坏死后的

基本解释 (translations)

postnecrotic

与坏死后的相关的网络例句 [注：此内容来源于网络，仅供参考]

Objective: To investigate the method for treating necrotic wound after large area skin avulsion.

目的：探讨大面积皮肤撕脱伤后皮肤坏死创面的治疗方法。

Inside the source apply color to develop process in the tooth in formative of, such as four wreath vegetables deposition is in the tooth essence, will make tooth yellow, brown or dark gray, be called four wreaths plain tooths;If containing fluorine in the drinking water is excessive, may also cause fluorine spot tooth, the tooth noodles presents a white chalk color, the palm tree brown spot piece;If the tooth nerve is bad clay-cold, hemoglobin and germ resolve an outcome to combine to also make tooth black.

内源性着色是在牙齿发育过程中形成的，如四环素沉积在牙本质内，就会使得牙齿变成黄色、棕色或暗灰色，称为四环素牙；如果饮用水中含氟过多，也可能导致氟斑牙，牙面呈白粉笔色、棕褐色斑块；如果牙神经坏死后，血色素与细菌分解产物结合也可使牙齿变黑。

Right frontal encephalomalacia The frontal pole is largely replaced with the high signal of cerebrospinal fluid left after liquifaction necrosis in this old infarct.

右额叶脑软化额极大部分被旧梗塞灶液化坏死后留下的高信号脑脊液替代。

HISI treatment prolong the survival time of mice in toxic condition induced by sodium nitrite and duration of electrocardiograph in trachea closed mice .HISI could obviously improve the ischemic electrocardiogram T-wave induced by Pituitrin.ST segment drifts on electrocardiogram in acute myocardial ischemia rats induced by Isoprotereno were obviously opposed by administration of HISI. The myocardial infarction areas, releasement of lactic dehydrogenase 、 creatine phosphokinase significantly increased in myocardial ischemia dogs, HISI treatment prevented these effects .

实验结果表明：苦碟子氯化钠注射液能显著延长小鼠常压缺氧环境及亚硝酸中毒情况下的存活时间，延长小鼠气管夹闭后心电消失时间；可显著改善垂体后叶素诱发的大鼠急性心肌缺血心电图T波异常；明显对抗异丙肾上腺素诱导的急性心肌缺血大鼠心电图ST段的偏移；显著改善冠脉结扎所致急性心肌缺血犬的心外膜心电图缩小其梗死心肌面积，降低心肌耗氧量，减少心肌坏死导致的心肌酶如乳酸脱氢酶、肌酸磷酸激酶的漏出。

Six weeks after drug intervention,we found,in ADM+Res group,the mean tumor volume of MCF-7/ADM cells implanted tumor was 1.203±0.338 cm~3 with statistical significance to compare with other groups(P＜0.05),decreased about 50.58%compared with the mean tumor volume of ADM alone group1.979±0.307 cm~3the result of the mean tumor weigh was alike to the mean tumor volume; compared with the control,Res could relieve weigh loss(P＜0.05,and the toxicity of treatment was not obvious;Microscopic examination showed that there was massive area of cytoclasis and apoptosis in the tumor tissue of the combination group comparing with other groups.

实验结果提示用药6W后，ADM+Res组移植瘤体积(1.203±0.338)cm~3明显小于其余各组，抑瘤率为50.58%，与单用ADM组(1.979±0.307)cm~3相比有差异P＜0.05肿瘤重量的结果与瘤体结果类似；未观察到用药后的毒副作用；经不同处理后，单独注射Res组小鼠体重较生理盐水组及ADM组有所增加(P＜0.05，而ADM+Res组治疗前后体重无差异P＞0.05HE结果显示与其他组相比，ADM+Res组肿瘤组织中出现大片坏死区域。

The thickness of bovine liver in vitro ranged from 4 to 10 cm. Ultrasound energy was continuously delivered to ablate the liver with a frequency of 1.6MHz, and spatial peak pulse average acoustic intensity of 12000 W/cm^2. The characteristics of complete coagulation necrosis and exposure time were measured after FUSC exposure, and pathological changes in treated bovine liver tissue were observed.

在超声频率为1.6MHz，声强12000W/平方公分条件下，对厚度4~10cm离体牛肝进行双面连续扫描辐照后，记录牛肝达到贯通性凝固性坏死的辐照时间，测量离体牛肝凝固性坏死性状的形状和特征，观察组织病理学的变化。

Objective To investigate the feasibility and effectiveness of radiofrequency ablation for the treatment of secondary splenomagely and hypersplenism in dog.

RFA后的脾脏CT病灶呈节段性毁损，包括高密度的坏死区和低密度的梗死区；梗死区在RFA后 4～ 6周内消失。

There were nanobacteria in the nucleus and intracellar and extracellar endothelial cells under transmission electron microscopy after co-culture. The cell vacuolar degeneration, membrane structural necrosis, mitochondrin edema, mitochondrin vacuolar degeneration and lysosome myelinization were found in the endothelial cells.

混合培养后的内皮细胞透射电镜观察见细胞间、细胞内均有纳米细菌存在，细胞核内也可见纳米细菌，细胞空泡变，膜性结构类似坏死，部分线粒体水肿，空泡变，溶酶体呈现髓鞘图像。

After 20, 40, 60 or 90 minutes ischemia and 120 minutes reperfusion, the ratio of necrotic to no-necrotic liver were 3.9%, 13.5%, 66.4% or 82.7%, of those the percentage necrosis during reperfusion 120 minutes was 7.7%, 11.1%, 68.1% or 67.2% respectively; The liver volume of blood flow decreased to 94.7%, 85.3%, 55.9% or 43.5% of normal values in end of ischemia; Liver sinusoidal obstruction by blebs originating from the hepatocytes, fragments of sinusoidal endothelial cell damage and aggregation of blood cells was observed in group of 60, 90 minutes ischemia.

血流阻断20、 40、 60和90 min组再开放2 h，肝总坏死范围分别为3.9%、 13.5%、 66.4%和82.7%，其中肝血流开放后的肝坏死范围分别占肝总坏死范围的7.7%、 11.1%、 68.1%和67.2%；肝门静脉恒压灌流量于阻断期末分别下降5.3%、 14.7%、 44.1%和56.5%；肝血窦在阻断20、 40 min组无阻塞现象，阻断60和90 min组出现血细胞，膜浆泡和纤维束积滞，阻塞血窦面积占肝血窦总面积的50%。

It is now known that seere hypoxia-ischemia may not cause immediate cell death, but may precipitate a complex biochemical cascade leading to the delayed deelopment of neuronal loss. These phases include a latent phase after reperfusion, with initial recoery of cerebral energy metabolism but EEG suppression, followed by a secondary phase characterized by accumulation of cytotoxins, seizures, cytotoxic edema, and failure of cerebral oxidatie metabolism from 6 to 15 h post insult.

现在认识到严重的局部缺氧有可能不立即引起细胞坏死，但是可能促成一种复杂的生化级联反应，这可能导致神经元丢失，进而导致延缓发育，这些时期包括再灌注后的潜伏期，该期出现早期的脑能量代谢复苏但脑电图却呈抑制状态，紧接着第二期表现为损伤后6－15个小时细胞毒素，癫痫，细胞毒性水肿，脑氧化功能衰竭的累积。

与坏死后的相关的网络解释 [注：此内容来源于网络，仅供参考]

White blood cell：白细胞

本研究采用家兔胆总管结扎致胆总管梗阻动物模型,观察胆总管梗阻不同时间及梗阻解除后的家兔体温(temperature)、白细胞(white blood cell)、胆囊组织内巨噬细胞(macrophage)的数量,以及血清及组织中的肿瘤坏死因子-α(TNF-α)数值的变化,

hemorrhagic infarct：出血性脑梗死

3.出血性脑梗死(hemorrhagic infarct)是由于脑梗死供血区内动脉坏死后血液漏出继发出血,常发生于大面积脑梗死之后. 4.多发性脑梗死(multiple infarct)是指两个和两个以上不同的供血系统脑血管闭塞引起的梗死,多为反复发生脑梗死的后果.

involucrum：包壳

坏死和由此诱发的修复反应(骨质增生)同时并存,早期以破坏和坏死为主,皮质骨内层接受干骺端的血液供应,血供受损后,骨质坏死,肉芽组织将其与存活的骨分开,形成死骨片,骨膜反应生成新骨称为包壳(involucrum),包裹感染骨和坏死骨,

ischemia：缺血

即最早的轻度缺血(ischemia)致T波倒置(缺血部心室肌动作电位时限延长,即复极时限延长). 接着是损伤期(injury)的ST后上升(损伤心肌细胞不能完全复极,复极后仍处于部分除极状态,与正常复极后的心肌间存在电位差). 最后是心肌细胞陷于坏死,

Hibernating myocardium：冬眠心肌

然而,近年来的研究已证明由于心肌缺血的速度、程度不同,以及缺血心肌有无再灌注或侧支循环供血,心肌梗死(MI)后的RWMA区域除了坏死心肌外,还可能有下列存活心肌(viable myocardium)存在:1.顿抑心肌(stunned myocardium);2.冬眠心肌(hibernating myocardium);3.伤残心肌(m

caseous necrosis：干酪性坏死

(三)感染 结核菌进入体内后经过约 6至8 周的繁殖,数量达1,000至10,000只之后,其抗原即足以激发宿主产生细胞型免疫反应(host cellular mediated immunity),引发乾酪性坏死(caseous necrosis),即所谓迟发型过敏反应(delayed-type hypersensitivity,

phlegmon：蜂窝织炎

另外过去文献上尚有胰腺蜂窝织炎(phlegmon)一词. 目前认为此时胰腺并无明显坏死或大块坏死,可能有小的散在的坏死灶,胰腺肿大,硬韧,以炎性浸润为主,周围和腹膜后组织水肿明显,远较单纯水肿严重,有大量渗出,造成液体积聚,无被膜包裹,

sunburn：日晒伤

日晒伤(sunburn)又称晒斑,是强烈日光照射后引起的局部急性红斑,水肿性皮肤炎症. 是由日光中的中波紫外线照射过度引起的急性炎症反应. 中波紫外线作用表浅,仅限于表皮,强烈照晒造成表皮角朊细胞坏死,释放的介质导致真皮血管扩张,

neuronophagia：噬神经细胞现象

坏死细胞周围常有小胶质细胞围绕并有中性粒细胞浸润形成噬神经细胞现象(neuronophagia). 脑实质肿胀. 软化灶形成后可发生钙化或形成空洞. 胶质细胞增生主要是小胶质细胞增生,呈弥漫性或灶性分存在血管旁或坏死崩解的神经细胞附近.

Osteonecrosis unspecified：股骨头缺血性坏死

分类于他处的其他疾病引起的骨坏死 Osteonecrosis in other diseases classified elsewhere | 股骨头缺血性坏死 Osteonecrosis unspecified | 脊髓灰质后骨病 Osteopathy after poliomyelitis