- 更多网络例句与活化相关的网络例句 [注：此内容来源于网络，仅供参考]
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In this thesis, two electrochemical activation methods and two activation solution were used to obtain four types of the activated GC electrodes. Effects of electrochemical activation procedure on the structure, the size, the distribution and the type of electron transfer site on electrode surface have been investigated. Application of electrochemical activation GC electrode has been exploited in electroanalysis of metal ions. Meanwhile, the essence structure and character of electron transfer site of sp2 hybridized carbon material have been explored.
在本论文研究中，通过采用酸性和碱性两种活化溶液，分别以两种不同的电化学活化方法，获得了四种不同的活化电极表面；调查了电化学活化方法对电极表面的电化学活性点的种类、尺度大小、结构以及分布的影响规律；拓展了电化学活化玻碳电极在电分析领域中的应用；探索了sp2杂化类碳材料电极在电子传导点的性质和结构上存在的本质共同点。
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It is included that 4-5 types of active sites exist in the catalytic system and the active site produced polymer with low molecular weight is easy to be activiated while TEA is used as the cocatalyst. While TIBA, the active site produced polymer with high molecular weight and seldom active site produced polymer with low one is prone to be activiated. MAO tends to activiate the active site produced polymer with middle-level molecular weight. In TEA-TIBA catalytic system, TEA and TIBA activiate the corresponding active site and the weight of every active site gradationally varied with the change of the ratio of the two cocatalysts. And the activity of the active site produced polymer with high molecular weight is proportion to the concentration of TIBA.
发现：TEA作为助催化剂时产生低分子量产物的活性中心易于活化，且体系中具有活性的活性中心有4～5种；TIBA易于活化产生高分子量的活性中心，基本上不能活化产生低分子量产物的活性中心；MAO对产生中等分子量产物的活性中心活化能力较强；在TEA-TIBA混合物体系中，TEA和TIBA各自对活性中心的作用表现为明显的互补现象，各活性中心的相对强度随着两种助催化剂之间的比例改变呈现渐次变化，其中产生高分子量产物的活性中心的相对活性(来源：c5AB3fC论文网www.abclunwen.com)与TIBA的浓度有明显的正相关性。
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Results Analysis of the expression of proteins of JAK2 in leukemic blasts, there was no detectable constitutive expression of phosphorylated JAK2 in all the AML samples analyzed; Furthermore, we could detect a constitutive activation of STAT 3 by immunoprecipitation of the targeted protein in primary leukemic blasts from 3 of 11 AML patients without any cytokine treatment; Pretreatment clinical characteristics, and treatment regimens did not differ significantly between patients with and without constitutive STAT3 activity, there was not significant relation between the constitutive activation of STAT3 and the FAB subvariety and chromosome karyotype; However, the 3 patients with constitutive STAT3 activity seemed to have adverse treatment outcome, one did not show remission, the other two patients relapsed shortly after remission; Consistent with the above-mentioned result, leukemic blasts from the 3 samples with constitutive expression of STAT3 showed higher percentage of spontaneous apoptosis and S-phase cells.
在实验研究基础上，本研究进一步运用免疫沉淀和Western Blot印迹方法，检测AML患者原代细胞JAK2和STAT3活化情况，并分析了STAT3活化与AML患者预后有关指标改变的关系。结果我们的初步研究发现，11例AML患者中，有3例呈现STAT3组成型活化（STAT3阳性，下同），无一例呈JAK2组成型活化。上述3例STAT3组成型活化与FAB分型和染色体核型改变未见明显相关性；在此3例患者中，1例未取得缓解，另外2例虽然取得短暂缓解，但均在二个月内复发。上述3例STAT3阳性AML患者S期白血病细胞百分率数值较组高，而凋亡率数值较低。
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Get high purity DCs by Cultured plastic-adherent monocytes isolated from healthy human peripheral blood with GM-CSF and IL-4 for 7 days. To observe the morphology of DCs by inverted phase contrast microscope ,electron microscope and laser confocal microscope. Analyse phenotype of DCs with flow cytometry. Investigate the endocytosis ability of DCs as a group by Horseradish peroxidase endocytosis assay. To appraise allogeneic mixed lymphocytes reaction of DCs by MTT reduction assay. Analyse the levels of IL-12 and TNF in liquids of cultured medium by ELISA and MTT reduction assay respectively. Soluble antigens of HCCs was obtained by 3 freeze-and-thaw cycles. Biological characteristics of HC soluble antigens pulsed DCs were monitored by flow cytometry. According to MTT reduction assay estimated the cell proliferation of self lymphocytes activated by HC antigens pulsed DCs. Get high purity BCG HSP 70 protein by SDS-PAGE electrophoresis and determined its biological activity with ELISA. Analyse phenotype of antigen pulsed DCs primed by BCG HSP70 with flow cytometry. By MTT reduction assay estimated the cell proliferation of self lymphocytes and the MLR of DC based vaccine. Analyse expression of HLA-DR molecule on surface of HCC lines. The IFN-γ mRNA in lymphocytes after actived by DC vaccine and the Fas-L expression on DC and DC vaccine primed lymphocytes were detected by in situ hybridization and flow cytometry respectively. Specific cytotoxity lysis of T lymphocytes and nonspecific inhibition of liquids in culture medium against HCC lines were also tested. Detect expression of hAFP on four HCC lines with Cell-ELISA. Induce apoptosis of HCCs with actinomycin-D. Interaction of DCs and apoptotic cells was observed under transmission electron microscope. Growth inhibition test of DC against HCC lines was also performed. Establish the nude mouse model bearing human HC xenografts and indentify the characteristic of tumour by histochemistry and immunohistochemistry techniques. Prevent and treat transplanted human HC on nude mouse with Freezing and anabiotic HC specific lymphocytes.
用GM-CSF和IL-4从健康人外周血诱导DC；分别用倒置相差显微镜、电子显微镜及激光共聚焦显微镜观察DC形态；流式细胞术检测DC表型；HRP吞噬实验测定DC的群体内吞能力；MTT法检测同种异体混合淋巴细胞反应；ELISA法和MTT法分别测定DC培养上清液中IL-12和TNF水平；冻融法制备肝癌细胞可溶性抗原；流式细胞术检测负载肝癌可溶性抗原后DC的生物学特性；MTT法检测DC负载肝癌抗原后对自身淋巴细胞增殖的影响；SDS-PAGE制备电泳纯化BCG HSP70并鉴定纯度，ELISA测定活性；流式细胞术检测负载抗原DC经BCGHSP 70活化后的表型；MTT法检测肝癌DC疫苗对自身淋巴细胞增殖的影响和混合淋巴细胞反应；流式细胞术检测肝癌细胞表面HLA-DR表达；MTT法检测肝癌DC疫苗对自身淋巴细胞的活化；原位杂交法检测肝癌DC疫苗活化后的淋巴细胞IFN-γmRNA表达；流式细胞术检测DC和肝癌DC疫苗活化后淋巴细胞表面Fas-L；MTT法分别检测肝癌DC疫苗活化的淋巴细胞和其培养上清对肝癌细胞的特异性杀伤和非特异性抑制作用；Cell-ELISA检测人肝癌细胞hAFP表达；MTT法检测负载AFP表位肽和凋亡肝癌细胞DC对自身淋巴细胞增殖的影响；ELISA法和MTT法分别测定活化后淋巴细胞培养上清中TNF和IL-12水平；肝癌细胞凋亡的诱导和检测；DC吞噬凋亡肝癌细胞后的电子显微镜观察；DC对肝癌细胞的生长抑制试验；人肝癌裸鼠皮下移植瘤动物模型的建立及其组织学和免疫组织化学鉴定；DC及肝癌特异性淋巴细胞预防和治疗人肝癌裸鼠皮下移植瘤；冻存和复苏后的肝癌特异性淋巴细胞预防和治疗人肝癌裸鼠皮下移植瘤。
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The activated or leached process and their influence elements are studied by 1DREACT software package of coupled mass/heat transfer and chemical reaction dynamics of water-rock interaction, according to the geological and geochemical characters of fine disseminated gold deposits.(1) Water-rock reaction time is not the important influence element of activating gold;(2) At first, activating capability of hydrothermal solution increases with the increasing of total sulfur activity; after lga〓≥-4, the content of activated gold in solution is mainly related to original content of gold in wall rock, and does not visibly dependent on total sulfur activity.(3) It is a complicated nonlinear process of influence of activating gold capacity of hydrothermal solution with the change of temperature, in general, 220 ℃ is most favorable to gold activation.(4) The influence of oxygen fugacity on gold activation has a multiple functions coupling nonlinear effect, in general, Igfo〓=-41 is most favorable to gold activation.(5) Solubility of gold in fluid decreases with the increasing of velocity of flow, the product of velocity and aurous solubility reaches maximum when velocity is 0.2～0.5m〓m〓. yr〓, i. e. this scope of velocity is most favorable to gold activation.
利用1DREACT水-岩相互作用反应-输运耦合动力学软件包，根据微细浸染型金矿床地质地球化学特征，计算机模拟研究了金的活化、浸取过程及其影响因素，发现：(1)金活化过程中水-岩反应时间不是其主要制约因素；(2)热液对金的活化能力开始随总硫活度的增高而增高，当lg a〓≥-4后，热液中活化金的含量将主要与围岩中金的初始丰度有关，而对总硫活度无明显依赖关系；(3)温度对热液浸金能力的影响是一个复杂的非线性过程，总体而言，220℃最有利于金的活化；(4)氧逸度对金活化的影响呈现出一种多因素叠加的非线性效应，总体而言，lgfo〓＝-41最有利于金的活化与浸取；(5)流体中金的浓度随流体流速的加快而降低，流速与金浓度的乘积在流速为0.2～0.5m〓m〓。yr〓时达到极大，即0.2～0.5m〓m〓。yr〓的流速范围最有利于本类矿床的金的活化。
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The major results of this proposal are as follows:(1) ABA induces a rapid, substantial accumulation of apoplastic H2O2 in mesophyll and bundle sheath cells of maize leaves, and the accumulation of apoplastic H2O2 is involved in the induction of the chloroplastic and cytosolic antioxidant enzymes.(2) ABA-induced H2O2 production activates a 46 kDa MAPK, which in turn induces the expression of antioxidant genes and up-regulates the activities of antioxidant enzymes. The activation of MAPK also enhances H2O2 production, forming a positive amplification loop.(3) Water stress also induces the activation of a 46 kDa MAPK, which is dependent on the accumulation of ABA and H2O2 production induced by water stress and involved in the up-regulation of the expression and the activities of antioxidant enzymes.(4) ABA-induced H2O2 production mediates NO generation, which in turn activates a 46 kDa MAPK and results in the up-regulation in the expression and the activities of antioxidant enzymes in ABA signaling. NO-independent signaling is also involved in ABA- and H2O2-induced antioxidant defense.
本项目的主要研究结果如下：（1）ABA诱导的H2O2产生主要出现在叶肉细胞与维管束鞘细胞的质外体中，质外体H2O2的积累能够上调叶绿体与细胞溶质中抗氧化酶的活性；（2）ABA诱导的H2O2产生活化一个46kDa的MAPK，由此而导致编码抗氧化防护酶基因的表达与酶活性的上调；而MAPK的活化也能增强H2O2的产生，从而形成一个正的反馈调节环；（3）水分胁迫也能诱导一46kDa MAPK活化，这一MAPK活化依赖于水分胁迫诱导的ABA积累以及H2O2的产生，同时参与水分胁迫诱导的抗氧化防护基因的表达与抗氧化酶活性的上调；（4）ABA诱导一个依赖于H2O2的NO产生，NO活化一个46kDa的MAPK，从而导致抗氧化防护基因的表达以及抗氧化酶活性的上调；同时一个不依赖于NO的信号转导途径也存在于ABA诱导的抗氧化防护过程中。
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Results are as followed:1 Exposure of HELF cells to BP caused c-Jun activation,and increased the activity of MAPK,PI-3K,p53 and cyclin D1 pathway.2 BP-induced c-Jun activation was inhibited by dominant negative mutants of extracellular signal-regulated protein kinase or c-Jun NH_2-terminal kinase,but not by p38,impling that JNK and ERK pathways medicate c-Jun activation induced by BP.3 Overexpression of dominant-negative mutants PI-3K and Akt potently blocked phosphorylations of c-Jun and ERK,but not JNK in response to BP,suggesting that PI-3K/Akt pathway positively regulates BP-induced c-Jun activation through ERK.4 Inhibition of p53 by its chemical or molecular inhibitor markedly increased the phosphorylation levels of c-Jun,Akt and ERK upon BP stimulation,indicating that p53 negatively medicates BP-induced c-Jun activation through PI-3K/Akt/ERK pathway.5 The cell lines expressed TAM67 exhibits no significant affecting normal cell growth properties.6 TAM67 was able to significantly block G_1-S transition and subsequent cell proliferation,suggesting that c-Jun is essential for cell cycle alternations elicited by BP.7 Overexpression of TAM67 impaired BP-induced cyclin D1 activation,decreasing expression of E2F1 and pRb,indicating that c-Jun participates in the modulation of BP-induced activation of cyclin D1/pRb/E2F1 pathway.8 Stably expression of TAM67 led to the increases in the expression levels of p53 and p21,elevating phosphorylation level of p53,clearly indicating that c-Jun regulates p53/p21 pathway activation induced by BRCollectively,PI3K/Akt/ERK pathway mediated BP-induced c-Jun activation through p53-dependent mechanism.
结果显示：1BP刺激细胞可促进c-Jun活化，并伴随着MAPK、PI-3K、p53和cyclinD1通路各组成成分的活性增强。2利用MAPK通路的显性失活突变体分别阻断细胞外信号调节激酶和c-Jun氨基末端激酶活性，均可明显抑制BP诱导的c-Jun活化，但阻断p38活性对BP引起的c-Jun活化无明显影响，提示JNK和ERK通路参与调控BP诱导的c-Jun活化。3过表达PI-3K和Akt的显性失活突变体也可显著抑制BP诱导的c-Jun活化，并降低磷酸化ERK的表达水平，但对磷酸化JNK的表达水平无明显影响，说明PI-3K/Akt通路通过ERK正性调控了BP诱导的c-Jun活化。4p53的化学/分子抑制剂能使BP作用的细胞内c-Jun活性明显增加，并同时诱导Akt和ERK的磷酸化水平的升高，表明p53可通过PI-3K/Akt/ERK通路对BP诱导的c-Jun活化进行负性调控。5随后观察转染细胞的生长情况，发现TAM67对细胞正常生长和形态无明显影响。6稳定表达TAM67可有效抑制BP诱导的S期细胞数的增加，提示c-Jun在BP致细胞周期改变的过程中发挥了重要作用。7TAM67过表达能够抑制BP诱导的cyclin D1活化，降低磷酸化Rb以及E2F1蛋白表达水平，表明c-Jun参与调控BP诱导的cyclin D1/Rb/E2F1通路的活化。8过表达TAM67可使BP刺激的细胞中p53、p21总蛋白以及p53磷酸化的表达水平明显升高，可见c-Jun也参与调控BP诱导的p53/p21通路活化。
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In the case of granite, observation results indicate that the extending routes of active cracks are mainly intercrystalline, transcrystalline, inter-transcrystalline, and intracrystalline. When the strain rate is low, the long intercrystalline cracks are prevailing, and with increasing of the strain rate, the short transcrystalline and intracrystalline cracks gradually increase. The statistic results show that the active crack density increases with increasing of the strain rate, and the mean length of active cracks diminishes with increasing of the strain rate, that is, the induced damage in comminuted product increases with increasing of the strain rate. For the first time this dissertation integrates macroscopic results of comminution with active crack growth under impact loading. It elucidates why the dynamic comminution strength of mineral beds increases with increasing of the strain rate through the active crack extending, and analyzes the influence of structure and constitution of minerals on their damage under impact stress. It is authenticated that both the size distribution and the damage population of comminuted product can be characterized by the fractal.
为分析料层颗粒在冲击下的细观损伤效果，采用德国生产的ASM68K半自动图象分析仪对粉碎生成物中的损伤情形进行了观测，以花岗岩为例，观测结果表明花岗岩颗粒在冲击应力下的活化裂纹扩展路径主要表现为沿晶、穿晶、沿穿晶和晶内等形式，在低应变速率下，活化裂纹以沿晶裂纹较多，随着应变速率的提高，其它裂纹形式大量繁衍，统计结果表明活化裂纹密度随着应变速率的增大而增大，活化裂纹的平均长度随着应变速率的增大而减小，综合表现为颗粒的细观损伤程度随着应变速率的增大而增大；首次将应力作用下的活化裂纹演化特征与粉碎的宏观效果相联系，并就料层动态粉碎强度随应变速率增大而提高的现象从裂纹演化特征的角度进行了解释；分析了矿岩构造结构特性对损伤产生和分布的影响。
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The liquid radioactive contamination comes from the radioactive substances produced by the circulatory cooled water activated after neutron radiation;the solid radioactive contamination comes from the match plate in the hot working chamber,the collimation equipment...
液态放射性污染主要指加速器冷却循环水受中子照射被活化形成放射性物质；固体放射性污染为热室工作箱内的模板，退役加速器离子源、准直器和靶周围被活化的器件及与放射性制剂分装、注射相关的用品；放射性气态污染，主要指中子使空气活化产生核素11C、13N、15O、41Ar，空气中其它成份被中子活化产额低，多为气溶胶状态；另外放射性表面污染主要指加速器厅内被活化的固体器件，冷却水破裂或放射性药物意外污染，有关传递通道和贮源容器的表面放射性污染。
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This paper deals with and summarizes the research and importance of ductile shear zones and presents the future direction and aim for the research of the ductile shear zones around the world. Four aspects of studies of the ductile shear zones have been proposed in this paper as follows:(1) The stress environment including the mineral assemblages, deformation, stress-shearing parameters;(2) The major element sequence and activation under condition of stress;(3) The variations of trace elements and REE, their transportation distributions under strong natural deformation as well as variations of mineral crystal parameters, which can also control the element changes during the ductile deformation;(4) The relationship between element migrations, activation and stress, which will present the new evidences for the studies of dynamic diagenesis and mineralization as well as the studies of evolution of ductile shear zones developed in middle and low levels.(5) The analysis of elements and isotopes in mylonites on the micro-scale are crucial for the understanding of component migrations during the mylonization.
对韧性剪切带及其变形岩石的研究现状和研究意义进行系统的综述，提出了未来韧性剪切带及其糜棱岩的研究方向和目标：①系统研究糜棱岩中主要造岩矿物组合及其变形特征，计算剪切变形岩石的应力－应变参数，搞清韧性剪切带所处的应力应变环境；②系统研究韧性剪切带岩石在天然分强剪切应力作用条件下常量元素迁移机制及活化转移的应力排序问题；③系统研究剪切变形作用过程中岩石化学组成的微量和稀土元素变化，讨论强变形条件下岩石中微量元素活化和迁移规律，深入探讨微量元素迁移的动力控制，包括稀土元素配分变化的应力制约以及应变矿物晶格化学变化行为及其对其寄主的变形岩石元素在应变过程中迁移变化的制约和影响；④从理论上探讨天然强剪切应变条件下岩石中组分活化、转移与应力的因果联系，为深入探讨韧性剪切带动力成岩作用提供理论的科学依据，为探讨中、下地壳中韧性剪切带的形成和演化提供科学依据，同时为韧性剪切变形作用条件下成岩、成矿地球化学作用提供理论和实验依据；⑤现代分析技术如激光同位素原位分析以及激光ICP-MASS分析技术对研究变形域内的岩石的元素和同位素的活化迁移规律，对深刻揭示糜棱岩化过程中的元素活化迁移机制提供更高质量的地球化学证据具有重要的作用。
- 更多网络解释与活化相关的网络解释 [注：此内容来源于网络，仅供参考]
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activated complex：活化配合物;活化复体
activated clay 活性粘土 | activated complex 活化配合物;活化复体 | activated earth 活性土
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activated complex theory：活化络合物理论
活化控制 activation control | 活化络合物理论 activated complex theory | 活化能 activation energy
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activated sludge：活化淤泥
"activated sintering","活化烧结" | "activated sludge","活化淤泥" | "activated state","活化态"
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activation analysis：活化分析
活化态;致活作用 activation | 活化分析 activation analysis | 活化能 activation energy
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activated bauxite：活化铝矾土
"activated atom","活化原子" | "activated bauxite","活化铝矾土" | "activated carbon","活化碳;活性碳"
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catalyst deactivation：触媒去活化;催化剂去活化
catalyst circulation 触媒循环;催化剂循环 | catalyst deactivation 触媒去活化;催化剂去活化 | catalyst deterioration 触媒变质;催化剂变质
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activation overpotential：活化过电位
activation logging 活化测井 | activation overpotential 活化过电位 | activation polarization 活化极化
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activated petroleum coke：活化石油焦
"activated montmorillonite clay","活化微晶高岭土" | "activated petroleum coke","活化石油焦" | "activated process","活化法"
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RAS：网状活化系统
这种活化作用是通过位于人脑根部的被称为网状活化系统(RAS)的次皮下单元的刺激而产生的. 通过一些装置就可以测量出人的活化作用水平,并以此为依据量化其情感警觉的程度,这样就完成了对感觉的测量.
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activable：可活化的;能被活化的
action limit 处理界限 | activable 可活化的;能被活化的 | activate 触发;对...起作用;开动