其内皮

基本解释 (translations)

soapbark

与其内皮相关的网络例句 [注：此内容来源于网络，仅供参考]

Objective: To investigate the possibility of pathologic scars treated in endostatin , by observing the effects of endothelial cell and neogenesis capillary of pathologic scars treated in endostatin , through establishing models of hyperplastic scar in rabbit ear and injecting Endostatin .

目的：建立兔耳增生性瘢痕的动物模型，并采用内皮抑素局部在体干预，观察其对病理性瘢痕新生毛细血管及血管内皮细胞的影响，以探讨内皮抑素防治病理性瘢痕的可行性。

The transter and expression of endostatin gene was analyzed by PCR, the effects of inhibition on proliferation of endothelial cell by endostatin in vitro by MTT method, and the effects on the growth of LoVo、 SGC-7901 in vitro was surveyed, and the growth of expressing endostatin LoVo、SGC-7901 cell line_in subcutaneously of nude mice was also measured.

用MTT法检测内皮抑素对内皮细胞体外增殖的抑制作用，并观察其对LoVo细胞和SGC-7901细胞体外生长的影响，观察表达内皮抑素基因的LoVo和SGC-7901细胞在裸鼠皮下种植的生长。

Wenxin capsule can promote endolethial cells releasing CO and synthesing 〓, enhance activity of HO, induce angiectasis through activating guanylic cyclase and adenglate cyclate of intracellar of vessel smooth muscle cell to enhance content of intracellar cGMP and AMP. 3. Wenxin capsule can inhibit releasing of platelet activiting factor 〓 which inhibit platelet aggregation and have antithrombosis function. 4. Wenxin capsule can promote activity of t-PA, reduce activity of PAI-1, strengthen fibrinolytic function, therefore inhibit fibrin disposition. 5. Wenxin capsule can inhibit synthesis of smooth muscle cellular DNA. Its function don't kill it directly but prevent transformation from period Go to peried S. 6. Wenxin capsule is an effective prescription of anti-coronary atherosclerosis atherosis. The results of image pattern analysis tastifies that area of lipid plaque in treating group reduce 54% and 58% in contrast with that in control group. The results of light, electro- microscopy show that structure of endolethial cell and smooth muscle cell in treating group is more integral than that in control group.

其作用机制之一是由于该药具有良好的调脂降脂作用，其不仅能明显降低血清LDL-c及升高HDL-c含量，且能有效阻止TC沉积于血管壁，从而保护内皮细胞结构完整；此外，温心胶囊尚具有明显的抗氧化作用，该药不仅能明显降低血浆LPO含量，且能提高SOD活性，同时其对血红素在细胞内代谢的调节作用也参与了抗氧化过程。2 温心胶囊能明显促进内皮细胞释放CO及合成〓，提高HO活性，通过激活血管平滑肌细胞内鸟苷酸环化酶及腺苷酸环化酶引起细胞内cGMP、AMP浓度增加而引起血管舒张。3 温心胶囊能明显抑制血小板激活因子〓的释放，从而抑制血小板聚集，抗血栓形成。4 温心胶囊能明显提高t-PA活性，降低PAI-1活性，增强纤溶功能，从而抑制纤维蛋白沉积。5 温心胶囊能明显抑制平滑肌细胞DNA合成，其抑制作用不是直接杀伤抑制，而是阻止细胞由Go期向S期转化。6 温心胶囊是抗冠状动脉粥样硬化的有效制剂，图象分析结果证实，该药治疗组脂斑面积比对照组缩小了54％和58％，光电镜结果显示内皮细胞及平滑肌细胞结构较为完整。

The hypercholesterolemic rats were selected to imitate early stage of human atherosclerosis The effects and mechanism of n-3 PUFA on endothelium were investigated with the following parameters: the morphological appearance and arrangement of endothelium stained by Ag, in vitro endothelium-dependentrelaxation of aortic ring, endothelial permeability test using Even′s Blue, blood lipid and body weight in one-week intervals, excitative and emissive spectrum of RBC membrane labeled by DPH (1, 6-Diphenyl-1, 3, 5-hexatriene), fluidity of RBC membrane, plasma and RBC membrane LPO

在整体和离体水平，选用大鼠高脂血症模型模拟动脉粥样硬化早期状况。用114只大鼠多指标着重研究n-3多不饱和脂肪酸在高脂血症状态下对内皮的保护作用，以探讨其预防动脉粥样硬化的机理。所采用指标包括：浸银法观察内皮细胞排列与形态；离体血管环观察内皮依赖性舒张；伊文氏蓝染料法观察内皮通透性。为探讨作用机理，进行了血脂和体重的动态监测、DPH(1，6-二苯基-1，3，5，乙三烯)标记红细胞膜的荧光光谱测定、红细胞膜流动性测定、血浆和红细胞膜脂质过氧化物测定。

The index of left ventricle pachynsis was lowered after treatment in both groups. The decrease of that in the treating group and diastolc function mprovement were more obvious (P.01). Conclusion: Bu Shen He Luo Fang has explicit hypotensive effects and it can depress ventricle remodeling.One of the mechanisms,we conjectured,are maybe local externalization of HGF.

&补肾和络&细颗粒剂具有明确的降血压及抑制高血压左室重塑的作用，推测其机制之一是通过改变局部HGF的分泌实现的，即通过促进组织HGF分泌，恢复其自身修复能力而逆转心室重塑，发挥其心血管保护作用；同时改善内皮功能，维护心肌组织与内皮的稳定性。

These indicated that endothelial cells of the mural coronary artery are free from atherosclerosis due to higher shear stress caused by myocardial bridge. The intima of the section proximal to mural coronary artery was likely to be sclerosed due to the lower and oscillatory shear force.

提示 壁冠状动脉内皮细胞受到心肌桥压迫导致血流切变力增高产生适应性反应，对内皮细胞有保护作用，而其近段内皮细胞由于切变力较低容易被损伤，成为动脉粥样硬化发生的基础。

Our results indicated that (1)SO_2 derivatives could dose-dependently relax the isolated aorta rings with or without endothelium precontracted by NE, no difference was found for the rings with and without endothelium;(2)Levels of cAMP and 6-Keto(PGI_2) in isolated aortic rings were increased by SO_2 derivatives in a dose-dependent way, which were independent of endothelium;(3)SO_2 derivatives decreased cGMP levels in the rings, but not significantly; The decrease of cGMP levels was also independent of endothelium;(4)No changes of TXB_2 in rings with or without endothelium were observed;(5)Whether presence or absence of endothelium, cAMP/cGMP and 6-Keto/TXB_2 ratios were increased significantly.

结果：①SO_2衍生物对去甲肾上腺素预收缩的内皮完整和去除内皮的大鼠胸主动脉血管环均呈浓度依赖性舒张作用，且其张力变化在内皮完整与去内皮的血管环之间无显著性差异；②SO_2衍生物可引起血管组织中cAMP、6-Keto(即PGI_2)含量剂量依赖性增加，其增加程度与内皮无关；③SO_2衍生物对血管环cGMP含量呈降低趋势但在统计学上无显著意义，且对去除内皮血管无显著影响；④SO_2衍生物对去内皮或内皮完整血管环的TXB_2含量均无明显影响；⑤无论是否去除内皮，SO_2衍生物均可引起cAMP／cGMP和6-Keto／TXB_2比值显著升高。

The entire process of PMN rolling on, adhering to, and transmigrating across the EC monolayer was recorded under both phase-contrast and fluorescence optics.

我们发现当单一PMN开始穿过内皮细胞层，会引发紧邻的内皮细胞 [Ca2+]i上升，但对其附近未直接接触此一PMN的内皮细胞，则没有影响。

Results VEGF-C and VEGFR-3 are all expressed on both mesenchymal cells and vascular endothelium of the embryos during early period. In middle and latter period, VEGFR-3mRNA is mainly expressed on cell plasm of alimentary canal, but it has no expression on vascular and lymphatic endothelium; The expression of VEGFR-3 is extensive and positive expression is seen on the vascular and lymphatic endothelium.

在胚胎中、后期消化道组织中VEGF-C主要表达于消化管壁组织细胞以及肠系膜内，在血管和淋巴管内皮细胞则无表达；VEGFR-3亦表现为广泛的表达，于血管和淋巴管内皮细胞可见阳性表达，并随着胚胎的发育，其在淋巴管内皮细胞的阳性表达率明显增加。

Methods Chromosome, HE stainings and autoradiography were made for the cornea of human and monkey.

目的 探讨灵长目动物角膜内皮细胞的再生能力及方式方法对猴及人角膜做染色体染色、HE染色及放射自显影结果成年创伤活体猴角膜内皮细胞有再生能力成年及幼年灵长目动物角膜内皮细胞在生理和病理条件下的再生方式均为无丝分裂结论灵长目动物角膜内皮细胞在一定条件下有再生能力，其再生方式为无丝分裂

与其内皮相关的网络解释 [注：此内容来源于网络，仅供参考]

dysfunction：机能障碍

ed)内皮机能障碍(dysfunction)之证据以肱(brachial)都普勒测量之对内皮功能或脂肪型态(lipid profile)没有影响同时以 DE S 处理以延迟其精子生成(spermatogenesis)高金雀花碱(Genistein)对无大豆饮食之抵销效果(reversed effect)血浆中 FSH 浓度符合精子生成之比例(proportional to spermatogenesis)骨特异(Bone specific)之

endothelial cell：内皮细胞

尽管其作用机制有几种不同类型,但都包括了细胞内Ca2+浓度的升高、钙依赖性钾通道(calcium-activated potassium channels,KCa)的开放及内皮细胞(endothelial cell)的超极化,最终导致了血管平滑肌细胞(smooth muscle cell,

endothelial cell：血管内皮细胞

五,血管内皮细胞 血管内皮细胞(endothelial cell)为衬贴在心血管内皮腔面的单层扁平细胞,大小较一致,为多边形, 细胞及其长轴沿血管长轴排列,其细胞器不太发达.内皮细胞更新较慢,细胞很少发生分裂,内皮损伤 或丧失时,内皮细胞可能经成纤维细胞,

Corneal endothelium：角膜内皮

(5)角膜内皮(corneal endothelium)又称角膜后上皮为单层扁平上皮,细胞具有分泌、合成蛋白质的作用. 内皮损伤时,可由其邻近细胞的扩展进行修复,而不是通过细胞分裂进行修复. 2)巩膜(sclera)主要由致密结缔组织构成,具有保护和支持眼球的作用.

monolayer：单层

模孔增大,原具有屏障作用的细胞脂层被剥脱,其通透性增加,当内皮毒素等有害物质进入血液循环后,首先损伤肺泡毛细血管内皮细胞(pulmonary cpillary endothelial cells,PCEC),使内皮细胞单层(monolayer)渗透性增加,收缩,死亡,

soapbark：皂皮树/其内皮

soaking /使人湿透的/ | soapbark /皂皮树/其内皮/ | soapberry /无患子属植物/其子实/

soapbark：其内皮

soap 胰 | soapbark 其内皮 | soapbark 皂皮树

soapberry：无患子属植物/其子实

soapbark /皂皮树/其内皮/ | soapberry /无患子属植物/其子实/ | soapbox /肥皂盒/临时演说台/街头演说的/

venule：微静脉

1.微静脉 微静脉(venule)管腔不规则,管径50~200μm,内皮外的平滑肌或有或无,外膜薄. 紧接毛细血管的微静脉称毛细血管后微静脉,其管壁结构与毛细血管相似,但管径略粗、内皮细胞间的间隙较大,故通透性较大,也有物质交换功能.

postcapillary venule：毛细血管后微静脉

弥散淋巴组织中有毛细血管后微静脉(postcapillary venule),其特征是内皮为单层立方或矮柱状,故又称高内皮微静脉(high endothelial venule),是淋巴细胞由血液进人淋巴组织的重要通道.