作用周期

词组短语

action cycle

与作用周期相关的网络例句 [注：此内容来源于网络，仅供参考]

To the later, this dissertation has been done transient dynamic analysis, calculated the stress intensity factors changing with the damp of asphalt pavement, the cycle of load action and the parameters of pavement structure.

应力强度因子最大值随荷载作用周期的变化呈倒"S"状变化，在高频和低频荷载作用时，逼近静载作用时的应力强度因子值，在一定的频率出现最大值和最小值，但应力强度因子的变化幅度不大。

Along with the fluconazole solution density rise,the experimental two kind of strain various glucose density is higher,showe d the glucose consumption are less,takes the logarithmof the medicine de nsity,discovered the logarithm the medicine density and each glucose den sity presents the linear relations;Carries on the analysis comparison to under the fluconazole function two kind of strain linear relations,disc overed the relations of the two strains has the nonuniformity.3 Compare the fluconazole induction reaiatance SC5314 strain and sens itive strain compares,its difference gene expression mainly concentrates in:The code proteinase body and the protein hydroltyic enzyme gene,in the code sugar fat metabolism process is connected the protein gene,the cell cycle correlation gene,the duplication and the translation adjustme nt correlation gene,the stress response correlation gene,the line plast ochondria correlation gene,the cell wall function related gene.4 Candida albicans SC5314 induction resiatance strain was processed b y Xianglian solution,its expression change gene mainly is:Code stress re sponse family protein gene,biomembrane relevant gene,a code proteinase body gene race,code cell cycle related protein gene,duplication and tra nslation adjustment related protein gene.5 The clinical reaiatance strain Candida albicans was processed by Xi anglian solution,its expression change gene mainly is:Codes the hot sho ck protein gene,the serine/threonine protein activating enzyme gene,the proteinase body family gene,the regulation copies and translates the ge ne.

随着氟康唑药液的浓度上升，试验的两种菌株各孔葡萄糖浓度越高，说明葡萄糖消耗越少，经过药物浓度取对数后进行分析，发现取对数后的药物浓度和每孔中葡萄糖浓度者呈现线性关系；对氟康唑作用下的两种菌株的线性关系进行分析比较，发现对两种菌株作用具有不一致性。3氟康唑诱导的耐药SC5314菌株与诱导前的敏感株相比，其差异基因表达主要集中在：编码蛋白酶体及蛋白水解酶的基因，编码糖脂代谢过程中相关蛋白的基因，细胞周期相关基因，转录及翻译调节相关基因，应激反应相关基因，线粒体相关基因，细胞壁功能相关基因。4白念珠菌SC5314诱导耐药株经香莲外洗液作用后，其表达变化的基因主要是：编码应激反应家族蛋白的基因，生物膜相关性基因，编码蛋白酶体基因一族，编码细胞周期相关蛋白基因，转录及翻译调节的相关蛋白基因。5白念珠菌临床耐药菌株经香莲外洗液作用后，其表达变化的基因主要是：编码热休克蛋白基因，丝氨酸/苏氨酸蛋白激酶基因，蛋白酶体家族基因，调控转录及翻译基因。

Secondly, we analyzed the interaction between business cycle and finance cycle. The former is the latter's external variable, and the latter is the formers internal variable.

其次，分析财务周期与经济周期之间的关系——互为作用与反作用关系，并且后者是前者的外生变量；财务周期与企业生命周期之间的关系——后者是前者的内生变量，并且具有互为作用与反作用关系。

The theory of accretion disks are important for many astrophysical phenomena, including Quasars, active galatic nucleic, X-ray binary stars, and young stellar objects. The most important branch of accretion djsk theory is the stability properties of djsks. It is widely considered that the luminosity variations of various astronomical objects are associated with the instability of accretion disk. The viscosity plays an important role in the accretion disk theory, and magnetic field exists commonly in astronomical phenomena. A more realistic analysis of the stabihty of the accretion disk should include both the radjal and azimuthal perturbations because of the strong shear of viscous flow.In this paper, according to the luminosity Variations of Various astronomical objects, we study the radial-azimuthal instability of an isothermal with magnetic field. We find that the radial and azimuthal fields B〓,B〓 are mainly responsible for enhancing the instability of the magneto-acoustic modes and non-axisymmetric modes,and that pulsational oscillations are larger in the radial than in the Z direction. The model may be useful in explaining the periodic light variation of FU orionis and. T Tauri stars.

在许多天体物理现象中，例如活动星系核，X-射线双星和年青的恒星理论中，吸积盘理论起着重要的作用，而吸积盘理论中的一个重要分支是吸积盘的不稳定性理论，普遍认为活动天体的周期，准周期光变现象与吸积盘的不稳定性有关，在吸积盘理论中，粘滞起着重要的作用，而磁场是天体物理中普遍存在的现象，对于几何薄吸积盘不稳定性分析，由于强剪切流的缘故，应该包括径向和环向扰动两个方面，本文结合天体物理中的各种周期，准周期光变现象，研究了含磁场的等温薄吸积盘的径向-环向振荡不稳定性，结果表明：磁场的径向和环向分量对磁声模和非轴对称模的不稳定性的增加起着主要的作用，且径向振荡大于轴向振荡，所得结果有利于解释FU Orionis和T Tauri Stars的周期光变现象。

The stable clones are further identified by RT-PCR and Western blot; 6 MTT assay is used to investigate the effect of ZNRD1 on the cell growth of cells (AGS, SGC7901, MKN28, NIH3T3, GES-1); 7 Soft agar assay is used to investigate the effect of ZNRD1 on the clonality of cells (AGS, MKN28); 8 Nude mice assay is used to investigate the effect of ZNRD1 on the cell growth of gastric cancer cells (AGS, MKN28); 9 Flow cytometry is used to investigate the effect of ZNRD1 on the cell cycle distribution of cells (AGS, MKN28, NIH3T3, GES-1); 10 Flow cytometry is used to investigate the effect of ZNRD1 on the cell apoptosis of cells (AGS, MKN28, NIH3T3); 11 MTT assay is used to investigate the effect of ZNRD1 on the drug sensitivity of cancer cells (SGC7901, SGC7901/VCR, HL-60, HL-60/VCR) in vitro; 12 SRCA is used to investigate the effect of ZNRD1 on the drug sensitivity of gastric cancer cells (SGC7901, SGC7901/VCR) in vivo; 13 Flow cytometry is used to investigate the effect of ZNRD1 on adriamycin accumulation of cancer cells (SGC7901, SGC7901/VCR, HL-60, HL-60/VCR); 14 Transmission electron microscope is used to investigate the effect of ZNRD1 on the sensitivity of SGC7901 cells towards drug-induced apoptosis; 15 Flow cytometry and DNA ladder assay are used to investigate the effect of ZNRD1 on the sensitivity of cells (SGC7901, SGC7901/VCR, HL-60/VCR) towards drug-induced apoptosis; 16 Microarray is used to investigate the profiling of ZNRD1-responsive genes in gastric cancer cells (AGS, MKN28, SGC7901, SGC7901/VCR); 17 RT-PCR and Western blot are used to identify the results of microarray; 18 Reporter gene assay is used to investigate the effect of ZNRD1 on the transcriptional activity of cyclin D1; 19 Reporter gene assay is used to investigate the effect of ZNRD1 on the transcriptional activity of MDR1; 20 Kinase assay is used to investigate the effect of ZNRD1 on the activity of cyclin E-CDK2 kinase; 21 The antisensenucleic acids of p21 is used to inhibit the expression of p21, and flow cytometry is used to investigate the effect of p21 on ZNRD1-induced cell cycle arrest in gastric cancer cells; 22 The antisensenucleic acids of p27 is used to inhibit the expression of p27, and flow cytometry is used to investigate the effect of p27 on ZNRD1-induced cell cycle arrest in gastric cancer cells; 23 Liposome is used to up-regulate the expression of Skp2, and flow cytometry is used to investigate the effect of Skp2 on ZNRD1-induced cell cycle arrest in gastric cancer cells; 24 Western blot is used to investigate the effect of ZNRD1 on the stability of Skp2 and p27 in gastric cancer cells; 25 MVD assay is used to investigate the effect of ZNRD1 on the angiopoietic activity of gastric cancer cells; 26 ELISA is used to investigate the effect of ZNRD1 on the expression of VEGF165 in gastric cancer cells; 27 The roles of DARPP-32 in MDR of gastric cancer cells are investigated using gene transfection, MTT assay, SRCA, flow cytometry and DNA ladder assay.

应用杂交瘤技术制备ZNRD1的首个单克隆抗体；2）利用RT-PCR、Western blot和免疫组化检测ZNRD1在胃癌组织、胃炎组织、正常胃上皮组织、胃癌细胞和正常胃组织上皮细胞中的表达；3）构建ZNRD1的小干扰RNA载体，并测序鉴定；4）利用脂质体将ZNRD1的真核表达载体及其空载体转染胃癌细胞（AGS、SGC7901、MKN28）和小鼠成纤维细胞（NIH3T3），G418筛选后进行鉴定；5）利用脂质体将ZNRD1的小干扰RNA载体及其空载体转染药敏胃癌细胞（SGC7901）、正常胃组织上皮细胞（GES-1）、对长春新碱耐药的胃癌细胞（SGC7901/VCR）、药敏白血病细胞（HL-60）、对长春新碱耐药的白血病细胞（HL-60/VCR），G418筛选后进行鉴定；6）利用MTT实验检测ZNRD1高/低表达对细胞（AGS、SGC7901、MKN28、NIH3T3、GES-1）生长的影响；7）通过软琼脂克隆形成实验检测上调ZNRD1对AGS、MKN28细胞克隆形成能力的影响；8）通过裸鼠成瘤实验检测上调ZNRD1对AGS、MKN28细胞体内成瘤性的影响；9）通过流式细胞仪分析ZNRD1高/低表达对细胞（AGS、MKN28、NIH3T3、GES-1）的细胞周期的影响；10）通过流式细胞仪分析上调ZNRD1对细胞（AGS、MKN28、NIH3T3）的凋亡的影响；11）通过MTT实验检测ZNRD1高/低表达对细胞（SGC7901、SGC7901/VCR、HL-60、HL-60/VCR）体外药物敏感性的影响；12）通过肾包膜下移植法检测ZNRD1高/低表达对细胞（SGC7901、SGC7901/VCR）体内药物敏感性的影响；13）通过流式细胞仪分析ZNRD1高/低表达对细胞（SGC7901、SGC7901/VCR、HL-60、HL-60/VCR）内阿霉素蓄积和泵出的影响；14）通过透射电镜检测上调ZNRD1对SGC7901细胞凋亡敏感性的影响；15）通过流式细胞仪和DNA梯度试验检测ZNRD1高/低表达对细胞（SGC7901、SGC7901/VCR、HL-60）凋亡敏感性的影响；16）通过基因芯片检测ZNRD1高/低表达对胃癌细胞内基因表达谱的影响；17）利用RT-PCR、Western blot对基因芯片的结果进行鉴定；18）利用报告基因实验检测ZNRD1对cyclin D1的启动子活性的调节作用；19）利用报告基因实验检测ZNRD1高/低表达对MDR1的启动子活性的调节作用；20）利用激酶试验检测ZNRD1对cyclin E-CDK2 激酶活力的影响；21）利用反义核酸技术抑制p21的表达；通过流式细胞仪检测抑制p21对ZNRD1介导的细胞周期阻滞的影响；22）利用反义核酸技术抑制p27的表达；通过流式细胞仪检测抑制p27对ZNRD1介导的细胞周期阻滞的影响；23）利用脂质体转染法上调Skp2的表达；通过流式细胞仪检测上调Skp2对ZNRD1介导的细胞周期阻滞的影响；24）利用Western blot检测ZNRD1对p27和Skp2的蛋白稳定性的影响；25）利用微血管密度实验检测ZNRD1对AGS、MKN28细胞裸鼠移植瘤微血管形成的影响；26）利用ELISA检测ZNRD1对AGS、MKN28细胞培养上清和移植瘤匀浆中VEGF165含量的影响；27）利用脂质体转染法、MTT实验、肾包膜下移植法、流式细胞仪和DNA梯度试验检测新耐药相关分子DARPP-32对细胞（SGC7901、SGC7901/VCR、对阿霉素耐药的胃癌细胞SGC7901/ADR）多药耐药表型的影响；利用脂质体转染法和MTT实验检测下调ZNRD1对DARPP-32介导的胃癌多药耐药的调控作用。

Finite element software ANSYS was employed to investigate the mechanical response of asphalt pavement structure under the axisymmetrical dynamic loading,and different loading period,loading amplitude and loading form were discussed in detail.

运用有限元程序ANSYS，采用轴对称模型，求出了沥青路面在动荷载作用下的力学响应，并针对不同的荷载作用周期、荷载幅值以及不同的荷载作用形式进行了详细的分析与讨论。

It analyses the essential factors, structure, environment as well as functions of high-tech corporation system and compares the similarities and differences between high-tech corporation system and traditional corporation system; it analyses characteristics in each stage of high-tech corporation lifecycle as well as management roots which lead to the declination of high-tech corporation and researches into the similarities and differences between high-tech corporation lifecycle and other lifecycles, including traditional corporation lifecycle, industry lifecycle, product lifecycle and ecology lifecycle; it studies functional coupling in each stage of high-tech corporation lifecycle and puts forward ways to realize internal coupling of high-tech corporation system and external environment coupling; based on fundamental concepts of dissipative structure, it summarizes six inherent laws of system evolution and on the basis of those laws it advances six principles of high-tech corporation lifecycle management, it poses four sequential parameters of high-tech corporation system, analyses coordination and competition between them and then it advanced four principles of high-tech corporation management; it researches into various cycles in high-tech corporation and puts forward five measures of high-tech corporation management from the point of hypercycle theory; it studies the relations between carious innovation of high-tech corporation innovation system and sets up models of innovation motive forces and resistances, after that it analyses the alternation laws of innovation motive forces and resistances in each stage of high-tech corporation lifecycle, puts forward some measures to strengthen motive forces and lessen resistances and then from the angle of dissipative structure theory, it researches into relevant questions of high-tech corporation innovation management; it studies the location, identification, cultivation, cultivation and protection of high-tech corporation core competence and researches into the key elements of core competence management in each stage of lifecycle and then from the angle of synergetic, it discusses several questions of high-tech corporation core competence management; it studies seven common characteristics of successful high-tech corporation culture and puts forward the ways and measures to cultivate and innovate high-tech corporation culture after that it researches into the characteristics of corporation culture in each stage of lifecycle and discusses relevant questions of high-tech corporation culture management from the point of hypercycle theory; it studies interplay of high-tech corporation system and environment and researches into the development and management of high-tech corporation ecology system after that it analyses the construction and disintegration of high-tech corporation strategic alliance.

剖析了高新技术企业系统的要素、结构、环境与功能，比较了高新技术企业系统与一般企业系统的异同；剖析了高新技术企业在生命周期各阶段的特点及衰退的管理根源，探讨了高新技术企业生命周期与一般企业生命周期、产业生命周期，产品生命周期和生物生命周期的异同；研究了高新技术企业生命周期各阶段的功能耦合，提出了实现高新技术企业系统内部耦合及系统与外部环境耦合的途径；以耗散结构理论的基本概念为基础，总结归纳出系统演化的六大内在规律，据此提出了高新技术企业生命周期管理的六个原则；提出了高新技术企业系统的四个序参量，剖析了生命周期各阶段序参量的协同与竞争，进而提出了高新技术企业管理的四大原则；研究了高新技术企业中的各种循环，从超循环理论的角度提出了高新技术企业管理的五大措施；研究了高新技术企业创新系统中各种创新的关系，建立了创新动力和创新阻力模型，剖析了生命周期各阶段创新动力与阻力的变化规律，提出了增强创新动力与减小创新阻力的措施，从耗散结构理论的角度研究了高新技术企业创新管理的相关问题；研究了高新技术企业核心竞争力的定位、识别、培养及保护，剖析了生命周期各阶段核心竞争力管理的要点，从协同理论的角度研究了高新技术企业的核心竞争力管理的几个问题；剖析了成功的高新技术企业文化的七个共同特点，提出了培养和创新高新技术企业文化的途径及措施，研究了生命周期各阶段企业文化的特征，从超循环理论的角度探讨了高新技术企业文化管理的相关问题；研究了高新技术企业系统与环境的交互作用，探讨了高新技术企业生态系统的开拓与管理，剖析了高新技术企业战略联盟的组建与解体。

Itanalyses the essential factors, structure, environment as well as functions of high-techcorporation system and compares the similarities and differences between high-tech corporationsystem and traditional corporation system; it analyses characteristics in each stage of-high-techcorporation lifecycle as well as management roots which lead to the declination of high-techcorporation and researches into the similarities and differences between high-tech corporationlifecycle and other lifecycles, including traditional corporation lifecycle, industry lifecycle,product lifecycle and ecology lifecycle; it studies functional coupling in each stage of high-techcorporation lifecycle and puts forward ways to realize internal coupling of high-tech corporationsystem and external environment coupling; based on fundamental concepts of dissipativestructure, it summarizes six inherent laws of system evolution and on the basis of those laws itadvances six principles of high-tech corporation lifecycle management; it poses four sequentialparameters of high-tech corporation system, analyses coordinfition and competition between themand then it advanced four principles of high-tech corporation management; it researches intovarious cycles in high-tech corporation and puts forward five measures of high-tech corporationmanagement from the point of hypercycle theory; it studies the relations between cariousinnovation of high-tech corporation innovation system and sets up models of innovation motiveforces and resistances, after that it analyses the alternation laws of innovation motive forces andresistances in each stage of high-tech corporation lifecycle, puts forward some measures tostrengthen motive forces and lessen resistances and then from the angle of dissipative structuretheory, it researches into relevant questions of high-tech corporation innovation management; itstudies the location, identification, cultivation, cultivation and protection of high-tech corporationcore competence and researches into the key elements of core competence management in eachstage of lifecycle and then from the angle of synergetic, it discusses several questions ofhigh-tech corporation core competence management; it studies seven common characteristics ofsuccessful high-tech corporation culture and puts forward the ways and measures to cultivate andinnovate high-tech corporation culture after that it researches into the characteristics ofcorporation culture in each stage of lifecycle and discusses relevant questions of high-techcorporation culture management from the point of hypercycle theory;' it studies interplay ofhigh-tech corporation system and environment and researches into the development andmanagement of high-tech corporation ecology system after that it analyses the construction anddisintegration of high4ech corporation strategic alliance.

剖析了高新技术企业系统的要素、结构、环境与功能，比较了高新技术企业系统与一般企业系统的异同；剖析了高新技术企业在生命周期各阶段的特点及衰退的管理根源，探讨了高新技术企业生命周期与一般企业生命周期、产业生命周期，产品生命周期和生物生命周期的异同；研究了高新技术企业生命周期各阶段的功能耦合，提出了实现高新技术企业系统内部耦合及系统与外部环境耦合的途径：以耗散结构理论的基本概念为基础，总结归纳出系统演化的六大内在规律，据此提出了高新技术企业生命周期管理的六个原则；提出了高新技术企业系统的四个序参量，剖析了生命周期各阶段序参量的协同与竞争，进而提出了高新技术企业管理的四大原则；研究了高新技术企业中的各种循环，从超循环理论的角度提出了高新技术企业管理的五大措施；研究了高新技术企业创新系统中各种创新的关系，建立了创新动力和创新阻力模型，剖析了生命周期各阶段创新动力与阻力的变化规律，提出了增强创新动力与减小创新阻力的措施，从耗散结构理论的角度研究了高新技术企业创新管理的相关问题；研究了高新技术企业核心竞争力的定位、识别、培养及保护，剖析了生命周期各阶段核心竞争力管理的要点，从协同理论的角度研究了高新技术企业的核心竞争力管理的几个问题；剖析了成功的高新技术企业文化的七个共同特点，提出了培养和创新高新技术企业文化的途径及措施，研究了生命周期各阶段企业文化的特征，从超循环理论的角度探讨了高新技术企业文化管理的相关问题；研究了高新技术企业系统与环境的交互作用，探讨了高新技术企业生态系统的开拓与管理，剖析了高新技术企业战略联盟的组建与解体。

The effects and mechanism of GABAergic neurons, NOergic neurons, opioid peptide and cyclic adenosine monophosphate in the nucleus reticularis thalami on sleep-wakefulness cycle of rats and the effects and mechanism of the 5-HTergic nerve fibers project from the nucleus raphes dorsalis to RT on sleep-wakefulness cycle of rats were investigated with the methods of brain stereotaxic, nucleus spile, microinjection and polysomngraphy.1. The effects of GABAergic neurons in RT on sleep-wakefulness cycle of rats1.1 Microinjection of 3-mercaptopropionic acid (3-MP, a kind of glutamate decarboxylase inhibitor) into RT. On the day of microinjection, sleep only decreased a litter. On the second day, sleep marked decreased and wakefulness marked increased. On the third and fourth day, sleep and wakefulness stages resumed to normal.1.2 Microinjection of gamma-amino butyric acid (GABA 1.0μg) into RT enhanced sleep and reduced wakefulness compared with control; while microinjection of L-glutamate (L-Glu, 0.2μg) decreased sleep and increased wakefulness; microinjection of bicuculline (BIC, 1.0μg), a GABAA receptor antagonist, enhanced wakefulness and reduced sleep; microinjection of baclofen (BAC, 1.0μg), GABAB receptor agonist, had the same effects as GABA.2. The effects of NOergic neurons in RT on sleep-wakefulness cycle of rats2.1 Microinjection of L-arginine (L-Arg, 0.5μg) into RT decreased sleep compared with control, but there were on statistaical difference between L-Arg group and control; while microinjection of sodium nitroprusside (SNP, 0.2μg), a NO donor into RT, sleep marked decreased and wakefulness marked increased. Microinjection of nitric oxide synthase inhibitor, N-nitro-L-arginine (L-NNA, 2.0μg) into RT enhanced sleep and reduced wakefulness.2.2 After simultaneous microinjection of L-NNA (2.0μg) and SNP (0.2μg) into RT, SNP abolished the sleep-promoting effect of L-NNA compared with L-NNA group; after simultaneous microinjection of L-NNA (2.0μg) and L-Arg(0.5μg) into RT, we found that L-NNA could not blocked the wakefulness-promoting effect of L-Arg.3. The effects of opioid peptide in RT on sleep-wakefulness cycle of rats3.1 Microinjection of morphine sulfate (MOR, 1.0μg) into RT increased wakefulness and decreased sleep compared with control; while microinjection of naloxone hydrochloride (NAL, 1.0μg), the antagonist of opiate receptors, into RT, enhanced sleep and reduced wakefulness.3.2 After simultaneous microinjection of MOR (1.0μg) and NAL (1.0μg) into RT, the wakefulness-promoting effect of MOR and the sleep-promoting effect of NAL were not observed compared with control.4. The effects of cAMP in RT on sleep-wakefulness cycle of rats Microinjection of cAMP (1.0μg) into RT increased sleep and decreased wakefulness compared with control; microinjection of methylene blue (MB,1.0μg) into RT enhanced sleep and reduced wakefulness compared with control.5. The effects of the 5-HTergic nerve fibers project from DRN to RT on sleep-wakefulness cycle of rats5.1 When L-Glu (0.2μg) was microinjected into DRN and normal sodium (NS,1.0μg) was microinjected into bilateral RT. We found that sleep was decreased and wakefulness was increased compared with control; when L-Glu (0.2μg) was microinjected into DRN and methysergide (MS,1.0μg), a non-selective 5-HT antagonist, was microinjected into bilateral RT, We found that sleep was enhanced and wakefulness was reduced compared with L-Glu group.5.2 When p-chlorophenylalanine (PCPA, 10μg) was microinjected into DRN and NS (1.0μg) was microinjected into bilateral RT, We found that sleep was increased and wakefulness was decreased compared with control; microinjection of 5-hydroxytryptaphan (5-HTP, 1.0μg), which can convert to 5-HT by the enzyme tryptophane hydroxylase and enhance 5-HT into bilateral RT, could block the effect of microinjection of PCPA into DRN on sleep-wakefulness cycle.

本研究采用脑立体定位、核团插管、微量注射、多导睡眠描记等方法，研究丘脑网状核(nucleus reticularis thalami，RT)中γ-氨基丁酸(gamma-amino butyric acid ，GABA)能神经元、一氧化氮(nitrogen monoxidum，NO)能神经元、阿片肽类神经递质、环一磷酸腺苷(cyclic adenosine monophosphate，cAMP)及中缝背核(nucleus raphes dorsalis，DRN)至RT的5-羟色胺(5-hydroxytryptamine，5-HT)能神经纤维投射对大鼠睡眠-觉醒周期的影响及其作用机制。1 RT内GABA能神经元对大鼠睡眠-觉醒周期的影响1.1大鼠RT内微量注射GABA合成关键酶抑制剂3-巯基丙酸(3-MP，5μg)，注射当天睡眠时间略有减少，第二日睡眠时间显著减少，觉醒时间明显增多，第三、四日睡眠和觉醒时间逐渐恢复至正常。1.2大鼠RT内微量注射GABA受体激动剂GABA( 1.0μg)后，与生理盐水组比较，睡眠时间增加，觉醒时间减少；而RT内微量注射L-谷氨酸(glutamic acid， L-Glu， 0.2μg)后，睡眠时间减少，觉醒时间增加；RT内微量注射GABAA受体阻断剂荷包牡丹碱(bicuculline，BIC，1.0μg)后，睡眠时间减少，觉醒时间增加；RT内微量注射GABAB受体激动剂氯苯氨丁酸(baclofen，BAC，1.0μg)后，产生了与GABA相似的促睡眠效果。2 RT内NO能神经元对大鼠睡眠-觉醒周期的影响2.1大鼠RT内微量注射NO的前体L-精氨酸(L-Arg，0.5μg)后，与生理盐水组对比，睡眠时间略有减少，但无显著性意义；而RT内微量注射NO的供体硝普钠(Sodium Nitroprusside，SNP，0.2μg)后可明显增加觉醒时间，缩短睡眠时间；微量注射一氧化氮合酶抑制剂L-硝基精氨酸(L-arginine，L-NNA，2.0μg)后，引起睡眠时间增多，觉醒时间减少。2.2大鼠RT内同时微量注射L-NNA(2.0μg)和SNP(0.2μg)后与L-NNA组比较发现SNP逆转了L-NNA的促睡眠作用；RT内同时微量注射L-NNA(2.0μg)和L-Arg(0.5μg)后，与L-NNA(2.0μg)组比较发现L-Arg可以增加觉醒而缩短睡眠，其促觉醒作用未能被NOS的抑制剂L-NNA所逆转。3 RT内阿片肽对大鼠睡眠-觉醒周期的影响3.1大鼠RT内微量注射硫酸吗啡(morphine sulfate，MOR，1.0μg)后与生理盐水组对比，睡眠时间减少而觉醒时间增加； RT内微量注射阿片肽受体拮抗剂盐酸纳洛酮(naloxone hydrochloride，NAL，1.0μg)后与生理盐水组比较，睡眠时间增加而觉醒时间减少。3.2大鼠RT内同时微量注射MOR(1.0μg)和NAL(1.0μg)后，与生理盐水组对比，原有的MOR促觉醒效果和NAL的促睡眠效果都没有表现。4 RT内环一磷酸腺苷信使对大鼠睡眠-觉醒周期的影响大鼠RT内微量注射cAMP(1.0μg)后与NS(1.0μg)组比较，睡眠时间增多而觉醒时间减少；RT内微量注射亚甲蓝(methylene blue，MB，1.0μg)后，与NS组比较，睡眠时间增多而觉醒时间减少。5中缝背核投射到丘脑网状核的5-羟色胺能神经纤维对大鼠睡眠-觉醒周期的影响5.1大鼠DRN内微量注射L-Glu(0.2μg)，同时在双侧RT内微量注射NS (1.0μg)后，与对照组(DRN和双侧RT注射NS， 0.2μg)比较，睡眠时间减少，觉醒时间增多；大鼠DRN内微量注射L-Glu(0.2μg)，同时在双侧RT内微量注射二甲基麦角新碱(methysergide， MS， 1.0μg )后，与对照组(DRN注射L-Glu 0.2μg，双侧RT注射NS 1.0μg)比较，睡眠时间增多，觉醒时间减少。5.2大鼠DRN内微量注射对氯苯丙氨酸(p-chlorophenylalanine，PCPA，10μg)，同时在双侧RT内微量注射NS (1.0μg)后，与对照组(DRN和双侧RT注射NS， 1.0μg)比较，睡眠时间增多，觉醒时间减少；大鼠DRN内微量注射PCPA(10μg)，产生睡眠增多效应后，在双侧RT内微量注射5-羟色胺酸(5-hydroxytryptaphan ， 5-HTP， 1.0μg )后，与对照组(DRN注射PCPA 10μg，双侧RT注射NS 1.0μg)比较，睡眠时间减少，觉醒时间增多。

The role of the magnetic field potential impact of time on the mother liquor cyclical changes in the role cycle of 60min.

磁场的作用时间对母液电位影响呈周期性变化，作用周期均为60min。

与作用周期相关的网络解释 [注：此内容来源于网络，仅供参考]

action cycle：作用周期

Action 作用 | Action Cycle 作用周期 | Action Line 作用线

action message：作用讯息

Action Line 作用线 | action message 作用讯息 | Action Period 作用周期

economic cycle：经济周期

这种短期权衡取舍关系在分析经济周期中起着关键作用,经济周期(economic cycle)是用生产的物品与劳务量或雇用的人数来(作者 唐渊 中国致公出版社)衡量的,经济活动中无规律的、大部分无法预测的波动.

cyclothem：周期沈积

圆口纲 Cyclostomata | 周期沈积 cyclothem | 循回沈积(作用) cyclotheric sedimentation

inaction period：无作用期间,钝化周期

ice period 冰[河]期,结冰期 | inaction period 无作用期间,钝化周期 | inoperative period 非运行期

menstrual cycle：经周期

卵巢因失去这两个刺激素的作用, 雌激素及黄体素的分泌也因而锐减, 终导致子宫内膜的剥落而开始下一个月经周期.从开始阴道流血的第一天算起, 这也是整个月经周期(menstrual cycle)的第一天, 出血的天数在3-5 天之间,

photoperiodism：光周期现象,光周期性

photooxidation 光氧化(作用) | photoperiodism 光周期现象,光周期性 | photoperoid 光周期

Restricted transduction：局限转导[在细菌细胞生活周期中易于接受外源DNA的某种暂时状态下出现]

restorpin 网状内皮(作用)素 | restricted transduction 局限转导[在细菌细胞生活周期中易于接受外源DNA的某种暂时状态下出现] | retardin 阻滞素,延缓素

chronotropic action：变时性作用, 周期变更作用

moor 沼地, 荒野 停泊, 固定, 系住 | chronotropic action 变时性作用, 周期变更作用 | vitalize 激发

chronotropic action：周期变更作用

连续照相 chronophotograph | 精微测时器 chronoscope | 周期变更作用 chronotropic action