增殖

基本解释 (translations)

proliferate  ·  proliferation  ·  propagation  ·  proliferative  ·  proliferated  ·  proliferates

与增殖相关的网络例句 [注：此内容来源于网络，仅供参考]

The double membrane vesicle, which was produced by budding of the outer membrane of amyloplast, accumulated starch to form new amyloplast. The double membranes of amyloplast invaginated and the inner membrane dilated to form new amyloplast in the amyloplast.

淀粉质体增殖产生新淀粉质体有多种方式：出芽增殖、缢缩增殖、中间隔板增殖、被膜向内出泡或内陷增殖、被膜形成双层膜小泡再积累淀粉增殖，它们均是淀粉质体被膜的一种膜行为。

It turned out reproduction rete of chrysanthemum was raised,the regular,non-plant diseases and insect pests could be offered to the market. Shoot tip were used as explants to study the effects of different sterilization methods, illumination intensity and concentrations of 6-BA and NAA on shoot propagation. Rooting with perlite, vermiculite and river sand instead of agar was studied. In the process of preliminary multiplication, the effect of using 0.1% HgCl_2 in sterilization of explants was better than using saturated bleaching solution. The highest multiplication rate was found under whole illumination. MS media with 6-BA from 0.2 mg·L~(-1) to 5 mg·L~(-1) and NAA from 0.05 mg·L~(-1) to 2 mg·L~(-1) were tested and as a result, the highest differentiation rate and the multiplication rate was reached with 0.2 mg·L~(-1) 6-BA, 0.05 mg·L~(-1) NAA.

本实验以菊花的茎尖为外植体，研究了不同灭菌方法，光照强度，全光照、半光照和弱光处理以及不同6-BA和NAA浓度及配比对于试管苗增殖的影响，并研究了珍珠岩、蛭石和河砂等琼脂替代物对于菊花生根的影响，结果表明：在初代培养物建立的过程中采用0.1％升汞进行表面灭菌的效果好于饱和漂白粉的灭菌效果；在全光照条件下外植体的增殖倍数最高；在试管苗增殖培养的过程中，以MS为基本培养基，并在培养基中添加0.2～5mg·L~(-1)6-BA和0.05～2mg·L~(-1)NAA，其中0.2mg·L~(-1)6-BA和0.05mg·L~(-1)NAA最适于外植体的分化和增殖，其分化率为100％，增殖倍数为12。

The additional supplements including glucose,ascorbic acid and insulin had the enhancement effects on C.andersoni development,while folic acid had no significantly effect on C. andersoni development and calcium pantothenate had inhibited effect.When added 50 mmol/L glucose,50μg/ml ascorbic acid,0.3 U/ml insulin into 10%FCS RPMI-1640 medium,the proliferation of C.andersoni had a great significantly increase,and the gene copies of C.andersoni were 8-fold compared with that in 10%FCS RPMI-1640 medium.

冻融法+蛋白酶K法+声裂法提纯隐孢子虫卵囊DNA效果最佳，其次为冻融法+声裂法和蛋白酶K法+声裂法，冻融法+蛋白酶K法效果最差。10%FCS是培养安氏隐孢子虫的最佳FCS浓度，葡萄糖、维生素C和胰岛素都具有促安氏隐孢子虫增殖作用，泛酸钙无促虫体增殖效果，而叶酸则抑制虫体增殖。10%FCS RPMI-1640培养液中加入50 mmol/L葡萄糖，50μg/ml维生素C和0.3 U/ml胰岛素促安氏隐孢子虫增殖效果最显著，虫体增殖8倍。

Ointment extracted from Plastrum Testudinis was separated by silica gel column chromatography, gradiently eluted by petroleum ether-ethyl acetate, and 16 (Ts-1～Ts-16) components were obtained, which were biologically evaluated by MTT assay and flow cytometry on the proliferation of rat marrow-derived mesenchymal stem cells. Results showed that Ts-12 induced proliferation compared with the control group ( p ＜0.05), while Ts-4 showed inhibitive effect. Other groups showed no significance ( p ＞0.05). GC-MS was used to analyze the chemical components in different groups.

中药龟板提取物浸膏经硅胶柱层析，用石油醚-乙酸乙酯作洗脱剂，梯度洗脱，得到16个组分；采用MTT法和流式细胞技术研究了它们对鼠骨髓间充质干细胞的增殖作用，实验结果显示组分Ts-12具有促进rMSCs增殖的作用( p ＜0.05)，而组分Ts-4则表现出抑制rMSCs增殖的作用( p ＜0.05)，其它样品对rMSCs的增殖作用不显著( p ＞0.05)，不具统计学意义。

To investigate the effect of p38MAPK signal conducting pathway on livercancinoma cell's malignant phenotype induced by VEGF,we take theexperiment with cell growth test,scanning microscope and laser scanningconfocal microscope so that observe effect of p38MAPK signal conductingpathway on liver cancinoma cell growth,pseudopodium formation andframework of cytoskeleton induced by VEGF.results indicate that the cellbecame ellipse and there were more and thick pseudopodium in the cell'ssurface after being treated by VEGF,and destroyed framework ofcytoskeleton,which can be blocked by pretreated with a special inhibitor ofp38MAPK SB203580 so that VEGF promote metastasis by enhancing livercell migration and movement via p38MAPK signal conducting pathway,butVEGF promote cell growth without p38MAPK signal conducting pathway.

为进一步探讨VEGF通过p38信号传导通路诱导肝癌细胞转移时，对肝癌细胞恶性表型的影响，采用细胞增殖实验、扫描电子显微镜、激光扫描共聚焦显微镜观察VEGF对肝癌细胞增殖作用、细胞伪足形成、细胞骨架微丝结构的影响以及p38MAPK信号通路调控作用。结果显示，VEGF能够不通过p38信号通路促进肝癌细胞增殖；VEGF诱导肝癌细胞丝状伪足增多、增长，使细胞骨架微丝结构破坏、甚至消失。阻断p38MAPK信号通路，可以抑制VEGF诱导的肝癌细胞伪足形成，细胞骨架丝状结构呈束状，排列较规整。上述结果表明，VEGF可以通过p38信号传导通路诱导肝癌细胞伪足增多、增长，促使细胞骨架微丝结构破坏，使肝癌细胞迁移、运动能力增加，促进肿瘤转移。VEGF并不通过p38信号通路诱导肝癌细胞增殖作用。

He CD4〓 T-cell viability determined by the trypan blue test was over 90%. PBMCs from the recipients, after inactivated, were added in 96-well culture plates with donor CD4〓 T-cell cocultured at 37℃ for 24 hours, TJU103 and CTLA4-Ig were given for additional 48 hours coculture. The CD4〓 T-cell was collected and washed as anergic CD4〓 T-cell.

JU103联合CTLA4-Ig应用对供者T细胞增殖活性的影响根据TJU103有剂量限制性特点，选择TJU103最大抑制效应浓度50μg/ml，与不同浓度CTLA4-Ig（1μg/ml、10μg/ml、50μg/ml、100μg/ml）联合，对供者T细胞的增殖抑制率分别为（59.5％、90.5％、93.4％、95.8％），TJU103（50μg/ml）与低浓度CTLA4-Ig（10μg/ml）联合，增殖抑制率在90％以上，已接近最大增殖抑制率，为二者联合应用的最佳组合，与单独应用组比较，有显著的统计学差异（p＜0.05）。

The inflammation reaction hyperplactic stage and lymphatic nodular stage are of nonspecific morphological changes and should be diagnosed with the help of positive tuberculin test and obvious increase of adenosine deaninase ; tuberculous nodular stage has a great number of epithelioid cells,and Langhans cells, caseous necrosis stage is characterized by a great deal of necrotic tissue and debris,a small number of fragmented epithelioid cells, and mainly by antiacid bacteria fibrinous hyperplastic stage is featured by a few fibrous tissue, cells and mucous oweing to hardness to get puncture, neans tuberculous restoration,and scar formation.

结核初期-炎性增殖期60例，占5.5%；结核早期-淋巴结节期130例，占11.9%；结核中期-结核性结节期有590例，占54.1%；结核晚期-干酪样脓样坏死期有280例，占25.7%；结核恢复期-纤维素增殖期30例，占2.8%。炎性增殖反应期非特异性形态学变化，需要结合结核抗体阳性和腺苷酸脱氨酶明显增高有助于诊断，结核结节期主要可有较多类上皮样细胞及郎罕氏细胞；而干酪样脓样坏死主要见大量坏死组织及碎屑、少数残碎不全类上皮样细胞，此期主要能查到抗酸菌为特点；纤维增殖期，抽出物难取，仅见少数纤维组织、纤维细胞和黏液间质为其特征，提示结核恢复、瘢痕形成所致。

Proliferating cell nuclear antigen is usually regarded as anindex of determinating cell proliferating and its expression levelin the nuclear is correlated with with the activity of cellproliferation. The activity of tumor cell proliferation can beknown by examinationing PCNA of tumor cells.

增殖细胞核抗原(proliferating cell nuclear antigen，PCNA)常作为测定细胞增殖水平的指标，其在细胞核内阳性表达的高低与细胞增殖活性密切相关，通过检测肿瘤细胞的PCNA ，可了解和评价肿瘤组织的增殖活性。

Methods: The model of evaluating T cell proliferation stimulated with polyclonal activators, ConA was established by vital dye carboxyl fluorescin diacetate succinmidyl ester labeling technique. The effect of a specific inhibitor for p38 MAPK, SB253580, on T cell proliferation on different doses and in different time was estimated by flow cytometry. At the same time, the percentage of proliferating T cells and proliferation index were measured by CellQuest and SPSS10.0 for Windows softwares.

以活体染料羧基荧光素乙酰乙酸琥珀酰亚胺酯染色，建立了在多克隆刺激剂刀豆蛋白A刺激下评价小鼠T细胞增殖的模型，通过流式细胞术分析p38丝裂原活化蛋白激酶的特异性抑制剂SB203580在不同剂量、不同时间对T细胞增殖的作用，并应用CellQuest和SPSS10.0 for Windows软件分析增殖细胞各代所占比例和增殖指数。

1、Cur inhibits K562 cells growth and induces cell apoptosis may be correlated with the down-regulation of p210~、inhibition of protein tyrosine phosphorylation and the signaling molecules such as p-Erk1/2、c-myc which are relevant with cell growth and apoptosis; 2、Cur synergizes STI571 to inhibit K562 cell growth and induce cell apoptosis may be correlated with the down-regulation of p210~、inhibition of protein tyrosine phosphorylation and the signaling molecules such as Hsp90、PKC which are relevant with cell growth and apoptosis; 3、Cur reverses the resistance of K562/G01 cells to STI571, and synergizes STI571 to inhibit K562/G01 cell growth and induce cell apoptosis; 4、Cur inhibits human originated CML CD34~+ cell growth、induces cell apoptosis, and enhances STI571 to down-regulate the expression of p210~, finally inhibit cell growth and induce cell apoptosis.

从以上实验结果我们得出如下结论： 1、Cur抑制K562细胞增殖、诱导细胞凋亡的作用可能与其下调p210~、蛋白酪氨酸磷酸化水平以及抑制下游p-Erk1/2、c-myc等信号分子有关； 2、Cur协同STI571抑制K562细胞增殖、诱导细胞凋亡的作用可能与其下调p210~、蛋白酪氨酸磷酸化水平以及抑制Hsp90和下游PKC等信号分子有关； 3、Cur可逆转K562/G01细胞对STI571的耐药性，并与STI571协同抑制K562/G01细胞增殖和诱导凋亡，其抑制K562/G01细胞增殖、诱导细胞凋亡的作用可能与其下调p210~、蛋白酪氨酸磷酸化水平以及抑制下游Procaspase-3和NF-κB等信号分子有关； 4、Cur可抑制来源于CML患者骨髓的CD34~+细胞的增殖并诱导其凋亡，还可协同STI571下调CML CD34~+细胞p210~表达，进而协同抑制细胞增殖、诱导细胞凋亡。

与增殖相关的网络解释 [注：此内容来源于网络，仅供参考]

Immortalization：无限增殖化,永生化[使细胞长期不断维持增殖状态]

idiotope 独特位 | immortalization 无限增殖化,永生化[使细胞长期不断维持增殖状态] | immunoadhesin 免疫粘附素

sporogony：孢子增殖

多分裂形式多样,疟原虫的裂体增殖(schizogony),孢子增殖(sporogony)和某些阿米巴、...生殖方式包括无性和有性两类. 无性生殖有裂体增殖(schizogony)产生裂殖子,以及孢子增殖(s... 详情...

adenotome：增殖体切除刀/增殖体切除器

adenosis /腺病/ | adenotome /增殖体切除刀/增殖体切除器/ | adenous /腺的/

adenotome blade：增殖体切除器刀片

adenotome 增殖腺刀,增殖体切除器 | adenotome blade 增殖体切除器刀片 | adenotomy 增殖体切除术

Cementum hyperplasia; Cemental hyperplasia：齿骨质增殖; 垩质增殖

Cementoclasy 齿骨质破坏 | Cementum hyperplasia; Cemental hyperplasia 齿骨质增殖; 垩质增殖 | Cenadelphus 完全对称深双畸胎

Desquamative conjunctival epitheliosis; Conjunctival epithelial hyperplasy：脱屑性结合膜上皮增殖; 结合膜上皮增殖

Desquamation; Peeling; Scaling 脱屑; 脱皮 | Desquamative conjunctival epitheliosis; Conjunctival epithelial hyperplasy 脱屑性结合膜上皮增殖; 结合膜上皮增殖 | Destructive adenoma 毁坏性腺瘤

proliferous polymerization：增殖聚合作用 增殖聚合

progestogens; progestins 孕激素 孕激素类 | proliferous polymerization 增殖聚合作用 增殖聚合 | promoter 促進劑(啟動子) 促进剂(启动子)

proliferous polymerization：增殖聚合[反应]

"proliferation","增殖" | "proliferous polymerization","增殖聚合[反应]" | "proliferous polymer","增殖聚合物"

immortalized cell：无限增殖化细胞

immortalization 无限增殖化,永生化[使细胞长期不断维持增殖状态] | immortalized cell 无限增殖化细胞 | imnortalizing gene 无限增殖(化)基因[与无限增殖化有关的基因]

breeder plant：增殖[反应]堆电厂,增殖[反应]堆电站

breeder pin 增殖燃料细棒,转换区燃料细棒 | breeder plant 增殖[反应]堆电厂,增殖[反应]堆电站 | breeder reactor 增殖[反应]堆