- 更多网络例句与癫痫发作后的相关的网络例句 [注：此内容来源于网络，仅供参考]
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The incidence of early epileptic seizures after intracerebral hemorrhage was high. The cortical hemorrhage and the medical history of intracerebral hemorrhage, electrolyte disbalance were the important related factors for the incidence of early epileptic seizures after intracerebral hemorrhage. The cortical hemorrhage was the independent risk factor.
脑出血后早发性癫痫发作的发生率较高，皮质出血、脑出血史以及电解质紊乱是影响脑出血后早发性癫痫发作的重要因素，皮质出血为独立的危险因素。
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Univariate analysis revealed that a high incidence of early epileptic seizures after intracerebral hemorrhage ocurred in patients with cortical hemorrhage, the medical history of intracerebral hemorrhage, electrolyte disbalance. Logistic regression analysis revealed that the position of hemorrhage and times of hemorrhage and electrolytic states were positively correlated with the incidence of early epileptic seizures after intracerebral hemorrhage. The cortical hemorrhage was the independent risk factor for the early epileptic seizures after intracerebral hemorrhage.
单因素分析显示，皮质出血、再发脑出血、电解质紊乱者易发生脑出血后早发性癫痫发作；Logistic回归分析显示，出血部位、出血次数和电解质情况与脑出血后早发性癫痫发作的发生有关，其中皮质出血为与脑出血后早发性癫痫发作有关的独立危险因素。
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Already have experiment evidence indicate, the spontaneity that brain inner nerve pathological mechanism changes worsens being able to not be controlled , hippocampus nerve basic degeneration appears to escape from loss , galotinous cicatrices takes form and moss shape fibre sprouts waiting for plasticity changing being connected with after this may flare up with acute falling sickness very much.
已有实验证据表明，这很可能与急性癫痫发作后，脑内神经病理变化的自发性恶化不能被控制，出现海马神经元变性脱失，胶质瘢痕形成以及苔状纤维发芽等可塑性改变有关。
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Result: 1、Ethology result:50 rats were injecting convulsive dose kainic acid, showing prodrome such as gaze, wet dog shake in the 0-30 minutes; 1-5 degree epileptic attack in the 30-60minutes; 4-5 degree repteately spontaneous epileptic attack in 60-90 minutes; then status epilepticus,SE-epileptic attack endurance became longer and interval became shorter with bulk salivas and various kinds psychomotor symptoms.
结果：1、行为学结果：SD大鼠(n=50)在注射惊厥剂量(10mg/kg)KA后，0-30min内动物出现凝视，湿狗样抖动(wet dog shake，WDS)等前驱症状；30-60min内动物出现1-5级癫痫发作；60-90min内动物反复自发出现4-5级癫痫发作；其后动物癫痫发作持续时间延长，间隔变短伴有大量唾液分泌，出现各种精神运动症状，呈现SE，SE后10天，用阈下剂量(5mg/kg)KA检测动物对癫痫刺激的敏感性。
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This effect of ACET can be enhanced after treating again within 7 to 14 days later.
在癫痫发作后的中期(7d～14d)内予二次埋线治疗对疗效有续效叠加的作用。
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ACET inhibit the descending of Bax,prolong its descending period andthen promote its expression,in order to weaken the inhibition of the apoptosis during 4 to 10 days after seizure and then promote the apoptosis during 10 to 14 days after.
埋线治疗通过抑制Bax蛋白表达的下降、延长其降至低峰值的时程，致使在癫痫发作后的中期(4d～10d)抑制大鼠海马神经元凋亡的作用减弱；继而通过促进Bax蛋白表达的回升，致使在癫痫发作后的远期(10d～14d)出现促进凋亡的作用。
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Observed the dynamic changes of the common condition of the rats,it suggested that ACET can help the kindling model rats to get well,the effect was significant after 7 days.To treat again after 7 days can also enhance the effect.
结果 1。动态观察大鼠一般状态的变化，提示埋线治疗有助癫痫发作后的一般状态恢复，以一周后的远期效果较为明显，一周后再行埋线治疗则疗效有所叠加。
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The dynamic changes of the the neuron apoptosis and correlative genes in hippocampus of ACET-groups and twice-ACET-groups suggested that ACET can slower the apoptosis by inhibiting p~(53)and prolong the ascending period of p~(53) in 4 days after the seizure,this effect would weaken after 4 days.ACET may inhibit the descending of Bax,prolong its descending period and then promote its expression,in order to weaken the inhibition of the apoptosis during 4 to 10 days after seizure and then promote the apoptosis during 10 to 14 days after.This effect of ACET can be enhanced after treating again within 7 to 14 days later.
埋线组和二次埋线组大鼠海马神经元凋亡及相关基因表达的动态变化结果提示：埋线治疗在癫痫发作后的早期(4d内)可通过抑制促凋亡基因p~(53)蛋白表达、延缓其达峰值的时程，从而减缓神经元凋亡的发生，此作用在4d后减弱；埋线治疗抑制大鼠海马神经元凋亡的作用在癫痫发作后的中期(4d～10d)减弱，甚至在远期(10d～14d)出现促进凋亡的作用，可能是通过抑制Bax蛋白表达的下降、延长其降至低峰值的时程，促进Bax蛋白表达的回升的结果；在中远期(7d～14d)内予二次埋线治疗对该效应有续效叠加的作用。
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Dr. Spitz listed several factors that can contribute to a delay in diagnosis. He observed that some physicians expect epilepsy patients to present with grand mal seizures, that comorbid conditions such as cardiac disease and cancer can mask seizure activity, and that patients can be extremely confused for a day or two after mild seizures."The recovery period from an epileptic seizure is longer and more dramatic in someone who is 70 as opposed to someone who is seven," he said.
Spitz医师开出几项会造成诊断延误的因子，他观察到一些医师认为癫痫病患会有重癫痫的发作，comorbid的症状如心脏疾病和癌症会掩盖发作活动，病患在轻微发作后一两天会极度困惑，他表示，70岁病患癫痫症发病到复原的期间比七岁病患更长和更惊人，Spitz进一步表示，一旦被诊断出，老年病患的发病通常是轻微的，并且能用药物治疗。
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Neural cell adhesion molecule is a membrane glucoprotein .
研究表明NCAM作为粘附分子中的一员，其在癫痫发作后有明显变化。
- 更多网络解释与癫痫发作后的相关的网络解释 [注：此内容来源于网络，仅供参考]
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posterior association area：后脑联想区
postepileptic 癫痫发作后的 | posterior association area 后脑联想区 | posterior column 后柱
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postictal automatism：癫痫发作后自动症
posthemorrhagic 出血后的 | postictal automatism 癫痫发作后自动症 | postictal paralysis 癫痫发作后瘫痪
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epileptic seizure：癫痫发作
而癫痫发作(epileptic seizure)是指脑神经元异常和过度超同步化放电所造成的临床现象. 癫痫病是完全可以治愈的一种疾病,关键是要找到一种科学有效的治疗方法,常规的方法是无法从根本上治好癫痫病的,北京癫痫病治疗中心万寿康临床基地通过数万例的患者的治疗总结后终于找到了癫痫病的一个
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postepileptic：癫痫发作后的
postdormitum 觉醒前时期 | postepileptic 癫痫发作后的 | posterior association area 后脑联想区