thrombase ['θrɔmbeis]
- thrombase的基本解释
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[化] 凝血酶, 纤维蛋白酶
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ocimum basilicum possesses an inhibitory effect on platelet aggregation induced by ADP and thrombase.
罗勒水提取物对ADP、凝水酶诱导的大鼠血小板聚集具有抑制作用。
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Methods: ADP and thrombase were used to induce platelet aggregation and plate function was evaluated by turbidimetry to observe ocimum basilicum effects on platelet aggregation.
60只Wistar大鼠随机分为6组:生理盐水组、8.8 mg/kg阿司匹林组、4.7 mg/kg丹参滴丸组及200、300、500 mg/kg罗勒水提取物3个剂量组,采用比浊法测定各组大鼠ADP、凝血酶诱导的血小板聚集率和抑制率。
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Therapy against nerve injury is rare. Hirudin was proved to be able to relieve the thrombase injury in ICH. There is no other therapy being accepted for ICH.
目前脑出血的治疗方法主要是针对颅内高压和脑出血的并发症,而针对继发性脑损害和脑损害的病理生理过程的治疗措施尚少。
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The research data showed that the many factors can trigger the secondary injury after cerebral hemorrhage,which includes secondary ischemia around the hematoma,releasing of thrombase,hemoglobinolysis,infiltration of inflammatory cells,and the expression of many cytokines.
研究发现,血肿周围继发性缺血,凝血酶的释放,血红蛋白分解,炎性细胞浸润及多种细胞因子的表达等均可能是脑出血后继发性损伤的触发因素。
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The path-physiological mechanism of ICH is complicated, which includes the high intracranial pressure caused by hematom mechanical pressure, the lesion by the decrease of regional cerebral blood flow surrouding the hematom, even regions adjacent to the hematom, Meanwhile, the materials released from hematom including ferrohemoglobin, thrombase, plasma protein and platelet, leucocyte, glusate not only directly injury neurons but also blood brain barrier.
脑出血的病理生理学机制复杂,其中包括血肿的机械压迫所造成的持续的脑缺血性损害;血肿分解后产生的血红蛋白、凝血酶、血浆蛋白、血小板和白细胞、谷氨酸、自由基等毒性物质不仅可直接损伤神经元,而且可损害血脑屏障加重脑水肿。
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- 更多网络解释与thrombase相关的网络解释 [注:此内容来源于网络,仅供参考]
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thrombase:凝血酶
throat plate 针板 | thrombase 凝血酶 | thrombin 凝血酶
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thrombase:凝血酶 纤维蛋白酶
thromballosis 静脉血凝固 静脉血凝固 | thrombase 凝血酶 纤维蛋白酶 | thrombasthenia 血小板衰弱症 血小板无力症
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thrombase:纤维蛋白酶
thromballosis 静脉血凝固 | thrombase 纤维蛋白酶 | thrombasethrombin 凝血酶