- 更多网络例句与脑水肿相关的网络例句 [注：此内容来源于网络，仅供参考]
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Results The brain ATP production in bilirubin induced neurotoxicity guinea-pig decreased within 4 hours after exposure to bilirubin, and secondary brain edema could be found 8 hours later; GAPA could prevent the secondary brain edema effectively ,but could not prevent the ATP decreasing significantly.
沉积于脑组织胆红素可抑制神经元能量代谢，致脑细胞水肿；兴奋性氨基酸受体拮抗剂GAPA可减轻脑水肿，但不影响能量代谢变化，其作用环节介于能量代谢变化与脑水肿之间。
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One of main manifestations of SBI is cerebral edema which is one of the most serious complications after head injury, cerebral edema can promote intracranial pressure and lead to malposition of brain tissue, even endanger life because of brain hernia.
SBI主要表现之一为脑水肿，脑水肿是颅脑损伤后最严重的并发症之一，它使颅内压增高和加重，引起脑组织移位甚至脑疝而危及生命。
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Objective To observe the role of thrombin in cerebral edema formation following intracerebral hemorrhage and the effect of hirudo powder treatment.
目的 探讨凝血酶在脑出血后脑水肿形成机制中的作用以及水蛭粉治疗脑水肿的疗效。
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Results In these patients who suffered acute mountain sickness, 5.4％had coma in different grades, 5％ had abalienation, 4％ had lethargy, 1％ had paresthesia cacesthesia, 1.48％ had eyeground haemorrhage or eyeguound edema, 1％ had aberrated in intracranial CT.
结果 急性高原病患者中5.4%有不同程度昏迷，5%有精神异常，4%有嗜睡，1%有感觉异常，1.48%有眼底出血或水肿，1%有颅内CT异常；在这部分病人中高原脑水肿占总病例数的10%，在高原脑水肿病例中脑功能障碍比例明显增高，其中精神行为异常者占51%，有不同程度昏迷者占51.5%。
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Results: The brain fabric was normal without edema, hemorrhage and necrosis in groupⅠ. The brain parenchyma were loose, perivascular canal became widen, some neurocytes bulk and degenerate in groupⅡ. In groupⅢ, the brain injury aggravateed accompanying the lapse of survival time with extreme cerebral edema at 24 h, and colliquative necrosis in the brain parenchyma aparted at 40 h.
结果：Ⅰ组脑组织结构清晰，无水肿、出血、坏死病灶；Ⅱ组脑实质疏松，小血管周隙增宽，部分神经细胞体积增大变性；Ⅲ组随溺水后存活时间的推移，脑损伤程度逐渐加重，其中24h脑水肿最重，40h个别脑实质小灶性液化性坏死。
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Thrombin may play an important role in the development of edema formation following intracerebral hemorrhage; Hirudo powder may have a significant effect on brain edema following intracerebral hemorrhage.
凝血酶可能在脑出血后脑水肿形成机制中起重要作用；水蛭粉对大鼠全血和凝血酶导致脑水肿有显著影响。
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TSRP could decrease brain hydrated amount and markedly lower permeability of blood-brain barrier subjected to 2 h MACO followed by 24 h reperfusion, and this may be a mechanism of TSRP alleviating brain edema during I/R.
茅莓总皂苷各剂量组显著降低缺血2h再灌注6，24，48，72 h脑水肿和其对血脑屏障通透性，这可能是其减轻I/R时脑水肿的机制之一。
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The effect of erythrocytes on edema was mainly at the third day after intracerebral hemorrhage, as was probably relevant to the hemoglobin released by the lysed red blood cells. Its effect on edema formation was later than that of thrombin. These results provide a theoretical basis for therapeutic intervention.
红细胞对脑水肿形成的影响主要在脑出血后第3天，可能与红细胞溶解释放血红蛋白有关，由于其对脑水肿形成的影响在时间上比凝血酶要晚，从而在理论上能为治疗干预提供可行依据。
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"CE may occur in the absence of acute changes on head computed tomograms. Early detection of CE at the bedside using an evidence-based protocol permits intervention in time to prevent permanent brain damage," the authors write. The diagnostic criteria for CE proposed in this study require prospective validation before they can be considered the standard of care.
研究人员认为，脑水肿可能会发生在头部电脑断层扫描没有急性改变的情况下，利用一个实证医学为根据的方案，在临床早期侦测出脑水肿，可避免永久性的脑部损害；在这项试验里提出的脑水肿诊断标准，在被考虑作为标准之前，仍必须进行前瞻性的确效试验。
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The authors reviewed a training sample of 26 occurrences of DKA complicated by severe CE and 69 episodes of uncomplicated DKA, along with head computed tomograms, and they incorporated signs of neurologic disease into a bedside evaluation protocol. They then applied this protocol to an independent test sample of 17 patients previously reported to have developed symptomatic CE during treatment for DKA.
研究人员回顾了26个发生糖尿病酮酸血症并发严重脑水肿的训练样本，与69个单纯糖尿病酮酸血症的个案，合并头部电脑断层扫描、神经疾病的病徵融入一个临床评估方案中；接著他们将这个方案应用到一个有17位之前在治疗糖尿病酮酸血症期间，产生脑水肿症状病患的独立试验样本中。
- 更多网络解释与脑水肿相关的网络解释 [注：此内容来源于网络，仅供参考]
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anasarca：全身水肿
水肿按分布范围可分全身水肿(anasarca)和局部水肿(local edema). 也可按发生部位命名,如脑水肿、肺水肿、皮下水肿等. ②抗利尿激素:在全身水肿形成中,抗利尿激素(ADH)增多的滞水作用也有一定意义. 有效循环血量或心排血量下降,
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cerebral contusion：脑挫伤
1.脑挫裂伤 脑挫伤(cerebral contusion)病理为脑内散在出血灶,静脉瘀血、脑血肿和脑肿胀;如伴有脑膜、脑或血管撕裂,则为脑裂伤(laceration of brain). 二者常合并存在,故统称为脑挫裂伤. CT图像上,低密度脑水肿区内,散布斑点状高密度出血灶,
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cerebral dysgenesis：脑发育障碍
cerebral diaplegia 脑性双侧瘫痪 | cerebral dysgenesis 脑发育障碍 | cerebral edema 脑水肿
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edema：脑水肿
文章摘要:脑出血(Intracerebral Hemmorhage ICH)系指原发性非外伤性脑实质内出血,是一种发病率、致残率及病死率都很高的急性脑血管病,至今仍无有效的治疗方法.ICH后引起机体和脑组织局部一系列病理反应,其中脑水肿(edema)是自发性脑出血后最重要的继发性病理变化之一,
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cerebral edema：脑水肿
脑水肿(cerebral edema)是指脑组织内液体异常聚积,从而使颅内压升高而引起的一系列临床症状的综合征. 脑水肿(cerebral edema)是指脑组织内液体异常聚积,从而使颅内压升高而引起的一系列临床症状的综合征.
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brain edema：脑水肿
方法 将近年来我院住院糖尿病周围神经病变患者246例随机分为治疗组(126....中文摘要: 脑水肿(brain edema)是颅脑损伤(traumatic brain injury, TBI)后所继发的一系列神经细胞变性、水肿以至坏死的病理过程,轻者引起神经功能障碍,
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intracerebral hematoma：脑水肿
脑出血:intracerebral heamorrhage | 脑水肿:intracerebral hematoma | 脑血肿:intracerebral hematoma
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hydrocephalus：脑水肿
[生物通讯]美国环境卫生科学研究所的科学家识别出一个导致小鼠出生缺陷--脑水肿(hydrocephalus)的基因--RFX4. 丢失一个拷贝该基因,小鼠的脑脊髓液就无法从脑腔中排出,从而引起脑水肿.
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inflammatory mediators：脑水肿
炎性反应:inflammatory | 脑水肿:inflammatory mediators | 聚类分析:systemic cluster
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Cerebral oedema：脑水肿
The Circle of Willis 脑底动脉环 | Cerebral oedema 脑水肿 | Temporal pole 颞极