- 更多网络例句与神经胶质过多相关的网络例句 [注：此内容来源于网络，仅供参考]
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Iron in the form of ferritin is highly toxic and may cause neural death and reactive gliosis. Astrocytes and microglia are responsible for uptake of excessive CSF iron in superficial siderosis.
星形胶质细胞和小胶质细胞可摄取脑脊液中过多的铁，而铁蛋白中铁的毒性又可导致神经元坏死和反应性神经胶质增生。
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A clear cause-and-effect relationship between excessive myocardial MMP-1 activity, increased mysial collagen degradation, and progression to systolic HF has been shown experimentally through the use of transgenic models and the use of pharmacological MMP activators.53,54 In addition, experimental evidence has been provided showing that following chronic neurohormonal activation, increased synthesis and release of MMPs into the local extracellular matrix of the cardiomyocyte occurs,55 which in turn could contribute to endomysial and perimysial collagen degradation and disruption.
一个明显的因果过多的心肌MMP-1关系活动， mysial增加胶原蛋白的降解和进展收缩性心力衰竭已被证明试验，通过使用转基因模型和使用的MMP药理作用此外，activators.53，54实验证据下面被提供显示慢性神经激活时，增加的MMPs合成和释放到当地的细胞外基质的心肌细胞发生，55反过来有助于人肌内膜和 perimysial胶原退化和破坏。
- 更多网络解释与神经胶质过多相关的网络解释 [注：此内容来源于网络，仅供参考]
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gliosis：神经胶质过多症
一些神经元在此过程中最终被疤痕组织所取代,称为神经胶质过多症(gliosis). 整个在海马回的变化,极似在患颞叶癫痫(temporal lobe epilepsy)成人大脑中所发现到的. 在这项研究中,Xrcc1的减少,也导致小鼠癫痫发作.