脓蛋白

基本解释 (translations)

pyin

与脓蛋白相关的网络例句 [注：此内容来源于网络，仅供参考]

One hand mechanical obstruct led to the increase of veinous resistance and the obstacle of microcirculation, the other hand the adhesive PMN was activated in excess, the white blood cells released a lot of enzymes, in which PMN-elastase can decompose the components of cell and many albumens, inclusive of immunoglobulin、alexin and fibrication. These components induced the injury of the pancreatic capillary vessels and cell and lysosome enzy made the tissue protein hydrolyze and produced unsaturated fatty acids, which destroyed the structure and function of cellar membrane. The inflammatory cellar factors activate other immunocytes to produce the injury and necrosis of tissue, which aggravated the pathological injury and led to shock、pyaemia and MODS. So ICAM-1 and LFA-1 played an important role in SAP. Frossard found that the expression of ICAM-1 in the rat model, especially in serum、pancreas and lung. All these showed ICAM-1 is an important factor in AP and concomitant lung injury.

胰腺小叶组织局部血管EC首先被激活，ICAM-1表达升高，与被激活的PMN表面表达的LFA-1相结合，"PMN-EC"相互作用加剧，一方面机械性阻塞毛细血管导致静脉阻力增加、微循环障碍；另一方面粘附的PMN过度吞噬或激活，当白细胞吞噬的颗粒不能被封闭隔离，连同细胞内的酶被释放出来，其中的PMN-elastase能够降解细胞基质中各种成分，水解多种蛋白，加重胰腺的毛细血管内皮细胞和腺泡的损伤；释放的溶酶体酶使组织蛋白水解，产生的不饱和脂肪酸引发脂质过氧化方应，破坏细胞膜的结构和功能；释放的炎性细胞因子，激发其他的免疫细胞的功能，导致进一步的组织损伤和坏死，加重SAP的病理损伤，最终导致休克、脓毒血症及多器官功能障碍等严重后果。

Methods:The rats with sepsis was established by administering endotoxin into the peritoneal cavity.The rats were randomly divided into 4 groups as follows:2,6,12 and 24 hours after administration of endotoxin,each group included normal control.We use the in vitro muscle incubation system with sufficient oxygen supply as well as amino acid automatic analyzer for detection of the proteolytic rate of extensor digitorium longus and soleusmuscle.

大鼠腹腔注射内毒素制成脓毒症模型，随机分成内毒素攻击后2、6、12和24小时组，每组设正常对照；利用骨骼肌充分氧供离体孵育系统，采用氨基酸全谱分析仪测定脓毒症大鼠伸趾长肌和比目鱼肌蛋白降解率。

We use the in vitro muscle incubation system with sufficient oxygen supply as well as amino acid automatic analyzer for detection of the proteolytic rate of extensor digitorium longus and soleusmuscle.

大鼠腹腔注射内毒素制成脓毒症模型，随机分成内毒素攻击后2、6、12和24小时组，每组设正常对照；利用骨骼肌充分氧供离体孵育系统，采用氨基酸全谱分析仪测定脓毒症大鼠伸趾长肌和比目鱼肌蛋白降解率。

RESULTS We successfully obtained human caspase-3 gene, the genes and expression vectors of reversed caspase-3 and its fusion protein with part of the translocation domain of Pseudomonas exotoxin A. Compared with the control group, the expression of both genes caused in a similar degree the decrease of living cell numbers and massive occurrence of apoptosis.

结果成功获得了caspase-3基因，构建了重构型caspase-3基因和重构型caspase-3与绿脓杆菌外毒素转膜结构域部分肽段的融合蛋白基因及其表达载体，与对照组相比，在转染后1～4 d内，两种基因的表达均导致HeLa细胞存活数减少，大量细胞发生凋亡，且二者活性相当。

That in fact, the puss and swelling is caused by indoxyl created by the rotten gut of the protein eaters that dissobey every rule of Nature that God ever gave us humans to follow and as a result, some have resulted to drinking their own urine; something we would expect the monkey in the cage to do out of bordom or maybe he is wanting to commit suicide and is just not able...

事实上，脓和肿胀是因为吃腐朽内脏蛋白的人建造吲哚酚引起的，自然的每个孤独规定，上帝曾经给了我们人类去追求和作为结果，有些结果是去喝他们自己的尿，有些事情我们应该去期待，在笼子中的猴子跑出或大概他没有畏罪自杀和恰好不能。。。

Methods The activity of AT-Ⅲ was measured by chromogenic substrate method.

目前有许多证据表明，在由SIRS发展到MODS整个过程中凝血机制异常起到很重要作用，采用活化蛋白-C治疗重症脓毒症也取得

Methods use monoclonal antibody against mammiferous hsp70 to recognize the related antigens of Bacillus pyocyaneus,Bacillus proteus,Bacillus coli,Staphylococcus aureus and Klebsiella pneumoniae with modified western blotting.

用抗哺乳类热休克蛋白（heat shock protein，hsp）70抗体识别绿脓杆菌、变形杆菌、大肠杆菌、金黄色葡萄球菌和肺炎克雷白杆菌抗原，进行免疫转印分析，测试正常豚鼠膜迷路中hsp70的表达水平。

One hand mechanical obstruct led to the increase of veinous resistance and the obstacle of microcirculation, the other hand the adhesive PMN was activated in excess, the white blood cells released a lot of enzymes, in which PMN-elastase can decompose the components of cell and many albumens, inclusive of immunoglobulin、alexin and fibrication. These components induced the injury of the pancreatic capillary vessels and cell and lysosome enzy made the tissue protein hydrolyze and produced unsaturated fatty acids, which destroyed the structure and function of cellar membrane. The inflammatory cellar factors activate other immunocytes to produce the injury and necrosis of tissue, which aggravated the pathological injury and led to shock、pyaemia and MODS. So ICAM-1 and LFA-1 played an important role in SAP. Frossard found that the expression of ICAM-1 in the rat model, especially in serum、pancreas and lung. All these showed ICAM-1 is an important factor in AP and concomitant lung injury.

胰腺小叶组织局部血管EC首先被激活，ICAM-1表达升高，与被激活的PMN表面表达的LFA-1相结合，&PMN-EC&相互作用加剧，一方面机械性阻塞毛细血管导致静脉阻力增加、微循环障碍；另一方面粘附的PMN过度吞噬或激活，当白细胞吞噬的颗粒不能被封闭隔离，连同细胞内的酶被释放出来，其中的PMN-elastase能够降解细胞基质中各种成分，水解多种蛋白，加重胰腺的毛细血管内皮细胞和腺泡的损伤；释放的溶酶体酶使组织蛋白水解，产生的不饱和脂肪酸引发脂质过氧化方应，破坏细胞膜的结构和功能；释放的炎性细胞因子，激发其他的免疫细胞的功能，导致进一步的组织损伤和坏死，加重SAP的病理损伤，最终导致休克、脓毒血症及多器官功能障碍等严重后果。

Objective To investigate the effect of berberine on serum high mobility group box 1 (HMGB1) level and survival rate in sepsis mice.

目的 观察小檗碱对脓毒症小鼠血清高迁移率族蛋白B1(HMGB1)水平和存活率的影响。

Results MIF mRNA and protein expressions in heart tissue significantly increased at the twelfth hour, peaking at the thirty-sixth hour, and a high level was maintained till the forty-eighth hour after CLP. But in the kidney tissue of models, the content of MIF reached peak at the twenty-forth hour and started to decrease at the forty-eighth hour after CLP.

结果 与对照组比较，MIF在脓毒症小鼠心脏组织中的表达增加，在CLP术后12 h开始升高(P.05)，36 h达峰值(P.01)，48h仍维持较高水平(P.05)；肾组织中MIF mRNA的表达在CIP术后12 h开始增加(P.05)，24h达峰值(P.01)，48 h后才开始减少，MIF蛋白则只在CLP术后24 h和36 h增加明显(P.05)。

与脓蛋白相关的网络解释 [注：此内容来源于网络，仅供参考]

mucoprotein：黏蛋白

\\"黏多醣类\\",\\"mucopolysaccharide\\" | \\"黏蛋白\\",\\"mucoprotein\\" | \\"黏液脓性分泌物\\",\\"mucopurulentive discharge\\"

pyin：脓蛋白

pyic 脓的 | pyin 脓蛋白 | pyjama jacket 睡衣上装

pyin：脓蛋白 脓素

pyidostigminebromide麦斯提龙 | pyin脓蛋白 脓素 | pyknictype粗短体型 肥短型 矮胖体型

pyuria：脓尿

尿色:(color)受以下代谢产物影响:(3) 脓尿(pyuria)呈云雾状. (4) 胆红素尿(bllirubinuria) 振荡后泡沫呈黄色. (5) 乳糜尿(chyluria)呈乳白色. 由于干化学法检测球蛋白(globulin)敏感性为白蛋白(albumin)的1/50-1/100,

fluorescin：荧光蛋白,荧光素[如见于绿脓杆菌]

fluorescence yield 荧光产额 | fluorescin 荧光蛋白,荧光素[如见于绿脓杆菌] | fluorination 氟化

mucopurulent：粘液脓性的 粘液浓的

mucoprotein 粘蛋白 粘蛋白 粘蛋白质 | mucopurulent 粘液脓性的 粘液浓的 | mucopurulentbloodystool 粘液脓血便

mucopus：粘液性脓

mucoid 类粘蛋白 | mucopus 粘液性脓 | mucormycosis 毛霉菌病

physometra：输卵管气脓

\\"子宫气肿\\",\\"Physocephalus sexalatus\\" | \\"输卵管气脓\\",\\"physometra\\" | \\"植物白蛋白\\",\\"physostigmine salicylate\\"

pyic：脓的

pygmyisn 像矮人的 | pyic 脓的 | pyin 脓蛋白

pyocele：脓囊肿

或因黏膜分泌物中的蛋白含量过多而引起的一系列生化和免疫反应所致. 本病多发生于筛窦和额窦,蝶窦和上颌窦少见,一般为单侧发病. 2.脓囊肿(pyocele)由于继发化脓性感染而形成,易引起窦壁骨质的吸收破坏,并可向颅内扩展并发颅内感染.