- 更多网络例句与内皮细胞层相关的网络例句 [注：此内容来源于网络，仅供参考]
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More recently, it has become clear that endothelial desquamation is not common and that an intact endothelial cell layer covers developing atherosclerotic lesions.
最近，可以确定的是内皮剥落并不常见，而且，完整的内皮细胞层会覆盖住正在形成的动脉粥样硬化病灶。
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HBV transplacental transmission route may be realized by cell to cell transfer in placenta, which means that HBVs in maternal blood and/or intervillous space infect DCEC and/or DC and/or TC, then transmit to VMC, then to VCEC, and finally to fetus.
HBV经胎盘感染胎儿的路径可能是通过胎盘细胞至细胞的传递而实现的。即母血和/或绒毛间隙HBV→蜕膜毛细胞管内皮细胞和/或蜕膜细胞和/或滋养层细胞→绒毛间质细胞→绒毛毛细血管内皮细胞→胎儿。
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Vascular endothelium plays an important role in regulating the transendothelial migration of polymorphonuclear leukocytes.
中文摘要血管内皮细胞层在调节白血球能否穿过血管进入组织的过程中扮演著重要的角色。
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Vascular endothelium plays an important role in regulating the transendothelial migration of polymorphonuclear leukocytes.
血管内皮细胞层在调节白血球能否穿过血管进入组织的过程中扮演著重要的角色。
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This hindrance is thought to be due to limited proliferative capacity of the endothelial layer.
产生这一障碍的原因是内皮细胞层的功能受到抑制或内皮细胞的增殖受到抑制而无法修复损伤。
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These include physical characteristics, such as repulsive forces resulting from the electronegative charge of both platelet and endothelial cell membranes, and biochemical attributes, such as the synthesis and release of inhibitors of platelet activation. Platelets respond rapidly to alterations of endothelial cells and to exposure of subendothelial structures by attaching firmly to the site of the lesion.
正常情况下，循环中的血小板与血管壁内皮细胞层紧密接触但不发生黏附，是因为血管内皮细胞具有一定的活性功能，包括一些物理特性如血小板和内皮细胞膜均带有负电荷产生相互排斥作用以及生化作用如合成和释放血小板活化抑制剂。
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The entire process of PMN rolling on, adhering to, and transmigrating across the EC monolayer was recorded under both phase-contrast and fluorescence optics.
我们发现当单一PMN开始穿过内皮细胞层，会引发紧邻的内皮细胞 [Ca2+]i上升，但对其附近未直接接触此一PMN的内皮细胞，则没有影响。
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VE-cadherin moved away from the transmigration site to different ends of it, whereas PECAM-1 opened to surround the periphery of a transmigrating PMN.
我们发现在PMN穿过内皮细胞层时，这两种连接体蛋白都不会突然消失，而是发生了渐进的相对移动--相邻内皮细胞间原本彼此接合的VE-cadherin会沿著细胞间的界线移向PMN两端；而PECAM-1的变化则是打开原本彼此间的键结，改而围绕在PMN周边。
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Some cell dropped into the cavity and became free. Thrombosis or part organization could be seen. The internal elastic layer became thin, disappear or broken. In internal and middle layer existed fibroblasts, fibrocytes and collagen. Some of the wall indicated hyaline change, soomth muscle cell decreased greatly. The massive inflammatory cells invaded the middle and external layer. There were many foam cells in the capsule tissue. Cytoplasm was filled with fatty tissue and cholesterol. some cavities were full of thrombosis. Some thrombosis was fibrosis, the bottom was organization. The surface of the thrombosis existed red blood cell and librae.(2)Elatic fibrila staining: the internal elastic menbrane almost completely disappeared, the intact internal elastic menbran could be seen in the new small vessels.
动脉瘤管壁厚薄明显不均，全层或局部区域显著变薄向外膨出，内皮细胞空泡变性或坏死脱落，部分内皮细胞剥离并突入管腔成游离状，可见血栓形成及部分血栓机化；内弹力板变薄、消失或突然中断；在内膜及中膜部位主要为纤维母细胞、纤维细胞和大片胶原；部分动脉瘤壁呈均质状玻璃样变，平滑肌细胞明显减少；中膜和外膜可见大量的炎性细胞浸润；瘤壁组织有纤维母细胞、纤维细胞、大片胶原成分及较多泡沫细胞，胞浆内充满脂类物质及胆固醇结晶；部分动脉瘤腔内充满血栓，有的血拴已经纤维化，血栓基部机化，血栓表面有红细胞和纤维素。
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The morphology and structure of reconstructed tissue was detected by microscope and scanning electron microscope.Results:(1) Compared with the control group, the cellular proportion of laminin group increased in 62 ~M phase, and decreased in Go~Gi phase significantly. As shown by the microscope, the cells of control group were in low density. The cells in mass connected tightly. The microfilament appeared reticular formation. The nucleus were the same in size. The cells of laminin group were in high density and put out so many lamellipodia, filopodia, which connected with the surrounding cells. The microfilament increased, elongated, and changed from reticulodromous to sarciniform, which reached to the pseudopods. The nucleus were different in size .(2) As shown by the inverted microscope and the cell growth curve, comparing with the controlgroup, cells of each test group increased evidently. The cellular proportion of each test group increased in S phase and G2 ~M phase, and decreased in Go~Gi phase significantly, but there was no considerable interations between LN and EGF;(3) As shown by the morphological observations, the cultured cat corneal endothelial cells formed an integrated membrane, and attached to the Descemets membrane closely, which was similar to the natural tissue. The cells connected tightly to each other, and some of them arranged in hexagon approximately.
结果：(1)层粘连蛋白组处于G_2～M期细胞比例较对照组显著提高，Go～G_1期细胞比例显著下降，提示层粘连蛋白促进内皮细胞DNA合成，及细胞分裂增殖；光镜下，对照组细胞分布成团状，细胞密度较低，细胞间连接紧密，细胞内微丝结成网状，细胞核大小一致；与对照组相比，层粘连蛋白组细胞生长旺盛，细胞密度高，向周边伸出大量板状及丝状伪足，细胞内微丝增多、拉长、集结成束，伸入伪足中，细胞核形状大小不一致；(2)倒置显微镜观察及细胞生长曲线显示，各组细胞数目随时间增加而明显增多，各实验组较对照组增生显著，EGF和LN联合应用组各时间点细胞数目最高；实验组处于S期和G_2～M期细胞数目增加，Go～G_1期细胞数目减少；提示EGF、LN单独及联合应用均可促进细胞增殖，但尚不能认为二者有交互作用；(3)倒置显微镜下，组织培养的猫角膜内皮细胞排列成密集的单层，细胞间连接紧密；组织学观察发现，培养的猫角膜内皮细胞形成完整的内皮层，贴附于脱水基质的后弹力膜上，与正常的角膜内皮组织结构相似；扫描电镜下，组织培养的猫角膜内皮细胞间紧密镶嵌排列，可见某些细胞呈近似六边形排列，细胞大小不甚一致，胞核清晰。
- 更多网络解释与内皮细胞层相关的网络解释 [注：此内容来源于网络，仅供参考]
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endoderm, entoderm：内胚药,内胚层
细胞内异物 endocyte | 内胚药,内胚层 endoderm,entoderm | 内皮 endodermis
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endothelium：内皮细胞层
④后弹力层 Descemet's membrane | ⑤内皮细胞层 Endothelium | (2)巩膜 Sclera
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endotherm：吸热线
endothelium 内皮细胞层 | endotherm 吸热线 | endothermalendothermic 吸热性的
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fenestra：窗孔
正常肝血窦没有基底膜,在结节多步癌变过程中,血窦基底膜出现,并且内皮细胞间的窗孔(fenestra)减少或消失,内皮细胞的抗原表达也发生一定程度的改变. CD34、CD31等内皮细胞抗原及Coll Ⅳ、层粘连蛋白Lam等基底膜标记物表达常常增强.
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Harmonic mean：调和均数
测定GBM厚度的方法目前有算术均数(arithmetic mean)和调和均数(harmonic mean)法二种. 算术均数法需选择适宜测量的GBM节段摘要:同时见到内皮细胞窗孔紧贴GBM内层呈线状分布和上皮细胞足突间隙,此目的是为避开正切面(tangential cuts),
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hydration：水化(作用)
(1)角膜混浊前色素膜的长期炎症,几乎都会引起后弹力层皱褶.当炎症累及角膜内皮细胞,破坏角膜水化作用(hydration)则引起角膜上皮水肿,出现大泡和小泡样改变,持久性水肿会引起血管翳和角膜实质层周边血管新生.当角膜内皮细胞严重水肿和广泛脱落时,
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tunica intima：内膜
(1)内膜 内膜(tunica intima)是管壁的最内层,由内皮和内皮下层组成,是三层中最薄的一层. 1.内皮为衬贴于血管腔单层扁平上皮. 内皮细胞长轴多与血液流动方向一致,细胞核居中,核所在部位略隆起,细胞基底面附着于基板上. 电镜观察,
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monolayer：单层
模孔增大,原具有屏障作用的细胞脂层被剥脱,其通透性增加,当内皮毒素等有害物质进入血液循环后,首先损伤肺泡毛细血管内皮细胞(pulmonary cpillary endothelial cells,PCEC),使内皮细胞单层(monolayer)渗透性增加,收缩,死亡,
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myosin：肌浆球蛋白
表12-1 软组织细胞可用的标记抗体 抗原名称 标记细胞 (1)角蛋白(keratin) 上皮与肌上皮细胞 (2)波纹蛋(vimentin) 间叶分化细胞、内皮细胞 (3)结蛋白(desmin) 肌组织分化细胞 (4)肌动蛋白(actin) 肌细胞,肌上皮细胞,肌纤维 (5)肌浆球蛋白(myosin) 肌母细胞,部分内皮细胞及肌细胞 (6)层粘蛋白(laminin) 平滑肌与雪旺氏细胞,
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mesenchymal cell：间叶细胞
Tripethi认为中央部角膜的缺陷是由于在胚胎10~14mm时,视杯边缘第一和第二间叶细胞(mesenchymal cell)停止向内生长;角膜后弹力层异常和内皮细胞的丧失,是由于原发性间叶细胞的缺陷,这一缺陷可能进一步受到晶状体泡分离延迟或分离不全的影响;