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And There was no serious complication;②In the course ofinterventional therapy, Direct portal vein angiography demonstrated vena coronaria ventriculi(100%)andgastricveins(65.26%)andvenagastricaposterior38.43%, Angiography demonstrated venacoronariaventriculi communicate esophagus varicose veins, gastric veins and vena gastrica posterior communicategastric varicose veins. vena coronaria ventriculi had only a small percentage of double vein, about30.57%. The sites of vena coronaria ventriculi arising from the portal vein, splenic vein, portosplenic junction, were found in 52.06%、27.39%、20.55% respectively.③12 extrahepaticprotosystemic shunts were found in these patients. Include gastro-nephrosshuntof 3 cases, 7 caseswere splenetic- nephros shunt and 2 cases shown recanalization of umbilical vein .④The averageportal pressure before and after the procedure were 3.87±1.82kPa and 3.64±1.14kPa in 73patients, but to the time of rebleeding, it was 3.96±0.23kPa in the 11 cases.⑤There werethree kinds of variceal outcome: disappearance (54,low degree (19).⑥Spearman logisticanalyse and ANOVAtest shown liver function class, variceal degree of the splenic necrosis area,the blood direction in portal vein before operation and remain small collateral routes were thesignificant factors concerning outcome of varices.⑦The bleeding volume and portalhypertensive gastropathy are main risk factors of rebleeding.⑧The course of livercirrhosis is the risk factor of survival and extrahepatic portosystemic shunt , fine varices are thebeneficial factors to survival.⑨During all cases'followed-up data, the 1, 2, 3, 4, 5 yearcumulative survival rates and rebleeding rates were 17.81%, 28.77%, 38.36%, 43.84%, 47.95%and93.15%,91.78%,86.30%,83.56%,80.82%respectively. Conclusion The interventional disconnection treatment for liver cirrhosis and portalhypertension was designed suitability. It rapidlycontrol bleeding,butpressure of portal vein was notobvious high, perfusion was not low .it was compared with surgery therapeutic that interventionaldisconnection treatment was safe and had a significant clinical effect to hemorrhage and preventfrom rebleeding.
结果:①术后一过性发热62例(84.9%),腹痛腹胀48例(65.8%)是介入断流术常见的并发症,未发生严重并发症;②门静脉造影显示胃冠状静脉、胃短静脉和胃后静脉的曲张分流的出现率是100%、65.26%和38.34%;显示食管静脉曲张主要由胃冠状静脉供血,胃静脉曲张主要由胃短静脉和胃后静脉供血;胃冠状静脉大多数为单支,少数为双支,其双支的出现率分别为30.57%;胃冠状静脉开口于门静脉主干的为52.06%,开口于脾静脉主干的为27.39%和开口于门脾静脉交汇处的为20.55%;③发现胃肾分流3例,脾肾分流7例、腹膜后门腔静脉分流2例,以及CTA检查发现脐静脉开放者2例;④73例患者介入断流术前和术后平均自由门静脉压力分别为3.87±1.82kpa和3.64±1.14kpa,前后比较存在显著性差异;11例再次介入手术患者的术前、术后和复发后的自由门静脉压力分别为4.02±0.24kpa、3.82±0.25kpa和3.93±0.23kpa ,前后比较发现首次术前与术后存在显著性差异,首次术前和复发出血术前门静脉压力比较无显著性差异;⑤介入术后复查曲张静脉转归基本消失54例,轻度19例;⑥Spearman相关分析和Logistic多因素回归分析,肝功能分级、静脉曲张程度、门脉血流方向和残存小侧支四个因素对曲张静脉转归有影响;Spearman相关分析和Logistic多因素回归分析门脉高压性胃病和出血量等因素对复发出血时间有影响;⑦COX回归分析,门体分流和曲张静脉转归两个因素对术后生存有影响;⑧术后随访6-70月,1、2、3、4、5年的累计复发出血率和累计生存率分别为17.81%、28.77%、38.36%、43.84%、47.95%和93.15%、91.78%、86.30%、83.56%、80.82%;结论:介入断流术治疗门脉高压食管胃底静脉曲张有独特的优点,可以快速直接控制曲张静脉出血而门静脉压力无显著增高,保证了肝脏灌注;与外科分流术相比适应证广、损伤轻、术后恢复快,不易遗漏曲张静脉;肝功能分级、曲张静脉程度、门脉血流方向和残存侧支血管对食管胃曲张静脉转归有影响;门脉高压性胃病对复发出血时间有影响;门体分流和曲张静脉转归对生存时间有影响。
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Assessment of Gastric Sensitivity, Compliance and Accommodation: The stomachs were distended isobarically by barostat via a double-lumen polyvinyl tube with an adherent plastic bag, gastric sensitivity was measured by perception threshold and gastric compliance was calculated as the slop of the pressure-volume curve.
胃敏感性、顺应性和容受性的检测:用barostat及双腔气囊导管对胃进行等压扩张,以患者的不适阈代表胃对扩张的敏感性,以压力-容量曲线的斜率反映胃顺应性;将气囊压力恒定为MDP+2mmHg,以进食前后胃内平均容量的差值来反映近端胃的容受性。7。
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dry plate:干版
dry objective 干物镜 | dry plate 干版 | dual-bag and three-cavity stomach catheter 双囊三腔胃导管