- 更多网络例句与剂量相关的网络例句 [注：此内容来源于网络，仅供参考]
-
Result:As compared with the model group, the effect of small dose group and middle dose group of osteopractic total flavone on TBV% of shankbone increased significantly; the effect of small dose group and middle dose group of osteopractic total flavone on TRS% of shankbone reduced significantly and TFS%, AFS%, MAR, BFR of shankbone reduced obviously; the effect of middle dose group of osteopractic total flavone on OSW and mAR reduced obviously, the effect of small dose group of osteopractic total flavone on them had the tendency of reducing, but there was no statistical significance.
结果 ：对胫骨骨小梁体积百分比的影响，与模型组比较，骨碎补总黄酮小剂量组和中剂量组的TBV %明显增高；对胫骨骨小梁吸收表面百分比的影响，与模型组比较，骨碎补总黄酮小剂量组、中剂量组的TRS %显著降低；对胫骨骨小梁形成表面百分比，活性生成表面百分比，骨小梁矿化率和骨小梁骨生成率的影响，骨碎补总黄酮小剂量组、中剂量组的TFS %，AFS %，MAR ，BFR较模型组皆明显降低；对胫骨类骨质平均宽度和骨皮质矿化率的影响，骨碎补总黄酮中剂量组的OSW和mAR与模型组比较，明显降低；骨碎补总黄酮小剂量组与之相比，有下降的趋势，但无统计学意义。
-
Results The UV-B treatment caused the declines of chlorophyll content, soluble protein content and water content of leaves, besides, dose-effect was existed. The decline of 30 μW/cm^2 treatment was bigger than that of 15 μW/cm^2 treatment. The influences of UV-B radiation on carotenoid content and anthocyanidin content were similar; the change trends were declined firstly then increased. The UV-B treatment with two doses restrained the electron transport of PSⅡ, particularly; the inhibitory effect was biggest after treated 2h, and then this effect was declined in 4, 6 and 8 h, so the dose-effect was existed. The UV-B radiation with two doses restricted photosynthetic rate and the inhibitory effect increased with the increase of treatment time. The high dose treatment caused huge damage to membrane system, while the result of low dose treatment was not obvious.
结果］UV-B处理导致叶绿素、可溶性蛋白含量以及含水量都降低，且存在剂量效应，30μW/平方公分处理的降幅大于15μW/平方公分；UV-B辐射处理对类胡萝卜素含量的影响与花青素相同，均表现为先下降后上升的趋势，2种剂量间差异不大；2种剂量的UV-B处理均抑制了PSⅡ的电子传递，尤其是处理2 h的抑制作用最大，4、6和8 h的抑制作用有所缓解，且存在剂量效应；2种剂量的UV-B辐射均抑制了叶片的光合速率，随着照射时间的延长抑制效应加大；高剂量UV-B处理对膜系统的损伤较大，低剂量UV-B处理结果不明显。
-
It will provide theoretical and experimental base for the research of syndrome of traditional Chinese medicine. Method: 60 depuratory and healthy SD female rats with normal estrous cycle(4~5days),3-month in age, 230±20g in body weight,were divided into 6 groups randomly, normal group,model group,low-dose group,middle-dose group, high dose group,control group.
选用动情周期正常(4～5天)的健康清洁级SD雌性大鼠60只，鼠龄3个月，体重230±20g，随机分为6组，每组10只：即正常对照组，模型对照组，模型加加减温经汤低剂量组，模型加加减温经汤中剂量组，模型加加减温经汤高剂量组(简称为&高剂量组&，低剂量组、中剂量组、高剂量组又合称为&治疗组&)，桃红四物汤组。
-
Results: Tyroserleutide can significantly increase the life span of H22 tumor-bearing mice by 50-70% in dosages of 20ug/kg/d-80ug/kg/d,specially the high dosage of 80ug/ml can significantly increase the life span by 69.24%; Tyroserleutide can inhibit the growth of transplanted hepatocellular tumor BEL-7402 in nude mice,the rate of tumor inhibition was25-50% in dosages of 40-320ug/ml ,the inhibition rate of 160ng/ml was 44.03%; Tyroserleutide could inhibit the growth of H22 and BEL-7402 tumor in a dose-dependent manner. Simultaneously, tumoricidal activity of tyroserleutide against BEL-7402 cell line in vitro was observed hinger when compared with the control group(P.05).The inhibition effect of 72hrs was higher than 24hrs,48hrs,96hrs.And specially the high dosage of 160ug/ml can significantly inhibit growth of tumor cell by 19.36%. Tyroserleutide can activated PEM and marked enhance cytotoxicity andphagocytosis functions in vitro and in vivo. The OD values of cytotoxicity were observed hinger when compared with the control group(P.05).The cytotoxicity of macrophages activated by tyroserleutide against BEL-7402 and B16-F10 was 35.58%,61.2% in vitro and21.39%,47.63% in vivo. The cytotoxicity rate of nude mice PEM was 32.86%,73.07% in vivo. Furthermore, tyroserleutide alone could stimulated the production of IL-1B TNF- a and NO by M . Tyroserleutide and LPS could synergistically activated M producing more cytotoxicity effectors. Conclusion: Tyroserleutide had inhibition functions against hepatoma carcinoma .Its possible mechanisms were related to the affect that Tyroserleutide could inhibit tumor cell directively and induce tumor cells apoptosis or death effectively.
结果：酪丝亮肽能显著延长腹水型肝癌H_(22)小鼠的生存时间，给药剂量为80μg／kg／d时疗效最显著，达到69.24％，在20μg／kg／d-80μg／kg／d剂量范围内生命延长率为50-70％，给药剂量与荷瘤鼠生存时间呈现一定量效关系；酪丝亮肽能显著抑制人肝癌BEL-7402移植瘤裸鼠的肿瘤生长，给药剂量为160μg／kg／d时疗效最显著，抑制率为44.03％，并且在40-320μg／kg／d剂量范围内抑制率为25-50％，给药剂量与肿瘤抑制率呈现一定量效关系；酪丝亮肽体外对人肝癌BEL-7402细胞生长有一定的抑制作用，在作用72hrs时各浓度酪丝亮肽对肿瘤细胞的抑制作用较24hrs、48hrs、96hrs明显，其中浓度为100μg／ml时抑制率达19.36％；酪丝亮肽体内外均能增强小鼠腹腔巨噬细胞对肿瘤细胞的杀伤：体外作用中巨噬细胞对BEL-7402、B16-F10的杀伤功能明显增强，与效应细胞对照组相比有显著性差异(P＜0.05)杀伤率分别达到35.58％、61.2％；体内作用中巨噬细胞对BEL-7402、B16-F10的杀伤功能明显增强，与生理盐水对照组相比有显著性差异(P 。05)，杀伤率分别达到21.39%、47.63%；裸鼠腹腔巨噬细胞经酪丝亮肤作用后对BEL一7402、B 16一F10杀伤功能明显增强，与生理盐水对照组相比有显著性差异(P.05)，最高杀伤率分别达到32.86%、73.07%；酪丝亮肤能增强单核巨噬细胞系统的吞噬功能，吞噬指数与生理盐水组比较有显著性差异(P.05)；酪丝亮肤体外作用能促进小鼠腹腔巨噬细胞分泌合成细胞毒效应分子IL一lp、TNF一Q和NO，与效应细胞对照组相比有显著性差异(P.05)；酪丝亮肤体内作用能促进小鼠腹腔巨噬细胞分泌合成细胞毒效应分子IL一lp、TNF一Q和NO，与生理盐水对照组相比有显著性差异(P.05)；酪丝亮肤能促进鼠巨噬细胞株R戌W264.7分泌合成IL一1p和NO，IL一1日、NO水平分别在酪丝亮肤作用24hrs、12hrs时达到高峰，酪丝亮肤单独应用能提高巨噬细胞的分泌合成功能，而且酪丝亮肤能与LPS协同作用刺激巨噬细胞的细胞毒效应分子分泌合成。
-
Methods Eighty Kunming mice were equally randomized into 8 groups: normal group, model group, vitamin E 90 mgkg^(-1d^(-1) group, high-and low-dose RPM protein 20 and 5 mgkg^(-1d^(-1)respectively groups, high-and low-dose RPM anthraquinone glycoside 28 and 7 mgkg^(-1d^(-1) respectively groups, combination (low-dose RPM protein and low-dose RPM anthraquinone glycosides) group. Except the normal group, the mice in other groups received subcutaneous injection of D-galactose to indue the aging model.
选用昆明种小鼠80只，随机分为8组：正常组，模型组，维生素E组（剂量为90mgkg^(-1)d^(-1)），何首乌蛋白质高、低剂量组（剂量分别为20、5mgkg^(-1)d^(-1)），何首乌蒽醌苷高、低剂量组（剂量分别为28、7mg kg^(-1)d^(-1)），AGRPM与PRPM混合组（混合组，均取两者低剂量）；除正常组外，其他组均采用长期（7周）皮下注射D-半乳糖复制衰老小鼠模型。
-
Results The micronuclear rate in medium dosage group and high dosage group were significantly higher than that of control group(P＜0.01). The ratio of sperm malformation in middle group and high group were higher than those of controls (P＜0.01).The ratio of sperm malformation increased in dose-dependent manner with dose, a dose-response relationship was found. Conclusion The DBP contamination presents the positive results under the certain dosage to the mouse marrow micronucleus experiment and the spermatozoon abnormal experiment.
结果 中、高剂量组微核率与阴性对照组相比明显升高(P＜0.05)，且各剂量组微核率随染毒浓度的增加而升高，呈现剂量-反应关系(r=0.937，P＜0.01)；中、高剂量组精子畸形率与阴性对照组比较差异有统计学意义(P＜0.05)，且各剂量组精子畸形率随染毒浓度的增加而升高，呈剂量-反应关系(r=0.904，P＜0.01)，畸形精子中以无钩为最多，其次是香蕉形、尾折叠、双头，分别为4.5%、3.09%和1.32%，结论 DBP染毒在一定剂量下对小鼠骨髓微核试验和精子畸形试验呈现阳性结果。
-
The microkemel and sperm cell abnomalities were analyzed. Results The micronuclear rate in medium dosage group and high dosage group were significantly higher than that of control group(P＜0.01). The ratio of sperm malformation in middle group and high group were higher than those of controls (P＜0.01).The ratio of sperm malformation increased in dose-dependent manner with dose, a dose-response relationship was found. Conclusion The DBP contamination presents the positive results under the certain dosage to the mouse marrow micronucleus experiment and the spermatozoon abnormal experiment.
结果 中、高剂量组微核率与阴性对照组相比明显升高(P＜0.05)，且各剂量组微核率随染毒浓度的增加而升高，呈现剂量-反应关系(r=0.937，P＜0.01)；中、高剂量组精子畸形率与阴性对照组比较差异有统计学意义(P＜0.05)，且各剂量组精子畸形率随染毒浓度的增加而升高，呈剂量-反应关系(r=0.904，P＜0.01)，畸形精子中以无钩为最多，其次是香蕉形、尾折叠、双头，分别为4.5%、3.09%和1.32%，结论 DBP染毒在一定剂量下对小鼠骨髓微核试验和精子畸形试验呈现阳性结果。
-
79Cm and 3. 78cm from M,, M2 to M3 respectively which cannot reach 1/3 height of the control averagely. The seedlings irradiated by 80 X1015 NYcmgave birth to some flower buds without floral shoots and their seed setting rate was lower than that of control obviously. Some M3 plants irradiated by 60X1015 NVcm2grow into bunches which did not appear in M, and M2. Moreover, some phenotypic variations could be inherited stably in subsequent generations. For instance, the dwarf character in the plants treated with the dose of 80 X1015 was stable in inheritance in consecutive three generations.
再次，对不同剂量的低能N~+处理的拟南芥的M_2代试管苗的叶片外植体愈伤组织的诱导和耐盐性进行了比较和分析，发现经不同剂量的低能离子处理的M_2代试管苗的叶片外植体，离子处理影响其出愈率，高剂量处理会抑制愈伤的诱导，但低剂量离子注入对愈伤组织的增殖有一定的促进作用；愈伤组织的耐盐性也与剂量相关，低剂量的耐盐能力比对照的低，但是高剂量处理反而提高愈伤的耐盐性，这间接表明低能离子引起了拟南芥的生理状态的变化，产生了生理效应。
-
The point doses at the inferior beam edge for the two energies were 31.2% and 32.4%, respectively. There were 47-54% dose increase for superior beam edge and 37.6-35.2% dose decrease for inferior beam edge, as compared to the 50% field edge dose with the static dose distribution. There were 8.5 and 7.5 mm range increase for superior beam penumbras and 5.5 and 4.5 mm range increase for inferior beam penumbras, an important result corresponding to treatment volume.
结果：以正常人的平均值为例，在一个照射时距内，使用6 MV及18 MV光子射束， 10X10cm2照野10cm深度之中心轴下，照野上缘的剂量，分别为照野中心点剂量的73.5 %及77.2 %，照野下缘的剂量则分别为中心点剂量的31.2 %及 32.4 %，此剂量反映出因治疗中的呼吸位移，造成照野上缘较照野中心点有47 –54 %的剂量增加，及照野下缘有37.6 %- 35.2 %的剂量减少，相对照於静止态下照野边缘为中心点50 %剂量的状态。
-
The accumulated self-exciting dose for Na-24 is only 1.12 mGy during its average life-time for irradiation of total thermal neutron fluence of 3.6E12 nth/cm^2. Conclusions: Because of the self-exciting dose reduced from neutron activation can be neglected for gamma dose measurement in neutron and gamma mixed field, and the threshold of linear response is 2 Gy, the FWT-70-40 dosimeter not only can be used for high dose rate Co-60 irradiation field and the high energy electron beam for medical purpose, but also can be used for gamma dose measurement in an epithermal neutron beam for boron neutron capture therapy.
型号FWT-70-4OM）剂量计於BNCT高中子通量与剂量的中子、加马混合辐射场所产生的自激剂量，对加马剂量度量干扰可忽略，且对加马的最低剂量限值为2Gy，因此除可应用於钴60加马高剂量辐射场与放射治疗用高能量、高剂量电子射束的剂量度量外，亦可应用於BNCT照射治疗用高能量、高剂量的中子、加马混合辐射场的加马剂量度量。
- 更多网络解释与剂量相关的网络解释 [注：此内容来源于网络，仅供参考]
-
dose rate meter：剂量率计
dose-rate dosimeter 剂量率剂量计 | dose-rate meter 剂量率计 | dose-rate monitor 剂量率监测器
-
local dose rate：局部剂量率;当地剂量率
局部剂量 local dose | 局部剂量率;当地剂量率 local dose rate | 区域落尘 local fall-out
-
dose equivalent：剂量当量
单位是伦琴(R),1R=2.58×104C/公斤;(4)辐照剂量率(exposure ra-te):系指单位时间内的照射剂量;(5)剂量当量(dose equivalent):基于辐射防护目的,把不同射线的校正系数和在受同位素内照射时的体内分布系数与吸收剂量相乘之积以rem表示即为剂量当量;
-
threshold dose：低限剂量
低限剂量(threshold dose)是指受辐射照射者所受的剂量在此值下无显著的作用发生,换句话说,所受的剂量在低限剂量之下,将不会造成辐射伤害. 假若任何的辐射剂量都会产生一些伤害,更假设此伤害无法恢复,那将没有低限剂量存在,
-
neutron dosimeter：中子剂量仪
neutron dose 中子剂量 | neutron dosimeter 中子剂量仪 | neutron dosimetry 中子剂量测定法,中子剂量定法
-
radiation dosimetry：辐射剂量测定法,辐射剂量测量学
radiation dose 辐射剂量 | radiation dosimetry 辐射剂量测定法,辐射剂量测量学 | radiation excitation 辐射激发
-
neutron dosimetry：中子剂量测定法,中子剂量定法
neutron dosimeter 中子剂量仪 | neutron dosimetry 中子剂量测定法,中子剂量定法 | neutron drop 中子滴
-
LD：致死剂量
致死剂量(LD)乃笼统地表示一化学物对实验动物引起死亡的剂量. 引起死亡的浓度称致死浓度(LC). 此剂量或浓度的在最小致死量(MLD)或(MLC)与绝对(100%)致死量(LD100或LC100)之间. 引起起90%实验动物死亡的剂量(浓度)则用LD90(LC90)表示之;
-
dose protraction：连续弱剂量;剂量延伸
剂量修饰因数 dose modifying factor | 连续弱剂量;剂量延伸 dose protraction | 剂量率 dose rate
-
dosis：剂量 服药量 一次剂量 一剂
dosiology剂量学 | dosis剂量 服药量 一次剂量 一剂 | dosistolerata耐受量 耐药量