炎症

基本解释 (translations)

inflammation  ·  phlegmasia

与炎症相关的网络例句 [注：此内容来源于网络，仅供参考]

objective to raise the level of regarding congnition,prevention,diagnosis and therapy on multiple organ dysfunction syndrome.methods report the process and correlative therapy on one case who occured with mods involving 6 organs after hemorrhagic shock lead to heartbeat and respiration stopping suddenly and resuscitated resulting in left nephrostome titanium nip falled off accidentally from left kidney lymphatic fastening.at the same time,the author reviewed correlative literature and introduce the investigative progress about mods.results one case who happened with mods following hemorrhagic shock,heartbeat and respiration stopped suddenly was diagnozed and resuscitaed.6 organ dysfuncted including brain,heart,lung,liver,kidney and the digestive system.after being given therapy integrated chinese and western medicines,allopathy and nutritional treatment,all circadian parameters of the case came back in gear and discharged at last.conclusion the mechanism about mods is very complicated.because of serious infection,hurt and oxygen-lack,excessive inflammatery reaction activates multifarious cell factors and inflammatery mediums,which improve the happen-rate of mods.wiping off pathogeny,providing life-sustain treatment and recognising sirs and mods in time and adopting corresponding therapy,such as treatment integrated chinese and western medicines,can improve the hit-rate of cure on mods.

作者：王静恩，蔡金芳，王志华，谢晓洪多器官功能障碍综合征；中西医结合；治疗；休克，出血性目的提高对多器官功能障碍综合征（multiple organ dysfunction syndrome，mods）的认识、预防、诊断、治疗水平。方法报告成功抢救1例经腹腔镜左肾淋巴管结扎手术后左肾静脉钛夹意外脱落失血性休克，导致心跳呼吸骤停复苏后再手术并发mods累及6个脏器功能衰竭过程及有关治疗，并复习相关文献，介绍目前关于mods的研究进展。结果确诊1例因失血性休克心跳呼吸骤停复苏后并发mods，累及脑、心、肺、肝、肾、血液、胃肠等6个脏器。经积极采取中西医结合对症、支持救治，各生理指标全部恢复正常，康复出院。结论 mods的发生机制非常复杂，在严重感染、创伤、缺氧等打击下，失控的过度炎症反应激活多种细胞因子和炎症介质的释放，使mods发生率升高。尽早去除病因，尽早给予各种生命支持治疗，尽早对可能发生全身炎症反应综合征、mods识别并给予干预治疗，包括中西医结合治疗能改善mods的救治成功率。

Objective:With environmental pollution and invasion of the inflammation induced by some materials, bronchia asthma and chronic obstructive pulmonary disease have become the important social public problems in recent years.

近年来，在支气管哮喘和慢性阻塞性肺疾病的治疗药物中，肾上腺皮质激素可对抗炎症或过敏介质，通过对慢性阻塞性肺疾病者的前炎症因子、炎症细胞、氧化应激的影响而发挥良好的疗效。

The medicine prepared with licoflavone has high curative effect on inflammatory enteropathy, wide safety range and other advantages.

用甘草黄酮制备的抗炎症性肠病的药物对炎症性肠病具有疗效好，安全范围大等优点，可以作为抗人炎症性肠病的药物。

Eosinophile, mast cell , basophils, lymphocyte and many kinds of cytokine are involved in this inflammation.

是以气道高反应性和慢性气道炎症为特征的变态反应性疾病，是由嗜酸性粒细胞、肥大细胞、T淋巴细胞等多种炎症细胞和细胞因子参与的气道慢性炎症。

ResultsMacroscopic examination showed no excrescence,thrombus formation,arm fractures and corrosion.The devices were covered with collagen fibrosis and discrete endocardial cells,apparent inflammatory infiltration in the devices and around the devices 1 month after implantation.The implants were nearly endothelialized,while the inflammatory reaction relieved gradually,with myocardial cells ingrowth at the edges of the device 3 months after implantation.The devices were completely covered with endocardium and fibrous tissue.Moreover,endothelial cells could be found on the smooth microscrew adaptor.The inflammatory reaction diminished with a few chronic inflammatory cells existing.Neovascularization and lymphatic vessels ingrowth could be observed 6 months after implantation.

结果所有封堵装置表面均没有发现赘生物、血栓形成、支架发生断裂及被腐蚀；术后1个月，封堵装置表面被胶原纤维和散在内皮细胞所覆盖，大量炎症细胞浸润，封堵装置边缘有小灶性炎症细胞浸润；术后3个月，封堵装置表面几乎被内皮细胞所覆盖，炎症细胞较1个月时明显减少，封堵装置内见纤维化，封堵装置边缘心肌细胞浸入；术后6个月，封堵装置表面完全被心内膜和纤维组织所覆盖，伞尖表面光滑并有内皮细胞上爬，炎症反应明显消散，但仍有少量慢性炎症细胞存在，装置内有新生的血管、淋巴管长入。

Large dosage of shang lixio by oral or inunction can inhibit twisting reaction by acetic acid,but hasn't specific analysict effect,can inhibit inflammation of animal by croton seed.

无论口服还是外用，在大剂量时可抑制醋酸所致的扭体反应，但无特异性镇痛作用；在灌胃及炎症部位外用时，均可明显抑制动物巴豆油耳廓炎症及角叉菜胶所致后跖炎症。

Lungs of conclusion children heart turns stream operation of orthoptic congenital heart disease can bring about the hurried inflammation He Kangyan such as TNF-α, IL-6, IL-8, IL-10, CRP inside the heart of disease medium release, turn in heart lungs the likelihood in shedding the systemic inflammation that cause to react produces main effect.

结论小儿心肺转流心内直视先天性心脏病手术可导致TNF-α、IL-6、IL-8、IL-10、CRP等促炎症和抗炎症介质的释放，在心肺转流诱发的全身炎症反应中可能发挥重要功能。

ABSTRACT］ Objective To study the pharmacological effects of oral medical ice and to provide for its evidence for clinical application. Methods The anti-inflammatory effect of OMI was determined on three inflammatory models: swelling of mice ear induced by dimethylbenzene, hind paw edema and back granuloma caused by hypodermic injection of agar in rats. The an- algesic action of OMI was studied by observing the mice's reaction to the heat simulation. The effect of OMI on bleeding time was tested by tail-cutting method.

摘要］ 目的探讨口腔医用冰的药理作用，临床用药提供依据方法制备二甲苯致小鼠耳部炎症、琼脂致鼠足肿胀急性炎症模型，大鼠琼脂肉芽肿慢性炎症模型，观察口冰对炎症的作用；采用热板法进行小鼠热刺激性疼痛试验，断尾法测定小鼠出血时间，观察口冰对小鼠疼痛及出血时间的影响。

Methods the anti-inflammatory effect of omi was determined on three inflammatory models: swelling of mice ear induced by dimethylbenzene, hind paw edema and back granuloma caused by hypodermic injection of agar in rats. the an- algesic action of omi was studied by observing the mice's reaction to the heat simulation. the effect of omi on bleeding time was tested by tail-cutting method.

制备二甲苯致小鼠耳部炎症、琼脂致大鼠足肿胀急性炎症模型，大鼠琼脂肉芽肿慢性炎症模型，观察口冰对炎症的作用；采用热板法进行小鼠热刺激性疼痛试验，断尾法测定小鼠出血时间，观察口冰对小鼠疼痛及出血时间的影响。

Inflammation per se may be responsible for mild airflow limitation.7 8 9 Indeed, it has been suggested that inflammation may lead to functional bronchiolar constriction by releasing mediators that may act directly on bronchiolar smooth muscle.10 The chronicity of inflammation would, in turn, produce other changes such as airways fibrosis, and may increase the smooth muscle mass either directly as a result of inflammation or indirectly as a result of chronically increased muscle tone.

炎症本身导致了气流限速轻微下降。而事实上它提示了由于释放的介质直接作用于细支气管平滑肌上，炎症引起了功能性细支气管的缢痕。慢性炎症反多来又会产生气道纤维化等其他改变，这样无论是炎症反应的直接影响结果，还是慢性肌紧张增高的直接影响结果，都会增加平滑肌质量。

与炎症相关的网络解释 [注：此内容来源于网络，仅供参考]

catarrhal inflammation：卡他性炎症

hemorrhagic inflammation 出血性炎症 | catarrhal inflammation 卡他性炎症 | inflammatory granuloma 炎性肉芽肿

inflammation：炎症

从现代医学角度讲,中医所说的"上火"和西医讲的"炎症"(inflammation)有着密不可分的关系. 汉语里,火上加火就是"炎",英语中"发炎"的动词形式inflame本意就有燃烧、火焰之意. 医学研究发现,上火或炎症发生的共同原因就是外来或内在的细菌、病毒以及毒素在体内积存,

chronic inflammation：慢性炎症

②继发第二节 急性炎症 炎症通常可依病程经过分为两大类:急性炎症(acute inflammation)和慢性炎症(chronic inflammation). 急性炎症起病急骤,持续时间短,仅几天到一个月,以渗出病变为其特征,炎症细胞浸润以粒细第八节 上皮性肿瘤 从上皮组织(包括覆盖上皮与腺上皮)发生的肿瘤最为常见,

acute inflammation：急性炎症

一般而言,炎症可分为二个基本临床类型:急性炎症(acute inflammation)和慢性炎症(chronic inflammation). 急性炎症往往以渗出性和变质性病变为主,而慢性炎症则以增生性改变为主.

inflammatory edema：炎症性水肿

化脓性炎症 inflammation,suppurative | 炎症性水肿 inflammatory edema | 吹张反射 inflation reflex

inflammatory mediator：炎症介质

在炎症过程中由细胞释放或体液中产生的参与、介导炎症反应得化学物质称为炎症介质(inflammatory mediator). 这些介质包括细胞释放的炎症介质、白细胞产物和体液产生的炎症介质. 这些介质通过各种途径作用于血管,使血管扩张、通透性增加,引起渗出,

fibrinous inflammation：纤维素性炎症

2.纤维素性炎症 纤维素性炎症(fibrinous inflammation)时以纤维蛋白原渗出并在炎症灶内形成纤维素为主. 光镜下,苏木素伊红染色可见大量红染的纤维素交织呈网状,间隙中有中性粒细胞及坏死细胞的碎屑. 大片纤维素在镜下表现为片状、红染、质地均匀的物质.

cardinal s's：(炎症)主征:炎症的體征,包括紅、腫、熱、痛及功能喪失,見

Cardarelli's sign Cardarelli征:喉及氣管橫向搏動,見於主動脈瘤及主動脈擴張 | cardinal s's (炎症)主征:炎症的體征,包括紅、腫、熱、痛及功能喪失,見inflammation | cogwheel sign 齒輪征:見phenomenon

inflammatory mediators：炎症介质

[炎症介质]炎症介质(inflammatory mediators)是指在致炎因子作用下,由局部组织或血浆产生 和释放的,参与或引起炎症反应的化学活性物质,故亦称化学介质.炎症介质有外源性(细菌及其产物) 和内源性两大类,但主要是后者.内源性炎症介质又可分为细胞源性和血浆源性两类,

inflammation mediums：炎症介质

炎症反应:anti-inflammation | 炎症介质:inflammation mediums | 低度炎症:lower-grade inflammation