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The concentration of GLY increased gradually with increasing hypoxia runs,but contents of GLY after four runs exposure reduced in telencephalon and diencephalon.
各脑区L-GLY含量均随着缺氧次数的增多而增高，端脑、间脑的含量在缺氧4次组有明显回降。
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Our result showed that P450scc expression was observed in hippocampus, habenular nucleus, neuron nucleus of thalamus and hypothalamus, and cerebral neocortex. The colocalization of P450scc and NeuN demonstrated that most of P450scc exist in neuron cell in these regions. In order to clarify the transcriptional regulation of CYP11A1 gene, established the transient SCC-iCre transgenic mice model by using R26R system to study the promoter activity of human CYP11A1 gene in the brain.
为了证明内生性P450scc在这些脑区的表现，本研究利用免疫组织染色分析，说明了在小鼠海马回结构，间脑内的上视丘疆核、视丘神经核、下视丘神经核，与新皮质等区域中皆可侦测到，且主要表现在神经细胞。
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One-sample t test was used in profoundly congenital hearing loss infants and control group,the activated brain areas were:bilateral transverse temporal gyri, superior temporal gyrus,middle temporal gyrus,bilateral insular lobe,bilateral precentral gyrus,postcentral gyrus,bilateral cingulate gyrus,bilateral inferior parietal lobule,bilateral superior parietal lobule,bilateral superior frontal gyrus.
在对极重度感音神经性耳聋患儿和对照组进行组间比较发现，病人组数据减去对照组数据时可见激活的脑区主要有：双侧颞横回、双侧颞上回、双侧颞中回、双侧岛叶、双侧中央前、后回、双侧扣带回、双侧顶下小叶、双侧顶上小叶、双侧额上回等，提示极重度感音神经性耳聋患儿在接受刺激后动用了更多的脑区而且激活强度明显增加。
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There were no statistically differences between the 8th~ 14thday, the 15th - 21th day post-BC and the control group. During the rt~24tkday post-BC , pycnosis degeneration or necrosis neurons of the locus cerebral cortex, dorsal hippocampus, dentate fornix were significantly increased, then decreased gradually, but recovered to normal by the 24thday after BC, and especially in parietal cortex and piriform cortex the necrosis neurons were significantly increased than temporal cortex , and there were no statistically difference berween the left and the right side. Pycnosis degeneration or necrosis neurons in the brainstem reticular formation were markedly increased in the 4thday after BC, and there were no statistically difference among the other groups and the control group.
与对照组相比，BC后在大脑皮质、背侧海马和齿状回部位，固缩变性和不完全坏死细胞数先显著增加(P＜0.05)，然后逐渐减少，至24d基本恢复正常；顶叶、梨状皮质比颞叶皮质变性坏死细胞多，差异有高度显著性(P＜0.01)；大脑左侧比右侧变性坏死细胞稍少但差异无显著性；在BC后4d，脑桥核、斜方体核平面的脑干网状结构中固缩变性和不完全坏死细胞数明显增多(P＜0.05)，其它组间无显著性差异。
- 更多网络解释与脑回间的相关的网络解释 [注：此内容来源于网络，仅供参考]
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gyrus：回
范艾森认为,大脑皮质上两个区域间有许多轴突相连,在发育期间会因为系缚的轴突产生拉力而拉近,形成隆起的脑回(gyrus);相对的,连结较弱的区域就会被拉开,分隔在脑沟(sulcus)的两侧.
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intergyral：脑回间的
intergroup relation 群际关系 | intergroup?contact hypothesis 团体间接触假说 | intergyral 脑回间的