- 更多网络例句与缺氧相关的网络例句 [注：此内容来源于网络，仅供参考]
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Methods］By observing the cytoplasmic Ca2+ concentration change of single cardiocyte in I/R status and the effect of Na+/H+ permutoid retarder to Ca2+ concentration in different phases (diabetes pure oxygen-lack deficit group called DM group,the oxygen deficit/reaeration on entire journey for medicine group called DM-EIPA group, the first group giringmedicine before reaeration on called DM-EIPA 1 group, and the second group giringmedicine before reaeration on called DM-EIPA 2 group) to detect the protection mechanism of Na+/H+ permutoid retarder to diabetic mouse cardiac muscle cell oxygen deficit/reaeration injury.
方法］ 通过观察糖尿病鼠单个心肌细胞在缺氧/复氧时细胞胞浆Ca2+浓度的动态变化以及Na+/H+交换体阻滞剂在不同时相（糖尿病单纯缺氧组即DM组，缺氧/复氧全程给药组即DM-EIPA组，复氧前给药1组即DM-EIPA 1组，复氧前给药2组即DM-EIPA 2组）对Ca2+浓度的影响来研究Na+/H+交换体阻滞剂对糖尿病鼠心肌细胞缺氧/复氧损伤的保护机制。
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The experiments showed a significant increase in Bax mRNA levels (P.01) in neurons cultured at severe hypoxia condition compared with those cultured at normoxia. In hypoxia groups, tolbutamide increased Bax mRNA expression (P.05), while diazoxide reduced it (n=5 for each).
缺氧+二氮嗪组中基因Bax的mRNA表达水平与单纯缺氧组相比下降(P.01)，缺氧＋甲苯磺丁脲组中基因Bax的mRNA表达水平相对于单纯缺氧组也有提高(P.05)。
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Through researching the mechanism of biological nitrification and denitrification and actual project application of the technology and related literature materials, the obtained results are as follows:(1) Anoxybiosis is effective to the removal of COD; after the anoxic process, BOD/COD of the outflow increases, it shows that the degradation ability of wastewater is improved; anoxybiosis is insensitive when shock loading of inflow wastewater varies greatly;(2) It is demonstrated by the operation of inoculation and cultivating that biofilm takes two weeks to succeed in cultivating on packings and the removal ratios of NH3-N and COD get 50% and 70% respectively, when gas-water ratio is 6:1, HRT is 7.3 hour, water temperature is above 15°C;(3) It is demonstrated that gas-water ratio, hydraulic loading, HRT, temperature, pH etc are important factors which influence the removal effect of pollutants, when the gas-water ratio is 6:1, hydraulic loading is about 0.38kgNH3-N/m~3.d, water temperature ranges from 15°C to 27°C, pH ranges from 7.5 to 8.0, the BIOFOR removal effect of pollutants is the best;(4) The operation indicated that, this technology has strong ability of nitrification and denitrification; it has extremely vital significance for reducing the water body eutrophication;(5) It is confirmed the feasibility and the usability of preanoxic-BIOFOR process to treat L-lactic acid production wastewater. The experiment indicated that the treatment effect of this technology is very good and also shows that the technology has many advantages, such as small volume, high treatment efficiency, good effluent quality and strong endurance to load variation, this technology is a new and economical wastewater treatment process.
通过对生物脱氮机理的研究和该组合技术的实际工程应用，并结合有关文献资料，取得如下结论：(1)预缺氧池对L-乳酸生产废水中的COD去除具有一定的效果；废水中难降解的高分子物质经过缺氧工段分解为易于氧化降解的物质，浮状有机物在水解菌的作用下，将不溶性有机物水解为溶解性物质；预缺氧池具有较强的抗冲击负荷能力；(2)BIOFOR接种挂膜过程表明：HRT为7.3h、气水比为6：1、水温保持在15℃以上，经过半个月生物膜即以形成，NH_3-N和COD的去除率分别达到50％和70％左右；(3)研究表明：气水比、水力负荷、HRT、温度、pH等是影响污染物去除效率的重要因素，在气水比为6：1，NH_3-N容积负荷为0.38kgNH_3-N／m~3·d，温度在15℃-27℃，pH在7.5-8.0之间的条件下，BIOFOR对污染物的去除效率达到最佳；(4)运行表明，该组合技术具有较强的脱氮能力，对于减少水体富营养化具有十分重要的意义；(5)经过半年多实际工程的现场调试与运行，验证了利用预缺氧—BIOFOR组合工艺处理L—乳酸生产废水的可行性和实用性，得出该组合技术具有占地面积小，处理效果好，运行费用省，耐冲击负荷能力强等优点，是一项新型、经济的污水处理技术。
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Methodological design, synthesis is aimed at anoxic and revulsive factor - the nucleotide of few of particularity short cable length of 1 α is acerbity, compose builds anoxic and revulsive factor - of ShR-NA of 1 α particularity recombine plasmid, stability turns catch; of cell of colonic cancer SW 480 to use chloridize cobalt preparation is anoxic and revulsive education radical, imitate tumour is anoxic condition.
方法设计、合成针对缺氧诱导因子-1α的特异性短链寡核苷酸，构建缺氧诱导因子-1α特异性shR-NA的重组质粒，稳定转染结肠癌SW 480细胞；采用氯化钴制备缺氧诱导培养基，模拟肿瘤缺氧状态。
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Methods 20 rats were randomly divided into A, B, C, D and E group. The models of cerebral anoxia in group A and C were made by hypopiesia and hypoxia, and the models of hypoxia-reoxgenation were made in group B and D. 24h and 1h before establishment of models, ginsenoside Rb3 was injected peritoneally in rats of group C and D, respectively. The number and form of GABA-like immunoreactive neurons in hippocampus CA1 area of rats after brain hypoxia-reoxgenation were investigated with immunohistochemistry.
將大鼠隨机分为A、B、C、D、E5组，A、C组以低压低氧暴露製作大鼠脑缺氧模型，B、D组为缺氧－复氧模型。C、D组动物分别於制模前24h、1h 2次腹腔注射人参皂甙Rh3溶液；採用免疫组化法，观察各组大鼠在缺氧及缺氧－复氧处理后海马CA1区GABA免疫反应阳性细胞形態及数量的变化，並与正常对照组比较。
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Hypoxia-ischemia for 10 minutes group , 20 minutes group: our experiment showed that at 24 hours after cerebral hypoxia-ischemia, expression of MMP-9mRNA was maximally increased, individually, in the hypoxia-ischemia for 10 minutes group, MMP-9mRNA was greatly expressed within superolateral surface cortex of cerebrum, whereas in the hypoxia-ischemia for 20 minutes group, the labeling of hybridization for MMP-9mRNA was strongly appeared within the cortexof cerebrum both the cortex in the subsequence of medial surface at the periphery of the cerebral longigudinal fissure and the cortex in superolateral surface where also there showed a few stained fibers.
3缺氧缺血10分钟、20分钟组：缺氧缺血后24小时脑内MMP9 mRNA阳性表达至最高峰，缺氧缺血10分钟组大量表达在端脑背外侧面皮质内，缺氧缺血20分钟组在端脑内侧面后份大脑纵裂旁周围皮质及背外侧面皮质内呈强阳性表达，少数纤维呈亦阳性染色。
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METHODS: While hypoxia-reoxygenation groups were not given any medicine intervention as control, the treatment of xionggui diwan 1×10-6mol/L was subdivided into xionggui diwan 1 × 10-6 mol/L + non-hypoxia and xionggui diwan 1 × 10-6 mol/L + hypoxia groups, and the treatment of xionggui diwan 10 × 10-6 mol/L was subdivided into xionggui diwan 10 × 10-6 mol/L + non-hypoxia and xionggui diwan 10 × 10-6 mol/L + hypoxia groups.
缺氧-复氧前后组分为两个亚组(即缺氧-复氧前、缺氧-复氧后)，均不进行药物干预；芎归滴丸1×10-6 mol/L组分为两个亚组(即芎归滴丸1.0×10-6mol/L+不缺氧组、芎归滴丸1×10-6mol/L+缺氧组)；芎归滴丸10×10-6mol/L组分为两个亚组(即芎归滴丸10×10-6 mol/L+不缺氧组、芎归滴丸10×10-6mol/L+缺氧组)。
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Adult Wistar rats, supplied by the Animal Center of that university,were divided by random lotting into control group, acute hypoxia group, and chronic hypoxia group.Hypobaric chamber was used to simulate hypoxia for 2and 30 days at an altitude of
成年Wistar大鼠，校动物中心提供，按随机抽签法分为对照组、急性缺氧组和慢性缺氧组，低压舱模拟海拔5 000 m连续缺氧2，30 d，复制动物急性和慢性缺氧模型。
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objective to investigate whether hypoxia preconditioning exist in adipose tissue,and construct the hypoxemic model of adipose tissue.methods after injected different density cocl2 to rat's subcutaneously of groin,use immunohistochemical method detect hif-1α protein,and use rt-pcr detect hif-1α mrna.results after injected different density cocl2,the adipose tissue of rat's groin express different degree of hif-1αprotein and hif-1αmrna.,and reach the peak when the density is 20-40 mg/kg and the time is 3 h.conclusion hypoxia preconditioning exist in adipose tissue,and the effect of preconditioning is most evidence when the density is 20-40 mg/kg and the time is 3 h.
目的 探讨脂肪组织中是否存在缺氧预适应现象，构建cocl2致缺氧的缺氧模型。方法不同浓度的cocl2大鼠腹股沟区皮下层浸润注射，分别用免疫组织化学法和rt-pcr法检测该区脂肪hif-1α蛋白和hif-1α mrna的表达情况。结果用不同浓度的cocl2行化学缺氧后在不同时间点观察有不同程度的hif-1α蛋白和hif-1α mrna表达，其中，在浓度为20～40 mg/kg，时间为3 h时脂肪组织中hif-1α表达达高峰。结论脂肪组织存在缺氧预适应现象；cocl2的浓度选择20～40 mg/kg，时间选择3 h时缺氧预适应效果较佳。
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Results The changes of ［Ca2+］, the contents of CaM, cAMP were significantly higher in hypoxic and soman intoxicated group than in soman intoxicated group and control group under hypoxia; but the activity of Ca2+/CaM-PK Ⅱ were significantly decreased.
结果 缺氧中毒组PC12细胞cAMP、CaM含量在中毒后24 h明显高于单纯中毒组、缺氧对照组和正常对照组；缺氧中毒组PC12细胞Ca2+/CaM-PK Ⅱ活性在中毒后24 h明显低于单纯中毒组、缺氧对照组和正常对照组。
- 更多网络解释与缺氧相关的网络解释 [注：此内容来源于网络，仅供参考]
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anemic hypoxia：贫血性缺氧症
hypoxic hypoxia 缺氧性缺氧症 | anemic hypoxia 贫血性缺氧症 | individual 个体
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anoxia,anemic：贫血性缺氧
\\"缺氧症\\",\\"anoxia\\" | \\"贫血性缺氧\\",\\"anoxia,anemic\\" | \\"乏氧性缺氧\\",\\"anoxia,anoxic\\"
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ischemic anoxia：缺血性缺氧
由于动脉狭窄或阻塞,致动脉血灌流不足而引起的缺氧,又称缺血性缺氧(ischemic anoxia);由于静脉血回流受阻,血流缓慢,微循环淤血,导致动脉血灌流减少而引起的缺氧,称淤血性缺氧(stagnant anoxia).
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hypoxic-ischemic brain damage：缺氧缺血性脑损伤
缺氧缺血性脑损伤:hypoxic-ischemic brain injury | 缺氧缺血性脑损伤:hypoxic ischemic brain damage | 缺氧缺血性脑损害:hypoxic-ischemic brain damage
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hypoxic-ischemic brain damage：缺氧缺血性脑损害
缺氧缺血性脑损伤:hypoxic ischemic brain damage | 缺氧缺血性脑损害:hypoxic-ischemic brain damage | 缺氧缺血性脑损伤:hypoxic-ischemic brain damage
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hypoxic hypoxia：缺氧性缺氧
(1)换气问题:肺泡内氧气不足造成的缺氧现象称为缺氧性缺氧(hypoxic hypoxia)其可能造成的原因有:A:贫血性缺氧(hypemic hypoxia)贫血、出血或是血红素异常可使血液携氧能力降低,造成贫血性缺氧.
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hypoxic hypoxia：缺氧性缺氧症
progressive 逐渐的 | hypoxic hypoxia 缺氧性缺氧症 | anemic hypoxia 贫血性缺氧症
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anoxemic：缺氧血症的, 血缺氧的
anoxemia /血缺氧/缺氧血症/ | anoxemic /缺氧血症的/血缺氧的/ | anoxia /缺氧症/
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congestive hypoxia：淤血性缺氧
循环性缺氧还可以分为缺血性缺氧( ischemic hypoxia )和淤血性缺氧( congestive hypoxia ). 缺血性缺氧是由于动脉供血不足所致;淤血性缺氧是由于静脉回流受阻所致. 组织性缺氧( histogenous hypoxia )是指由于组织、细胞利用氧障碍所引起的缺氧.
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hypokinetic hypoxia：低动力性缺氧
74.循环性缺氧(circulatory hypoxia)指组织血流量减少使组织氧供应减少所引起的缺氧,又称低动力性缺氧(hypokinetic hypoxia). 75.缺血性缺氧是指 ...74.循环性缺氧(circulatory hypoxia)指组织血流量减少使组织氧供应减少所引起的缺氧,