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During the curing process, first,to inject the Harmony No.1 at focus of patient directly to let medicine go inside of tumour, and after 24hours, the medicine will damage and dissolve the tumour cells; second, prick the area has been injected and make it as cinquefoil, use minus-pressure puller to drain gore of dead tumour cell out of body; third,to inject Harmony No.1 at focus again, then inject in mainline to clear virus and tumour cells haven't been formed in blood.
在对病人治疗过程，首先对肿瘤癌症病灶部位直接注射和谐一号核子注射液，使药物直接进入肿瘤内部，经过24小时药物对肿瘤细胞的杀死破坏及溶解；第二步，对注射处扎针，使其呈梅花状，用负压吸引器把含有坏死肿瘤细胞的瘀血排出体外；第三步，给病人病灶部位注射和谐一号注射液，再对病人注射静脉针液，用来清除血液中的病菌病毒及杀死血液中未形成的肿瘤细胞。
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The method is highly sensitie, he says: as few as fie percent of the cells within the tumor mass needed to be induced into the lytic cycle in order to be detected.
这个方法的敏感性很高，他还说，在肿瘤中只要有5%的肿瘤细胞能被诱导转入溶解期，这个方法就能检测出肿瘤来。
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Human neurotropic JC virus is able to infect human brain glial cells. The causative agent of the fatal human demyelinating disease progressive multifocal leukoencephalopathy, that cytolyses and destroys oligodendrocytes, the myelin-producing cells of the central nervous system. Also results from several experiments have indicated the JCV is able to induce neoplasms.
人类嗜神经性JC病毒可感染人类脑细胞是引起进行性多病灶脑白质病的因素，为一种致命性的脱髓鞘疾病，其导致神经系统产髓质细胞的溶解破坏，但JCV也在许多动物实验上显示其有促成肿瘤的潜在性。
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Results Cells cultured in serum-free medium AIMV could proliferate,kill target cellsand express CD11 c well enough to substitute the cells cultured in standard serum-containing medium.Proliferation,cytotoxicity and expression of CD11 c of A-NK were better than nonadherent natural killer cells.Death patterns of tumor target cells were necrosis and apoptosis.
结果 与完全培养基培养细胞相比，AIMV培养细胞增殖能力、杀伤肿瘤细胞的能力、表达CD11 c 的能力与之相当；在同一培养条件下A-NK细胞的增殖能力、杀伤活性及表达CD11 c 的能力明显强于NA-NK细胞；被A-NK细胞杀伤的肿瘤细胞死亡形式是溶解坏死和凋亡。
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Results Cells cultured in serum-free medium AIMV could proliferate,kill target cellsand express CD11 c well enough to substitute the cells cultured in standard serum-containing medium.Proliferation,cytotoxicity and expression of CD11 c of A-NK were better than nonadherent natural killer cells.Death patterns of tumor target cells were necrosis and apoptosis.
结果 与完全培养基培养细胞相比，AIMV培养细胞增殖能力、杀伤肿瘤细胞的能力、表达CD11 c 的能力与之相当；在同培养条件下A-NK细胞的增殖能力、杀伤活性及表达CD11 c 的能力明显强于NA-NK细胞；被A-NK细胞杀伤的肿瘤细胞死亡形式是溶解坏死和凋亡。
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Results The BMC surrounding tumor mass and strands showed multi-layered or dissolution, pseudopodia were seen extended from tumor cells, and contained numerous microvesicles.
瘤细胞伸出伪足样突起，且胞浆内含大量微泡，该处基膜消失，胶原原纤维溶解，肿瘤细胞围绕血管部位的血管基膜增厚，纤维组织增生。
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Materials and Methods Thirty SD rats, feeding in clearing grade standard, were classified into five groups at random. Application of transmission electron microscopy, scanning electron microscopy, light microscopy and immunohistological staining meth- ods, normal morphology of outflow pathways was observed and component of extra cel- lular matrix was defined. The effects of 50 IU of TNF-α given intracamerally on the morphology of outflow pathways and other tissues of anterior segments, expression of trabecular stromelysin and the change of components of trabecular extra cellular matrix were observed in 24h, 3days, 7days and 14days after injection.
中文题名TNFα对鼠眼房水外流影响的形态学观察副题名外文题名 The effect of TNFα on morphology of outflow pathways in rat eyes 论文作者王大博导师王竫华教授学科专业眼科学研究领域\研究方向学位级别博士学位授予单位青岛大学学位授予日期2002 论文页码总数90页关键词青光眼房水外流通道肿瘤坏死因子-α馆藏号BSLW /2003 /R775 /1 目的在观察SD大鼠眼房水外流通道形态及确定其房水外流通道细胞外基质成份的基础上，探讨房水中肿瘤坏死因子α浓度的增加是否能诱导小梁细胞基质溶解素的表达和分泌，进而启动小梁细胞外基质的重塑过程。
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oncolytic：溶瘤细胞的
oncolysis 溶癌作用 | oncolysis 肿瘤细胞溶解 | oncolytic 溶瘤细胞的
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oncolysis：瘤细胞溶解
oncogeography 肿瘤地理学 | oncolysis 瘤细胞溶解 | one crop system 单作
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oncolysis：肿瘤细胞溶解 溶癌作用
oncology 肿瘤学 | oncolysis 肿瘤细胞溶解 溶癌作用 | oncolytic 溶瘤细胞的