增多

基本解释 (translations)

manifold  ·  manifolded  ·  manifolds  ·  multiplications

词组短语

grow in number or quantity

与增多相关的网络例句 [注：此内容来源于网络，仅供参考]

When TiO〓 content is increased, the surfacial energy formed by crystals may be reduced, the devitrification degree, strengthened and the content of wollastonite in crystal phase raised; if Cr〓O3〓 content is increased, the crystal size is enlarged, and the content of diopside in crystal phase, raised; if the sulphide content is increased; the reduction atmosphere is heightened and the content of wollastonite become bigger.

晶核剂浓度研究表明，晶核剂浓度不同，对基础玻璃析晶强度，晶相含量和晶体尺寸有一定的影响，当TiO〓含量增加时，可以降低晶体形成的表面能，析晶强度增大，晶相中硅灰石含量增多；Cr〓O〓含量增加，晶体尺寸增大，晶相中透辉石含量增加；硫化物增多，还原气氛增大，同时硅灰石含量增多。

Cases of eosinophilia were collected and chromosome specimens of bone marrow cells were prepared by 24hour culture, and Gbanding technique was used for karyotyping.

为探讨染色体异常克隆在嗜酸性粒细胞增多症诊断和鉴别诊断中的意义及克隆性嗜酸性粒细胞增多症涉及的染色体异常，收集了65例嗜酸性粒细胞增多患者的骨髓标本，培养24小时，采用G显带进行核型分析。

The aim of study was to investigate the importance of chromosome aberration in differential diagnosis of eosinophilia and the chromosomal aberrations involved in patients with clonal eosinophilia. 65 cases of eosinophilia were collected and chromosome specimens of bone marrow cells were prepared by 24hour culture, and Gbanding technique was used for karyotyping.

为探讨染色体异常克隆在嗜酸性粒细胞增多症诊断和鉴别诊断中的意义及克隆性嗜酸性粒细胞增多症涉及的染色体异常，收集了65例嗜酸性粒细胞增多患者的骨髓标本，培养24小时，采用G显带进行核型分析。

With increasing aluminum content in the alloys, the fraction of primary phase α increases and the eutectoid decreases.

随着铝含量的增多，合金中的初生α相增多，共析体减少；同时合金的硬度和耐磨性随铝含量的增多而增大；当铝含量大于30%时，合金的二次枝晶臂间距变化不明显。

Terrain heightened can enhance blocking effect, which lead to strengthen water vapor flux divergence and vertical convection, and cloud water increases but centralizes in the middle-low level. This change intensify accretion of snow by collecting more supercooled cloud water, and rain water converts into graupel by collecting more snow, and dry growth of graupel increase at the same time. Rain water augment because of more graupel melting, and rain water continue to increase by collecting cloud water in the low level.

地形高度增加后，阻挡作用加强，使水汽通量辐合加强，垂直气流增大，中低层云水增多，导致雪撞冻过冷云水增多，促使了雨水撞冻雪转化为霰和霰干增长过程的加强，霰融化为雨水增多，低层雨水碰并云水加强，冷云过程得到增强。

Histological structures of bursas of Fabricius were observed at the different developmental stages from chicken embryoes to chickens. The results were as follows. The bursa of Fabricius began to develop on the 13th day of post-hatching and its cell aggregated into the nodosity clique underneath the epithelium mucosae. On the 15th day of post-hatching, those cell cliques were surrounded by the flat epithelium and formed the basic structure of folliculus lymphaticus. The number of lymphocytes in the folliculus lymphaticus increased gradually, and the medulla and cortex of a majority of folliculus were not discriminated obviously. Until on the 7th day for chickens, the medulla and cortex of folliculus formed completely and the number of folliculus increased significantly. From 21 to 35 days old, the folliculus lymphaticus transformed from polygon into rectangle. Mucosa epithelial cells increased gradually and their cytoblasts arranged compactly into aline in the basilar part on the 35th day.

本研究观察了从鸡胚到出壳后的不同生长发育阶段法氏囊的组织结构，结果表明：胚胎13日龄时，法氏囊开始发育，粘膜上皮下形成结节状细胞集团；胚胎15日龄，聚集的细胞集团形成初始的淋巴滤泡；胚胎18日龄到出壳后1日龄，滤泡内淋巴细胞数量增多，多数滤泡髓质和皮质界限仍不明显；7日龄雏鸡法氏囊髓质皮质分界清晰，滤泡数量显著增多；21日龄到35日龄，滤泡由多边形逐渐转变成长方形；在35日龄时，粘膜上皮细胞逐渐增多，排列紧密，细胞核紧密排列成直线位于基底部。

The results of immuhistochemistry show: compared with normal retina, more 3-NT-positive cells and iNOS-positive cells appear in inner nucleus layer of diabetic retina; less eNOS-positive cells appear in inner nucleus layer and vascular endoderm of diabetic retina; less nNOS -positive cells appear in inner nucleus layer of diabetic retina; more ET-positive cells, ETRA- positive cells, ETRB- positive cells appear in inner nucleus layer of diabetic retina; moreα- synuclein- positive cells appear in ocular cone and rod layer of diabetic retina. Conclusions 1. RFDD-PCR is an efficient technique for research diseases genomics as a mass screening to complete gene expression with the identifying of candidate gene related to disease.

免疫组织化学结果显示：13-NT和NOS：与正常视网膜相比，8周糖尿病大鼠视网膜中，INL的3-NT和iNOS免疫阳性细胞明显增多，INL和血管内皮层的eNOS阳性细胞明显减少，INL的nNOS阳性细胞也明显减少；2ET及ETR：与正常视网膜相比，8周糖尿病大鼠视网膜中，ET、ETRA、ETRB免疫阳性细胞明显增多，增多的阳性细胞主要集中于INL，而在血管内皮层增多不明显；3α-synuclein：与正常视网膜相比，8周糖尿病大鼠视网膜中，α-synuclein免疫阳性细胞明显增多，增多的阳性细胞主要集中于视网膜视锥视杆层。

Methods Constructed the model of fetal scarless wound healing and investigated the expression of collagen in fetal and adult rat wounds by immunohistochemistry and in situ hybridization.

染色，Ⅰ、Ⅲ型胶原免疫组化，Ⅰ、Ⅲ型胶原原位杂交。结果胎鼠伤口愈合快，组织结构再生完全，没有瘢痕形成，胶原呈网状正常排列，Ⅰ、Ⅲ型胶原蛋白及表达均增多，同时伴细胞数增多，Ⅲ型胶原首先形成网状排列；成年伤口胶原纤维沉积慢，平行排列的胶原逐渐呈束状填塞伤口以致瘢痕形成，伤口内Ⅰ、Ⅲ型前胶原mRNA表达均增多，细胞数目增多不明显。

The number of abnormal structure of thyroid gland of B group was significantly higher than that of A and D groups(P .05).The abnormal number by blood biochemistry detection of A, B, C group was significantly higher than that of D group(P.05). Conclusions : The thyroid gland volume and blood biochemistry abnormal number of patients of type 2 diabetes was obvious increase than normal control group.

结果：A组、B组、C组甲状腺体积较D组明显增加(P.05)，B组甲状腺体积较A组、C组明显增加(P.05)；B组甲状腺肿大例数较A组、C组、D组明显增多(P.05)；C组甲状腺结构异常例数较A组、B组、D组明显增多(P.05)，B组甲状腺结构异常例数较A组、D组明显增多(P.05)；A组、B组、C组的血液生化指标异常数较D组明显增多(P.05)。

The effects and mechanism of GABAergic neurons, NOergic neurons, opioid peptide and cyclic adenosine monophosphate in the nucleus reticularis thalami on sleep-wakefulness cycle of rats and the effects and mechanism of the 5-HTergic nerve fibers project from the nucleus raphes dorsalis to RT on sleep-wakefulness cycle of rats were investigated with the methods of brain stereotaxic, nucleus spile, microinjection and polysomngraphy.1. The effects of GABAergic neurons in RT on sleep-wakefulness cycle of rats1.1 Microinjection of 3-mercaptopropionic acid (3-MP, a kind of glutamate decarboxylase inhibitor) into RT. On the day of microinjection, sleep only decreased a litter. On the second day, sleep marked decreased and wakefulness marked increased. On the third and fourth day, sleep and wakefulness stages resumed to normal.1.2 Microinjection of gamma-amino butyric acid (GABA 1.0μg) into RT enhanced sleep and reduced wakefulness compared with control; while microinjection of L-glutamate (L-Glu, 0.2μg) decreased sleep and increased wakefulness; microinjection of bicuculline (BIC, 1.0μg), a GABAA receptor antagonist, enhanced wakefulness and reduced sleep; microinjection of baclofen (BAC, 1.0μg), GABAB receptor agonist, had the same effects as GABA.2. The effects of NOergic neurons in RT on sleep-wakefulness cycle of rats2.1 Microinjection of L-arginine (L-Arg, 0.5μg) into RT decreased sleep compared with control, but there were on statistaical difference between L-Arg group and control; while microinjection of sodium nitroprusside (SNP, 0.2μg), a NO donor into RT, sleep marked decreased and wakefulness marked increased. Microinjection of nitric oxide synthase inhibitor, N-nitro-L-arginine (L-NNA, 2.0μg) into RT enhanced sleep and reduced wakefulness.2.2 After simultaneous microinjection of L-NNA (2.0μg) and SNP (0.2μg) into RT, SNP abolished the sleep-promoting effect of L-NNA compared with L-NNA group; after simultaneous microinjection of L-NNA (2.0μg) and L-Arg(0.5μg) into RT, we found that L-NNA could not blocked the wakefulness-promoting effect of L-Arg.3. The effects of opioid peptide in RT on sleep-wakefulness cycle of rats3.1 Microinjection of morphine sulfate (MOR, 1.0μg) into RT increased wakefulness and decreased sleep compared with control; while microinjection of naloxone hydrochloride (NAL, 1.0μg), the antagonist of opiate receptors, into RT, enhanced sleep and reduced wakefulness.3.2 After simultaneous microinjection of MOR (1.0μg) and NAL (1.0μg) into RT, the wakefulness-promoting effect of MOR and the sleep-promoting effect of NAL were not observed compared with control.4. The effects of cAMP in RT on sleep-wakefulness cycle of rats Microinjection of cAMP (1.0μg) into RT increased sleep and decreased wakefulness compared with control; microinjection of methylene blue (MB,1.0μg) into RT enhanced sleep and reduced wakefulness compared with control.5. The effects of the 5-HTergic nerve fibers project from DRN to RT on sleep-wakefulness cycle of rats5.1 When L-Glu (0.2μg) was microinjected into DRN and normal sodium (NS,1.0μg) was microinjected into bilateral RT. We found that sleep was decreased and wakefulness was increased compared with control; when L-Glu (0.2μg) was microinjected into DRN and methysergide (MS,1.0μg), a non-selective 5-HT antagonist, was microinjected into bilateral RT, We found that sleep was enhanced and wakefulness was reduced compared with L-Glu group.5.2 When p-chlorophenylalanine (PCPA, 10μg) was microinjected into DRN and NS (1.0μg) was microinjected into bilateral RT, We found that sleep was increased and wakefulness was decreased compared with control; microinjection of 5-hydroxytryptaphan (5-HTP, 1.0μg), which can convert to 5-HT by the enzyme tryptophane hydroxylase and enhance 5-HT into bilateral RT, could block the effect of microinjection of PCPA into DRN on sleep-wakefulness cycle.

本研究采用脑立体定位、核团插管、微量注射、多导睡眠描记等方法，研究丘脑网状核(nucleus reticularis thalami，RT)中γ-氨基丁酸(gamma-amino butyric acid ，GABA)能神经元、一氧化氮(nitrogen monoxidum，NO)能神经元、阿片肽类神经递质、环一磷酸腺苷(cyclic adenosine monophosphate，cAMP)及中缝背核(nucleus raphes dorsalis，DRN)至RT的5-羟色胺(5-hydroxytryptamine，5-HT)能神经纤维投射对大鼠睡眠-觉醒周期的影响及其作用机制。1 RT内GABA能神经元对大鼠睡眠-觉醒周期的影响1.1大鼠RT内微量注射GABA合成关键酶抑制剂3-巯基丙酸(3-MP，5μg)，注射当天睡眠时间略有减少，第二日睡眠时间显著减少，觉醒时间明显增多，第三、四日睡眠和觉醒时间逐渐恢复至正常。1.2大鼠RT内微量注射GABA受体激动剂GABA( 1.0μg)后，与生理盐水组比较，睡眠时间增加，觉醒时间减少；而RT内微量注射L-谷氨酸(glutamic acid， L-Glu， 0.2μg)后，睡眠时间减少，觉醒时间增加；RT内微量注射GABAA受体阻断剂荷包牡丹碱(bicuculline，BIC，1.0μg)后，睡眠时间减少，觉醒时间增加；RT内微量注射GABAB受体激动剂氯苯氨丁酸(baclofen，BAC，1.0μg)后，产生了与GABA相似的促睡眠效果。2 RT内NO能神经元对大鼠睡眠-觉醒周期的影响2.1大鼠RT内微量注射NO的前体L-精氨酸(L-Arg，0.5μg)后，与生理盐水组对比，睡眠时间略有减少，但无显著性意义；而RT内微量注射NO的供体硝普钠(Sodium Nitroprusside，SNP，0.2μg)后可明显增加觉醒时间，缩短睡眠时间；微量注射一氧化氮合酶抑制剂L-硝基精氨酸(L-arginine，L-NNA，2.0μg)后，引起睡眠时间增多，觉醒时间减少。2.2大鼠RT内同时微量注射L-NNA(2.0μg)和SNP(0.2μg)后与L-NNA组比较发现SNP逆转了L-NNA的促睡眠作用；RT内同时微量注射L-NNA(2.0μg)和L-Arg(0.5μg)后，与L-NNA(2.0μg)组比较发现L-Arg可以增加觉醒而缩短睡眠，其促觉醒作用未能被NOS的抑制剂L-NNA所逆转。3 RT内阿片肽对大鼠睡眠-觉醒周期的影响3.1大鼠RT内微量注射硫酸吗啡(morphine sulfate，MOR，1.0μg)后与生理盐水组对比，睡眠时间减少而觉醒时间增加； RT内微量注射阿片肽受体拮抗剂盐酸纳洛酮(naloxone hydrochloride，NAL，1.0μg)后与生理盐水组比较，睡眠时间增加而觉醒时间减少。3.2大鼠RT内同时微量注射MOR(1.0μg)和NAL(1.0μg)后，与生理盐水组对比，原有的MOR促觉醒效果和NAL的促睡眠效果都没有表现。4 RT内环一磷酸腺苷信使对大鼠睡眠-觉醒周期的影响大鼠RT内微量注射cAMP(1.0μg)后与NS(1.0μg)组比较，睡眠时间增多而觉醒时间减少；RT内微量注射亚甲蓝(methylene blue，MB，1.0μg)后，与NS组比较，睡眠时间增多而觉醒时间减少。5中缝背核投射到丘脑网状核的5-羟色胺能神经纤维对大鼠睡眠-觉醒周期的影响5.1大鼠DRN内微量注射L-Glu(0.2μg)，同时在双侧RT内微量注射NS (1.0μg)后，与对照组(DRN和双侧RT注射NS， 0.2μg)比较，睡眠时间减少，觉醒时间增多；大鼠DRN内微量注射L-Glu(0.2μg)，同时在双侧RT内微量注射二甲基麦角新碱(methysergide， MS， 1.0μg )后，与对照组(DRN注射L-Glu 0.2μg，双侧RT注射NS 1.0μg)比较，睡眠时间增多，觉醒时间减少。5.2大鼠DRN内微量注射对氯苯丙氨酸(p-chlorophenylalanine，PCPA，10μg)，同时在双侧RT内微量注射NS (1.0μg)后，与对照组(DRN和双侧RT注射NS， 1.0μg)比较，睡眠时间增多，觉醒时间减少；大鼠DRN内微量注射PCPA(10μg)，产生睡眠增多效应后，在双侧RT内微量注射5-羟色胺酸(5-hydroxytryptaphan ， 5-HTP， 1.0μg )后，与对照组(DRN注射PCPA 10μg，双侧RT注射NS 1.0μg)比较，睡眠时间减少，觉醒时间增多。

与增多相关的网络解释 [注：此内容来源于网络，仅供参考]

eosinophilia：嗜酸性粒细胞增多症

嗜酸性粒细胞增多症(eosinophilia)的病因、临床表现及实验室资料,加深对本病的认识.方法:对1995年1月至2005年7月广州市花都区人民医院儿科收住院的27例嗜酸性粒细胞增多症患儿的病因、临床表现、实验室检查、治疗等进行回顾性分析.结果:嗜酸性粒细胞增多症病因主要是寄生虫感染(11例)、变态反应性疾病(8例)、原因不明(8例),

eosinophilia：酸性粒细胞增多

2.嗜酸性粒细胞增多(eosinophilia) (1)过敏性疾患:如在支气管哮喘、血管神经性水肿、食物过敏、血精病时均可见血中嗜酸性粒细胞增多. 肠寄生虫抗原与肠壁内结合IgE的肥大细胞接触时,使后者脱颗粒而稀放组胺,导致嗜酸性粒细胞增多.

tropical eosinophilia：热带嗜曙红细胞增多,热带嗜曙红细胞增多症

tropical ecology 热带生态学 | tropical eosinophilia 热带嗜曙红细胞增多,热带嗜曙红细胞增多症 | tropical eosinophilic asthma 热带嗜曙红细胞增多性气喘

erythroblastosis：成红细胞增多症,有核红细胞增多症

erythroblast成红细胞,有核红细胞 | erythroblastosis成红细胞增多症,有核红细胞增多症 | erythrochromia脊(髓)液血色症

erythroblastosis：成红细胞增多 成红细胞增多病

erythroblastomatosis 成红细胞瘤病 | erythroblastosis 成红细胞增多 成红细胞增多病 | erythroblastosisfetalis 胎儿成红细胞增多

erythrocytosis：红细胞增多症

瘤伴发红细胞增多症(erythrocytosis)者罕见患者多无症状主要的诊断依据是血红蛋白与红细胞计数增高除子宫肌瘤外找不到其他引起红细胞增多症的原因肿瘤切除后血红蛋白与红细胞均降至正常国内吴葆桢于1964年报道一例子宫肌瘤合并红细胞增多症患者35岁腹部胀大如足月妊娠剖腹探查全腹为一巨大的分叶状实质肿物充满表面静脉迂曲

erythrocytosis megalosplenica：巨脾性红细胞增多,巨脾性红细胞增多

erythrocytic stage 红细胞内期 | erythrocytosis megalosplenica 巨脾性红细胞增多,巨脾性红细胞增多 | erythrodermie pityriasique enplaques disseminees 斑块状类牛皮癣

relative erythrocytosis：相对性红细胞增多,红细胞相对增多

relative error of the period 相对周期误差 | relative erythrocytosis 相对性红细胞增多,红细胞相对增多 | relative excessiveness of yin and yang 阴阳偏胜

hyperpigmentation：色素增多

(26)色素增多:色素增多(hyperpigmentation)是指表皮基底及真皮上部黑素颗粒增多,见于riehl黑变病、黄褐斑等. (27)色素减少:色素减少(hypopigmentaion)是指表皮基底层内黑素颗粒减少或缺如,见于白癜风、炎症后色素脱失等.

infectious lymphocytosis：传染性淋巴细胞增多症

传染性淋巴细胞增多症(infectious lymphocytosis)是一种传染病,主要发生于儿童,少数散发于成人. 1941年由Smith将其与传染性单核细胞增多症区别开来本病的特征为外周血中白细胞总数增多,其中以淋巴细胞增多为主,持续时间较长,症状较轻且为非特异性,