肺泡内的

词组短语

intra-alveolar

与肺泡内的相关的网络例句 [注：此内容来源于网络，仅供参考]

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概要： 隐源性机化性肺炎是一类以临床病理命名的间质性肺炎，COP是1985年Epler等[1]提出的病理表现为肺泡内、肺泡管、呼吸性细支气管及终末细支气管腔内有肉芽组织形成的独立病种，又称特发性闭塞性细支气管炎伴机化性肺炎，近20年来报道逐渐增多，易和肺部感染、结核、肿瘤、结节病、其他间质性肺疾病等混淆，现将我院2007年至2008年收治的支气管镜肺活检获得病理证实的8例病例分析如下。

The honeycomb vascular networks were various in size and form at a low magnification. The venule connected with the pulmonary capillary networks directly. In some fields of view, the pulmonary alveolar networks were plate-like and sieve-like.

低倍镜下肺实质内的微血管网呈现大小不等的蜂窝状，肺实质内微静脉与肺泡毛细血管之间联系紧密；在局部区域，肺泡毛细血管网可呈现板块状或筛网状。

Intrapulmonary pressure is the pressure in pulmonary alveoli.

肺内压是指肺泡内的压力。

These results suggested that the lung Fib were activated and produced excess HA, resulting in the excessive accumulation of HA within the lung at the stage of alveolitis during the process of pulmonary fibrosis. However, increased HA synthesis of lung Fib and increased HA levels in BALF merely reflect the intensity of alveolitis and the disease activity rather than the degree of pulmonary fibrosis.

以上结果提示：肺Fib在肺纤维化的早期肺泡炎阶段就处于活化状态，产生HA增多，导致肺内HA的过多聚积，肺Fib产生HA增多和BALF中HA水平的增高可反映肺泡炎的强度和疾病的活动性，但并不能反映肺胶原代谢或纤维化的程度。

The main pathological findings include a desquamative interstitial pneumonia-like reaction with intra-alveolar macrophages and numerous large multinucleated histiocytes that ingested inflammatory cells were admixed with macrophages.

GIP主要病理表现有脱屑性间质性肺炎样反应，即在肺泡腔内有巨噬细胞和大量的多核巨细胞聚集，位于肺泡腔内的多核巨细胞内可见被吞噬的炎性细胞，是GIP的特点。

Because of little and not well-distributed suction power of gas molecule to liquid molecule, there is surface tension between the liquid and gas in alveoli(a kind of suction power or concentric force souced from surface).

肺泡内液气界面存在着表面张力（一种表面能来源与分子间的吸引力或内聚力），是由于气体分子对肺泡内面表面液气分子吸引力很小，液体分子各向受力不匀的表现。

Some exudation and bleedings happen in the pulmonary alveoli, some inflammatory cells infiltrate into lung tissues; in the serum AMY, ALP, TNF-α, IL-6 increases obviously high, COX-2 expression increase; The expression of NF-κB in big rat lung of SAP increase, and the nucleus is thick dyed, it explain that the NF-κ moved into the nucleus; Stimulate the inflamatory reaction.

我们的实验中见到SAP组的大鼠肺脏组织细胞腔扩大、肺泡壁断裂，有的出现肺大泡，肺泡内伴有渗出及少量非出血，肺组织可见咽细胞浸润；血清中AMY、ALP、TNF-α、IL-6明显增高；在SAP组大鼠肝细胞、肾小管上皮细胞、肺脏小气管及肺泡上皮上COX-2表达增加；NF-κB在SAP大鼠肺脏中表达增加，并且出现核浓染，说明NF-κB前移入核；启动炎症反应等一系列反应SAP大鼠受损伤的肺组织中AQP-1无论在mRNA水平上、蛋白水平上还是免疫组化染色病理学检测上都明显下降。

The alveolar cavities were made of single-layer capillary networks and the alveolar cavities varied in size. The alveolar cavities were half-hollow spheres, concave to outside and base to inside.

单层毛细血管网围成肺泡腔，腔体大小不一，伸展状态较好的肺泡呈现出凹面向外，底面朝内的半空心球状结构。

The causes of pneumomediastinum in DKA patients may be a result of vomiting or Kussmaul's respiration, where increased intra-alveolar pressure permits air to dissect along the perivascular sheath into the mediastinum.

造成气纵膈的可能原因，酮酸中毒病人会发生呕吐或库氏呼吸(Kussmaul's rcspiration)，此会增加肺泡内压力，使得空气可剥离肺泡血管周围鞘进入纵膈腔内。

The cytochemistrialstaining was used to detect the activities of PAMs.The results showed that the activities of ACPaseat the early stage of postinoculation,the activities of glycogen or glycoprotein,ATPase and ANAEin PRRSV-infected PAMs were markedly decreased.

用酶组织化学染色法对感染PRRSV的仔猪肺泡巨噬细胞内的细胞化学成分进行了动态研究，发现PRRSV急性感染早期肺泡巨噬细胞ACPase活性减弱，感染后各时期肺泡巨噬细胞内糖类、ATPase和ANAE活性均减弱。

与肺泡内的相关的网络解释 [注：此内容来源于网络，仅供参考]

alveolus：肺泡

肺脏内被网状隔膜分隔成许多小室,称为肺泡(alveolus),以此增大肺脏与空气的接触面积;肺泡壁密布着肺动脉和肺静脉的微血管,使动物体的气体交换在肺内得以顺利进行.

atelectasis：肺膨胀不全 肺不张

肺萎陷(collapse of lungs)又称肺膨胀不全,肺不张(atelectasis),是指肺泡内空气含量减少甚至消失,以致肺泡呈塌陷关闭的状态. 肺不张或肺膨胀不全一般是指先天性的,即肺组织从未被空气扩张过,由于呼吸道被胎粪、羊水、黏液阻塞或胎儿呼吸中枢发育不成熟等原因,

BAL：支气管肺泡灌洗

在实际工作中,病人一旦建立人工气道,在第一时间内,我们给予病人进行气管镜检查通过带保护套的标本刷(PSB)及支气管-肺泡灌洗(BAL)方法,获取下呼吸道分泌物标本,提高病原菌诊断的阳性率,有的放矢的使用抗生素,

bleb：气泡

7 肺大泡 CT 表现① 常于肺中下部 ; ② 表现为圆形 , 椭 病理 肺实质内的空气腔称为肺大泡 ( bulla ) , 如位于肺边缘由多数肺泡融合而成 , 则称气泡 ( bleb) 或肺气瘤 (pneumatocele ) .其壁由肺泡壁及纤 维组织构成 ,

carnification：肉质变

使相邻肺泡内的纤维素网互相连接 图9-16 大叶性肺炎 左下叶实变,呈灰白色,肺叶明显肿胀 合并症 (1)肺肉质变:因吞噬细胞数量少或功能缺陷,渗出物不能被完全吸收清除时,则由肉芽组织予以机化(图9-17),病变部位肺组织变成褐色肉样纤维组织,称肉质变(carnification).

alveolar macrophage：肺泡巨噬细胞

有的巨噬细胞游走入肺泡腔内,称肺泡巨噬细胞(alveolar macrophage). 肺巨噬细胞的吞噬、免疫和分泌作用都十分活跃,有重要防御功能. 吸入空气中的尘粒、细菌等异物进入肺泡和肺间质,多被巨噬细胞吞噬清除,故细胞胞质内常见尘粒、细菌等物进入肺泡和肺间质,

maldevelopment：发育不良

2.肺血管发育不良(maldevelopment) 指在宫内表现为平滑肌从肺泡前(prealveoli)生长至正常无平滑肌的肺泡内(intra-alveoli)动脉,而肺小动脉的数量正常.

PAP：肺泡蛋白质沉积症

肺泡蛋白质沉积症(PAP),以肺泡和细支气管腔内充满过碘酸-雪夫(PAS)染色阳性的不可溶性的富磷脂蛋白质物质为其特征. 好发于青中年,男性发病约2倍于女性. 病因未明,

surfactant：表面活性物质

(1)表面活性物质(surfactant)缺乏:肺表面活性物质由Ⅱ型肺泡上皮细胞生成是一种磷脂蛋白质复合物起主要作用的是二棕搁酰卵磷脂表面活性物质衬覆在肺泡内面具有降低肺泡的气-液交界面表面张力的作用有稳定肺泡防止肺泡萎陷的功能如果表面活性物质缺乏则肺泡表面张力增大肺泡回缩力增加肺泡即萎陷造成很多处微型肺不张(microa

intrapulmonary pressure：肺内压

当呼吸肌收缩或舒张,引起胸廓扩大或缩小使胸腔容积增大或缩小,肺也随之扩大或缩小,导致 肺泡内的压力即肺内压(intrapulmonary pressure)降低或升高. 由于肺与外界直接相通,肺内压低于大气压时气体进肺即吸气;肺内压高于大气压时气体出肺即呼气.