- 更多网络例句与纤维胶质相关的网络例句 [注：此内容来源于网络，仅供参考]
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Rat in Group B was set an air bladder which was empty and not perfused water. Water of 0.6 ml, 0.8 ml, 1.0 ml, 1.2 ml, 1.4 ml and 1.5 ml was perfused respectively into the air bladders which were set in the colon of rats in group C, D, E, F, G and H. Then the changes of ethology of the rats were recorded in interval of an hour.
应用疼痛行为学的观察方法，观察1h各组动物的行为学改变，并采用免疫荧光化学的方法，观察大鼠DCN中神经元被激活的标志c-fos及胶质细胞的胶质原纤维酸性蛋白(glial fibrillary acidic protein， GFAP)和小胶质细胞特异性补体OX42的表达情况，空白对照组是在疼痛高峰点(1.2 ml)处死取材。
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The GFAP is a specific marker of astrocyte, its expression is more higher in the activity astrocyte, and finally the GFAP become the main composition of scar formations. The S -100 is a kind of acidity, dissolubilites, low molecular quantity and calcium hydronium conjugated protein, and it is mainly existed in neuroglial cell and schwann cell. It can promote the growth of axon, glial hyper-plasia and nerve divide and calcium's stability inside of cell, thus regulatin g the shape and metabolism of astrocyte . The quantity of S -100 protein and the degree of ischemia have direct proportion .
胶质纤维酸性蛋白是星形胶质细胞的特异性标记物，在活性星形胶质细胞中GFAP的表达相对更高，且最后GFAP成为胶质疤痕的主要成份。S-100蛋白是一种酸性、可溶性、低分子量的钙离子结合蛋白，主要存在于胶质细胞和雪旺细胞中，它可促进轴突生长、胶质增生、神经分化和细胞内钙的稳定，从而调节星形胶质细胞的形态和代谢。S-100蛋白与缺血的程度是成正比的。
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Glia; gliogenesis; glioma; glial cells; central nervous system; CNS; glial progenitor cells; glial fibrillary acidic protein; ependymocytes; ependoma; cytokeratins; vimentin; tanycytes; ion transport; lining cells; cerebrospinal fluid; CSF; ventricular system; floor plate; axon guidance; choroidal cells; choroid plexus epithelium; Chiari malformations; congenital aqueductal stenosis; neuroblast migration disorders; gap junctions
神经胶质；gliogenesis；神经胶质瘤；胶质细胞；中枢神经系统；CNS；神经胶质祖细胞；胶质原纤维酸性蛋白；室管膜细胞；ependoma；细胞角蛋白；波形蛋白；tanycytes；离子转运；衬细胞；脑脊髓液；CSF；脑室系统；底板；；脉络膜细胞；脉络丛上皮；恰里畸形；先天性导水管狭窄；成神经细胞迁徙扰乱；缺隙连接点
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INTERVENTIONS: 4 vessel occlusion(4VO) brain ischemic models in rats stained with thionine staining and GFAP immunohistochemistry staining. were used. Sixty-four rats, of which bilateral vertebral arteries were occluded permanently by electrocautery, were divided into the following 8groups: sham operation group, cerebral ischemic preconditioninggroup, ischemic insult group; BIT group; MTPG + sham operation group;MTPG+BIT group; MTPG+ischemia group and -4C3HPG+BIT coup. All the rats were killed 7 days after the operation or the final ischemic treatment. Cerebral sections were selected and stained with thionine staining and GFAP immunohistochemistry staining.
干预：采用大鼠四血管闭塞全脑缺血模型，应用硫堇染色和胶质纤维酸性蛋白免疫组化法。64只大鼠椎动脉凝闭后分为假手术组、单纯预处理组、单纯缺血组、脑缺血耐受组，MTPG+假手术组、MTPG+脑缺血耐受组、MTPG+缺血组和-4C3HPG+脑缺血耐受组，所有动物均在手术后或末次缺血后7 d处死取材，行脑组织切片，硫堇染色和胶质纤维酸性蛋白免疫组化染色。
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Recombinant apoE〓 protein could inhibit the death of the neurocyte cell of mice brain induced by beta amyloidal peptide 25-35 fragment and the death of the SK-N-SH-neuroblastoma cell induced by beta amyloidal peptide 25-35 fragment and 1-40 fragment.
重组apoE〓蛋白对Aβ 25—35多肽诱导的胎鼠大脑皮质神经细胞凋亡及Aβ25-35和A β1-40多肽诱导的神经成纤维胶质瘤细胞生存力下降均具有明显的拮抗作用，表明apoE对神经退化性疾病中的细胞死亡具有拮抗作用。
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The pcDNA〓-apoE〓 plasmid was transfected to SK-N-SH Neuroblastoma cell by liposome. It could be screened by G〓. The immunohistochemical assay was shown that neuroblastoma cell transfected by pcDNA〓-apoE〓 expressed the recombinant apoE protein. The intracellular expressed recombinant protein could inhibit the death of neuroblastoma cell induced by beta amyloidal peptides 25-35 fragment.
用pcDNA〓—apoE〓真核表达载体与脂质体共转染神经成纤维胶质瘤细胞，用G〓选择压力筛选，细胞免疫荧光检测表明，pcDNA〓—apoE〓转染的神经成纤维胶质瘤细胞表达了apoE〓重组蛋白，这种内源性的apoE〓重组蛋白对25μM Aβ25—35多肽诱导的神经成纤维胶质瘤细胞的死亡具有拮抗作用。
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The antibodies to cathepsin D and glial fibrillary acid protein and immunohistochemical PAP technique were used to observe the changes in microglia and astrocytes in the cerebella from NPC and normal mice of different ages.
应用组织蛋白酶D和胶质原纤维酸性蛋白抗体以及免疫组织化学方法，观察生后不同年龄的NPC和正常小鼠小脑内小胶质细胞和星形胶质细胞的变化。
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Methods HE and immunohistochemistry were used to observe the changes of glial fibrillary acidic protein and CD34 in the surrounding areas after cerebral infarction in human.
方法采用HE染色观察星形胶质细胞和微血管的变化，免疫组化技术检测胶质纤维酸性蛋白和CD34在星形胶质细胞和微血管的表达。
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This pattern is characterized by the combination of pro-MMP-2 and membrane type 1 (MT1)-MMP expression, which drie pericellular generation of actie MMP-2 and local degradation of normal lier matrix. In addition there is a marked increase in expression of TIMP-1 leading to a more global inhibition of degradation of fibrillar lier collagens by interstitial collagenases (MMP-1/MMP-13). These pathways play a significant role in the progression of lier fibrosis.
其降解肝正常基质，同时抑制引起肝纤维化的纤维胶原降解，这种情况的特点是前明胶酶A合成和膜型1－MMP表达，使得小叶内细胞激活MMP-2和降解当处的正常肝基质，此外血浆1型组织基质金属蛋白酶抑制剂（TIMP-1）明显升高引起广泛的抑制间隙胶原酶(MMP-1/MMP-13)对纤维肝脏胶原的降解。
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After the cell growth curves was recorded, RPE cells of the 3-5th passages were utilized. 2、Three different siRNA (siRNAl,siRNA2,siRNA3) targeting against human cx43 gene and one negative control siRNA were designed and transfected into cultured human RPE cells via liposome reagent. The most effective siRNA can be determined by semi-quantitative reverse transcription PCRRT-PCR. 3、To the most effective siRNA, after transfected into human RPEs with different concentration, the cellular proliferate activities were messured by MTT colorimetry ; the percentages of RPE in different cell circle phase was assayed by FCM; the changes of phenotypical properities were observed with SCM; the protein expression of cx43 was studied through immunocytochemistry stain and Weston blot; the communication intercellular was calculated with FRAP; and the ability of recovery was assessed by using an in vitro wound healing model.4、The total proteins of siRNA1 and RPE were seperated by two-dimensional gel electrophoresis and visualized by silver staining. Proteins with significant expression alterations were selected and their peptide mass fingerprints (PMFs were obtained by matrix-assisted laser desorption/ionization time of flying mass spectrometry (MALDI-TOF-MS).The PMFs were used to search NCBInr database by Auto MS-Fit software.
实验方法：1、培养原代的人RPE细胞，经过细胞角蛋白、S-100和神经胶质原纤维酸性蛋白免疫细胞化学鉴定后，通过AO/PI染色技术确定培养细胞的存活率，描记其生长曲线，第3-5代用于以下细胞实验2、生物合成针对人cx43基因的三条小干扰RNA和一条阴性RNA通过脂质体转染RPE细胞后，通过RT-PCR的方法确定抑制效率最高的干扰片断3、将该片段以不同浓度通过阳离子脂质体转染培养的人RPE细胞后，采用MTT法观察其对细胞的增殖力的作用；通过流式细胞仪观察其对细胞周期的影响；通过扫描电镜观察其对细胞形态的影响；通过免疫细胞化学和Weston blot观察其对cx43蛋白表达的作用；采用激光共聚焦和荧光淬灭恢复技术观察荧光恢复速率平均百分率，评价其对细胞间通讯功能的影响；通过制作RPE细胞损伤模型，观察其对损伤修复能力的作用4、分离纯化转染siRNA的RPE组和正常对照组RPE细胞的全部蛋白质，应用等电聚焦电泳和SDS-PAGE双向电泳技术，银染显示分离出的蛋白质斑点，经凝胶图像分析软件对两个样本进行胶图分析，寻找差异蛋白点。
- 更多网络解释与纤维胶质相关的网络解释 [注：此内容来源于网络，仅供参考]
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astrocyte：星形胶质细胞
1.星形胶质细胞 星形胶质细胞(astrocyte)是胶质细胞中体积最大的一种,与少突胶质细胞合称为大胶质细胞(macroglia). 细胞呈星形,核圆形或卵圆形,较大,染色较浅. 星形胶质细胞可分两种:①纤维性星形胶质细胞(fibrous astrocyte),
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fibrous astrocyte：纤维性星形胶质细胞
根据胶质丝的含量以及胞突的形状可将星形胶质细胞分为两种:纤维性星形胶质细胞(fibrous astrocyte)多分布在脑脊髓的皮质,突起细长,分支较少,胞质中含大量胶质丝,又称蜘蛛细胞(spider cell);原浆性星形胶质细胞(protoplasmic astrocyte),
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fibrous astrocyte：纤维性星型胶质细胞
astrocyte星型胶质细胞 | fibrous astrocyte纤维性星型胶质细胞 | glial filament胶质丝
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collagen fibril：生胶质原纤维
collagen 生胶质 | collagen fibril 生胶质原纤维 | collagen-fibers 生胶质纤维
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glia fiber：(神经)胶质(细胞)纤维
gliadin 麦醇溶蛋白 | glia fiber (神经)胶质(细胞)纤维 | glial growth factor 胶质(细胞)生长因子
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astroblast：成星形胶质细胞
成胶质细胞首先分化为各类胶质细胞的前体细胞,即成星形胶质细胞(astroblast)和成少突胶质细胞(oligodendroblast). 然后,成星形胶质细胞分化为原浆性和纤维性星形胶质细胞,成少突胶质细胞分化为少突胶质细胞. 最近有人在体外培养的研究中发现,
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fibroglia：纤维胶质
fibrocartilage 纤维软骨 | fibroglia 纤维胶质 | fibroid 纤维状的
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glial filament：胶质丝
有此突起末端扩大形成脚板(end feet)在脑和脊髓表面形成胶质界膜(glial liroltans)(图7-11,图7-15),或贴附在毛细血管壁上,构成血-脑屏障.胞质内含有胶质丝(glial filament),胶质丝的生化性质是一种胶质原纤维酸性蛋白(glial fibrillary acidic protein GFAP),
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inoglia：纤维胶质
inogenesis 纤维组织形成 | inoglia 纤维胶质 | inohymenitis 纤维膜炎
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gelatinous fiber;mucilaginous fiber：胶质纤维
胶化 gelatinize | 胶质纤维 gelatinous fiber;mucilaginous fiber | 胶质地衣 gelatinous lichen