胰腺

基本解释 (translations)

pancreas  ·  pancreases

与胰腺相关的网络例句 [注：此内容来源于网络，仅供参考]

The results showed that the method of rubbing the kerf then injection of retrovirus suspension into pancreas could more effectively mark the pancreatic cells than the caudal vein injection and the intraperitoneal injection did in vivo. Furthermore, this study also found that some acinus cells could accept injury stimulus signals to regenerate through resuming mitosis after pancreatic injury.

结果发现在活体条件下，与尾静脉注射及腹腔注射法相比，胰腺伤口涂抹并胰腺内注射反转录病毒的方法能够更有效的标记胰腺细胞；而且，通过对标记细胞的世系追踪研究证明，在胰腺损伤后，胰腺腺泡细胞能够接受损伤信号刺激发生再生。

Also, few centers perform pancreatectomy for nonmalignant disease because of problems with fragile diabetes, especially in alcoholics.

个人评论：单纯就其统计学意义来说，研究人员将接受手术的胰腺癌和慢性胰腺炎患者的死亡率进行了比较，统计表明手术治疗慢性胰腺炎手术死亡率低于胰腺癌患者，但这仍不能作为建议手术治疗慢性胰腺炎的依据，因为没有对慢性胰腺炎患者中手术治疗和非手术治疗患者的预后和死亡率进行对比，我们还无法认定手术治疗慢性胰腺炎效果优于非手术治疗。

Ap with hyperlipaemia model was prepared by both triton wr1339 and cerulein simultaneously. human albumin was used to treat the ap accompanied with hyperlipaemia. in each group, the histological score, volume of ascites, ratio of pancreatic wet/dry weigh, serum amylase and pancreatic acinar cell apoptosis in each group were compared. the level of protein kinase c membrane translocation in pancreatic tissue was detected by western blot.

分别用triton wr1339、cerulein制作大鼠高脂血症和急性胰腺炎（acute pancreatitis，ap）模型，同时应用两者制作伴有高脂血症的ap模型，应用白蛋白治疗伴有高脂血症的ap，比较各组胰腺病理损害评分、腹水量、胰腺湿/干比、血清淀粉酶和胰腺组织的凋亡；western blot检测胰腺组织pkc的膜转位。

Methods Hyperlipaemia and AP model were established with Triton WR1339 and Cerulein respectively. AP with hyperlipaemia model was prepared by both Triton WR1339 and cerulein simultaneously. Human albumin was used to treat the AP accompanied with hyperlipaemia. In each group, the histological score, volume of ascites, ratio of pancreatic wet/dry weigh, serum amylase and pancreatic acinar cell apoptosis in each group were compared. The level of protein kinase C membrane translocation in pancreatic tissue was detected by Western blot.

分别用Triton WR1339、Cerulein制作大鼠高脂血症和急性胰腺炎（acute pancreatitis，AP）模型，同时应用两者制作伴有高脂血症的AP模型，应用白蛋白治疗伴有高脂血症的AP，比较各组胰腺病理损害评分、腹水量、胰腺湿/干比、血清淀粉酶和胰腺组织的凋亡；Western blot检测胰腺组织PKC的膜转位。

They were male in 8 eases and female in 14. Pancreatic cystoadenoma was in 14 cases (63.30%) and pancreatic cystoadenocarcinoma in 8(36.3%). It was 1.7:1 between two groups. Tumor at the head of pancreas was 6 cases (27.21%). at body-tail 12 (54.5%). at tail 4 (18.1%). Local resection was performed in one case, pancreatic body-tailectomy pancreatoc1uodenectoni in 3, central pancreatectomy in 2 cases.

男8例，女14例。14例(63.3%)为胰腺囊腺瘤（浆液性6例、黏液性8例），8例(36.3%)为胰腺囊腺癌，两者之比为1.7：l肿瘤位于胰腺头部6例(27.2%)，胰腺体尾部12例(54.5%)，胰腺尾部4例(181%)行胰腺肿瘤局部切除术l例，肤腺体尾部切除术14例，胰头十二指肠切除术3例，胰腺中段切除术2例，囊肿－空肠Roux-Y吻合术1例，T管架桥胆－肠内引流术1例。

Results The DNA was obtained from PPJ through a nasopancreatic tube. Aberrant p16 methylation and K- ras gene mutation were detected in the same samples of PPJ. Sensitivity,specificity,positive predictive values,negative predictive values and accuracy of HE staining for pancreatic cancer were 40%,100%,100%,45.4% and 60.0%,respectively. Of the 20 cases of pancreatic cancer,K- ras gene mutation was detected in 14 (70%) and the p16 gene was shown to be methylated in 7 (35%). Of the 8 cases of chronic pancreatitis,K-ras gene mutation was detected in 2 (25%). Of the 2 cases of mucinous cystoadenoma of pancreas,K-ras gene mutation was detected in 1 (50%). Aberrant methylation of p16 was not detected in pancreatic juice samples from patients with chronic pancreatitis and mucinous cystoadenoma of pancreas.

结果 所有胰液标本均成功抽提出DNA，30例胰腺疾病病人胰液标本同时进行了K-ras基因突变和p16基因启动子区5′CpG岛甲基化检测，其中20例胰腺癌病人胰液中K-ras基因突变率为70%（14/20），p16基因甲基化率为35%（7/20），8例慢性胰腺炎中K-ras基因突变率为25%（2/8），2例胰腺囊腺瘤病人中K-ras基因突变率为50%（1/2），慢性胰腺炎和胰腺囊腺瘤病人胰液中无p16基因甲基化。

American Atlanta conference edited in September 1992 pancreatitis of serious illness acute definition is: AP companion has local complication (cyst of pancreatic and necrotic, false sex, pancreatic abscess) or the organ fails (function of shock, lung not complete, kidney declines, gastrorrhagia, sow medicinal powder the cruor inside sexual blood-vessel and serious metabolization obstacle), or both holds concurrently.

概要： 急性胰腺炎为临床常见急腹症，是由于胰液和溶蛋白酶漏入到胰腺实质和胰腺四周组织而引起。1963年，法国马赛会议以病理形态学为基础，将AP分为水肿性胰腺炎和出血、坏死性胰腺炎。1992年9月美国亚特兰大会议修订重症急性胰腺炎定义为：AP伴有局部并发症(胰腺坏死、假性囊肿、胰腺脓肿)或器官衰竭(休克、肺功能不全、肾衰、胃出血、播散性血管内凝血和严重的代谢障碍)，或二者兼有。

Result:the incidence of AP went up year by year;(2)bile tract disease is the major reason,but hyperlipdemic AP should be attach importance to for its higher and higher incidence;(3)mean age of biliary AP is higher than alcohlolic and hyperlipdemic AP;(4)the difference of sex is significant in biliary AP and alcohlolic AP;(5)the incidence of local omplications is lower than alcohlolic and hyperlipdemic AP,but the difference is of no significance in view of systemic complications;(6)lungis of the most liability for dysfunction,and renal dysfunction is most closely related to death ;(7)ESS is associated with AP and related to the severity and prognosis .

结果表明：(1)急性胰腺炎发病率呈逐年上升趋势；(2)胆道疾病仍是急性胰腺炎的首要病因，高脂血症性胰腺炎明显增多，应受到重视；(3)胆源性胰腺炎患者年龄高于酒精性和高血脂性等其它原因的胰腺炎；(4)性别差异在胆源性、酒精性胰腺炎中显著；(5)胆源性胰腺炎出现局部并发症几率少于酒精性和高血脂性胰腺炎，而在全身并发症上无明显差异；(6)重症急性胰腺炎中，肺脏最易发生功能障碍，肾功能障碍与死亡关系最密切；(7)急性胰腺炎易伴发正常甲状腺病态综合症，并与其严重程度和预后相关。

The percentage of TGF-β〓 positive cell in the pancreatic cancer tissue (43.8±5.2)％ was significantly higher than that in adjacent pancreatic tissue (28.7±3.6)％, P＜0.05. The worse the cancer cells differentiated and lymphy node metastasis, the more over-expression of TGF-β〓. 2. The percentage of Tr positive cell in the pancreatic cancer tissue (41±4)％ was significantly higher than that in adjacent pancreatic tissue (23±3)％, P＜0.05. The worse the cancer cells differentiated and lymphy node metastasis, the more over-expression of Tr, but the expression of Tr protein was not correlated with lymphy node metastasis (P＞0.05). 3. The percentage of β-GCD positive cell in the pancreatic cancer tissue (62.5± 4.1)％ was significantly higher than that in adjacent pancreatic tissue (33.5±2.8)％, P＜0.05. The worse the cancer cells differentiated and lymphy node metastasis, the more over-expression of β-GCD in pancreatic cancer tissue, but the expression of β-GCD protein was not correlated with lymphy node metastasis. P＞0.05. 4. the expression of β-GCD was significantly correlated with TGF-β〓 and Tr in the pancreatic cancer tissue.

结果如下：1、胰腺癌组织TGF-β〓阳性细胞百分率(43.8±5.2)％明显高于癌旁胰腺组织(28.7±3.6)％，P＜0.05；且癌细胞分化愈差或有淋巴结转移TGF-β〓过度表达愈多。2、胰腺癌组织Tr阳性细胞百分率(41±4)％，明显高于癌旁胰腺组织(23±3)％，P＜0.05；且不同分化程度胰腺癌组织Tr表达强度不同，分化程度愈低，表达强度愈高，P＜0.05；但胰腺癌Tr表达强度与淋巴结是否转移无关，P＞0.05.3、对于胰腺癌组织TGF-β〓和Tr表达，检测胰腺癌组织(32例)β-GCD阳性细胞百分率分别为(62.5±4.1)％或(62±4)％，分别明显高于癌旁胰腺组织β-GCD阳性细胞百分率(33.5±2.8)％或(43±3)％，P＜0.05；不同分化程度胰腺癌组织β-GCD表达强度不同，分化程度越低，表达强度越高，P＜0.05；但胰腺癌组织β-GCD表达强度与淋巴结是否转移无关，P＞0.05.4、TGF-β〓、Tr和β-GCD在胰腺癌组织中的表达随着分化程度的改变，呈现一致性的关系，而且TGF-β〓与淋巴结转移有关，Tr和β-GCD与淋巴结是否转移无关。

Second is studying the traumatic condition changes in a choosey pressure at different post-PT phases. Moreover, we detected model PLA_2 activity, express of PLA_2 subtype mRNA by fluorescent quantitative RT-PCR technique, protein express and site by Western blot and IHC at different post-PT phases. At last, we studied the PLA_2 inhibited effects and model protection of Variabilin in vivo.

研究在不同压强的外力作用下，模型指定时相点的一般情况和存活率、大体、光镜及电镜下胰腺组织的病理变化和常规酶学变化，得到稳定建模致伤压强；然后，固定致伤压强，研究胰腺创伤后不同时相点的伤情变化(包括一般情况、胰腺组织的病理变化、AMS和LPS活性变化、血常规、血生化、胰腺细胞凋亡以及胰腺组织细胞生长周期变化情况)；再次，采用3H-花生四烯酸标记大肠杆菌膜作为底物的方法检测大鼠胰腺创伤后不同时相点的PLA_2活性，利用荧光定量RT-PCR技术检测其亚型mRNA的表达，利用Western blot和IHC技术检测其蛋白表达量和蛋白组织定位；最后，将Variabilin体内应用于急性胰腺创伤大鼠模型，研究其对模型的保护作用和对PLA_2的抑制效应。

与胰腺相关的网络解释 [注：此内容来源于网络，仅供参考]

Exocrine Pancreas：(胰腺的外分泌)

Endocrine Pancreas(胰腺的内分泌) | Exocrine Pancreas(胰腺的外分泌) | Exocrine Pancreatic Insufficiency(胰腺外分泌不足)

Exocrine Pancreas：胰腺外分泌[医]

胰腺石病[医]Pancreatolithiasis | 胰腺外分泌[医]Exocrine pancreas | 胰腺外瘘[医]external fistula of pancreas

Postpancreatectomy hyperglycemia：胰腺切除术后高血糖[医]

胰腺泡[医]Pancreatic acinus | 胰腺切除术后高血糖[医]Postpancreatectomy hyperglycemia | 胰腺石病[医]Pancreatolithiasis

pancreatitis：胰腺炎

胰腺炎(pancreatitis)是胰腺因胰蛋白酶的自身消化作用而引起的疾病. 急性胰腺炎是由于胰酶消化胰腺本身组织而引起的化学性炎症;慢性胰腺炎是指胰腺的复发性的或持续性的炎性病变. 胰腺炎发病原因与胆汁或十二指肠液反流人胰管或胰管梗阻.

acute pancreatitis：急性胰腺炎

急性胰腺炎(acute pancreatitis)是常见的急腹症之一,是胰酶在胰腺内被激活而发生胰腺自身消化的化学性炎症. 临床上通常有急性上腹痛和血清胰酶升高. 大多数属轻型,其病理基础是胰腺细胞的水肿,少数可伴轻微胰腺坏死病灶,故亦称急性单纯水肿性胰腺炎.

acute pancreatitis：胰腺炎

[概述] 急性胰腺炎(acute pancreatitis)是指胰腺分泌的消化酶引起胰腺组织自身消化的化学性炎症. 临床主要表现为急性上腹痛、发热、恶心、呕吐、血和尿淀粉酶增高,重症伴腹膜炎、休克等并发症. 本病可见于任何年龄,但以青壮年居多.

chronic pancreatitis：慢性胰腺炎

慢性胰腺炎(chronic pancreatitis)是由于慢性酒精中毒.胆道系统疾病等各种原因造成的胰腺组织结构和功能持续性损害.胰腺组织节段性或弥漫性的发生慢性进行性炎症,其病理学特点包括胰腺腺泡减少、腺体萎缩、纤维化、钙化及胰腺导管串珠样改变等不可逆的胰腺实质破坏.

Pancreatic steatorrhea：胰腺性脂泻[医]

胰腺性胸膜积液[医]Pancreatic pleural effusion | 胰腺性脂泻[医]Pancreatic steatorrhea | 胰腺炎高营养配方[医]Hyperalimentation formula for pancreatitis

Cauda pancreatis：胰腺尾

胰腺头 - Caput pancreatis | 胰腺尾 - Cauda pancreatis | 胰腺淋巴结 - Nodi pancreatici

heterotopic pancreas：胰腺异位

胰腺移植, pancreas transplantation | 胰腺异位, heterotopic pancreas | 胰腺周围脓肿, peripancreatic abscess