In the medium and high dose F0 groups, splenic germinal center and periarterial lymphatic sheaths were hyperplasia , companies with lymphopoiesis after DC ingestion for 7 months, splenic white pulp became more small and sparse after DC ingestion for 20 months.
结果 F0代的中、高剂量组大鼠摄入贫铀14个月后可见雄性的精曲小管萎缩，管壁增厚呈空虚网状，生精细胞层次减少，间质细胞增生，但仍见有精子生成；高剂量组可见到精子呈异型性改变，细胞核浓缩深染，且随着摄入时间延长改变愈趋明显；F1代大鼠摄入贫铀5个月后就有上述改变且更为严重。F0代中、高剂量组大鼠摄入贫铀14个月后肾小球轻度萎缩，间质增生明显，20个月时肾小球萎缩纤维化；F1代大鼠摄入贫铀5个月后就有上述改变。F0代中、高剂量组摄入贫铀7个月时脾脏生发中心和淋巴鞘增生，淋巴母细胞增生活跃，20个月时脾小体减少，生发中心稀疏；F1代大鼠摄入贫铀早期和晚期有类似改变。F0和F1代高剂量组摄入贫铀早期肝脏有炎症细胞浸润，晚期骨髓有核细胞减少，脂肪细胞增加。
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It is important to differentiate immune thrombocytopenia from nonimmune thrombocytopenia because of the different clinical treatment, but untill now there is no satisfactory laboratory tests could be used.
非免疫性血小板减少性紫癜，是指由非免疫因素引起的血小板减少，如化疗后骨髓抑制、急性白血病、脾功能亢进以及恶性肿瘤骨髓转移等引起的血小板减少。
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The diagnosis of ITP is made clinically by exclusion of other causes of thrombocytopenia, the absence of splenomegaly, and the finding of normal or increased megakaryopoiesis on bone marrow examination. The nonimmune thrombocytopenia is caused by some nonimmune factors, such as acute leukemia, hypersplenism and post-chemotherapic bone marrow suppression.
特发性血小板减少性紫癜的诊断仍只是一种排除性的临床诊断，其国内诊断标准为多次化验检查血小板计数减少，脾脏不增大或仅轻度增大，骨髓检查巨核细胞数增多或正常，有成熟障碍，并且具备以下五点中的任何一点：泼尼松治疗有效，切脾治疗有效，血小板相关抗体增多，血小板相关补体增多，血小板寿命测定缩短，同时应排除继发性血小板减少症。
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Such massive splenomegaly is usually indicative of some myeloproliferative disease such as chronic myelogenous leukemia or myelofibrosis.
这样的脾肿大通常预示有骨髓增生型疾病，例如慢性髓性白血病或骨髓纤维化。
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Such massive splenomegaly is usually indicative of some myeloproliferative disease such as chronic myelogenous leukemia or myelofibrosis .
这样的脾肿大通常预示有骨髓增生型疾病，例如慢性髓性白血病或骨髓纤维化。你知道它的英文吗？
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Subacut myeloid leukemia can be a defined type of leukemia. The common characteristics of subacute myeloid leukemia are as follows: the onset is insidious, and anemia and hemorrhage of skin and mucosa are the main clinical manifestations. Hepatosplenomegaly is rare. There are multilineage abnormalities in blood cell counts and increased blasts in the bone marrow with cytogenetical abnormal clone in most cases. The disease course is subacute and progressive.
1、亚急性髓性白血病是一个可以确定的白血病类型，其特征为：起病隐袭，以贫血或皮肤粘膜出血为主要症状，肝脾肿大的特征较少，血象为一系、二系或三系的异常，骨髓细胞形态学检查原早幼粒细胞异常增高，细胞遗传学检查多数有克隆性异常核型，病程中可有克隆演进，呈现亚急性而进行性的临床经过。
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ResultsCharacteristic features at the presentation of AITL included generalized lymphadenopathy, fever, splenomegaly, and skin rashes with polyclonal hyper-gammaglobulinemia and other hematological abnormalities (such as Coombs-positive hemolytic anemia), which often involved the bone marrow and had well-described histologic features.The positive rate for CXCL13 was 93.3%.
结果：AITL主要表现为淋巴结肿大、发热、脾大和皮疹，伴有多克隆性高球蛋白血症和其他血液学异常如白细胞升高、Coombs试验阳性，常累及骨髓，具有特征性的病理组织学表现，免疫组织化学实验显示B-淋巴细胞趋化因子CXCL13阳性率为93.3%。
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Polycythemia Vera：真性红细胞增多症
真性红细胞增多症(polycythemia vera)简称\真红\是一种克隆性的以红系细胞异常增殖为主慢性骨髓增生性疾病. 临床特征有皮肤粘膜红紫、脾肿大和血管及神经系统症状. 血液学的特征为红细胞和全血容量绝对增多,血粘滞度增高,常伴有白细胞和血小板增多.
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lienomedullary：脾骨髓的
lienomalacia 脾软化 | lienomedullary 脾骨髓的 | lienomyelogenousleukemia 脾骨髓性白血病
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cystinosis：胱氨酸病
系一种家族性类脂质代谢障碍性疾病,为常染色体隐性遗传因神经磷脂酶缺陷而引起神经磷脂在单核巨噬细胞系统中蓄积,肝脾、淋巴结、骨髓等处有大量的含神经磷脂的网状细胞(3)胱氨酸病(cystinosis):本病为常染色体隐性遗传性疾病,