突触的

基本解释 (translations)

synaptic

与突触的相关的网络例句 [注：此内容来源于网络，仅供参考]

Objective To count the number of ribbon synapses in cochlear inner hair cell using three-dimensional reconstruction model.

目的利用三维建模的方法对耳蜗内毛细胞带状突触进行计数分析，解决由于内毛细胞带状突触数量少、位置深在造成的计数困难，为内毛细胞带状突触的可塑性研究提供有效可靠的计数方法。

The relative timing between the presynaptic and postsynaptic spiking determined the direction and the extent of synaptic changes.

在前突触与后突触的相对尖突激发时间，决定了神经突触改变的程度与方向。

Sperry showed that the regenerated retinal axons always innervated the original sites of termination in the optic tectum. This experiment strongly disprove the resonance hypotheses proposed by Paul Weiss, who suggested that the nerve growth and synapse formation are largely random events, and the precision of connections between nerve cells emerged by selectively eliminating the inappropriate connections, only at later developmental stage.

Sperry的实验在支持Cajal假说的基础上，进一步明确发育过程中轴突的生长及突触的形成具有内在决定性，并且有力地反驳了当时占主流地位的由Paul Weiss于1930～40年代提出的resonance hypothesis，后者认为轴突的生长和突触的形成大多是一些随机事件，成年脑中的精确联系是通过对随机形成的不恰当联系进行选择地消除形成的。

Using the intracellular recording technique for monitoring the excitatory postsynaptic potentials of CA1 pyramidal neuron and delivering an activator of protein kinase C and/or inhibitors of PKC and 〓/calmodulin-dependent protein kinase Ⅱ into postsynaptic cell in hippocampal slices, we studied the role of postsynaptic PKC and CaMKⅡ in long-term potentiation induced by high frequency stimulation at the CA1 synapses made by Schaffer collateral/commissural and perforant pathway afferents.

用电生理学方法在大鼠海马脑片CA1区锥体细胞记录Schaffer侧枝和穿通通路到CA1神经元突触的兴奋性突触后电位，通过送蛋白激酶抑制剂和激活剂进入突触后细胞，我们研究了突触后蛋白激酶C和依赖钙/钙调蛋白的Ⅱ型蛋白激酶在高频刺激诱导的长时程增强产生和维持中的作用。

Based on above results, we conclude that the synaptic depression of thalamic input to the cortex contributes to rapid adaptation in cortical neuron, and both the thalamocortical and intracortical synaptic transmission could then enhance the degree of adaptation.

基于以上结果，我们认为，持续刺激引起的丘脑—皮层突触的抑制对皮层神经元的快速适应有贡献，而丘脑—皮层突触以及皮层—皮层突触的放大机制都可以将这一特性进一步增强。

To clarify the effect of lidocaine oncholinergic synapse, we reconstructed a cultured soma–somachemical synapse model consisting of two identified visceraldorsal 4 (VD4) and left pedal e-1 (LPeD1) neurons from the snail, Lymnaea stagnalis , in vitro , and used it to determine how lidocaineaffects cholinergic synaptic transmission.

为了阐明利多卡因对胆碱能突触的影响，我们从椎实螺属 stagnalis 的蜗牛离体重建了含两种已被鉴定的内脏背4（ VD4）和左足 e-1（ LPeD1）神经元的培养体细胞-体细胞化学突触模型，来研究利多卡因如何影响胆碱能突触传递。

Two kinds changes of neuron plasticity are confirmed being related to GAP-43:(1)synaptic structural change makes GAP-43 increase ;(2) changes of synaptic transduction efficacy that long term potential and long term depression can alter GAP-43 phosphatize state and mRNA expression.

突触素(synaptophysin，P38)系一相对分子质量为38000的膜结构蛋白，存在于突触前囊泡膜，与突触的结构和功能密切相关。P38作为突触前终末特异性标记物，常用来反映突触的密度和分布。

Ltp and ltd at excitatory neuronal synapses are observed to be induced by precise timing of pre- and postsynaptic events.

兴奋神经元突触的ltp和ltd是由突触前和突触后活动的精确定时所诱导。

Thus, correlated pre-and postsynaptic activity alters not only the strength of the activated input, but also its dendritic integration with other inputs.

因此，突触前后神经元的相关活动不仅影响被激化突触的传递效率，还影响树突的信号整合特性。

Many lines of physiological and pharmacological evidence show that AMPA receptor desensitization plays an important role in fast excitatory synaptic transmission by affecting transmission efficacy of individual synaptic bouton, integrative functions of neuron, and synapse plasticity.

大量的生理学和药理学证据表明，AMPA受体失敏在快速兴奋性突触传递过程中起着重要的作用，对单个突触的传递效率、经元的整合功能和突触的可塑性均有影响。

与突触的相关的网络解释 [注：此内容来源于网络，仅供参考]

synaptic cleft：突触裂隙

synaptic 突触的 | synaptic cleft 突触裂隙 | synaptic delay 突触延迟

synaptic cleft：神经链裂,突触空隙

\\"突触的,神经链的\\",\\"synaptic\\" | \\"神经链裂,突触空隙\\",\\"synaptic cleft\\" | \\"神经链泡,突触小囊\\",\\"synaptic vesicle\\"

inhibitory synapse：抑制性突触

可分为 (excitatory synapse)和抑制性突触 (inhibitory synapse). 突触的信息传递使突触后膜去极化,产生兴奋性的突触后电位的称为兴奋性突触. 突触的信息传递使突触后膜超极化,产生抑制性的突触后电位的称为抑制性突触.

synaptic plasticity：突触可塑性

之前的研究发现,神经突触可塑性(synaptic plasticity)与学习和记忆能力有关. 而且,N-甲基-天冬氨酸盐(NMDA)受体对于神经突触可塑性的启动是必需的. 然而,科学家发现,在最初的启动之后,NMDA会开始减弱神经突触的作用,这意味着,

synapsis：联会,胞突缠络,突触

\\"突触,神经链\\",\\"synapse,synapsis\\" | \\"联会,胞突缠络,突触\\",\\"synapsis\\" | \\"突触的,神经链的\\",\\"synaptic\\"

synaptic vesicle：突触囊泡

当神经冲动到来时,贮存于突触囊泡(synaptic vesicle)内的神经递质便进行排放,通过突触间隙扩散到突触后膜,引起突触后成分去极化或超极化. 因此,有兴奋性突触和抑制性突触之称. 神经冲动通过化学突触时有明显的突触延搁(synaptic delay)(约0.5-2 ms).

reciprocal synapses：交互性突触

还存在树突-树突式、树突-胞体式、树突-轴突式、胞体-树突式、胞体-胞体式、胞体-轴突式突触,以及两个化学性突触或化学性突触与电突触组合而成的串联性突触(serial synapses)、交互性突触(reciprocal synapses)和混合性突触(mi

serial synapses：串联性突触

此外,由于中枢存在由大量局部神经元构成的局部神经元回路(见后文),还存在树突-树突式、树突-胞体式、树突-轴突式、胞体-树突式、胞体-胞体式、胞体-轴突式突触,以及两个化学性突触或化学性突触与电突触组合而成的串联性突触(serial synapses)、交互性突触(re

mixed synapses：混合性突触

还存在树突-树突式、树突-胞体式、树突-轴突式、胞体-树突式、胞体-胞体式、胞体-轴突式突触,以及两个化学性突触或化学性突触与电突触组合而成的串联性突触(serial synapses)、交互性突触(reciprocal synapses)和混合性突触(mixed synapses)等(图10-3B)

synaptic：突触的

synapsis 联会 | synaptic 突触的 | synaptic cleft 突触裂隙