shield cell

shield cell的基本解释

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盾形细胞

与shield cell相关的网络例句 [注：此内容来源于网络，仅供参考]

Most of the turtle's shell and shield piece 12, between the carapace and the plastron is connected by sutures, from top to bottom were: throat shield, ribs shield, chest shield, belly shields, hip shield, anal shield.

大多数龟类的腹甲盾片为12枚，背甲和腹甲间借骨缝相连，从上到下依次为：喉盾、肋盾、胸盾、腹盾、臀盾、肛盾。

Due to the complexity of the cell jitter, the NonSynchronous Tining Recovery methods are currently not mature With the emphasis being given to the Class A CBR traffic, this paper analyzes the performance of the queueing delay and cell jitter at the source node and intermediate nodes, and discusses the Source Timing Recovery at the destination node in ATM networks Firstly, this paper presents a description of the cell jitter of CBR traffic, and gives the definitions of two kinds of cell jitter regarding the Source Timing Recovery for CBR traffic Then, by using exact mathematical models and analysis methods, this paper analyzes the impact of the factors, such as the capacity of the queueing buffer, the randomness, the deterministic nature and the correlation in cell arrivals of the background traffic sources, on the queueing delay and cell jitter performance of the CBR traffic through Statistical Multiplexitng To obtain an insight into the power spectral distribution and look for better schemes for the depression and filtering of the cell jitter, within the analyses we succeed deriving the power spectrum of the cell jitter for CBR traffic Hence, not only the power spectral distribution of the cell jitter can in the frequency domain be qualitatively understood, but also can the rms (root-meansquare) value of the cell jitter be quantitatively obtained so as to more accurately measure the amplitude of the jitter In the end-to-end performance analysis of the queueing delay and cell jitter, we propose a kind of quasi-periodic cell stream model to characterize the jittered CBR traffic, and present an initial queueing analysis of the CBR traffic following such a model at a generic intermediate node Additionally, we briefly discuss the buildout/playout and Source Timing Recovery functions of the destination node Finally, regarding the Source Timing Recovery of CBR traffic, this paper systematically discusses several important principles of the cell jitter filtering and depression reported in the literature, introduces several implementation schemes of the Source Timing Recovery e.

由于信元抖动的复杂性，非同步定时恢复方法目前还很不成熟。本文针对A类CBR业务流在ATM网络源节点和中间节点的排队时延和信元抖动性能，以及在目的节点的源定时恢复问题作了较为全面的研究。首先，文中描述了CBR业务流的信元抖动，并具体地给出了两种与CBR业务源定时恢复有关的信元抖动的定义。然后，采用了精确的数学模型和分析方法，有针对性地分析了业务背景中信元到达的纯随机性、确定性和相关性以及排队缓存器容量等因素对CBR业务流经过统计复用后的排队时延和信元抖动性能的影响。为了了解信元抖动的功率频谱分布和寻求更好的抑制和滤除抖动的方法，在性能分析中，我们成功地完成了CBR业务流信元抖动的功率谱分析，使得不但可以从频域定性地认识信元抖动的能量分布特性，而且还可以定量地求出信元抖动的均方根值(rms：root-mean-square)，以更为准确地衡量抖动的大小。在CBR业务流的多节点端-端排队时延和信元抖动性能分析中，我们提出了一种准周期性(quasi-periodic)信元流模型来描述感染了信元抖动的CBR业务流，并基于这一模型进行了CBR业务流中间节点的初步排队分析。

1、Cur inhibits K562 cells growth and induces cell apoptosis may be correlated with the down-regulation of p210~、inhibition of protein tyrosine phosphorylation and the signaling molecules such as p-Erk1/2、c-myc which are relevant with cell growth and apoptosis; 2、Cur synergizes STI571 to inhibit K562 cell growth and induce cell apoptosis may be correlated with the down-regulation of p210~、inhibition of protein tyrosine phosphorylation and the signaling molecules such as Hsp90、PKC which are relevant with cell growth and apoptosis; 3、Cur reverses the resistance of K562/G01 cells to STI571, and synergizes STI571 to inhibit K562/G01 cell growth and induce cell apoptosis; 4、Cur inhibits human originated CML CD34~+ cell growth、induces cell apoptosis, and enhances STI571 to down-regulate the expression of p210~, finally inhibit cell growth and induce cell apoptosis.

从以上实验结果我们得出如下结论： 1、Cur抑制K562细胞增殖、诱导细胞凋亡的作用可能与其下调p210~、蛋白酪氨酸磷酸化水平以及抑制下游p-Erk1/2、c-myc等信号分子有关； 2、Cur协同STI571抑制K562细胞增殖、诱导细胞凋亡的作用可能与其下调p210~、蛋白酪氨酸磷酸化水平以及抑制Hsp90和下游PKC等信号分子有关； 3、Cur可逆转K562/G01细胞对STI571的耐药性，并与STI571协同抑制K562/G01细胞增殖和诱导凋亡，其抑制K562/G01细胞增殖、诱导细胞凋亡的作用可能与其下调p210~、蛋白酪氨酸磷酸化水平以及抑制下游Procaspase-3和NF-κB等信号分子有关； 4、Cur可抑制来源于CML患者骨髓的CD34~+细胞的增殖并诱导其凋亡，还可协同STI571下调CML CD34~+细胞p210~表达，进而协同抑制细胞增殖、诱导细胞凋亡。

与shield cell相关的网络解释 [注：此内容来源于网络，仅供参考]

RED BLOOD CELL SHIELD：红血球盾

芭比盾 BARBIE SHIELD | 红血球盾 RED BLOOD CELL SHIELD | 戒指RING