分泌神经元

基本解释 (translations)

adenoneure

与分泌神经元相关的网络例句 [注：此内容来源于网络，仅供参考]

These results suggest that the activate spleen-energy method can achieve appetitive purpose by not only affecting the gastrointestinal tract function in periphery, but also regulating the process of ingestion in brain. The basic research about ingestion indicate: one hand, after food enters the gastro-intestinal tract, the endocrine cell of the intestinal tract is activated and secrete many kinds of brains intestines peptide, the level of the brain intestines peptide in blood as the periphery signal spreads to the central nervous system regions that control digest and feeding behavior. On the other hand, it has been shown that dorsal parabranchial neurons, containing CCK-8S, extend fibers to the VMH and are involved in the inhibition of feeding .

关于摄食控制的基础研究表明，一方面食物进入胃肠道后，激活肠道的内分泌细胞，分泌多种脑肠肽如CCK-8S，这些脑肠肽在血中的水平作为外周信号可通过"肠—脑轴"传入中枢神经系统介导消化和摄食行为的部位，影响摄食中枢神经元的活动调节动物的摄食行为；另一方面中枢神经元可合成释放内源性的神经肽直接作用于摄食中枢神经元调节摄食，如中枢鳃旁体神经元的神经纤维就可延伸到达VMH，并且合成释放内源性的CCK-8S。

The complexus of X organ and sinus gland was the main part of the neurosecretory of eyestalk.The X organ,which contained type I～VI neurosecretory cells,was located optic ganglion and was single nerve cell.

结果表明，克氏原螯虾眼柄神经内分泌系统主要组成部分为X器官-窦腺复合体。X器官分布在视神经节上，由I~VI型6种神经分泌细胞构成，均为单级神经元。I~VI型分泌细胞类型及数量在不同生长期存在差异。

Results After oral administration of ST pinocytosis vesicles began to increase in endothelial cells of blood vessel in 1 hour,apocrine of ependymal cells began to increase and myelin sheath degeneration appeared in 2 hours,extensive myelin sheath degeneration and mitochondrion degeneration and incisure and vacuoles in nucleus of neuron were found in 4 and 8 hours.

结果 ST灌胃后1 h血管内皮细胞吞饮囊泡开始增多，2 h组室管膜细胞的顶浆分泌增多，部分神经髓鞘变性；4 h和8 h组神经纤维广泛变性，神经元线粒体变性、核切迹和空泡出现；16 h组神经元胞浆内线粒体变性更加明显，并出现脂褐素。

Many studies reported suggest both neuronal precursor cells before innervation of target fields and the neurons of adult animal coexpress neurotrophic factors and their receptors, implying that autocrine and paracrine modes of NTFs exit. It has been observed that a number of neurotrophic factors and their receptors were expressed in cultured hippocampal cells and dorsal root ganglionic cells.

研究表明，无论是靶组织联系建立以前的神经元前体细胞，还是成年后的神经元，均有神经营养因子与其受体的共同表达，提示有神经营养因子自分泌和旁分泌存在的可能性。

These results suggest that the activate spleen-energy method can achieve appetitive purpose by not only affecting the gastrointestinal tract function in periphery, but also regulating the process of ingestion in brain. The basic research about ingestion indicate: one hand, after food enters the gastro-intestinal tract, the endocrine cell of the intestinal tract is activated and secrete many kinds of brains intestines peptide, the level of the brain intestines peptide in blood as the periphery signal spreads to the central nervous system regions that control digest and feeding behavior. On the other hand, it has been shown that dorsal parabranchial neurons, containing CCK-8S, extend fibers to the VMH and are involved in the inhibition of feeding .

关于摄食控制的基础研究表明，一方面食物进入胃肠道后，激活肠道的内分泌细胞，分泌多种脑肠肽如CCK-8S，这些脑肠肽在血中的水平作为外周信号可通过&肠—脑轴&传入中枢神经系统介导消化和摄食行为的部位，影响摄食中枢神经元的活动调节动物的摄食行为；另一方面中枢神经元可合成释放内源性的神经肽直接作用于摄食中枢神经元调节摄食，如中枢鳃旁体神经元的神经纤维就可延伸到达VMH，并且合成释放内源性的CCK-8S。

These results can lead to the following conclusion:〤GRP synthesized by the motoneurons may be a self-serving neurotrophic factor after axotomy.(2) CGRP may serve as a "injury signal" activating both the central and peripherical gli

本研究结果提示，运动神经轴突损伤后，运动神经元合成分泌的CGRP对神经元自身具有直接的神经营养作用，CGRP还可以作为损伤神经元分泌的&损伤信号&激活中枢和外周胶质细胞，从而为运动神经元的存活和轴突再生创造有利的微环境。

Kangdai Ⅰ has protective function to the damaged neurons and astrocytes: Main results:(1) It has direct protective function to the damaged neurons. It can increase the activity and survival rate, decrease the mortality and the transudation rate of LDH in cultured medium and the strong positive cell count of NOS expression of injured neurons.(2) It has also directly protective function to the damaged astrocytes. It can increase the activity and survival rate and protein content in conditioned medium.(3) It can strengthen the ability of BDNF, GDNF, bFGF, HSP and IL-6expression in damaged astrocytes.(4) It can also strengthen obviously the expressions of NSE, bFGF-receptor and bc1-2, lower the expression of bax and caspase-3.(5) It can indirectly protect and restore the damaged neurons by astrocytes. Because the effect of ACMK (ACM interfered by Kangdai Ⅰ) is stronger than ACM+K (ACM associated with Kangdai Ⅰ).

抗呆Ⅰ号对受损的神经元和星形胶质细胞均具有保护作用：主要表现为：(1)对受损神经元具有直接的保护作用，可提高受损神经元的活性和存活率，降低细胞培养液LDH的漏出率、细胞死亡率和NOS染色强阳性细胞的表达量；(2)对受损的星形胶质细胞也有直接的保护作用，可提高其活性、存活率以及培养液蛋白质的含量；(3)能增强受损星形胶质细胞分泌BDNF、GDNF、bFGF、HSP和IL-6的能力；(4)可明显增强受损神经元对NSE、bFGF的受体和bc1-2的表达，降低受损神经元对bax和caspase-3的表达；(5)抗呆Ⅰ号可通过星形胶质细胞间接地保护和修复受损的神经元，因为在多数实验组中经抗呆Ⅰ号作用的ACM的作用远大于ACM与抗呆Ⅰ号联合应用的作用，统计学上具有显著性差异。

Type II cells were mainly distributed in layer V of cortex. It appeared that these cells receive the CRF immunostaining fibers terminals afferent from other cells rather than synthesize CRF by themselves. It is probable that type II cells receive afferent fibers from both extracortex and type I cells within layers II -III cortex. Considered that Type I cells morphologically are inhibitory interneuron, we presumed that type I in layer II -III could inhibit activity of type II cells in layer V.

CRF阳性Ⅱ型细胞主要分布在皮层第Ⅴ层，形态学特征显示这类细胞更像是接收来自其它神经元的CRF纤维投射而非自身分泌CRF，证据显示Ⅱ-Ⅲ层CRF神经元纤维可进入第Ⅴ层，考虑到皮层的CRF神经元形态上为抑制性中间神经元，这些结果提示Ⅱ-Ⅲ层CRF神经元能够抑制第Ⅴ层神经元的活动。

It is widely known that during the development of nervous system, the synapse between neurons and target tissues plays an important role in the survival and maturation of neurons because target tissues or target cells can produce and secrete many kinds of neurotrophic factors and neuroinductive factors which can promote the growth, differentiation and migration of innervating neurons.

在神经系统发育中，神经元与相应靶组织建立的突触联系，是神经元存活和分化的关键因素，因为靶组织或靶细胞可以合成、分泌多种神经营养因子和神经诱向因子，促进支配神经元的生长、分化和迁移。

HCRT potentiates NMDAR-mediated somatic release from LC neuronsApplication of 100μM NMDA evoked a pronounced secretion.This response to NMDA was subsequently enhanced by treatment with 100 nM HCRT for 5 min.

HCRT增强LC神经元NMDA受体介导的胞体递质分泌给予100μM NMDA可以显著引起分泌信号，随后用100 nM HCRT处理5 min，再次给予100μM NMDA可引起更大的分泌信号(增加大约40%)。

与分泌神经元相关的网络解释 [注：此内容来源于网络，仅供参考]

contact area：接触面积

与控制组相比较,移植多巴胺神经元八周后,多巴胺神经元分泌多巴胺进而改善鼠脚掌压力强度 (intensity)、脚掌接触面积 (contact area)、四肢脚掌跨越速度(swing speed)以及减少了后肢脚掌的相对距离.

neuroendocrine cell：神经内分泌细胞

下丘脑神经内分泌细胞(neuroendocrine cell)是指下丘脑具有内分泌功能的神经元. 其分泌的激素称为神经激素. 本世纪60年代初Halasz提出在下丘脑基底部存在下丘脑促垂体区(hypophysiotropic area),主要包括正中隆起、弓状核、视交叉上核、腹内侧核、视周核等.

we've recorded：唔 我们发现 恋爱的时候

would say things like,|会说 | "Well, as we fall in love, we know, we've recorded|"唔 我们发现 恋爱的时候 | that there's additional serotonin release in neurons in the brain.|大脑的神经元开始分泌更多的...

encapsulated nerve ending：有被囊神经末梢

2.有被囊神经末梢 有被囊神经末梢(encapsulated nerve ending)外面均包裹有结缔组织被囊,它们的种类很多,常见的有如下几种:运动神经末梢(motor nerve ending)是运动神经元的长轴突分布于肌组织和腺内的终末结构,支配肌纤维的收缩和腺的分泌.

neurolemma tube：神经膜管

神经微管neurotubule | 神经膜管neurolemma tube | 神经元分泌neurocrine

merocrine gland：局浆分泌腺

局浆分泌merocrine | 局浆分泌腺merocrine gland | 局部环路神经元local circuit neuron