纤维组织原的

基本解释 (translations)

inogenous

与纤维组织原的相关的网络例句 [注：此内容来源于网络，仅供参考]

ResultsMacroscopic examination showed no excrescence,thrombus formation,arm fractures and corrosion.The devices were covered with collagen fibrosis and discrete endocardial cells,apparent inflammatory infiltration in the devices and around the devices 1 month after implantation.The implants were nearly endothelialized,while the inflammatory reaction relieved gradually,with myocardial cells ingrowth at the edges of the device 3 months after implantation.The devices were completely covered with endocardium and fibrous tissue.Moreover,endothelial cells could be found on the smooth microscrew adaptor.The inflammatory reaction diminished with a few chronic inflammatory cells existing.Neovascularization and lymphatic vessels ingrowth could be observed 6 months after implantation.

结果所有封堵装置表面均没有发现赘生物、血栓形成、支架发生断裂及被腐蚀；术后1个月，封堵装置表面被胶原纤维和散在内皮细胞所覆盖，大量炎症细胞浸润，封堵装置边缘有小灶性炎症细胞浸润；术后3个月，封堵装置表面几乎被内皮细胞所覆盖，炎症细胞较1个月时明显减少，封堵装置内见纤维化，封堵装置边缘心肌细胞浸入；术后6个月，封堵装置表面完全被心内膜和纤维组织所覆盖，伞尖表面光滑并有内皮细胞上爬，炎症反应明显消散，但仍有少量慢性炎症细胞存在，装置内有新生的血管、淋巴管长入。

The notable proliferation was not observed by eyes in the local of injection. The infiltration of inflammation cells and mild proliferation of fibrocyte around dura mater was observed by HE stained in 4 and 8 weeks after injection. Infiltration and exudation of inflammation cells was observed by HE stained in epidural nerve root. Compared with group A, no changes of group B, C and D were observed under specific stained. Proliferation of type Ⅱ collagen fibers around dura mater was seen under immunohistochemical stained in 4 and 8 weeks after injection. There is no significant demyelination changes under LFB stained. The thickness and shape of the myelin sheath in epidural nerve root was not regular under transmission electronic microscopy in 4 and 8 weeks after injection. Fibroblast was also seen there. In nerve endometrium, macrophage could be seen under TEM, myelinated nerve fiber changed significantly, but nonmyelinated nerve fiber changed mildly. When 8 weeks, the changes of group D is smaller than the group B and C.

给药局部肉眼观察未见明显的纤维组织增生；HE染色可见B、C、D三组给药后四周及八周时硬膜内外均有炎细胞浸润，纤维细胞轻度增生，硬膜外神经根内有炎细胞浸润及炎性渗出；特殊染色B、C、D三组同A组相比未见有脊髓及神经根的改变；免疫组化染色，给药后四周及八周时，硬膜内外均有Ⅱ型胶原纤维增生；固兰染色B、C、D三组未见有明显脱髓鞘改变，与A组相比无明显异常改变；电镜观察B、C、D三组在给药后的四周及八周时，表现为硬膜外神经根内髓鞘厚薄不一，形状不规则，可见成纤维细胞，神经内膜中可见有巨噬细胞；粗大的有髓神经纤维变化明显，无髓神经纤维受累较轻；八周时电镜下D组改变较B、C两组为轻。

In this article, we used automotive immunohistochemical stainer to check the expressions of HBsAg and HCV antigens in 100 cases of HCC and their adjacent tissues, mean while, we checked their histological activity index and hepatic fibrosis degree. We also used radioimmunoassay to detect serum levels of 4 serum fibrosis markers and serum AFP levels in these cases. Immunofluorescence labeling and flow cytometry were used to check the T-cell subpopulations and the activity of NK cells of these 100 cases. Then the correlations among these factors were studied.

本文采用全自动免疫组化仪对100例肝癌组织及癌旁组织中的HBsAg、HCV抗原表达进行了标记，同时对其进行肝组织活动指数、纤维化分期；同时采用放射免疫测定方法检测同期血清透明质酸、Ⅲ型前胶原蛋白、层粘连蛋白和Ⅳ型胶原蛋白等四项血清肝纤维化标志物和血清甲胎蛋白水平；采用免疫荧光标记技术和流式细胞仪对T细胞亚群及NK细胞活性测定，对各项指标的相关性进行系列对比研究。

Explore JIAWEISANRENTANG Decoction on liver fibrosis in rat tissue plasminogen activator inhibitor,tissue-type plasminogen activator impact.

探讨加味三仁汤对肝纤维化大鼠组织纤维酶原激活剂抑制剂、组织型纤维酶原激活剂的影响。

This compound staining method has multicolor and fine contrast, and can better show melanins, the red blood cells at hemorrhagic focus, and collagenic and elastic fibers.

能够较好的显示皮肤组织中含有黑素细胞颗粒、胶原纤维及出血部位的红细胞和弹力纤维组织成分。

This pattern is characterized by the combination of pro-MMP-2 and membrane type 1 (MT1)-MMP expression, which drie pericellular generation of actie MMP-2 and local degradation of normal lier matrix. In addition there is a marked increase in expression of TIMP-1 leading to a more global inhibition of degradation of fibrillar lier collagens by interstitial collagenases (MMP-1/MMP-13). These pathways play a significant role in the progression of lier fibrosis.

其降解肝正常基质，同时抑制引起肝纤维化的纤维胶原降解，这种情况的特点是前明胶酶A合成和膜型1－MMP表达，使得小叶内细胞激活MMP-2和降解当处的正常肝基质，此外血浆1型组织基质金属蛋白酶抑制剂（TIMP-1）明显升高引起广泛的抑制间隙胶原酶(MMP-1/MMP-13)对纤维肝脏胶原的降解。

Special staining methods, such as Masson and the Van Gieson staining were used to study the distribution of collogen fibers and elastic fibers. ResultsBy HE staining, the subepithelial connective tissues and vessels in the pterygium were more prominent than normal conjunctival tissues. An amorphous subepithelial superficial hyalinized zone and coarse eosinophilic granular materials were observed in the pterygia, but they were not found in normal conjunctival specimens. Coarse fibers were visible only in the deeper subepithelial connective tissues of pterygial samples. With Masson′s staining, the dense staining of collagen fibers was also more prominent in the pterygium than in the subepithelial connective tissues of normal conjunctiva. Abnormal collagen fibers were visible in the deeper sub-epithelial connective tissues of pterygial samples. With Van Gieson staining, abnormal collagen fibers were visible in the deeper subepithelial connective tissues. Dark coarse elastic fibers were found in the abnormal fibers only in the subepithelial deep connective tissues of pinguecula in the pterygia but not in the conjunctiva. With immunohistochemistry staining, MMP-3 was strong in the pterygial epithelium, moderate in fibroblast and absent from pterygial vascular walls. LN was strongly expressed in the blood vessel wall, moderately in the epithelial basement membrane and absent from the entire stroma.

结果HE染色：翼状胬肉组织上皮下基质中存在结缔组织的增生和血管形成；基质浅层存在一无定形物质透明区及粗糙的颗粒样嗜酸性物质，在翼状胬肉体部深层基质中存在粗糙的纤维组织；正常球结膜组织细胞排列整齐；基质为疏松结缔组织，胶原纤维平行排列，其间可见成纤维细胞，散在少量中性粒细胞、毛细血管；Masson染色：翼状胬肉浅层基质中存在致密的胶原纤维染色，深层基质中的胶原纤维存在变性样改变；VG染色：翼状胬肉组织深层基质中存在大量变性的胶原纤维，其间夹杂黑色的弹性纤维；免疫组化染色法：MMP-3在翼状胬肉上皮细胞中呈强表达，成纤维细胞中呈中等强度表达，血管内皮细胞中未见表达；LN在血管壁中呈强表达，在上皮细胞基底膜中呈中等强度表达，在整个基质中未见明显表达；col Ⅲ在整个翼状胬肉基质中呈强表达。

Results The BMC surrounding tumor mass and strands showed multi-layered or dissolution, pseudopodia were seen extended from tumor cells, and contained numerous microvesicles.

瘤细胞伸出伪足样突起，且胞浆内含大量微泡，该处基膜消失，胶原原纤维溶解，肿瘤细胞围绕血管部位的血管基膜增厚，纤维组织增生。

Objective: To explore the mechanisms resulting in the recurrence of urethral scar which make urethral strictures difficult to be cured, a series experiments were conducted to find potential effective factors involved in urethral scar formation and degradation, including the studies of extracellular matrix component of urethral stricture scar, the characteristics of urethral scar fibroblast, and the effects of urine on urethral fibroblast in vitro, as well as the studies to compare the difference of collagenase activity, type Ⅰ collagen and tissue inhibitor of metalloproteinase-1 (TIMP-1) in the tissues and cultured fibroblasts from normal urethra and strictured urethra respectively, and the studies to investigate the effect of antisense TIMP-1 oligodeoxyonucleotide on cell proliferation and collagenase activity of urethral scar fibroblast.

中文题名尿道瘢痕基础研究副题名胶原酶活性，TIMP-1的表达及其反义基因治疗外文题名 Experimental study on urethral scar-activity of collagenase，expression of TIMP-1，and antisense TIMP-1 gene transfection of urethral scar fibroblast 论文作者黄翔导师杨宇如教授学科专业外科学研究领域\研究方向学位级别博士学位授予单位四川大学学位授予日期2002 论文页码总数104页关键词尿道手术瘢痕胶原酶成纤维细胞尿道瘢痕馆藏号BSLW /2003 /R699 /12 目的：研究尿道瘢痕的细胞外基质的组成，尿道瘢痕成纤维细胞的生物学特性以及尿液对其生长的影响；比较胶原酶活性，金属蛋白酶组织抑制因子-1（TIMP-1）以及Ⅰ型胶原含量在尿道瘢痕和正常尿道组织及体外培养的成纤维细胞中的差异；研究反义TIMP-1寡核苷酸对尿道瘢痕成纤维细胞增殖以及胶原酶活性的影响。

A 46-year-old man presented with a gradually growing nodule on the left dorsal foot, which had been present for over 10 years. In the beginning, the lesion was treated as a keloid. Because of poor response to treatment, biopsy was arranged. Sections showed a dermal tumor composed of numerous foamy cells, hyalinized wiry collagen, spindle cells and entrapped collagen. The foamy cells were positive for KP1(CD68) and negative for S-100 protein and HMB-45. The histological and immunohistochemical results were consistent with the diagnosis of lipidized fibrous histiocytoma.

一位46岁男性从十年前开始於左足背上出现一个逐渐长大之结节，一开始被当作蟹足肿给予治疗，因为对治疗的反应不好，故安排了切片检查，病理显示一个由大量泡沫状细胞，坚硬而透明之胶原纤维，梭状细胞，胶原纤维束所构成的真皮肿瘤，免疫组织化学染色显示KP1(CD68)阳性，S-100 protein与HMB45阴性，综合病理与免疫组织化学染色结果，诊断为脂肪性纤维组织细胞瘤。

与纤维组织原的相关的网络解释 [注：此内容来源于网络，仅供参考]

borderline：边缘

局部侵袭能力很强,Dahlin称其为"边缘"(borderline)型肿瘤. 骨韧带样纤维瘤来源于纤维组织. 大体标本呈白色或灰白色,坚韧有弹性如硬橡皮样,瘤内一般无钙化或骨化组织,但偶有囊性软化区域. 镜下特点为丰富的胶原纤维束,

collagen fibril：胶原原纤维

而由胶原装配成胶原原纤维(collagen fibril)则是通过分子间的交联. 弹性蛋白是弹性纤维(elastic fibers)的主要成分. 弹性纤维主要存在于韧带和脉管壁. 弹性纤维与胶原纤维共同存在, 赋予组织以弹性和抗张能力. 层粘连蛋白是基膜(basal lamina)的主要结构成分.

myofibril：肌原纤维

肌肉细胞(muscle cell),又称为肌纤维(muscle fiber),含有肌原纤维(myofibril),是肌肉收缩的要素. 脊椎动物的肌肉组织可分为三种:(1)平滑肌(smooth muscle)又称不随意肌,分布於内脏器官,血管的管壁等;(2)横纹肌(striated muscle)又称随意肌,

scar tissue：瘢痕组织

瘢痕组织(scar tissue)的形成是肉芽组织逐渐纤维化的过程. 此时网状纤维及胶原纤维越来越多,网状纤维胶原化,胶原纤维变粗,与此同时纤维母细胞越来越少,少量剩下者转变为纤维细胞;间质中液体逐渐被吸收,中性粒细胞、巨噬细胞、淋巴细胞和浆细胞先后消失;

soybean protein：大豆蛋白 刺激细胞新生,产生胶原纤维及弹力纤维,增强肌肤支撑组织的弹性与紧实

Sorbitol 山梨醇 保湿剂 | Soybean Protein 大豆蛋白 刺激细胞新生,产生胶原纤维及弹力纤维,增强肌肤支撑组织的弹性与紧实 | Stearic Acid 硬脂酸 油脂剂

collagenic fiber：胶原纤维

即管平衡听泡形成后其四周的间质组织 (mesenchymal tissue) 即变成听囊迷路,其与膜性迷路之间形成外淋巴间隙(perilymphatic space) 并蜗部分的外淋巴间隙分成二个部分,即鼓阶(scala tympani)及前庭成,其中间的胶原纤维(collagenic fiber)则为第一第二鳃弓的

collagenous：成胶的, 产生成胶的

collagenous tissue | 胶原组织, 白纤维组织 | collagenous | 成胶的, 产生成胶的 | collagraph | 抽象派美术画

collagenous tissue：胶原组织, 白纤维组织

collagenous fibril | 胶原纤丝 | collagenous tissue | 胶原组织, 白纤维组织 | collagenous | 成胶的, 产生成胶的

fibrinous inflammation：纤维素性炎

2.纤维素性炎(fibrinous inflammation) 是以渗出物中含有大量纤维素为特征的渗出性炎症. 纤维素的大量渗出,提示毛细血管和小静脉损伤较重,通透性明显升高,大量纤维蛋白原渗出到血管外,在坏死组织释出的组织因子作用下,转化为纤维素,故有纤维素性炎之称.

picric acid：苦味酸

在进行人体正常和肿瘤子宫组织的形态学研究中,选用丽春红S(poncean S)、苦味酸(picric acid)和地衣红(orccin)进行组合染色,简称PPA-O法. 经反复实验后,能够同时分别显示组织中弹性纤维、胶原纤维和平滑肌的分布及变化情况,得到了较好的染色效果,