- 更多网络例句与脱氧作用相关的网络例句 [注：此内容来源于网络，仅供参考]
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Its subject coverage includes: types and effects of deoxidizing agents; deoxidant consumption; deoxidizing procedures; effect of deoxidizers on microstructure and product properties; and furnace and ladle deoxidation.
它的主题报导包括：脱氧剂的类型和作用；还原剂消耗量；脱氧规程；还原作用在微结构和产品特性中；熔炉和杓子去氧。
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The thermodynamic analysis is consistent with the experimental results,which leads to the affirmation of the deoxidation role of Mg during the nitridation of the alloys.
结合实验结果证实了热力学分析的正确性，确定了Mg在反应自生成AlN材料中的脱氧作用。
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In the present paper, the induction characteristic of lymphoblastoid, in-terferon in vitro is described. Results indicate that Namalwa cell line possesses a higher interferon-inducing ability than Raji and Yu-Nu cell lines. NDV B1 strain, F strain and Sendai virus can induce more interferon than BB1 virus and poly 1 : C. Priming of pretreating cells with homologous interferon or superinduction did not effect interferon production. The optimal cell concentration for interferon induction is about 106 cells/ml...
本文研究了类淋巴母细胞干扰素的诱导特性：Namalwa细胞系的干扰素诱生能力大于Raji和余奴系，而诱生剂中以NDV和仙台病毒诱生干扰素的能力较大，但"起动"和超诱导均无作用；诱生干扰素适宜的细胞浓度为10~6/毫升；增殖期的Namalwa细胞诱生能力较大；黄芪和5'-溴脱氧尿嘧啶核苷对Namalwa细胞干扰素的诱生系统有明显刺激作用，在最适条件下粗制干扰素平均滴度可达40，000单位/毫升。
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In the present paper, the induction characteristic of lymphoblastoid, in-terferon in vitro is described. Results indicate that Namalwa cell line possesses a higher interferon-inducing ability than Raji and Yu-Nu cell lines. NDV B1 strain, F strain and Sendai virus can induce more interferon than BB1 virus and poly 1 : C. Priming of pretreating cells with homologous interferon or superinduction did not effect interferon production. The optimal cell concentration for interferon induction is about 106 ce...
本文研究了类淋巴母细胞干扰素的诱导特性：Namalwa细胞系的干扰素诱生能力大于Raji和余奴系，而诱生剂中以NDV和仙台病毒诱生干扰素的能力较大，但"起动"和超诱导均无作用；诱生干扰素适宜的细胞浓度为10~6/毫升；增殖期的Namalwa细胞诱生能力较大；黄芪和5'-溴脱氧尿嘧啶核苷对Namalwa细胞干扰素的诱生系统有明显刺激作用，在最适条件下粗制干扰素平均滴度可达40，000单位/毫升。
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Objective to evaluate the functional recovery of acute spinal cord injuried rats treated with exogenous wnt-3a signal protein administration and to explore its mechanism.methods moderate spinal cord contusion injury was made in 40 adult sprague dawley rats at t10.twenty rats served as contusion controls(group 1).twenty rats were treated with wnt-3a for three days after injury (group 2).the functional recovery of the rats was observed through basso,beattie,bresnahan open field locomotor score.rats were killed at 14 or 28 days after injury,then spinal cords were removed for histopathological examinations,and the expression of the bromodeoxyuridine plus neural cell markers was stained with immunohistochemical method.results rats of two groups receiving a contusive injury recovered substantial function within 1 week.by 28 days,rats in groups 2 scored 7.0 points better on the bbb scores than rats in group 1 group 2=16.94,after 28 days vs.
目的 研究外源性wnt-3a信号蛋白对脊髓损伤的修复作用，并探讨其作用机制。方法取40只成年雌性sd大鼠在t10节段制备适度脊髓挫伤模型。随机从中取20只为损伤对照组(group 1)，另外20只为损伤治疗组(group 2)。脊髓损伤3天后用wnt-3a蛋白治疗。这些大鼠的功能恢复通过basso、beattie、bresnahan开放视野运动评分来观察。这些大鼠分批在损伤后14天或28天被处死，取出损伤节段脊髓用来组织病理学检查，同时用5-溴脱氧尿嘧啶核苷和神经细胞标记物进行免疫组化染色。结果两组大鼠在伤后一周运动功能有明显的恢复。不过，到损伤后28天，我们观察发现，损伤治疗组中的大鼠bbb评分比损伤对照组中的评分高出7.0分左右(group 2∶16.94±1.18，group 1∶9.89±1.29；p.05)，光镜和电镜检查发现wnt-3a蛋白对髓鞘形成和轴突再生有一定的修复效果。
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Result 1 Magnetic nanoparticles, magnetic nanoparticles modified with antisense oligodeoxynucleotide of human telomerase reverse transcriptase induced HL-60 tumor cells to apoptosis, we could see typical morphologic change of apoptosis cells: karyopyknosis, chromation"s condensing and aggregation in nuclear, forming crescent-shaped or annulus structures to lean on edge of cell nucleus"s membrane and posing apoptosis body by Atomic Force Microscope, Fluorescence microscope, transmission electron Microscope 2 There was a significant difference compared with control group(p.01), inhibition ratio had significant positive correlation with medication dosage and time ;during 0.8-8μM dosage amplitude, inhibition ratio accrescenced by dosages increasing. However, the inhibition ratio would decrease when dosage over 8-80μM.
结果 1 磁性纳米粒子、修饰有端粒酶反义寡核苷酸的磁性纳米粒子诱导HL-60细胞发生凋亡，原子力显微镜、光学显微镜、荧光显微镜和透射电镜下均观察到HL-60细胞呈现典型的凋亡细胞的形态变化：细胞核固缩，核内染色质浓缩、凝聚、形成新月形或环状结构紧靠在细胞核膜边缘，并形成凋亡小体。2 磁性纳米粒子、修饰有端粒酶反义寡聚脱氧核苷酸的磁性纳米粒子对HL-60肿瘤细胞的生长和增殖有明显的抑制作用，与对照组相比有显著性差异(p＜0.01)，在剂量为0.8-8μmol／L范围内，抑制率随剂量的增加而增加，当剂量超过8μmol／L时，抑制率反而下降；3 磁性纳米粒子、修饰有端粒酶反义寡聚脱氧核苷酸的磁性纳米粒子可增强p53基因的表达活性，引起DNA降解损伤，反向调节细胞周期活动，促使细胞从G0期进入G1期，抑制肿瘤细胞的生长。4 修饰有端粒酶反义寡聚脱氧核苷酸的量子点能通过内吞作用进入HL-60肿瘤细胞的细胞核，可以在细胞内进行定位和促进HL-60肿瘤细胞的凋亡。
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The above indicated that EWDs found in Lumbricus Bimastus had special enzymological characters and were new DNases, which were different from DnaseⅠand DNase II their catalytic mechanisms and classification wait to be studied and sorted.
这些提示：蚯蚓组织中发现的脱氧核糖核酸酶EWDs具有特殊的酶学特征，不同于DNaseⅠ和DNaseⅡ，是种新型的脱氧核糖核酸酶，但其作用机理和分类有待于进一步的研究和归类。
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Glioma is still one of refractory disease in the neurosurgical field; the development of new primary and adjuvant treatment is vital. Recently, the gene therapy of glioma is developed rapidly and there are many methods about the gene therapy that include: suicide gene therapy, immunologic gene therapy, drug resistangce gene therapy, angiostatin gene therapy and so on. The sucide gene therapy is the most potential approach of antitumer, these nonmammalian genes encode enzyme that convert nontoxic prodrugs into highly toxic metablites. Cells transfected with suicide genes are targeted for specific negative selection, witch can be induced by administrtion of the corresponding produg. Among the enzyme/produg combinations, two of the best characterized system are herpes simplex virus thymidine kinase /ganciclovir and Escherichia coli cytosine deaminase /5-flourocytosine (5-FC). The formor can convert the antiviral nucleoside analogs acyclovir , ganciclovir to their nucleoside monophosphate derivatives, the monophosphate forms are subsequently phosphorylated by endogenus cellular kinases to triphosphates, these molecules are potent inhibitors of DNA synthesis.
近年来脑胶质瘤的基因治疗发展迅速，应运而生的方法有自杀基因、免疫基因、多药耐药基因以及抗血管生成基因等，其中自杀基因被认为是最有前景的基因治疗方法，它又称病毒介导的酶／药物前体疗法，是利用转基因技术将哺乳动物细胞中所不含有的自杀基因转入到哺乳动物肿瘤细胞中，该基因表达的产物可将无毒的药物前体转化为毒性药物，从而选择性杀伤该肿瘤细胞，常用的自杀基因有单纯疱疹病毒-胸苷激酶基因和大肠杆菌胞嘧啶脱氨酶基因，前者催化无毒性抗病毒核苷类似物如丙氧鸟苷、无环鸟苷等成为单磷酸核苷衍生物，然后在内源性细胞激酶作用下转化为具有明显毒性的三磷酸核苷，作为DNA合成链的终止剂和DNA合成酶的抑制剂，干扰细胞DNA的合成；后者编码的胞嘧啶脱氨酶可催化5-氟胞嘧啶（5-FC）脱氨成为5-氟尿嘧啶（5-FU），然后代谢为有毒性的5-氟尿嘧啶-5′三磷酸（5-FUTP）和5-氟-2′脱氧尿嘧啶-5′磷酸（5-FdUTP），5-FUTP通过与UTP竞争性结合而抑制mRNA和tRNA的合成，5-FdUTP则作用于胸苷合成酶，导致TMP衰竭而阻止DNA的合成，最终诱导肿瘤细胞凋亡。
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At last, the effects of JA on the bile salt resistance of Bifidobacteria were studied.The test proved that:Deoxycholic acid Na-Salt had intensely toxical action on Blm and Bbm;Adding glucose and fructose in media could decrease the lexical action on Bbm.but inulinand JAP had not apparent effect.
初步研究了菊芋对双歧杆菌的胆盐耐受性的影响，实验表明，脱氧胆酸钠盐对Blm和Bbm有一定毒性作用；在培养基中补充一定量的葡萄糖和果糖可以改善Bbm的胆盐耐受性，而菊糖和菊芋粉对提高Blm和Bbm的胆盐耐受性没有明显作用。
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What is the function of DNA polymerase in DNA replication?
什麽是脱氧核糖核酸聚合酶的作用在脱氧核糖核酸复制的？
- 更多网络解释与脱氧作用相关的网络解释 [注：此内容来源于网络，仅供参考]
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deoxidation：脱氧(作用),去氧(作用)
deoiling 去油,去油的 | deoscillator 阻压器,减震器 | deoxidation 脱氧(作用),去氧(作用)
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deoxidization：脱氧 脱氧作用
deoxidiaer 去氧剂 | deoxidization 脱氧 脱氧作用 | deoxidize 脱氧 脱氧化膜 除酸
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deoxidization：脱氧(作用)
deoxide | 脱氧 | deoxidization | 脱氧(作用) | deoxidize | 脱氧
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deoxidisation;deoxidization：脱氧(作用);还原;除酸
deoxidation脱氧(作用) | deoxidisation;deoxidization脱氧(作用);还原;除酸 | deoxidizer脱氧剂;还原剂
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deoxidisation;deoxidization：脱氧[作用]
脱氧[作用] deoxidation | 脱氧[作用] deoxidisation, deoxidization | 脱氧剂 deoxidizer,deoxidant
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deoxygenation：脱氧作用
deoxycytidylicacid 胞嘧啶脱氧核苷酸 | deoxygenation 脱氧作用 | deoxygenation 去氧脱氧
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deoxygenation：脱氧作用;除氧
denitrification 脱氮作用 | deoxygenation 脱氧作用;除氧 | depletion 消耗;减损;损害
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deoxidate：脱氧
deoxidant 脱氧剂 去氧剂 | deoxidate 脱氧 | deoxidation 脱氧 脱氧作用 去氧化 去氧作用 除酸 还原
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deoxygenization：脱氧作用
deoxygenation脱氧作用 | deoxygenization脱氧作用 | deoxyribonucleicacid脱氧核糖核酸
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deoxidise：脱氧,脱氧
脱氧作用,脱氧作用 deoxidisation | 脱氧,脱氧 deoxidise | 脱氧作用 deoxygenation