外膜
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The adventitia might also play an important role in the formation of cerebral AVM formation. The destruction of collagen in the adventitia might be due to the high level of MMP-9 mRNA.
血管内膜的破坏与大脑AVM形成有关;外膜也参与了脑AVM的形成过程;外膜胶原纤维的破坏与外膜中MMP-9 mRNA高含量呈正相关。
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The long-term opening of mitochondria permeability transition pore results in the release of cytochrome C, apoptosis inducing factors, Ca2+ and other apoptosis factors into cytoplasm, which can destruct the whole cell structure and function by activating the main member of apoptosis protein family——caspase, or destroying the intranuclear chromoplasm independently, or affecting other protein relying on Ca2+, and finally lead to the formation of apoptotic body.
线粒体是真核生物能量和代谢的中心,也是细胞凋亡信号传导途径中起关键作用的细胞器,越来越多的研究表明,在凋亡信号的刺激下,线粒体发生膨胀,其外膜膜电位降低,外膜破裂后释放出膜间隙的诸多凋亡效应因子,在细胞凋亡中起着主开关的作用。
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The operation mainly used epineurium-epineurium coaptation, neuroma-epineurium coaptation and epineurium-tissue coaptation respectively. Therefore, the ruptured nerves were sutured without tension.
分别采用外膜-外膜法、神经瘤外膜-外膜法、外膜-组织床法等3种方法,使神经断端在无张力下缝合。
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The histological changes of the epimysium-reconstructed epineurium was similar to the intact epineurium.
结论肌外膜瓣重建神经外膜后其组织学改变与神经外膜极为相似,为临床应用提供了实验室依据。
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METHODS: Sixty Sprague-Dawley male rats were randomly divided into four groups(n = 15), four methods were designed on rats models of sciatic nerve compression. There were simple decompression as group A, internal neurolysis after decompression as group B, lemithason(0.5 mg/kg) injected in the epineurium after decompression as group C, and lemithason(0.5 mg/kg) injected around the epineurium after decompression and internal neurolysis as group D.
在大鼠坐骨神经卡压模型基础上,将60只SD成年雄性大鼠随机等分为四组。A组:仅去除卡压;B组:去除卡压后切开神经外膜;C组:去除卡压后神经外膜下周围注射利美达松(0.5mg/kg);D组:去除卡压后切开神经外膜,然后在神经周围注射利美达松(0.5mg/kg)。
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The proliferation of medial smooth muscle cells and collagenous fibers of pulmonary arteriole in patients with PH were observed by electron microscopy.
电镜下,中重度PH组肺细小动脉中膜平滑肌增生,外膜胶原纤维密集;轻度PH组肺细小动脉中膜平滑肌细胞和外膜胶原纤维增生轻于中重度PH组;无PH组肺细小动脉结构改变不明显。
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The operations used in this group include neurolysis,epineurial repair,epineurial-fascicular repair,fascicular repair and nerve grafting.Results:All cases were followed-up after 18 months to 60 months and the excellent rate was 80.4%.Conclusions:Those result indicated that the preceded microsurgical repair of peripheral nerve injury is an effective method for raising the excellent rate of peripheral nerve injuries,and for vary nerve,different section of nerve injured,very nature of nerve injury,the method are vary.
目的:分析应用显微外科技术在修复周围神经损伤中影响其疗效的因素及处理措施方法:从1987年至1997年,用显微外科技术修复周围神经损伤80例92条神经,方法包括神经松解术、神经外膜缝合术、外膜束组膜缝合术、束膜缝合术和神经移植术结果:经术后18个~60个随访,优良率80.4%结论:神经断端精确对合和及早修复可提高其疗效;同神经、同的损伤平面、损伤程度同,采用同的缝合方法
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He expression levels of TGF-β3 decreased in descending order of endotenon tenocytes (42.5%), tendon sheath fibroblasts (41.2%), and epitenon tenocytes (33.3%).
GF-β1 表达平均降低程度从大到小依次为腱鞘细胞(36.1%)、腱内膜细胞(31.2%)、腱外膜细胞(31.0%),TGF-β2依次为腱鞘细胞(37.9%)、腱外膜细胞(32.1%)、腱内膜细胞(27.0%),TGF-β3 依次为腱内膜细胞(42.5%)、腱鞘细胞(41.2%)及腱外膜细胞(33.3%)。
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Was simultaneously injected into cisterna magna at the second injection blood in the presence of subarachnoid blood. At 2 to 4 days, GFP was expressed in leptomeninges over the brain stem, cortex and cerebral arteries, smooth muscle cells of small vessels were occasionlly transduced. GFP was expressed in adventitia of spastic basilar artery on days 2 (day 5 after first injection blood) after injection 〓, but was undetectable by days 4, transgene was not expressed in medial or intimal layers. The prensence of subarachnoid blood can not prevent access of adenovirus to vessels and transgene expression.
注入腺病毒载体后2天(初次注血第5天)行荧光显微镜、免疫组织化学和RT-PCR检测,结果在颅内大血管如基底动脉的外膜可见外源基因的表达,而外源基因不能转移至血管中膜和内膜,4天时(初次注血第7天)基底动脉的外膜中外源基因表达消失;注入腺病毒载体后2~4天,外源基因可以有效转移至颅底软脑膜细胞,小血管外膜和平滑肌层也可见外源基因的表达,表明蛛网膜下腔内的血凝块不能阻止腺病毒载体介导外源基因转移至颅内血管。
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Before transecting inject 1ml Sodium Chloride in the epineurium. Annular tensile the proximal of epineurium with microscopic forceps annular shear the epineurium 7mm before broken ends with microscopic shear and stretch the epineurium until the two ends distance is 5mm.Use 8-0 suture line to bridge the nerve gap and make a 5 mm nerve tube at the proximal nerve end. Interrupted suture the epineurium tube and the distal nerve end make a 5 mm nerve mamuro for the injury nerve.Results: Postperative observation lasted for 6 weeks regeneration nerve through the 5 mm nerve mamuro.
在大鼠的坐骨神经距股方肌下缘15mm处,用1ml注射器向神经外膜下注入1ml生理盐水,然后于此处切断坐骨神经,用显微镊环形拉伸近端神经外膜,在距断端约7mm处用显微剪环形剪开神经外膜,并继续拉伸外膜使外膜两断端间距约5mm停止,用8-0的无损伤缝线桥接神经外膜两端,在神经近端形成约5mm的神经外膜管,将神经外膜管与神经远端间断缝合,使断伤的神经束间留有5mm的小间室。
- 更多网络解释与外膜相关的网络解释 [注:此内容来源于网络,仅供参考]
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adventitia:外膜,动脉外膜
Adventist | 耶稣复临论者 | adventitia | 外膜,动脉外膜 | adventitial cell | 外膜细胞, 周皮细胞(吞噬细胞)
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tunica adventitia:外膜
有类似成纤维细胞的功能.在动脉发育过程中,可分泌各种细胞外基质成分,如胶原纤维,弹性膜和基质.在病理状态下,动脉中膜的平滑肌可移入内膜增生并产生结缔组织,使内膜增厚,是动脉硬化发生的重要病理过程.3,外膜(tunica adventitia)由疏松结缔组织组成,
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tunica adventitia:外膜,动脉外膜
膜 tunica | 外膜,动脉外膜 tunica adventitia | 白膜 tunica albuginea
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ectosarc:原虫外膜
ectorhinal 鼻外的 | ectosarc 原虫外膜 | ectosarcous 原虫外膜
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epicardium:心外膜
3.心外膜 心外膜(epicardium)是心包膜的脏层,其结构为浆膜(serous membrane),它的表层是间皮,间皮下面是薄层结缔组织,与心肌膜相连. 心外膜中含血管和神经,并常有脂肪组织. 心包膜壁层衬贴于心包内面,也是浆膜,与心外膜连续.
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epicardium:心外膜 鳃囊管
epicardiectomy 心外膜切除术 | epicardiolysis 心外膜松解术 | epicardium 心外膜 鳃囊管
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epineurium:神经外膜
轴突最外层包绕神经内膜,若干轴突组成一个神经束,有束膜(Perineurium)包绕,若干神经束组成神经干,外包的结缔组织膜称为神经外膜(Epineurium). 神经内膜、束膜及外膜均有防神经受伤过度牵拉的作用,尤以后二者为重要.
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epicardial mapping:心外膜标测
epicardial injury 心外膜损伤 | epicardial mapping 心外膜标测 | epicardial pacing 心外膜起搏
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epicardial fat:心外膜脂肪垫
epicardial electrode 心外膜电极 | epicardial fat 心外膜脂肪垫 | epicardial injury 心外膜损伤
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perimetrium:子宫外膜
1.外膜 子宫外膜(perimetrium)于底部和体部为浆膜,其余部分纤维膜. 2.肌层 子宫肌层(myometrium)甚厚,由成束或成片的平滑肌组成,肌束间以结缔组织分隔. 肌层分层不明显,各层肌纤维互相交织,自内向外大致可分为粘膜下层、中间层和浆膜下层.