- 更多网络例句与分化异常相关的网络例句 [注：此内容来源于网络，仅供参考]
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The expression of Filaggrin was limited to stratum corneum in normal epidermis.
在扁平苔藓皮损中角质形成细胞存在着明显的增生和分化异常。2。
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Cases of the abnormal sex differentiated diseases in children were presented. Among them, there were 4 cases of Turner's syndrome, 1 Klinefelter's syndrome, 9 female pseudohermaphroditism, 2 male pseudohermaphroditism, and 4 true hermaphroditism.
本文报道小儿性分化异常疾病20例(包括先天性卵巢发育不全综合征4例、先天性睾丸发育不全综合征1例、女性假两性畸型9例、男性假两性畸型2例、真两性畸型4例)，对上述病例进行了细胞染色体核型分析，同时作了口腔上皮细胞性染色质、中性粒细胞核鼓植体及尿17酮类固醇、17羟类固醇的检查。
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This article mainly discusses the relationship of abnormal adipocyte differentiation to IR as well as its effects on NAFLD.
本文主要讨论脂肪细胞分化异常与IR的关系，及其对NAFLD的影响。
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Role of adipocyte disdifferentiation in insulin resistance and nonalcoholic fatty liver disease.
脂细胞分化异常与胰岛素抵抗的关系及其在非酒精性脂肪性肝病中的作用。
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This result indicates that WT1 gene plays an important role in differentiation and development of fetal kidney and may be the factor that promotes metanephric blastemal cell to differentiate into epithelial cell.
结果显示小胎龄肾组织中WT1蛋白在胚基细胞和幼稚肾小球细胞核表达而大胎龄组肾组织中WT1在肾小管细胞胞浆表达，阳性率分别为57.1％(8/14)和46.2%(6/13)，提示WT1基因在胚胎肾分化发育的过程中起着重要作用，WT1蛋白可能是促进后肾胚基细胞向上皮细胞分化的调控因子，其表达在时间上和空间上都受到严格的调控，WT1的表达异常可能导致胚基细胞分化停滞。17例肾母细胞瘤WT1蛋白表达阳性率为41.2%(7/17)，阳性部位在胚基型和上皮型肿瘤细胞核，表达部位和阳性率与早期胚胎肾相似，其中间质型肾母细胞瘤均为阴性，胚基型和上皮型肾母细胞瘤阳性率70%(7/10)，两组间阳性率有显著差异。
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Results: CK18 was not detected in normal and abnormal oral tissue sections. But in normal nonkeratinized mucosa, CK19 was detected in the basal cell layer dispersively. In epithelial dysplasia, CK19 was detected in the suprabasal cell layer and the number of CK19-positive cell layers was correlated with the dysplasia degree of epithelia. Furthermore, CK19 was detected in oral squamous cell carcinoma, especially in the poor-differentiated cancer cells.
结果：正常和异常的口腔粘膜均未检测到CK18；CK19可表达于正常非角化上皮的基底层细胞、异常增生上皮的基底上层细胞及口腔鳞状细胞癌中，阳性细胞数与上皮的异常增生程度及鳞癌的分化程度相关。
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Ultrasonographic scans, fetal genetic studies, and hormonal assays of amniotic fluid can diagnosis certain diseases, fetal sex differentiation disorders, fetal endocrinal disorders, and chromosome abnormality.
产前超声发现胎儿生殖器官异常可作为筛查指标之一，联合羊水中激素检查可早期诊断某些胎儿生殖器官异常、胎儿性分化异常、胎儿内分泌代谢异常、胎儿染色体异常相关疾病。
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Yelodysplastic syndromes,as a disease of dyspoiestichematopoietic stem cell,have a disturbance of DNA synthesis,cellsdifferentiation and some leudemic characterization.It's marrow cell displaylike megaloblastic change,so we postulated that folate receptor have adifference of both quantitative and quanlititive in megalolbastic anemia,MDSand lenkemia.
DS作为异常干细胞克隆性增生疾病，在DNA的合成及细胞分化异常，表现出白血病的某些特征和骨髓细胞具有不同程度的巨幼变，因而我们推测巨幼贫、白血病和MDS的叶酸受体不但有量的改变，而且有质的变化。
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Cytological observations of the anther development of pms15-16-2-3 mutant and the wild-type plant showed that the mutant middle layer degenerated later, both the tapetal cell morphology and the tetrads were abnormal, which resulted in few fertile pollen grains formed in the anther.
细胞学观察表明，突变体在花药发育的过程中，中层细胞延迟降解，绒毡层细胞形态分化异常，出现异常的四分体，导致最终只能形成少量的花粉。
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In this thesis, we find that dlg is indispensible in the establishment of anterior-posterior and dorsal-ventral polarity of drosophila oocyte. Removal of Dlg function from the posterior follicle cells using the FLP/FRT system leads to disruption of oocyte skeleton reconstruction that is elicited by the failure of those posterior cells to differentiate normally in mid-oogenesis. We demonstrate that abnormity of Notch, JAK-STAT and EGFR signal pathway in dlg mutants contributes to this aberrant differentiation. dlg null mutant also blocks the normal differentiation of two groups of anterior follicle cell-stretched cell and centripetal cell, but not border cell, with a lower penetrance. However unlike the result in posterior follicle cells, Notch and JAK-STAT signaling are both undisrupted in all mutant anterior follicle cells, implying other fate determinants may be involved.
我们的研究发现，后端滤泡细胞中的Dlg在果蝇卵子发生中期卵母细胞前后轴和背腹轴建立过程中也是必须的，PFC中dlg完全缺失型突变引起PFC的分化异常，导致卵子发生中期卵母细胞骨架重组异常，Stau、Grk等极性决定蛋白定位错误。dlg突变阻碍了Notch、JAK-STAT、EGFR等调节PFC分化的信号通路的激活。dlg突变的PFC也没有获得前端滤泡细胞命运。dlg突变不影响前端滤泡细胞群中边界细胞的分化，但是在一定程度上影响伸展细胞和向心细胞的分化，并且这种影响不依赖于前端滤泡细胞Notch或JAK-STAT信号激活的异常。
- 更多网络解释与分化异常相关的网络解释 [注：此内容来源于网络，仅供参考]
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biosynthesis：生物合成
表型上,这些男性是毫无疑问地最常见,且有潜力变得最严重的性别分化异常被证实是在具有皮质醇(cortisol)生物合成(biosynthesis)酵素缺陷的 46,XX 个体(38, I 17).
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social deviance：社会异常
social development of children 儿童社会性发展 | social deviance 社会异常 | social differentiation 社会分化
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hindbrain：菱脑
视黄醇类如VitA可激活这些基因中的某些,对正常的生长及组织分化都很重要(Soprano1995)致畸的遗传及生理机制-同源基因此机制与菱脑(hindbrain)与肢芽(limb buds)的异常有关丙戊酸可激活在近5\'终端调节头颅骨的形态的同源性基因,
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neoplasia：肿瘤形成
其形成过程,被称为肿瘤形成(neoplasia). 肿瘤细胞具有异常功能、代谢和结构,以及与机体不相协调的过度生长能力和分化不完全等特点. 肿瘤分为良性与恶性两大类. 良性肿瘤生长较慢,具有一定自限性,与周围组织分界明显,危害相对较小;
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seminiferous tubule：细精管
性腺组织必须表现出卵巢滤泡与细精管(seminiferous tubule). 透纳氏症(Turner's syndrome)的病人表现出性别分化异常,乃是因为一性染色体缺失症候群,其结果导致异常性腺发育(见表 16.1). 在这些病患,卵巢刚开始发育时拥有正常的卵母细胞(oocyte)存量(14).
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cecidology：蟲癭學
由于寄生昆虫的种类不同,在寄主上产生的虫瘿形伏也不同,所以可借此鉴别寄生昆虫的种类,这种鉴别一般称为虫瘿学(cecidology). 据认为这是由于寄生昆虫所产生的物质和侵入引起的伤害促进了细胞的分裂,招致增生和分化异常的结果.
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mononuclear cell：单一核细胞
1 银屑病的发病机理 银屑病的主要病理表现为角化不全、棘层增生、真皮RT血管扩张和单一核细胞(mononuclear cell)浸润. 因此,多年来人们一直认为角质形成细胞的异常增生和分化不全是银屑病的发病关键,并据此应用甲氨喋呤、PUVA、维A酸类等治疗并取得明显效果,
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prosopectasia：巨面
prosopalgia 三叉神经痛 | prosopectasia 巨面 | prosoplasia 分化异常
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prosoplasia：分化异常
prosopectasia 巨面 | prosoplasia 分化异常 | prosopodiplegia 两侧面瘫